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Atherosclerosis, ISSN 0021-9150, 2012, Volume 221, Issue 2, pp. 375 - 382
Highlights ► Liraglutide can normalize TNF-α-induced pro-oxidant production in HUVEC probably through reduced expression of NADPH oxidase subunit gp91phox and... 
Cardiovascular | Oxidative stress | NF-κB | Endothelial cell | Inflammation | Apoptosis | CHRONIC HEART-FAILURE | TRANSLOCATION | PROTEIN-KINASE-C | PENTRAXIN 3 | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | IN-VITRO | NF-kappa B | ACUTE MYOCARDIAL-INFARCTION | DEGRADATION | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | VASCULAR NAD(P)H OXIDASE | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Reactive Oxygen Species - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Membrane Glycoproteins - metabolism | Protein Kinase C-alpha - metabolism | Apoptosis - drug effects | Humans | NADPH Oxidases - metabolism | Human Umbilical Vein Endothelial Cells - immunology | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Dose-Response Relationship, Drug | C-Reactive Protein - metabolism | Transfection | I-kappa B Kinase - metabolism | Time Factors | Inflammation Mediators - metabolism | Superoxide Dismutase - metabolism | Recombinant Proteins - metabolism | Glutathione Peroxidase - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Anti-Inflammatory Agents - pharmacology | Glucagon-Like Peptide 1 - analogs & derivatives | Cells, Cultured | Glucagon-Like Peptide 1 - pharmacology | Antioxidants - pharmacology | NADPH Oxidase 2 | Protein Transport | Catalase - metabolism | NF-kappa B - genetics | Signal Transduction - drug effects | Serum Amyloid P-Component - metabolism | Oxidative Stress - drug effects | Liraglutide
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 2015, Volume 64, Issue 5, pp. 1444 - 1449
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2012, Volume 32, Issue 3, pp. 669 - 676
OBJECTIVE—Nicotinic acid (NA) treatment has been associated with benefits in atherosclerosis that are usually attributed to effects on plasma lipoproteins. The... 
atherosclerosis | macrophages | vascular biology | receptors | cholesterol-lowering drugs | LONG-TERM | INTEGRIN VLA-4 | ACTIVATION | DENDRITIC CELLS | KAPPA-B | FOAM CELLS | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | EXTENDED-RELEASE NIACIN | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Integrin alpha4beta1 - metabolism | Toll-Like Receptor 2 - agonists | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Human Umbilical Vein Endothelial Cells - immunology | Monocytes - metabolism | Monocytes - immunology | Niacin - pharmacology | Receptors, Prostaglandin - metabolism | Receptors, Immunologic - antagonists & inhibitors | Inflammation - metabolism | Transfection | Chemotaxis, Leukocyte - drug effects | I-kappa B Kinase - metabolism | RNA Interference | Inflammation Mediators - metabolism | Toll-Like Receptor 4 - agonists | Chemokine CCL2 - metabolism | Receptors, G-Protein-Coupled - drug effects | Interleukin-6 - metabolism | Receptors, Prostaglandin - antagonists & inhibitors | Receptors, Nicotinic - drug effects | Receptors, Nicotinic - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Cyclooxygenase 2 Inhibitors - pharmacology | Anti-Inflammatory Agents - pharmacology | Cells, Cultured | Inflammation - immunology | Toll-Like Receptor 2 - metabolism | Cell Adhesion - drug effects | Toll-Like Receptor 4 - metabolism | Monocytes - drug effects | Transcription Factor RelA - metabolism | Lipopolysaccharides - pharmacology | Inflammation - genetics | Receptors, G-Protein-Coupled - genetics | Pyrazines - pharmacology | Vascular Cell Adhesion Molecule-1 - metabolism | Receptors, Nicotinic - genetics | Receptors, Immunologic - metabolism
Journal Article
Circulation Research, ISSN 0009-7330, 03/2009, Volume 104, Issue 5, pp. 576 - 588
The major arteries and veins of the vertebrate circulatory system are formed early in embryonic development, before the onset of circulation, following de novo... 
