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The Journal of Infectious Diseases, ISSN 0022-1899, 1/2009, Volume 199, Issue 2, pp. 263 - 271
Journal Article
Lancet Neurology, The, ISSN 1474-4422, 2009, Volume 8, Issue 7, pp. 628 - 634
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 04/2017, Volume 120, Issue 9, pp. 1453 - 1465
RATIONALE:Angiogenesis occurs after ischemic injury to skeletal muscle, and enhancing this response has been a therapeutic goal. However, to appropriately... 
Erythrocyte | Microvascular dysfunction | smooth muscle | Angiogenesis | Microcirculation | CAPILLARY NETWORKS | CARDIAC & CARDIOVASCULAR SYSTEMS | angiogenesis | LIMB ISCHEMIA | OXYGEN-TRANSPORT | THERAPEUTIC ANGIOGENESIS | microcirculation | FLOW | erythrocyte | SKELETAL-MUSCLE | microvascular dysfunction | BLOOD-CELL DYNAMICS | IMAGE-ANALYSIS | IN-VIVO | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Microvessels - physiopathology | Venules - physiopathology | Hindlimb | Image Interpretation, Computer-Assisted | Male | Venules - diagnostic imaging | Cell Hypoxia | Time Factors | Arterioles - physiopathology | Microvessels - diagnostic imaging | Hypoxia - diagnostic imaging | Disease Models, Animal | Ischemia - diagnostic imaging | Muscle, Skeletal - blood supply | Microscopy, Video - methods | Capillaries - physiopathology | Mice, Inbred C57BL | Cellular Microenvironment | Ischemia - blood | Ischemia - physiopathology | Microscopy, Confocal - methods | Regional Blood Flow | Oxygen - blood | Hypoxia - blood | Animals | Erythrocytes - metabolism | Hypoxia - physiopathology | Arterioles - diagnostic imaging | Capillaries - diagnostic imaging | Neovascularization, Physiologic | Blood cells | Erythrocytes | Spatial discrimination | Smooth muscle | Skeletal muscle | Blood | Vascularization | Musculoskeletal system | Ischemia | Actin | Spatial heterogeneity | Hypoxia | Microvasculature | Circulatory system | Capillaries | Structure-function relationships
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 01/2013, Volume 304, Issue 1, pp. 33 - 41
Patients with autosomal dominant polycystic kidney disease have a high prevalence of hypertension and structural vascular abnormalities, such as intracranial... 
Ischemia-reperfusion injury | Peroxisome proliferator-activated receptor | Polycystic kidney disease 2 | Autosomal dominant polycystic kidney disease | Nitric oxide | CELLS | nitric oxide | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | RAT | PEROXYNITRITE | POLYCYSTIC KIDNEY-DISEASE | autosomal dominant polycystic kidney disease | HAPLOINSUFFICIENCY | ischemia-reperfusion injury | NITRIC-OXIDE | RESISTANCE VESSELS | PERIPHERAL VASCULAR DISEASE | MICE | peroxisome proliferator-activated receptor | EXPRESSION | polycystic kidney disease 2 | Venules - physiopathology | Calcium - metabolism | Capillary Permeability - drug effects | Endothelium, Vascular - drug effects | Male | Venules - drug effects | PPAR gamma - metabolism | Time Factors | Arterioles - physiopathology | Myocytes, Smooth Muscle - drug effects | Thiazolidinediones - pharmacology | Polycystic Kidney, Autosomal Dominant - drug therapy | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Endothelial Cells - metabolism | Genotype | Mice, Transgenic | Arterioles - drug effects | Disease Progression | TRPP Cation Channels - genetics | Hypertension - metabolism | Mesentery - blood supply | Polycystic Kidney, Autosomal Dominant - metabolism | Phenotype | Arterioles - metabolism | Endothelium, Vascular - metabolism | Mice | Oxidative Stress - drug effects | Vasodilation - drug effects | Polycystic Kidney, Autosomal Dominant - complications | Myography | Muscle, Smooth, Vascular - metabolism | Muscle, Smooth, Vascular - physiopathology | Renal Insufficiency - etiology | Hypertension - etiology | Dose-Response Relationship, Drug | Renal Insufficiency - physiopathology | Polycystic Kidney, Autosomal Dominant - genetics | Superoxides - metabolism | Myocytes, Smooth Muscle - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | TRPP Cation Channels - metabolism | Vasodilator Agents - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Endothelium, Vascular - physiopathology | Renal Insufficiency - metabolism | Antioxidants - pharmacology | Hypertension - physiopathology | Venules - metabolism | Animals | PPAR gamma - agonists | Peroxynitrous Acid - metabolism | Reperfusion Injury - physiopathology | Heterozygote | Polycystic Kidney, Autosomal Dominant - physiopathology | Nitric Oxide - metabolism | Endothelial Cells - drug effects | Dilatation | Physiological aspects | Blood vessels | Smooth muscle | Development and progression | Research | Health aspects | Polycystic kidney disease | Endothelium | Vascular Biology and Microcirculation
Journal Article