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Journal of neurochemistry, ISSN 1471-4159, 2009, Volume 110, Issue 1, pp. 400 - 411
... inhibitor, and proteinase K inhibited the internalization of extracellular monomeric α‐syn into BV‐2 cells, and the addition of monosialoganglioside GM1 ameliorated the inhibition... 
gangliosides | endocytosis | lipid raft | α‐synuclein | microglia | Endocytosis | Lipid raft | Gangliosides | α-synuclein | Microglia | CELLS | HUMAN PLASMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | alpha-synuclein | NEUROSCIENCES | CELLULAR UPTAKE | MULTIPLE SYSTEM ATROPHY | MEDIATED ENDOCYTOSIS | DEPENDENT ENDOCYTOSIS | INFLUENZA-VIRUS | LEWY BODIES | PARKINSONS-DISEASE | CHOLERA-TOXIN | Microglia - metabolism | Dynamins - metabolism | Dynamins - drug effects | Membrane Microdomains - metabolism | Coculture Techniques | Humans | Clathrin - drug effects | Extracellular Space - drug effects | Protein Transport - physiology | Caveolins - metabolism | Protein Transport - drug effects | Glucosyltransferases - metabolism | Extracellular Space - metabolism | Encephalitis - physiopathology | Encephalitis - metabolism | Parkinson Disease - metabolism | Caveolins - drug effects | Endopeptidase K - pharmacology | Glucosyltransferases - antagonists & inhibitors | Cell Line | Microglia - drug effects | Endocytosis - drug effects | Enzyme Inhibitors - pharmacology | Neurodegenerative Diseases - metabolism | Parkinson Disease - physiopathology | Clathrin - metabolism | G(M1) Ganglioside - pharmacology | Endocytosis - physiology | Animals | Neurodegenerative Diseases - physiopathology | Membrane Microdomains - drug effects | Endopeptidase K - metabolism | Cell Line, Tumor | Mice | alpha-Synuclein - metabolism | G(M1) Ganglioside - metabolism
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2013, Volume 110, Issue 43, pp. 17380 - 17385
Journal Article
Biochemical journal, ISSN 1470-8728, 2004, Volume 377, Issue 1, pp. 159 - 169
Non-phagocytic eukaryotic cells can internalize particles <1 µm in size, encompassing pathogens, liposomes for drug delivery or lipoplexes applied in gene delivery... 
particle size | COATED PIT FORMATION | CHOLESTEROL | TRANSFECTION | VIRUS ENTRY | MAMMALIAN-CELLS OCCURS | UMCG Approved | gene delivery | EPITHELIAL-CELLS | SIGNALING PATHWAY | microsphere uptake | endocytosis | CLASS-I MOLECULES | caveolae | CHOLERA-TOXIN
Journal Article
Journal Article
Molecules (Basel, Switzerland), ISSN 1420-3049, 2019, Volume 24, Issue 4, p. 723
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 5, p. e96977
Journal Article
Journal Article
Nature medicine, ISSN 1546-170X, 2011, Volume 17, Issue 5, pp. 589 - 595
...HCV is a major cause of liver cirrhosis and hepatocellular carcinoma. Current antiviral treatment is limited by drug resistance, toxicity and high costs (1... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | B TYPE-I | NEUTRALIZING ANTIBODIES | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | FACTOR RECEPTOR | CELL BIOLOGY | EPIDERMAL-GROWTH-FACTOR | CELL TRANSMISSION | HUMAN LIVER | INFECTION | ERLOTINIB | Erlotinib Hydrochloride | RNA, Small Interfering - genetics | Receptor, Epidermal Growth Factor - genetics | Hepatitis C - therapy | Humans | Virus Internalization - drug effects | RNA Interference | Membrane Proteins - physiology | Base Sequence | Hepacivirus - physiology | Hepatitis C - prevention & control | Receptor, Epidermal Growth Factor - physiology | Hepacivirus - drug effects | Cell Line | Antiviral Agents - pharmacology | Receptor, EphA2 - physiology | Tetraspanin 28 | Claudin-1 | Animals | Hepatitis C - virology | Receptor, EphA2 - genetics | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Antigens, CD - physiology | Hepatitis C - physiopathology | Ligands | Mice | Protein Kinase Inhibitors - pharmacology | Receptor, EphA2 - antagonists & inhibitors | Quinazolines - pharmacology | Host-Pathogen Interactions - physiology | Ephrin A | Antiviral agents | Care and treatment | Patient outcomes | Physiological aspects | Research | Hepatitis C | Hepatitis | Viruses | Drug therapy | Antiviral drugs | Receptor, EphA2 | Hépatology and Gastroenterology | Antigens, CD81 | Antiviral Agents | Quinazolines | Virus Internalization | Membrane Proteins | Host-Pathogen Interactions | Life Sciences | Human health and pathology | Protein Kinase Inhibitors | RNA, Small Interfering | Hepacivirus | Antigens, CD | Receptor, Epidermal Growth Factor | Liver | HCV escape variants | Cell-cell transmission | genetics | pharmacology | Antiviral | therapy | physiology | drug effects | prevention & control | antagonists & inhibitors | physiopathology | Phosphotyrosine kinase | virology
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 139, Issue 3, pp. 953 - 964.e4
Background & Aims Hepatitis C virus (HCV) infection is a challenge to prevent and treat because of the rapid development of drug resistance and escape... 
Gastroenterology and Hepatology | Genetic Barrier | Antiviral | Treatment | Receptor | Host Factor | B TYPE-I | LIVER-TRANSPLANTATION | SOURCE OUTBREAK | ESCAPE | PROTECT | RESPONSES | BROADLY NEUTRALIZING ANTIBODIES | GASTROENTEROLOGY & HEPATOLOGY | BINDING | VIRAL ENTRY | Antibodies, Monoclonal - toxicity | Antibody Specificity | Cricetulus | Antiviral Agents - toxicity | Humans | Hepacivirus - genetics | Hepacivirus - pathogenicity | Antiviral Agents - metabolism | Virus Internalization - drug effects | Dose-Response Relationship, Drug | Hepatitis C - immunology | Hepatitis C - prevention & control | Hepatocytes - drug effects | CHO Cells | Binding, Competitive | Hepacivirus - drug effects | Cell Survival - drug effects | Antiviral Agents - pharmacology | Cricetinae | Antibodies, Monoclonal - pharmacology | Genotype | Membrane Proteins - immunology | Epitopes | Hepatocytes - immunology | Claudin-1 | Hep G2 Cells | Animals | Membrane Proteins - antagonists & inhibitors | Hepatocytes - virology | Antibodies, Monoclonal - metabolism | Binding Sites, Antibody | Prevention | Antiviral agents | Disease transmission | Gastrointestinal agents | Immunoglobulin G | Fluorescence | Monoclonal antibodies | Drug resistance | Hepatitis C | Hepatitis C virus | Health aspects | Infection control | Hépatology and Gastroenterology | Cell Survival | Antiviral Agents | Antibodies, Monoclonal | Virus Internalization | Membrane Proteins | Life Sciences | Human health and pathology | Hepatocytes | Hepacivirus
Journal Article