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The Journal of biological chemistry, ISSN 1083-351X, 12/2009, Volume 284, Issue 51, pp. 35839 - 35849
...±-myosin heavy chain-Cre deletor strain. LKB1-KO mice displayed biatrial enlargement with atrial fibrillation and cardiac dysfunction at 4 weeks of age... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Protein Kinases - metabolism | Heart Atria - pathology | Protein Kinases - genetics | Hypertrophy, Left Ventricular - enzymology | Antibiotics, Antineoplastic - pharmacology | Atrial Fibrillation - enzymology | Organ Specificity - genetics | RNA, Messenger - biosynthesis | Peptide Elongation Factor 2 - genetics | Phosphorylation - genetics | Collagen Type I - genetics | Hypertrophy, Left Ventricular - genetics | Hypertrophy, Left Ventricular - pathology | Heart Atria - enzymology | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | Collagen Type II - biosynthesis | Atrial Fibrillation - pathology | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Gene Expression Regulation - genetics | RNA, Messenger - genetics | Carrier Proteins - antagonists & inhibitors | Protein-Serine-Threonine Kinases - genetics | Ribosomal Protein S6 Kinases, 70-kDa - genetics | Myocardium - pathology | Phosphotransferases (Alcohol Group Acceptor) - genetics | Signal Transduction - genetics | Atrial Fibrillation - genetics | Sirolimus - pharmacology | Collagen Type II - genetics | Mice, Knockout | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Gene Expression Regulation - drug effects | Carrier Proteins - genetics | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Collagen Type I - biosynthesis | Myocardium - enzymology | Animals | Carrier Proteins - metabolism | Signal Transduction - drug effects | Fibrosis | Mice | TOR Serine-Threonine Kinases | Peptide Elongation Factor 2 - metabolism | Index Medicus | Mechanisms of Signal Transduction
Journal Article
PloS one, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, pp. e85708 - e85708
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 2014, Volume 306, Issue 7, pp. H1025 - 31
Patients with heart failure (HF) have enhanced systemic IL-1 activity, and, in the experimental mouse model, IL-1 induces left ventricular (LV... 
Heart failure | Inflammation | Interleukins | Systolic dysfunction | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Humans | Heart Failure - physiopathology | Male | Systole | Heart Failure - blood | Ventricular Dysfunction, Left - chemically induced | Time Factors | Ventricular Dysfunction, Left - blood | Ventricular Dysfunction, Left - genetics | Interleukin-6 - blood | Interleukin 1 Receptor Antagonist Protein - pharmacology | Disease Models, Animal | Signal Transduction | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Interleukin-18 - deficiency | Heart Failure - genetics | Heart Failure - metabolism | Interleukin-1beta | Ventricular Dysfunction, Left - physiopathology | Mice, Knockout | Antibodies - pharmacology | Ventricular Dysfunction, Left - metabolism | Animals | Intercellular Signaling Peptides and Proteins - pharmacology | Mice | Interleukin-18 - genetics | Heart Failure - chemically induced | Interleukin-18 - metabolism | Cardiac patients | Physiological aspects | Research | Cardiovascular research | Health aspects | Protein-protein interactions | Plasma | Cytokines | Rodents | Index Medicus | heart failure | systolic dysfunction | Signaling and Stress Response | inflammation | interleukins
Journal Article
PloS one, ISSN 1932-6203, 04/2019, Volume 14, Issue 4, pp. e0215818 - e0215818
...), especially in relation to left ventricular (LV) dysfunction. Furthermore, transmission electron microscopy of the diseased tissue was carried out to investigate if the gene expression changes are translated into ultrastructural alterations... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Humans | Middle Aged | Male | Gene Expression Profiling | RNA, Messenger - metabolism | Case-Control Studies | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Cardiomyopathy, Dilated - drug therapy | Ventricular Dysfunction, Left - genetics | Myocardium - metabolism | Adult | Female | Autophagy - genetics | Nuclear Proteins - genetics | Autophagy-Related Proteins - genetics | Heart Ventricles - pathology | Adrenergic beta-Antagonists - therapeutic use | Cardiomyopathy, Dilated - genetics | RNA, Messenger - genetics | Gene Expression Regulation | Myocardium - pathology | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Cardiomyopathy, Dilated - metabolism | Ventricular Dysfunction, Left - physiopathology | Mineralocorticoid Receptor Antagonists - therapeutic use | Ventricular Dysfunction, Left - metabolism | Sequence Analysis, RNA | Ventricular Dysfunction, Left - drug therapy | Cardiomyopathy, Dilated - physiopathology | Adaptor Proteins, Signal Transducing - genetics | Diuretics - therapeutic use | Heart Ventricles - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Autophagy-Related Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Heart | Ethylenediaminetetraacetic acid | Medical research | RNA | Analysis | Genes | Medicine, Experimental | Aprotinin | Gene expression | Cardiomyopathy, Dilated | Usage | Cardiomyopathy | Genetic aspects | Research | Electron microscopy | Heart diseases | Left ventricular function | Biomedical research | Disease | Remodeling | Kinases | Tissues | Autophagy | Gene sequencing | Proteins | Cardiology | Heart failure | Ribonucleic acid--RNA | Hospitals | Transmission electron microscopy | Molecular modelling | Disease transmission | Dilated cardiomyopathy | Ventricle | Phagocytosis | Index Medicus | Ribonucleic acid
Journal Article
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 12/2015, Volume 66, Issue 6, pp. 1159 - 1167
Journal Article
Circulation research, ISSN 0009-7330, 04/2010, Volume 106, Issue 7, pp. 1253 - 1264
...) showed an obvious baseline cardiac phenotype at young ages. Tg-Nox4 gradually displayed decreased left ventricular (LV... 
