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Journal of the American College of Cardiology, ISSN 0735-1097, 06/2014, Volume 63, Issue 24, pp. 2734 - 2741
Cardiovascular | Internal Medicine | endothelial-mesenchymal transition | diastolic dysfunction | endothelium | NADPH (nicotinamide adenine dinucleotide phosphate) oxidase | angiotensin II | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Heart | Biological and medical sciences | Medical sciences | Heart Failure, Diastolic - genetics | Mesenchymal Stromal Cells - enzymology | Humans | Cardiomegaly - pathology | Male | Endothelium, Vascular - enzymology | Membrane Glycoproteins - physiology | Ventricular Dysfunction, Left - genetics | Ventricular Dysfunction, Left - pathology | NADPH Oxidases - genetics | Ventricular Dysfunction, Left - enzymology | Inflammation Mediators - physiology | Fibrosis - genetics | Fibrosis - enzymology | Cells, Cultured | Heart Failure, Diastolic - enzymology | Mice, Transgenic | Cardiomegaly - enzymology | Heart Failure, Diastolic - pathology | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Animals | Endothelium, Vascular - pathology | Mice | Fibrosis - pathology | Mesenchymal Stromal Cells - pathology | Cardiomegaly - genetics | NADPH Oxidases - physiology | Oxidases | Fibrosis | Stem cells | Endothelium | Hypertension | Heart failure | Genotype & phenotype | Stroke | Heart attacks | Pathogenesis | Rodents | Cardiomyocytes | Variance analysis | Index Medicus | Abridged Index Medicus
Journal Article
Circulation research, ISSN 0009-7330, 04/2010, Volume 106, Issue 7, pp. 1253 - 1264
Aging | Oxidative stress | Reactive oxygen species | Superoxide | Apoptosis | Hypertrophy | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Aconitate Hydratase - metabolism | Up-Regulation | Cysteine | Cell Proliferation | Uncoupling Agents - pharmacology | Oxidative Stress | Rats, Wistar | Ventricular Function, Left | Apoptosis - drug effects | Mitochondria, Heart - pathology | Humans | NADPH Oxidases - metabolism | Cardiomegaly - pathology | Mitochondria, Heart - drug effects | Myocytes, Cardiac - enzymology | Transfection | Ventricular Dysfunction, Left - genetics | Rotenone - pharmacology | Superoxides - metabolism | Ventricular Dysfunction, Left - pathology | NADPH Oxidases - genetics | Ventricular Dysfunction, Left - enzymology | Disease Models, Animal | Oxidation-Reduction | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mitochondria, Heart - enzymology | Cardiomegaly - physiopathology | Rats | Genotype | Mice, Transgenic | Cardiomegaly - enzymology | NADPH Oxidase 4 | Onium Compounds - pharmacology | NADH Dehydrogenase - metabolism | Ventricular Dysfunction, Left - physiopathology | Aging - pathology | Myocytes, Cardiac - pathology | Phenotype | Animals | Myocytes, Cardiac - drug effects | Fibrosis | Mice | Cardiomegaly - genetics | Aging - metabolism | Index Medicus | apoptosis | aging | superoxide | hypertrophy | oxidative stress
Journal Article
Circulation research, ISSN 0009-7330, 02/2013, Volume 112, Issue 4, pp. 675 - 688
fibroblast | MMP-28 | inflammation | macrophage phenotype | myocardial infarction | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Myocardial Infarction - blood | Cell Adhesion Molecules - genetics | Cicatrix - etiology | Extracellular Matrix Proteins - biosynthesis | Male | Matrix Metalloproteinases, Secreted - deficiency | Pulmonary Edema - etiology | Myocytes, Cardiac - enzymology | Myofibroblasts - metabolism | Ventricular Dysfunction, Left - enzymology | Ventricular Remodeling - genetics | Female | Myocardial Infarction - physiopathology | Transcription, Genetic | Cytokines - genetics | Heart Rupture - enzymology | Receptors, Cytokine - genetics | Matrix Metalloproteinase 9 - blood | Myocardial Infarction - enzymology | Macrophage Activation - physiology | Macrophages - classification | Cell Adhesion Molecules - biosynthesis | Receptors, Cytokine - biosynthesis | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Ventricular Dysfunction, Left - etiology | Inflammation | Ventricular Remodeling - physiology | Macrophages - enzymology | Mice, Knockout | Collagen - metabolism | Myocardial Infarction - complications | Heart Rupture - etiology | Animals | Pulmonary Edema - enzymology | Matrix Metalloproteinases, Secreted - genetics | Matrix Metalloproteinases, Secreted - physiology | Protein-Lysine 6-Oxidase - metabolism | Cicatrix - enzymology | Mice | Cytokines - biosynthesis | Index Medicus
