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Journal Article
Journal Article
Journal of the American Society of Echocardiography, ISSN 0894-7317, 2014, Volume 27, Issue 9, pp. 911 - 939
Journal Article
Journal Article
Basic Research in Cardiology, ISSN 0300-8428, 9/2017, Volume 112, Issue 5, pp. 1 - 14
Advanced glycation end-products (AGEs) have been associated with poorer outcomes after myocardial infarction (MI), and linked with heart failure. Methylglyoxal... 
Myocardial infarction | Methylglyoxal | Advanced glycation end-products | Medicine & Public Health | Extracellular matrix | Bone marrow cells | Glyoxalase-1 | Cardiology | CELLS | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | HEART-FAILURE | BONE-MARROW | DIABETIC CARDIOMYOPATHY | ENDPRODUCTS AGES | DISEASE | MICE | EXPRESSION | Ventricular Function, Left | Humans | Lactoylglutathione Lyase - genetics | Ornithine - analogs & derivatives | Stem Cells - metabolism | Pyruvaldehyde - metabolism | Ventricular Remodeling | Time Factors | Myocardial Infarction - pathology | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Myocardial Infarction - physiopathology | Disease Models, Animal | Imidazoles - metabolism | Genetic Predisposition to Disease | Collagen Type I - metabolism | Signal Transduction | Lactoylglutathione Lyase - metabolism | Ornithine - metabolism | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Myocardium - pathology | Myocardial Infarction - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Ventricular Dysfunction, Left - metabolism | Phenotype | Animals | Glycation End Products, Advanced - metabolism | Stem Cells - pathology | Human Umbilical Vein Endothelial Cells - pathology | Myocardial Infarction - prevention & control | Neovascularization, Physiologic | Apoptosis | Immunohistochemistry | Heart | Enzymes | Collagen | Genetic engineering | Mass spectrometry | Cells | Heart attack | Cell culture | Collagen (type I) | Heart attacks | Arterioles | Remodeling | Damage prevention | Recruitment | Angiogenesis | Rodents | Cell adhesion | Bone marrow | Heart diseases | Age | Advanced glycosylation end products | Transgenic mice | Mass spectroscopy | Glycosylation | c-Kit protein | Overexpression | Stem cells | Cells (biology) | Myocardium | Mice | Infarction | Pyruvaldehyde | In vitro methods and tests | Index Medicus
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 01/2010, Volume 69, Issue 1, pp. 218 - 221
Journal Article
Cardiovascular Research, ISSN 0008-6363, 12/2012, Volume 96, Issue 3, pp. 444 - 455
Age-related diastolic dysfunction has been attributed to an increased passive stiffness, which is regulated by extracellular matrix (ECM). We recently showed... 
Diastolic function | Extracellular matrix | Matrix metalloproteinase | Ageing | Collagen | CARDIAC & CARDIOVASCULAR SYSTEMS | TGF-BETA | ANGIOGENESIS | PROTECTION | HEART-FAILURE | FIBROBLASTS | MATRIX METALLOPROTEINASES | CARDIAC-HYPERTROPHY | PERIOSTIN EXPRESSION | REVEALS | Blood Pressure | Cell Adhesion Molecules - genetics | Phosphorylation | Age Factors | Diastole | Ventricular Function, Left | Male | Systole | Ventricular Dysfunction, Left - genetics | Matrix Metalloproteinase 8 - metabolism | Matrix Metalloproteinase 9 - genetics | Smad2 Protein - genetics | Ventricular Dysfunction, Left - pathology | Ventricular Dysfunction, Left - enzymology | Female | Real-Time Polymerase Chain Reaction | Matrix Metalloproteinase 8 - genetics | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Smad2 Protein - metabolism | Ventricular Dysfunction, Left - etiology | Genotype | Myocardium - pathology | Matrix Metalloproteinase 9 - deficiency | Cell Adhesion Molecules - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Aging - pathology | Mice, Knockout | Collagen - metabolism | Myocardium - enzymology | Phenotype | Animals | Transforming Growth Factor beta - genetics | Fibrosis | Connective Tissue Growth Factor - genetics | Mice | Transforming Growth Factor beta - metabolism | Connective Tissue Growth Factor - metabolism | Aging - metabolism | Index Medicus | Original
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 11/2017, Volume 174, Issue 21, pp. 3696 - 3712
Journal Article
Circulation Research, ISSN 0009-7330, 09/2011, Volume 109, Issue 7, pp. 783 - 793
RATIONALE:Pressure overload cardiac hypertrophy, a risk factor for heart failure, is associated with reduced mitochondrial fatty acid oxidation (FAO) and... 
heart failure | cardiac hypertrophy | mitochondria | gene expression | TRANSCRIPTIONAL COACTIVATOR PGC-1-ALPHA | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MITOCHONDRIAL | ENERGETICS | METABOLISM | FATTY-ACID OXIDATION | CARDIAC-HYPERTROPHY | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | MICE | CONTRACTILE FUNCTION | HEMATOLOGY | Blood Pressure | Mitochondria, Heart - metabolism | Oxidative Stress | Ventricular Function, Left | Heart Failure - physiopathology | Heart Failure - prevention & control | Ventricular Dysfunction, Left - genetics | Myocardium - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Trans-Activators - genetics | Ultrasonography | Aging | Heart Failure - diagnostic imaging | Energy Metabolism - genetics | Fatty Acids - metabolism | Heart Failure - etiology | Disease Models, Animal | Myocardial Contraction | Oxidation-Reduction | Gene Expression Regulation | Oxidative Phosphorylation | Ventricular Dysfunction, Left - etiology | Heart Failure - genetics | Heart Failure - metabolism | Disease Progression | Ventricular Dysfunction, Left - diagnostic imaging | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Animals | Trans-Activators - deficiency | Hypertrophy, Left Ventricular - complications | Glucose - metabolism | Mice | Transcription Factors | Hypertrophy, Left Ventricular - physiopathology | Hexokinase - metabolism | Hypertrophy, Left Ventricular - diagnostic imaging | Index Medicus
Journal Article
Journal Article