Venous specification | COUP-TFII | Arterial | VEGF | Notch | MURINE YOLK-SAC | CARDIAC & CARDIOVASCULAR SYSTEMS | arterial-venous specification | ENDOTHELIAL-GROWTH-FACTOR | NOTCH PATHWAY | BRANCHING MORPHOGENESIS | CARDIOVASCULAR DEVELOPMENT | EPH RECEPTORS | PROTEIN-COUPLED RECEPTOR | PERIPHERAL VASCULAR DISEASE | EMBRYONIC VASCULAR DEVELOPMENT | HEMATOLOGY | RECEPTOR TYROSINE KINASE | SONIC-HEDGEHOG | Veins - embryology | Receptors, Notch - metabolism | Humans | Hedgehog Proteins - metabolism | Arteries - embryology | Intracellular Signaling Peptides and Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Arteries - metabolism | Forkhead Transcription Factors - metabolism | Gene Expression Regulation, Developmental | Veins - enzymology | Cell Differentiation | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Endothelial Cells - metabolism | Signal Transduction - genetics | Ephrin-B2 - metabolism | Receptor, EphB4 - metabolism | Animals | Arteries - enzymology | Veins - metabolism | SOX Transcription Factors - metabolism | COUP Transcription Factor II - metabolism | Body Patterning - genetics | Endothelial Cells - enzymology | Neovascularization, Physiologic
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 03/2016, Volume 92, pp. 152 - 162
The effects of physiological oxygen tension on Nuclear Factor-E2-Related Factor 2 (Nrf2)-regulated redox signaling remain poorly understood. We report the... 
NQO1 | Bach1 | Solute Carrier Family 7-anionic amino acid transporter light chain xCT | Coronary artery | HO-1 | Redox signaling | Endothelial cells | Normoxia | GCL | Physiological oxygen tension | Mitochondria | Nuclear Factor-E2-Related Factor 2, Nrf2 | Glutathione reductase GR | Thioredoxin reductase-1 | Sequestosome-1 | Glutathione | Physiologicaloxygentension | NuclearFactor-E2-RelatedFactor2,Nrf2 | Redoxsignaling | transporterlightchainxCT | Solute CarrierFamily7-anionicaminoacid | Thioredoxinreductase-1 | REPRESSOR BACH1 | ENDOCRINOLOGY & METABOLISM | SMOOTH-MUSCLE-CELLS | HEME OXYGENASE-1 | MOLECULAR-BASIS | KEAP1-NRF2 SYSTEM | NQ01 | OXIDATIVE STRESS | STEM-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | VASCULAR ENDOTHELIUM | SENSING MECHANISMS | TRANSCRIPTION FACTOR NRF2 | Heme Oxygenase-1 - metabolism | Basic-Leucine Zipper Transcription Factors - metabolism | Reactive Oxygen Species - metabolism | Glutathione - metabolism | Antioxidants - metabolism | Humans | NAD(P)H Dehydrogenase (Quinone) - genetics | Oxygen - metabolism | Coronary Vessels - metabolism | DNA-Binding Proteins - metabolism | Kelch-Like ECH-Associated Protein 1 - genetics | Heme Oxygenase-1 - genetics | Fanconi Anemia Complementation Group Proteins - metabolism | Amino Acid Transport System y+ - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | NF-E2-Related Factor 2 - genetics | Glutamate-Cysteine Ligase - metabolism | Endothelial Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Fanconi Anemia Complementation Group Proteins - genetics | Kelch-Like ECH-Associated Protein 1 - metabolism | Basic-Leucine Zipper Transcription Factors - genetics | DNA-Binding Proteins - genetics | Veins - metabolism | NF-E2-Related Factor 2 - metabolism | NAD(P)H Dehydrogenase (Quinone) - metabolism | Genetic research | Physiological aspects | Genes | Endothelium | Surface active agents | RNA | Amino acids | DNA binding proteins
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2210 - 20
VEGFR-2/Notch signalling regulates angiogenesis in part by driving the remodelling of endothelial cell junctions and by inducing cell migration. Here, we show... 