Aging | Oxidative stress | Reactive oxygen species | Superoxide | Apoptosis | Hypertrophy | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Aconitate Hydratase - metabolism | Up-Regulation | Cysteine | Cell Proliferation | Uncoupling Agents - pharmacology | Oxidative Stress | Rats, Wistar | Ventricular Function, Left | Apoptosis - drug effects | Mitochondria, Heart - pathology | Humans | NADPH Oxidases - metabolism | Cardiomegaly - pathology | Mitochondria, Heart - drug effects | Myocytes, Cardiac - enzymology | Transfection | Ventricular Dysfunction, Left - genetics | Rotenone - pharmacology | Superoxides - metabolism | Ventricular Dysfunction, Left - pathology | NADPH Oxidases - genetics | Ventricular Dysfunction, Left - enzymology | Disease Models, Animal | Oxidation-Reduction | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mitochondria, Heart - enzymology | Cardiomegaly - physiopathology | Rats | Genotype | Mice, Transgenic | Cardiomegaly - enzymology | NADPH Oxidase 4 | Onium Compounds - pharmacology | NADH Dehydrogenase - metabolism | Ventricular Dysfunction, Left - physiopathology | Aging - pathology | Myocytes, Cardiac - pathology | Phenotype | Animals | Myocytes, Cardiac - drug effects | Fibrosis | Mice | Cardiomegaly - genetics | Aging - metabolism | Index Medicus | apoptosis | aging | superoxide | hypertrophy | oxidative stress
Journal Article
Circulation research, ISSN 1524-4571, 02/2013, Volume 112, Issue 4, pp. 675 - 688
...) have not been explored. OBJECTIVE:To determine the impact of MMP-28 deletion on post-MI remodeling of the left ventricle (LV... 
fibroblast | MMP-28 | inflammation | macrophage phenotype | myocardial infarction | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Myocardial Infarction - blood | Cell Adhesion Molecules - genetics | Cicatrix - etiology | Extracellular Matrix Proteins - biosynthesis | Male | Matrix Metalloproteinases, Secreted - deficiency | Pulmonary Edema - etiology | Myocytes, Cardiac - enzymology | Myofibroblasts - metabolism | Ventricular Dysfunction, Left - enzymology | Ventricular Remodeling - genetics | Female | Myocardial Infarction - physiopathology | Transcription, Genetic | Cytokines - genetics | Heart Rupture - enzymology | Receptors, Cytokine - genetics | Matrix Metalloproteinase 9 - blood | Myocardial Infarction - enzymology | Macrophage Activation - physiology | Macrophages - classification | Cell Adhesion Molecules - biosynthesis | Receptors, Cytokine - biosynthesis | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Ventricular Dysfunction, Left - etiology | Inflammation | Ventricular Remodeling - physiology | Macrophages - enzymology | Mice, Knockout | Collagen - metabolism | Myocardial Infarction - complications | Heart Rupture - etiology | Animals | Pulmonary Edema - enzymology | Matrix Metalloproteinases, Secreted - genetics | Matrix Metalloproteinases, Secreted - physiology | Protein-Lysine 6-Oxidase - metabolism | Cicatrix - enzymology | Mice | Cytokines - biosynthesis | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 1, pp. e0147269 - e0147269
While Huntington's disease (HD) is classified as a neurological disorder, HD patients exhibit a high incidence of cardiovascular events leading to heart... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Tissue Array Analysis | Myocardial Contraction - physiology | Myocardial Contraction - drug effects | Apoptosis - genetics | Gene Expression Profiling | Ventricular Dysfunction, Left - genetics | Ultrasonography | Aging | Adrenergic beta-Agonists - therapeutic use | Real-Time Polymerase Chain Reaction | Heart Ventricles - pathology | Huntington Disease - physiopathology | Disease Models, Animal | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Adrenergic beta-Agonists - pharmacology | Cardiomegaly - physiopathology | Mice, Transgenic | Multiplex Polymerase Chain Reaction | Huntington Disease - metabolism | Nerve Tissue Proteins - genetics | Trinucleotide Repeat Expansion | Ventricular Dysfunction, Left - diagnostic imaging | Ventricular Dysfunction, Left - physiopathology | Huntingtin Protein | Animals | Ventricular Dysfunction, Left - drug therapy | Isoproterenol - pharmacology | Fibrosis | Huntington Disease - genetics | Heart Ventricles - metabolism | Mice | Cardiomegaly - genetics | Heart | Drugs | Huntingtons disease | Laboratories | Cardiomyopathy | Genes | Genomes | Isoproterenol | Proteins | Ultrasonic imaging | Rodents | Fibroblasts | Physiology | Lesions | Heart diseases | Age | Heart failure | Cardiovascular system | Echocardiography | Health risks | Artificial chromosomes | Metabolism | Huntington's disease | Gene expression | Pathology | DNA microarrays | Cardiovascular diseases | Psychiatry | Apoptosis | Structure-function relationships | Index Medicus
Journal Article