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World Journal of Gastroenterology, ISSN 1007-9327, 07/2014, Volume 20, Issue 27, pp. 9154 - 9161
Journal Article
Pain, ISSN 0304-3959, 02/2018, Volume 159, Issue 2, pp. 331 - 341
T lymphocytes play a pivotal role in endogenous regulation of inflammatory visceral pain. The analgesic activity of T lymphocytes is dependent on their... 
T lymphocytes | Vaccination | Visceral sensitivity | OPIOIDS | RECEPTOR | RELEASE | NEUROSCIENCES | BETA-ENDORPHIN | CLINICAL NEUROLOGY | CELL RESPONSE | NEURONS | ANESTHESIOLOGY | ENDOGENOUS REGULATION | EXPRESSION | MODULATION | ANALGESIA
Journal Article
2007, ISBN 9780849328978, xv, 528
about the book Standing alone as the first definitive and comprehensive book on the subject, this guide describes the most recent studies on the brain-gut... 
Abdominal pain | Other branches of medicine | Neurology | Pain Management | Gastroenterology | Viscera - physiology | Pelvic pain | Chronic pain | Chest pain
Book
Journal Article
Seminars in Reproductive Medicine, ISSN 1526-8004, 01/2017, Volume 35, Issue 1, pp. 088 - 097
Journal Article
Journal Article
Pancreatology, ISSN 1007-9327, 2014, Volume 14, Issue 3, pp. S13 - S13
AIM: To systematically characterize specific pain patterns in the most frequent pancreatic diseases.METHODS: Pain in patients with chronic pancreatitis(n =... 
pain | Chronic | neoplasm | pancrea | Abdominal | Pancreatic | Endocrinology & Metabolism | Gastroenterology and Hepatology
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 148, Issue 1, pp. 148 - 157.e7
Background & Aims Chronic stress alters the hypothalamic–pituitary–adrenal axis, increases gut motility, and increases the perception of visceral pain. We... 
Gastroenterology and Hepatology | HPA | Epigenetic Modification | Pain Sensitivity | Visceral Hyperalgesia | METHYLATION | SENSORY FIBERS | WATER AVOIDANCE STRESS | GUT | RECEPTOR | TRPV1 | GASTROENTEROLOGY & HEPATOLOGY | EXPOSURE | Epigenesis, Genetic | E1A-Associated p300 Protein - genetics | Male | E1A-Associated p300 Protein - metabolism | Ganglia, Spinal - physiopathology | Receptors, Glucocorticoid - metabolism | Visceral Pain - diagnosis | RNA Interference | Time Factors | Histones | Transcription, Genetic | Acetylation | Disease Models, Animal | Abdominal Pain - diagnosis | DNA (Cytosine-5-)-Methyltransferase 1 | Visceral Pain - physiopathology | Abdominal Pain - metabolism | Rats, Sprague-Dawley | Visceral Pain - genetics | Receptor, Cannabinoid, CB1 - genetics | Hyperalgesia - genetics | Abdominal Pain - genetics | Pain Threshold | Chronic Disease | Stress, Psychological - complications | Stress, Psychological - physiopathology | Hyperalgesia - psychology | Stress, Psychological - psychology | DNA (Cytosine-5-)-Methyltransferases - metabolism | TRPV Cation Channels - metabolism | DNA Methylation | Stress, Psychological - metabolism | Visceral Pain - psychology | Hyperalgesia - diagnosis | Promoter Regions, Genetic | Hyperalgesia - metabolism | Abdominal Pain - physiopathology | Gene Expression Regulation | Visceral Pain - metabolism | TRPV Cation Channels - genetics | Hyperalgesia - physiopathology | Abdominal Pain - psychology | DNA (Cytosine-5-)-Methyltransferases - genetics | Receptor, Cannabinoid, CB1 - metabolism | Stress, Psychological - genetics | Animals | Receptors, Glucocorticoid - genetics | Pain Measurement | Ganglia, Spinal - metabolism | Stress (Psychology) | Epigenetic inheritance | Genes | epigenetic modification | pain sensitivity | Visceral hyperalgesia
Journal Article
Journal Article