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Journal of Clinical Investigation, ISSN 0021-9738, 05/2015, Volume 125, Issue 5, pp. 1901 - 1912
Reversal of HIV-1 latency by small molecules is a potential cure strategy. This approach will likely require effective drug combinations to achieve high levels... 
SUPPRESSIVE ANTIRETROVIRAL THERAPY | MEDICINE, RESEARCH & EXPERIMENTAL | VIRUS REACTIVATION | ACTIVATION | RESERVOIR | IN-VIVO | GENE-EXPRESSION | CD4(+) T-CELLS | HISTONE DEACETYLASE INHIBITORS | NF-KAPPA-B | LIFELONG PERSISTENCE | Anti-HIV Agents - pharmacology | Transcription, Genetic - drug effects | Lymphokines - secretion | Humans | Middle Aged | RNA, Messenger - analysis | Male | Virus Latency - drug effects | Anti-HIV Agents - administration & dosage | HIV-1 - physiology | Phorbol Esters - pharmacology | Adult | Anti-HIV Agents - therapeutic use | Female | CD4-Positive T-Lymphocytes - virology | Drug Evaluation, Preclinical | Bryostatins - pharmacology | HIV-1 - drug effects | Lymphocyte Activation | HIV Infections - virology | Protein Kinase C - drug effects | Cells, Cultured | RNA, Viral - analysis | Virion - metabolism | Azepines - pharmacology | Drug Synergism | Triazoles - pharmacology | CD4-Positive T-Lymphocytes - secretion | HIV Infections - drug therapy | Histone Deacetylase Inhibitors - pharmacology | Disulfiram - pharmacology | CD4-Positive T-Lymphocytes - drug effects | Physiological aspects | Research | RNA | T cells | Drug therapy | HIV infection | Studies | Statistical analysis | Cytokines | Acquired immune deficiency syndrome--AIDS | Lymphocytes | T cell receptors | Mathematical models | Gene expression | HIV | Infectious disease | AIDS
Journal Article
Science Translational Medicine, ISSN 1946-6234, 06/2013, Volume 5, Issue 190, pp. 190ra79 - 190ra79
Journal Article
Journal Article
Nature, ISSN 0028-0836, 07/2016, Volume 535, Issue 7610, pp. 169 - 172
Ebola viruses (EBOVs) are responsible for repeated outbreaks of fatal infections, including the recent deadly epidemic in West Africa. There are currently no... 
SYSTEM | NIEMANN-PICK C1 | ENTRY | CHOLESTEROL | SMALL-MOLECULE INHIBITORS | CRYSTAL-STRUCTURE | MULTIDISCIPLINARY SCIENCES | ANTIBODY | RECEPTOR | INFECTION | HIV-1 VIRION FUSION | Temperature | Humans | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Antiviral Agents - metabolism | Endosomes - metabolism | Protein Subunits - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Ebolavirus - drug effects | Ibuprofen - chemistry | Antiviral Agents - chemistry | Endosomes - drug effects | Viral Envelope Proteins - metabolism | Conserved Sequence | Marburgvirus - chemistry | Anti-Inflammatory Agents, Non-Steroidal - metabolism | Binding Sites | Membrane Fusion - drug effects | Toremifene - metabolism | Toremifene - pharmacology | Cell Line | Antiviral Agents - pharmacology | Ibuprofen - pharmacology | Models, Molecular | Protein Structure, Quaternary - drug effects | Ibuprofen - metabolism | Virus Attachment - drug effects | Ebolavirus - chemistry | Viral Envelope Proteins - antagonists & inhibitors | Protein Stability - drug effects | Viral Envelope Proteins - chemistry | Hydrophobic and Hydrophilic Interactions | Protein Binding | Ligands | Protein Subunits - chemistry | Toremifene - chemistry | Ebola virus | Glycoproteins | Host-virus relationships | Observations | Health aspects | Drug interactions | Drug therapy | Binding sites | Viral infections | Crystal structure
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2013, Volume 110, Issue 10, pp. 3991 - 3996
Journal Article
Nature, ISSN 0028-0836, 10/2015, Volume 526, Issue 7572, pp. 218 - 223
HIV-1 Nef and the unrelated mouse leukaemia virus glycosylated Gag (glycoGag) strongly enhance the infectivity of HIV-1 virions produced in certain cell types... 