TIP CELLS | PERMEABILITY | ELONGATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | RHO | RAC1 | BETA-CATENIN | ENDOCYTOSIS | FLOW | VASCULAR ENDOTHELIAL-CADHERIN | Human Umbilical Vein Endothelial Cells | Neovascularization, Physiologic - drug effects | Vascular Remodeling | Cadherins - metabolism | Humans | Actins - metabolism | rac GTP-Binding Proteins - metabolism | Actins - drug effects | Vascular Endothelial Growth Factor A - metabolism | Cell Movement - physiology | Antigens, CD - metabolism | Pseudopodia - drug effects | Actin-Related Protein 2-3 Complex - metabolism | Microtubules - metabolism | Microtubules - drug effects | Actin-Related Protein 3 - metabolism | Wiskott-Aldrich Syndrome Protein - metabolism | Actin-Related Protein 2 - metabolism | Cell Polarity - drug effects | Vascular Endothelial Growth Factor A - pharmacology | Cardiac Myosins - metabolism | Endothelial Cells - physiology | Signal Transduction | Wiskott-Aldrich Syndrome Protein Family - metabolism | Endothelial Cells - metabolism | Models, Cardiovascular | Intercellular Junctions - metabolism | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Antigens, CD - drug effects | Cadherins - drug effects | Cell Adhesion | Cell Movement - drug effects | Neovascularization, Physiologic - physiology | Pseudopodia - metabolism | Intercellular Junctions - drug effects | Cell Polarity - physiology | Myosin Light Chains - metabolism | Pseudopodia - physiology | Endothelium, Vascular | Endothelial Cells - drug effects | Cell junctions | Pseudopodia | Dephosphorylation | Cadherin | Remodeling | Contraction | Endothelial cells | Cell adhesion & migration | Vascular endothelial growth factor receptors | Prisons | Angiogenesis | Signaling | Cell activation | Actin | Microtubules | Myosin | Notch protein | Vascular endothelial growth factor | Cell migration | Elongation
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 02/2012, Volume 302, Issue 4, pp. C644 - C651
Borst O, Abed M, Alesutan I, Towhid ST, Qadri SM, Foller M, Gawaz M, Lang F. Dynamic adhesion of eryptotic erythrocytes to endothelial cells via... 
Scavenger receptor binding phosphatidylserine and oxidized low-density lipoprotein | Eryptosis | Phosphatidylserine | Endothelium | endothelium | CHEMOKINE LIGAND-16 | PHYSIOLOGY | CATION CONDUCTANCE | SR-PSOX | INFECTED ERYTHROCYTES | CELL BIOLOGY | phosphatidylserine | eryptosis | SCAVENGER RECEPTOR | DISINTEGRIN | scavenger receptor binding phosphatidylserine and oxidized low-density lipoprotein | RED-BLOOD-CELLS | PHOSPHATIDYLSERINE EXPOSURE | SUICIDAL DEATH | EXPRESSION | Endothelium, Vascular - cytology | RNA, Small Interfering - genetics | Antibodies - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Calcium - metabolism | Humans | Receptors, Scavenger - antagonists & inhibitors | Human Umbilical Vein Endothelial Cells - cytology | Erythrocytes - cytology | Cell Membrane - metabolism | Receptors, Scavenger - metabolism | Chemokines, CXC - antagonists & inhibitors | Chemokine CXCL16 | Rheology | Phosphatidylserines - metabolism | Gene Silencing | Cell Size | Chemokines, CXC - genetics | Cell Adhesion | Glucose - deficiency | Annexin A5 - metabolism | Cell Death - physiology | Receptors, Scavenger - genetics | Endothelium, Vascular - metabolism | Chemokines, CXC - metabolism | Erythrocytes - metabolism | Ionomycin - metabolism | Calcium Ionophores - metabolism | Cytosol - metabolism | Erythrocyte Count | RNA, Small Interfering - metabolism | Research | Properties | Cell adhesion | Erythrocytes
Journal Article
Journal Article
Diabetes, ISSN 0012-1797, 03/2016, Volume 65, Issue 3, pp. 598 - 609
Journal Article
by Peng, JH and Cui, T and Huang, F and Chen, L and Zhao, Y and Xu, L and Xu, LL and Feng, Q and Hu, YY
JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS, ISSN 0022-3565, 03/2013, Volume 344, Issue 3, pp. 646 - 654
Puerarin, an isoflavone component extracted from Kudzu (Pueraria lobata), has been demonstrated to alleviate alcohol-related disorders. Our study examined... 