Journal Article
Antioxidants & redox signaling, ISSN 1523-0864, 01/2013, Volume 18, Issue 1, pp. 5 - 18
Original Research Communications | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Up-Regulation | Hypertrophy, Left Ventricular - enzymology | Oxidative Stress | Cardiomyopathy, Dilated - enzymology | Male | Monoamine Oxidase - genetics | Heart Ventricles - enzymology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Ventricular Dysfunction, Left - pathology | Ventricular Dysfunction, Left - enzymology | Heart Ventricles - pathology | Oxidation-Reduction | Mice, Inbred C57BL | Cells, Cultured | Mitochondria, Heart - enzymology | Tumor Suppressor Protein p53 - metabolism | Enzyme Induction | Rats | Mice, Transgenic | Rats, Sprague-Dawley | Myocytes, Cardiac - pathology | Animals | Fibrosis | Myocytes, Cardiac - metabolism | Trans-Activators - metabolism | Mice | Transcription Factors | Chronic Disease | Necrosis - enzymology | Monoamine Oxidase - metabolism | Index Medicus | Life Sciences | Cellular Biology
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 2017, Volume 313, Issue 2, pp. H224 - H236
Remodeling | Tissue inhibitor of metalloproteinases 3 | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Myocardial Infarction - genetics | Up-Regulation | Cell Proliferation | Hypertrophy, Left Ventricular - enzymology | Ventricular Function, Left | Coronary Vessels - physiopathology | Humans | Male | Recovery of Function | Myocardial Infarction - therapy | Ventricular Remodeling | Hypertrophy, Left Ventricular - genetics | Time Factors | Ventricular Dysfunction, Left - genetics | Proteolysis | Tissue Inhibitor of Metalloproteinase-3 - biosynthesis | Adenoviridae - genetics | Ventricular Dysfunction, Left - enzymology | Myocardial Infarction - physiopathology | Disease Models, Animal | Myocardial Infarction - enzymology | Tissue Inhibitor of Metalloproteinase-3 - genetics | Coronary Vessels - enzymology | Transduction, Genetic | Signal Transduction | Hypertrophy, Left Ventricular - prevention & control | Mice, Inbred C57BL | Myocardium - pathology | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Myocardium - enzymology | Animals | Adenoviridae - metabolism | Hypertrophy, Left Ventricular - physiopathology | Matrix Metalloproteinases - metabolism | Endothelial Cells - pathology | Genetic Vectors | Endothelial Cells - enzymology | Neovascularization, Physiologic | Genetic Therapy - methods | Myocardial infarction | Regulators | Heart attacks | Echocardiography | Matrix metalloproteinases | Coronary artery | Effects | Density | Endothelial cells | Arteries | Angiogenesis | Matrix | Antiangiogenics | Rodents | Dilation | Protein expression | Disruption | Index Medicus
Journal Article
Basic research in cardiology, ISSN 0300-8428, 9/2017, Volume 112, Issue 5, pp. 1 - 11
ADMA | Medicine & Public Health | Cardiology | Nitric oxide | Ventricular hypertrophy | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Hypertrophy, Left Ventricular - enzymology | Ventricular Function, Left | Male | Tyrosine - analogs & derivatives | Arginine - analogs & derivatives | Myocytes, Cardiac - enzymology | Ventricular Remodeling | Hypertrophy, Left Ventricular - genetics | Ventricular Dysfunction, Left - genetics | Ventricular Dysfunction, Left - enzymology | Amidohydrolases - deficiency | Disease Models, Animal | Amidohydrolases - metabolism | Genetic Predisposition to Disease | Atrial Natriuretic Factor - metabolism | Signal Transduction | Amidohydrolases - genetics | Hypertrophy, Left Ventricular - prevention & control | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Mice, Knockout | Myocytes, Cardiac - pathology | Phenotype | Tyrosine - metabolism | Animals | Arginine - blood | Fibrosis | Hypertrophy, Left Ventricular - physiopathology | Nitric Oxide - metabolism | Heart | Physiological aspects | Heart enlargement | Analysis | Exercise therapy | Sarcolemma | Stresses | Homeostasis | Cardiomyocytes | Bioavailability | Endothelial cells | Eutrophication | Degradation | Rodents | Aging | Aorta | Mice | Nitrotyrosine | Ventricle | Heart diseases | Dimethylargininase | Hypertrophy | Plasmas (physics) | Index Medicus
