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1. Full Text Sphingolipid metabolites in inflammatory disease
by MacEyka, Michael and Spiegel, Sarah
Nature, ISSN 0028-0836, 2014, Volume 510, Issue 7503, pp. 58 - 67
Sphingolipids are ubiquitous building blocks of eukaryotic cell membranes. Progress in our understanding of sphingolipid metabolism, state-of-the-art... 
INTESTINAL INFLAMMATION | CERAMIDE BIOSYNTHESIS | SPHINGOSINE KINASE 1 | INFLUENZA-VIRUS INFECTION | APOLIPOPROTEIN M | PROTEIN-COUPLED RECEPTOR | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | AIRWAY INFLAMMATION | TNF-ALPHA | LYMPHOCYTE EGRESS | Lymphocytes - metabolism | Tumor Necrosis Factor-alpha - metabolism | Lysophospholipids - metabolism | Ceramides - metabolism | Signal Transduction | Humans | Sphingolipids - metabolism | Adipokines - metabolism | Lymphocytes - cytology | Inflammation - metabolism | Sphingosine - analogs & derivatives | Animals | Endothelium - metabolism | Inflammation - drug therapy | Sphingosine - metabolism | Autoimmune Diseases - metabolism | Autoimmune Diseases - pathology | Physiological aspects | Lipid research | Inflammation | Research | Lipid metabolism | Medical case management | Proteins | Enzymes | Lipoproteins | Multiple sclerosis | Metabolites | Disease | Lipids | Metabolism | Cholesterol | Inflammatory diseases
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2. Full Text Endoplasmic reticulum stress in liver disease
by Malhi, Harmeet and Kaufman, Randal J
Journal of Hepatology, ISSN 0168-8278, 2010, Volume 54, Issue 4, pp. 795 - 809
The unfolded protein response (UPR) is activated upon the accumulation of misfolded proteins in the endoplasmic reticulum (ER) that are sensed by the binding... 
Gastroenterology and Hepatology | Hepatosteatosis | Endoplasmic reticulum stress | Unfolded protein response | Liver injury | MESSENGER-RNA TRANSLATION | UNFOLDED-PROTEIN RESPONSE | TRANSCRIPTION FACTOR XBP-1 | NECROSIS-FACTOR-ALPHA | ISCHEMIA-REPERFUSION INJURY | PROGRAMMED CELL-DEATH | GLUCOSE-REGULATED PROTEINS | HEPATITIS-C VIRUS | HUMAN HEPATOCELLULAR-CARCINOMA | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | Hepatitis C, Chronic - metabolism | Fatty Liver - metabolism | Cholestasis - metabolism | eIF-2 Kinase - metabolism | Humans | Liver Diseases - pathology | Stress, Physiological | Endoplasmic Reticulum - metabolism | Hyperhomocysteinemia - metabolism | Hepatocytes - pathology | Hepatocytes - metabolism | Unfolded Protein Response | Liver Diseases, Alcoholic - metabolism | Activating Transcription Factor 6 - metabolism | Inflammation - metabolism | Animals | Models, Biological | Liver Neoplasms - metabolism | Non-alcoholic Fatty Liver Disease | Protein Conformation | Liver Diseases - metabolism | Reperfusion Injury - metabolism | Apoptosis | Carcinoma, Hepatocellular - metabolism | Inositol | Immunoglobulins | Stress (Physiology) | Ribonuclease | Glucose | Genetic translation | Dextrose | Messenger RNA | Proteases | Proteolysis | Analysis | Hepatitis C | Adenylic acid | Hepatitis C virus | Health aspects | Protein binding | Tumors
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3. Full Text Type I interferons in infectious disease
by McNab, Finlay and Mayer-Barber, Katrin and Sher, Alan and Wack, Andreas and O'Garra, Anne
Nature Reviews Immunology, ISSN 1474-1733, 01/2015, Volume 15, Issue 2, pp. 87 - 103
Type I interferons (IFNs) have diverse effects on innate and adaptive immune cells during infection with viruses, bacteria, parasites and fungi, directly... 
NATURAL-KILLER-CELLS | IFN-ALPHA-BETA | INFLUENZA-VIRUS INFECTION | NITRIC-OXIDE SYNTHASE | INNATE IMMUNE-RESPONSE | EXPERIMENTAL VIRAL-INFECTIONS | INTRACELLULAR BACTERIAL-INFECTION | IMMUNOLOGY | LYMPHOCYTIC CHORIOMENINGITIS VIRUS | CHRONIC MUCOCUTANEOUS CANDIDIASIS | CD8 T-CELLS | Virus Diseases - immunology | Signal Transduction | Humans | Communicable Diseases - genetics | Mycoses - genetics | Virus Diseases - metabolism | Interferon Type I - immunology | Communicable Diseases - metabolism | Bacterial Infections - genetics | Animals | Bacterial Infections - immunology | Mycoses - metabolism | Communicable Diseases - immunology | Interferon Type I - genetics | Interferon Type I - metabolism | Parasitic Diseases - immunology | Parasitic Diseases - genetics | Virus Diseases - genetics | Mycoses - immunology | Bacterial Infections - metabolism | Parasitic Diseases - metabolism | Physiological aspects | Development and progression | Interferon | Immune response | Research | Communicable diseases
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4. Full Text Inflammasomes in liver diseases
by Szabo, Gyongyi and Csak, Timea
Journal of Hepatology, ISSN 0168-8278, 2012, Volume 57, Issue 3, pp. 642 - 654
Summary Inflammation is a common element in the pathogenesis of most chronic liver diseases that lead to fibrosis and cirrhosis. Inflammation is characterized... 