CYTOPLASMIC DOMAIN | CELL-SURFACE CD4 | GLYCOSYLATED-GAG | VIRION FUSION | REPLICATION | PRIMARY T-LYMPHOCYTES | MULTIDISCIPLINARY SCIENCES | IMMUNODEFICIENCY-VIRUS TYPE-1 | CD4 DOWN-REGULATION | MURINE LEUKEMIA-VIRUS | OPTIMAL VIRAL INFECTIVITY | Humans | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - pharmacology | Virion - chemistry | Host-Pathogen Interactions - drug effects | Membrane Proteins - pharmacology | Neoplasm Proteins - metabolism | Receptors, Cell Surface - antagonists & inhibitors | Membrane Proteins - deficiency | HIV-1 - growth & development | HIV-1 - physiology | HIV-1 - chemistry | Gene Deletion | nef Gene Products, Human Immunodeficiency Virus - metabolism | Membrane Proteins - metabolism | Cell Line | Virus Replication - drug effects | nef Gene Products, Human Immunodeficiency Virus - deficiency | HIV-1 - drug effects | Down-Regulation | HIV Infections - virology | Virion - physiology | CD4-Positive T-Lymphocytes - metabolism | Receptors, Cell Surface - metabolism | Gene Products, gag - metabolism | Protein Transport | Virion - drug effects | Membrane Proteins - antagonists & inhibitors | Receptors, Cell Surface - deficiency | HIV Infections - drug therapy | Neoplasm Proteins - deficiency | Virion - growth & development | Leukemia Virus, Murine - chemistry | Development and progression | Genetic aspects | Virulence (Microbiology) | Viral proteins | Properties | HIV infection | Proteins | Genomes | Immunology | Mutation | Human immunodeficiency virus--HIV | Cells
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 3, p. e17225
Background: Japanese encephalitis virus (JEV) induces neuroinflammation with typical features of viral encephalitis, including inflammatory cell infiltration,... 
PROGENITOR CELLS | NEURAL STEM-CELLS | SUBVENTRICULAR ZONE NEUROGENESIS | NEURONAL DEATH | FOCAL CEREBRAL-ISCHEMIA | BIOLOGY | NITRIC-OXIDE | ADULT HIPPOCAMPAL NEUROGENESIS | CENTRAL-NERVOUS-SYSTEM | NECROSIS-FACTOR-ALPHA | BLOOD-BRAIN-BARRIER | Solubility - drug effects | Encephalitis, Japanese - complications | Inflammation - pathology | Neurons - pathology | Encephalitis, Japanese - virology | Minocycline - pharmacology | Brain - virology | Cell Lineage - drug effects | Inflammation - complications | Inflammation Mediators - metabolism | Microglia - pathology | Neurogenesis - drug effects | Neurons - drug effects | Disease Models, Animal | Chemokines - biosynthesis | Microglia - drug effects | Neural Stem Cells - drug effects | Encephalitis Virus, Japanese - physiology | Virion - metabolism | Neural Stem Cells - pathology | Virion - drug effects | Brain - drug effects | Cell Movement - drug effects | Animals | Encephalitis, Japanese - pathology | Cell Differentiation - drug effects | Brain - pathology | Encephalitis Virus, Japanese - drug effects | Cell Proliferation - drug effects | Mice | Virus diseases | Inflammation | Research | Neurons | Analysis | Japanese encephalitis | Neuropathogenesis | Cell culture | Animal models | Leukemia | Memory | Viruses | Inflammatory response | Infections | Kinases | Neurogenesis | Cell activation | Cell growth | Epidermal growth factor | Neuroblasts | Rodents | Human immunodeficiency virus--HIV | Neurospheres | Minocycline | Degeneration | Trends | Conditioning | Vector-borne diseases | Encephalitis | Cytokines | Complications | Microglia | Brain research | Nitric oxide | Stem cells | Infiltration | Chemokines | Animal cognition | Side effects | neurospheres | Differentiation | HIV | Human immunodeficiency virus
Journal Article
PLoS Biology, ISSN 1544-9173, 12/2012, Volume 10, Issue 12, p. e1001448
Journal Article
Antiviral Research, ISSN 0166-3542, 07/2017, Volume 143, pp. 176 - 185
Journal Article
Journal Article