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Journal of the American College of Cardiology, ISSN 0735-1097, 06/2014, Volume 63, Issue 24, pp. 2734 - 2741
Journal Article
Circulation research, ISSN 0009-7330, 04/2010, Volume 106, Issue 7, pp. 1253 - 1264
Aging | Oxidative stress | Reactive oxygen species | Superoxide | Apoptosis | Hypertrophy | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Aconitate Hydratase - metabolism | Up-Regulation | Cysteine | Cell Proliferation | Uncoupling Agents - pharmacology | Oxidative Stress | Rats, Wistar | Ventricular Function, Left | Apoptosis - drug effects | Mitochondria, Heart - pathology | Humans | NADPH Oxidases - metabolism | Cardiomegaly - pathology | Mitochondria, Heart - drug effects | Myocytes, Cardiac - enzymology | Transfection | Ventricular Dysfunction, Left - genetics | Rotenone - pharmacology | Superoxides - metabolism | Ventricular Dysfunction, Left - pathology | NADPH Oxidases - genetics | Ventricular Dysfunction, Left - enzymology | Disease Models, Animal | Oxidation-Reduction | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mitochondria, Heart - enzymology | Cardiomegaly - physiopathology | Rats | Genotype | Mice, Transgenic | Cardiomegaly - enzymology | NADPH Oxidase 4 | Onium Compounds - pharmacology | NADH Dehydrogenase - metabolism | Ventricular Dysfunction, Left - physiopathology | Aging - pathology | Myocytes, Cardiac - pathology | Phenotype | Animals | Myocytes, Cardiac - drug effects | Fibrosis | Mice | Cardiomegaly - genetics | Aging - metabolism | Index Medicus | apoptosis | aging | superoxide | hypertrophy | oxidative stress
Journal Article
Circulation research, ISSN 0009-7330, 02/2013, Volume 112, Issue 4, pp. 675 - 688
fibroblast | MMP-28 | inflammation | macrophage phenotype | myocardial infarction | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Myocardial Infarction - blood | Cell Adhesion Molecules - genetics | Cicatrix - etiology | Extracellular Matrix Proteins - biosynthesis | Male | Matrix Metalloproteinases, Secreted - deficiency | Pulmonary Edema - etiology | Myocytes, Cardiac - enzymology | Myofibroblasts - metabolism | Ventricular Dysfunction, Left - enzymology | Ventricular Remodeling - genetics | Female | Myocardial Infarction - physiopathology | Transcription, Genetic | Cytokines - genetics | Heart Rupture - enzymology | Receptors, Cytokine - genetics | Matrix Metalloproteinase 9 - blood | Myocardial Infarction - enzymology | Macrophage Activation - physiology | Macrophages - classification | Cell Adhesion Molecules - biosynthesis | Receptors, Cytokine - biosynthesis | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Ventricular Dysfunction, Left - etiology | Inflammation | Ventricular Remodeling - physiology | Macrophages - enzymology | Mice, Knockout | Collagen - metabolism | Myocardial Infarction - complications | Heart Rupture - etiology | Animals | Pulmonary Edema - enzymology | Matrix Metalloproteinases, Secreted - genetics | Matrix Metalloproteinases, Secreted - physiology | Protein-Lysine 6-Oxidase - metabolism | Cicatrix - enzymology | Mice | Cytokines - biosynthesis | Index Medicus
Journal Article
Antioxidants & redox signaling, ISSN 1523-0864, 01/2013, Volume 18, Issue 1, pp. 5 - 18
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 2017, Volume 313, Issue 2, pp. H224 - H236
Remodeling | Tissue inhibitor of metalloproteinases 3 | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Myocardial Infarction - genetics | Up-Regulation | Cell Proliferation | Hypertrophy, Left Ventricular - enzymology | Ventricular Function, Left | Coronary Vessels - physiopathology | Humans | Male | Recovery of Function | Myocardial Infarction - therapy | Ventricular Remodeling | Hypertrophy, Left Ventricular - genetics | Time Factors | Ventricular Dysfunction, Left - genetics | Proteolysis | Tissue Inhibitor of Metalloproteinase-3 - biosynthesis | Adenoviridae - genetics | Ventricular Dysfunction, Left - enzymology | Myocardial Infarction - physiopathology | Disease Models, Animal | Myocardial Infarction - enzymology | Tissue Inhibitor of Metalloproteinase-3 - genetics | Coronary Vessels - enzymology | Transduction, Genetic | Signal Transduction | Hypertrophy, Left Ventricular - prevention & control | Mice, Inbred C57BL | Myocardium - pathology | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Myocardium - enzymology | Animals | Adenoviridae - metabolism | Hypertrophy, Left Ventricular - physiopathology | Matrix Metalloproteinases - metabolism | Endothelial Cells - pathology | Genetic Vectors | Endothelial Cells - enzymology | Neovascularization, Physiologic | Genetic Therapy - methods | Myocardial infarction | Regulators | Heart attacks | Echocardiography | Matrix metalloproteinases | Coronary artery | Effects | Density | Endothelial cells | Arteries | Angiogenesis | Matrix | Antiangiogenics | Rodents | Dilation | Protein expression | Disruption | Index Medicus
Journal Article
Basic research in cardiology, ISSN 0300-8428, 9/2017, Volume 112, Issue 5, pp. 1 - 11
Journal Article
Cardiovascular research, ISSN 0008-6363, 2015, Volume 105, Issue 3, pp. 318 - 329
Journal Article
Cardiovascular research, ISSN 0008-6363, 12/2012, Volume 96, Issue 3, pp. 444 - 455
Journal Article