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Cardiovascular research, ISSN 1755-3245, 06/2018, Volume 114, Issue 7, pp. 954 - 964
Journal Article
Circulation research, ISSN 0009-7330, 04/2010, Volume 106, Issue 7, pp. 1253 - 1264
...) showed an obvious baseline cardiac phenotype at young ages. Tg-Nox4 gradually displayed decreased left ventricular (LV... 
Aging | Oxidative stress | Reactive oxygen species | Superoxide | Apoptosis | Hypertrophy | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Aconitate Hydratase - metabolism | Up-Regulation | Cysteine | Cell Proliferation | Uncoupling Agents - pharmacology | Oxidative Stress | Rats, Wistar | Ventricular Function, Left | Apoptosis - drug effects | Mitochondria, Heart - pathology | Humans | NADPH Oxidases - metabolism | Cardiomegaly - pathology | Mitochondria, Heart - drug effects | Myocytes, Cardiac - enzymology | Transfection | Ventricular Dysfunction, Left - genetics | Rotenone - pharmacology | Superoxides - metabolism | Ventricular Dysfunction, Left - pathology | NADPH Oxidases - genetics | Ventricular Dysfunction, Left - enzymology | Disease Models, Animal | Oxidation-Reduction | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Mitochondria, Heart - enzymology | Cardiomegaly - physiopathology | Rats | Genotype | Mice, Transgenic | Cardiomegaly - enzymology | NADPH Oxidase 4 | Onium Compounds - pharmacology | NADH Dehydrogenase - metabolism | Ventricular Dysfunction, Left - physiopathology | Aging - pathology | Myocytes, Cardiac - pathology | Phenotype | Animals | Myocytes, Cardiac - drug effects | Fibrosis | Mice | Cardiomegaly - genetics | Aging - metabolism | Index Medicus | apoptosis | aging | superoxide | hypertrophy | oxidative stress
Journal Article
Circulation. Cardiovascular imaging, ISSN 1941-9651, 05/2013, Volume 6, Issue 3, pp. 363 - 372
... associated with revascularization in patients with systolic dysfunction. Methods and Results-The extent of perfusion defects and metabolism-perfusion mismatch... 
Prognosis | Ischemia | Positron emission tomography | Viability | Revascularization | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Radiology, Nuclear Medicine & Medical Imaging | Science & Technology | Predictive Value of Tests | Ventricular Function, Left | Humans | Middle Aged | Male | Positron-Emission Tomography | Ventricular Dysfunction, Left - mortality | Recovery of Function | Patient Selection | Ventricular Dysfunction, Left - therapy | Time Factors | Ventricular Dysfunction, Left - pathology | Myocardial Ischemia - diagnostic imaging | Myocardial Ischemia - physiopathology | Female | Myocardial Ischemia - mortality | Myocardial Stunning - diagnostic imaging | Radiopharmaceuticals | Rubidium Radioisotopes | Myocardial Revascularization - mortality | Myocardial Stunning - physiopathology | Risk Assessment | Risk Factors | Proportional Hazards Models | Myocardial Stunning - therapy | Myocardium - pathology | Treatment Outcome | Chi-Square Distribution | Myocardial Stunning - mortality | Ventricular Dysfunction, Left - diagnostic imaging | Ventricular Dysfunction, Left - physiopathology | Myocardial Ischemia - pathology | Myocardial Perfusion Imaging - methods | Myocardial Stunning - pathology | Stroke Volume | Cardiovascular Agents - therapeutic use | Propensity Score | Myocardial Ischemia - therapy | Fluorodeoxyglucose F18 | Tissue Survival | Aged | Hemodynamics | Myocardial Revascularization - adverse effects | Index Medicus
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 03/2007, Volume 115, Issue 11, pp. 1398 - 1407
Journal Article
Circulation (New York, N.Y.), ISSN 1524-4539, 08/2010, Volume 122, Issue 7, pp. 717 - 728
Journal Article
Journal Article
Heart (British Cardiac Society), ISSN 1355-6037, 10/2016, Volume 102, Issue 20, pp. 1671 - 1679
Journal Article
Cardiovascular research, ISSN 1755-3245, 12/2012, Volume 96, Issue 3, pp. 456 - 465
.... Doxorubicin treatment caused left ventricular dilatation and dysfunction within 6 days. Cardiomyocyte autophagy appeared to be activated in the doxorubicin group, based... 
Heart failure | Autophagy | Doxorubicin | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | AMP-Activated Protein Kinases - metabolism | Microtubule-Associated Proteins - genetics | Ventricular Function, Left | Microtubule-Associated Proteins - metabolism | Heart Failure - physiopathology | Green Fluorescent Proteins - genetics | Ventricular Dysfunction, Left - chemically induced | Autophagy - drug effects | Autophagy-Related Protein-1 Homolog | Starvation - metabolism | Heart Failure - prevention & control | Hypertrophy, Left Ventricular - chemically induced | Time Factors | Hypertrophy, Left Ventricular - pathology | Adenosine Triphosphate - metabolism | Ventricular Dysfunction, Left - pathology | Protein-Serine-Threonine Kinases - metabolism | Green Fluorescent Proteins - metabolism | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Cells, Cultured | Rats | Mice, Transgenic | Heart Failure - metabolism | Antibiotics, Antineoplastic | Heart Failure - pathology | Cathepsin D - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Stroke Volume | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Animals | Energy Metabolism | Myocytes, Cardiac - metabolism | Mice | Ventricular Pressure | Hypertrophy, Left Ventricular - physiopathology | Heart Failure - chemically induced | Starvation - complications | Index Medicus
Journal Article
Journal Article