HEPATITIS | LIPOPOLYSACCHARIDE-BINDING PROTEIN | DISEASE | BARRIER | FATTY LIVER | RATS | ETHANOL | PHARMACOLOGY & PHARMACY | MICE | KAPPA-B | INTESTINAL PERMEABILITY | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Tight Junctions - genetics | Isoflavones - pharmacology | Male | Alanine Transaminase - blood | Alcohol-Related Disorders - genetics | Antigens, CD - genetics | Receptors, Immunologic - antagonists & inhibitors | Antigens, CD - metabolism | Liver Diseases, Alcoholic - drug therapy | Zonula Occludens-1 Protein - metabolism | Neutrophil Infiltration - drug effects | Disease Models, Animal | Tight Junctions - drug effects | Tight Junctions - metabolism | Zonula Occludens-1 Protein - genetics | Fatty Liver - metabolism | Portal Vein - metabolism | Liver - metabolism | Rats | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 2 - metabolism | Neutrophil Infiltration - genetics | Rats, Sprague-Dawley | Toll-Like Receptor 4 - metabolism | Fatty Liver - drug therapy | Triglycerides - metabolism | Microvilli - genetics | Aspartate Aminotransferases - blood | Diet | Microvilli - metabolism | Receptors, Immunologic - genetics | Intestines - drug effects | Tumor Necrosis Factor-alpha - metabolism | Liver - enzymology | Intestines - metabolism | Acute-Phase Proteins - genetics | Alcohol-Related Disorders - drug therapy | Lipopolysaccharide Receptors - metabolism | Alcohol-Related Disorders - metabolism | Liver Diseases, Alcoholic - genetics | Receptors, Immunologic - biosynthesis | Kupffer Cells - metabolism | Endotoxins - metabolism | Fatty Liver - genetics | Portal Vein - drug effects | Acute-Phase Proteins - metabolism | Endotoxins - genetics | gamma-Glutamyltransferase - metabolism | Antigens, Differentiation, Myelomonocytic - genetics | Liver Diseases, Alcoholic - metabolism | Kupffer Cells - drug effects | Membrane Glycoproteins - genetics | Carrier Proteins - genetics | Microvilli - drug effects | Endotoxins - antagonists & inhibitors | Animals | Carrier Proteins - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Lipopolysaccharide Receptors - genetics | Receptors, Immunologic - metabolism
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 09/2011, Volume 301, Issue 3, pp. C695 - C704
Wang Y, Zang QS, Liu Z, Wu Q, Maass D, Dulan G, Shaul PW, Melito L, Frantz DE, Kilgore JA, Williams NS, Terada LS, Nwariaku FE. Regulation of VEGF-induced... 
Antioxidants | Mitochondria | Reactive oxygen species | Oxidant signaling | Endothelial cells | NADPH OXIDASE | endothelial cells | SUPEROXIDE | ACTIVATION | PHYSIOLOGY | OXIDATIVE DAMAGE | antioxidants | mitochondria | TERMINAL KINASE | CELL BIOLOGY | oxidant signaling | reactive oxygen species | IN-VIVO | TARGETED ANTIOXIDANTS | GROWTH-FACTOR | DYSFUNCTION | STRESS | DNA Polymerase gamma | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Endothelium, Vascular - injuries | Humans | Cytoplasm - metabolism | Endothelium, Vascular - drug effects | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Cell Movement - physiology | DNA-Directed DNA Polymerase - genetics | Electron Transport Complex IV - metabolism | Endothelium, Vascular - physiology | Human Umbilical Vein Endothelial Cells - cytology | Superoxides - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Vascular Endothelial Growth Factor A - pharmacology | Carotid Artery Injuries - pathology | von Willebrand Factor - metabolism | Cell Line | Human Umbilical Vein Endothelial Cells - drug effects | Transduction, Genetic | Catalase - genetics | Mice, Inbred C57BL | Cells, Cultured | Mitochondria - metabolism | Mitochondria - drug effects | Regeneration - physiology | p21-Activated Kinases - metabolism | Hydrogen Peroxide - metabolism | Catalase - metabolism | Cell Movement - drug effects | Regeneration - drug effects | Animals | Cyclooxygenase 2 - metabolism | Endothelium, Vascular - pathology | Cell Proliferation - drug effects | Mice | DNA-Directed DNA Polymerase - metabolism | Cytoplasm - drug effects | Hydrogen Peroxide - antagonists & inhibitors | rac1 GTP-Binding Protein - metabolism | Vitamin E - pharmacology | rac1 GTP-Binding Protein - genetics | Care and treatment | Atherosclerosis | Physiological aspects | Diagnosis | Research | Vascular endothelial growth factor | Cell migration | Vascular Biology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2013, Volume 33, Issue 1, pp. 96 - 104
OBJECTIVE—Angiotensin-II (Ang-II) promotes the interaction of mononuclear cells with arterioles and neutrophils with postcapillary venules. To investigate the... 