Journal Article
Cardiovascular research, ISSN 0008-6363, 2015, Volume 105, Issue 3, pp. 318 - 329
Diastolic dysfunction | SERCA2a | TNF-α | Transcription | Simvastatin | Atp2a2 | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Inflammation - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Diastole | Rats, Wistar | Male | NF-kappa B - metabolism | Ventricular Dysfunction, Left - chemically induced | Lipopolysaccharides | Dose-Response Relationship, Drug | Myocytes, Cardiac - enzymology | Ventricular Dysfunction, Left - genetics | Ventricular Dysfunction, Left - enzymology | Transcription, Genetic | Binding Sites | Disease Models, Animal | Cell Line | Promoter Regions, Genetic | NF-kappa B - antagonists & inhibitors | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Signal Transduction | Anti-Inflammatory Agents - pharmacology | Down-Regulation | Ventricular Function, Left - drug effects | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Gene Expression Regulation, Enzymologic | Tumor Necrosis Factor-alpha - pharmacology | Animals | Myocytes, Cardiac - drug effects | NF-kappa B - genetics | Inflammation - genetics | Protein Binding | Inflammation - prevention & control | Mice | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Inflammation - enzymology | Inflammation - physiopathology | Index Medicus
Journal Article
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2015, Volume 86, pp. 1 - 8
Cardiovascular | Right ventricular failure | Creatine kinase | Diastolic sarcomere length | Monocrotaline | Pulmonary artery hypertension | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cell Biology | Diastole | Calcium - metabolism | Heart Failure - enzymology | Humans | Rats | Myocardium - pathology | Heart Failure - pathology | Ventricular Dysfunction, Right - enzymology | Pulmonary Artery - enzymology | Sarcomeres - pathology | Myocytes, Cardiac - enzymology | Myocytes, Cardiac - pathology | Myocardium - enzymology | Animals | Creatine Kinase - biosynthesis | Adenosine Triphosphate - metabolism | Creatine Kinase - metabolism | Sarcomeres - enzymology | Hypertension, Pulmonary - pathology | Hypertension, Pulmonary - enzymology | Pulmonary Artery - pathology | Ventricular Dysfunction, Right - pathology | Heart failure | Hypertension | Index Medicus | Original
Journal Article
Cardiovascular research, ISSN 0008-6363, 12/2012, Volume 96, Issue 3, pp. 444 - 455
Diastolic function | Extracellular matrix | Matrix metalloproteinase | Ageing | Collagen | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Medical sciences | Blood Pressure | Cell Adhesion Molecules - genetics | Phosphorylation | Age Factors | Diastole | Ventricular Function, Left | Male | Systole | Ventricular Dysfunction, Left - genetics | Matrix Metalloproteinase 8 - metabolism | Matrix Metalloproteinase 9 - genetics | Smad2 Protein - genetics | Ventricular Dysfunction, Left - pathology | Ventricular Dysfunction, Left - enzymology | Female | Real-Time Polymerase Chain Reaction | Matrix Metalloproteinase 8 - genetics | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Smad2 Protein - metabolism | Ventricular Dysfunction, Left - etiology | Genotype | Myocardium - pathology | Matrix Metalloproteinase 9 - deficiency | Cell Adhesion Molecules - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Aging - pathology | Mice, Knockout | Collagen - metabolism | Myocardium - enzymology | Phenotype | Animals | Transforming Growth Factor beta - genetics | Fibrosis | Connective Tissue Growth Factor - genetics | Mice | Transforming Growth Factor beta - metabolism | Connective Tissue Growth Factor - metabolism | Aging - metabolism | Index Medicus | Original
Journal Article