Gastroenterology and Hepatology | Drug induced liver injury | Viral hepatitis | Interleukin-1ß | Inflammasomes | Steatohepatitis | Fibrosis | Endotoxin | Ischaemiareperfusion | Caspase-1 | ANTHRAX LETHAL TOXIN | ACETAMINOPHEN-INDUCED HEPATOTOXICITY | INTERLEUKIN-1 RECEPTOR ANTAGONIST | INNATE IMMUNE-RESPONSE | NECROSIS-FACTOR-ALPHA | HEPATIC ISCHEMIA/REPERFUSION INJURY | Interleukin-1 beta | CASPASE RECRUITMENT DOMAIN | ISCHEMIA-REPERFUSION INJURY | III SECRETION APPARATUS | VIRUS CORE PROTEIN | GASTROENTEROLOGY & HEPATOLOGY | Calcium-Binding Proteins - metabolism | Inflammasomes - metabolism | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Caspase 1 - metabolism | Nuclear Proteins - metabolism | Apoptosis Regulatory Proteins - metabolism | CARD Signaling Adaptor Proteins - metabolism | Carrier Proteins - metabolism | Interleukin-1beta - metabolism | Liver Diseases - enzymology | Adaptor Proteins, Signal Transducing - metabolism | Liver Diseases - metabolism | DNA-Binding Proteins | Oxidases | Niacinamide | Interleukins | Acetaminophen | Carbon tetrachloride | Melanoma | Biological response modifiers | Hepatitis C virus | Muscle proteins | Hepatitis C | Mitogens | Adenosine triphosphate
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5. Full Text Neuropeptides and Microglial Activation in Inflammation, Pain, and Neurodegenerative Diseases
by Carniglia, Lila and Ramírez, Delia and Durand, Daniela and Saba, Julieta and Turati, Juan and Caruso, Carla and Scimonelli, Teresa N and Lasaga, Mercedes
Mediators of Inflammation, ISSN 0962-9351, 2017, Volume 2017, pp. 5048616 - 23
Microglial cells are responsible for immune surveillance within the CNS. They respond to noxious stimuli by releasing inflammatory mediators and mounting an... 
EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | GENE-RELATED PEPTIDE | VASOACTIVE-INTESTINAL-PEPTIDE | MELANOCYTE-STIMULATING HORMONE | IMPROVES FUNCTIONAL RECOVERY | ATTENUATES MECHANICAL ALLODYNIA | NECROSIS-FACTOR-ALPHA | MELANOCORTIN TYPE-3 RECEPTOR | IMMUNOLOGY | HUMAN-IMMUNODEFICIENCY-VIRUS | NF-KAPPA-B | CELL BIOLOGY | Microglia - metabolism | Vasoactive Intestinal Peptide - metabolism | Inflammation Mediators | Tachykinins - metabolism | Humans | Leptin - metabolism | Neuralgia - metabolism | Neuropeptides - metabolism | Adrenomedullin - metabolism | Neurodegenerative Diseases - metabolism | Pain - metabolism | Macrophage Activation | Ghrelin - metabolism | Neuropeptide Y - metabolism | Pro-Opiomelanocortin - metabolism | Inflammation - metabolism | Animals | Neuroglia - metabolism | Calcitonin Gene-Related Peptide - metabolism | Physiological aspects | Neuropeptides | Nervous system | Degeneration | Health aspects | Pain | Cytokines | Neurodegeneration | Rodents | Tumor necrosis factor-TNF | Inflammation | Kinases | Alzheimers disease | Chemokines | Apoptosis | Review
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6. Full Text Toll-like receptors and adaptor molecules in liver disease: Update
by Seki, Ekihiro and Brenner, David A
Hepatology, ISSN 0270-9139, 07/2008, Volume 48, Issue 1, pp. 322 - 335
Toll-like receptors (TLRs) are pattern recognition receptors that recognize pathogen-associated molecular patterns and signal through adaptor molecules,... 
INNATE IMMUNE-RESPONSES | I INTERFERON INDUCTION | NEGATIVE REGULATION | PRIMARY BILIARY-CIRRHOSIS | HEPATITIS-C-VIRUS | KILLER CELLS/INTERFERON-GAMMA | IFN-BETA PROMOTER | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | PROINFLAMMATORY CYTOKINE PRODUCTION | PATTERN-RECOGNITION RECEPTORS | Hepatitis C, Chronic - metabolism | RNA Helicases - metabolism | Acute Disease | Signal Transduction | Humans | Hepatocytes - metabolism | Receptors, Pattern Recognition - metabolism | Animals | Cytosol - metabolism | Toll-Like Receptors - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Liver Diseases - metabolism | Chronic Disease
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