mononuclear cells | angiotensin-II | arterial endothelium | endothelial dysfunction | CX3CL1 | Human Umbilical Vein Endothelial Cells - metabolism | Membrane Glycoproteins - metabolism | Atherosclerosis - genetics | Humans | Leukocyte Rolling | Tumor Necrosis Factor-alpha - genetics | NADPH Oxidases - metabolism | Male | NF-kappa B - metabolism | Venules - drug effects | Interferon-gamma - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Apolipoproteins E - metabolism | RNA Interference | Time Factors | NADPH Oxidases - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Receptors, Chemokine - genetics | Disease Models, Animal | Atherosclerosis - pathology | Signal Transduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Losartan - pharmacology | Chemokine CX3CL1 - genetics | Arteries - drug effects | Arterioles - drug effects | Cell Adhesion | Atherosclerosis - metabolism | Mice, Knockout | Arterioles - metabolism | Veins - metabolism | Mice | CX3C Chemokine Receptor 1 | Leukocytes - metabolism | Tumor Necrosis Factor-alpha - metabolism | Chemokine CX3CL1 - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Arteries - metabolism | Transfection | Female | Membrane Proteins - metabolism | Angiotensin II - metabolism | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Cells, Cultured | NADPH Oxidase 5 | NADPH Oxidase 4 | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Veins - drug effects | Venules - metabolism | Animals | Apolipoproteins E - genetics | Atherosclerosis - prevention & control | Endothelial Cells - drug effects
Journal Article
Circulation Research, ISSN 0009-7330, 02/2007, Volume 100, Issue 3, pp. 328 - 341
The AMP-activated protein kinase (AMPK) system acts as a sensor of cellular energy status that is conserved in all eukaryotic cells. It is activated by... 
Calcium signaling | Signaling pathways | Diabetes | Metabolism | Insulin | RAT-LIVER | CARDIAC & CARDIOVASCULAR SYSTEMS | insulin | 5-AMINOIMIDAZOLE-4-CARBOXAMIDE RIBONUCLEOSIDE | STIMULATED GLUCOSE-TRANSPORT | calcium signaling | N-TERMINAL KINASE | ACETYL-COA CARBOXYLASE | VEIN ENDOTHELIAL-CELLS | COENZYME-A CARBOXYLASE | HUMAN SKELETAL-MUSCLE | signaling pathways | FATTY-ACID OXIDATION | PANCREATIC BETA-CELLS | PERIPHERAL VASCULAR DISEASE | metabolism | HEMATOLOGY | diabetes | Consensus Sequence | Protein Subunits | Protein-Serine-Threonine Kinases - deficiency | Insulin - physiology | Obesity - drug therapy | Humans | Muscle Cells - drug effects | Adipocytes - drug effects | AMP-Activated Protein Kinases | Aminoimidazole Carboxamide - pharmacology | Multienzyme Complexes - deficiency | Calcium - physiology | Adenosine Triphosphate - metabolism | Energy Metabolism - physiology | Binding Sites | Peptide Hormones - physiology | Hypoglycemic Agents - therapeutic use | Amino Acid Sequence | Diabetes Mellitus - drug therapy | Models, Molecular | Rats | Hypoglycemic Agents - pharmacology | Mice, Knockout | Calcium-Calmodulin-Dependent Protein Kinase Kinase | Carbohydrate Metabolism - physiology | Aminoimidazole Carboxamide - analogs & derivatives | Diabetes Mellitus - therapy | Protein-Serine-Threonine Kinases - drug effects | Mice | Protein-Serine-Threonine Kinases - chemistry | Cell Cycle - drug effects | Energy Metabolism - drug effects | Lipid Metabolism - physiology | Multienzyme Complexes - drug effects | Carbohydrate Metabolism - drug effects | Phosphorylation | Metformin - therapeutic use | Molecular Sequence Data | Hepatocytes - metabolism | Ribonucleotides - pharmacology | Hepatocytes - drug effects | Adenosine Monophosphate - metabolism | Protein-Serine-Threonine Kinases - physiology | Metformin - pharmacology | Muscle Cells - metabolism | Protein-Serine-Threonine Kinases - genetics | Diabetes Mellitus - metabolism | Neoplasms - enzymology | Multienzyme Complexes - genetics | Enzyme Activation - drug effects | Protein Processing, Post-Translational - physiology | Multienzyme Complexes - chemistry | Obesity - metabolism | Sequence Homology, Amino Acid | Sequence Alignment | Animals | Oxygen Consumption - drug effects | Adipocytes - metabolism | Multienzyme Complexes - physiology | Lipid Metabolism - drug effects | Cell Cycle - physiology | Neoplasms - pathology
Journal Article