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PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e53338 - e53338
Photoreceptor degeneration is the most critical cause of visual impairment in age-related macular degeneration (AMD). In neovascular form of AMD, severe... 
RETINAL GANGLION-CELLS | MITOCHONDRIAL APOPTOSIS | BRILLIANT BLUE-G | P2X RECEPTORS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | INTRACEREBRAL HEMORRHAGE | MACULAR DEGENERATION | VITREOUS HEMORRHAGE | CHOROIDAL NEOVASCULARIZATION | RAT MODEL | Choroidal Neovascularization | Caspase 9 - genetics | Caspase 9 - metabolism | Apoptosis - drug effects | Coculture Techniques | Humans | Male | Purinergic P2X Receptor Antagonists - pharmacology | Receptors, Purinergic P2X7 - genetics | Adenosine Triphosphate - pharmacology | Retinal Pigment Epithelium | Vitreous Body - metabolism | Adenosine Triphosphate - metabolism | Photoreceptor Cells - pathology | DNA Fragmentation - drug effects | Choroid - pathology | Apoptosis Inducing Factor - metabolism | Choroid - metabolism | Retinal Hemorrhage - pathology | Choroid - blood supply | Rosaniline Dyes - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Apoptosis Inducing Factor - genetics | Macular Degeneration - metabolism | Animals | Photoreceptor Cells - drug effects | Photoreceptor Cells - metabolism | Vitreous Body - blood supply | Receptors, Purinergic P2X7 - metabolism | Vitreous Body - pathology | Mice | Primary Cell Culture | Retinal Hemorrhage - metabolism | Macular Degeneration - pathology | Macular degeneration | Hemorrhage | Analysis | Apoptosis | Cell culture | Neuroprotection | Central nervous system | Retina | Nervous system | Blood | Vascularization | Purine P2X receptors | Neurotoxicity | Mitochondria | Neurodegeneration | Rodents | Surgery | DNA fragmentation | Biocompatibility | Ion channels | Degeneration | Vascular endothelial growth factor | Age | Deoxyribonucleic acid--DNA | Visually handicapped people | Translocation | Retinal cells | Apoptosis-inducing factor | Blood vessels | Caspase | Nuclear transport | Studies | Blood circulation | Cell death | Caspase-9 | Retinal degeneration | Photoreceptors | Nuclei (cytology) | ATP | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Journal Article
Journal Article
Journal Article
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 09/2012, Volume 53, Issue 10, pp. 6495 - 6503
Journal Article
Biomaterials, ISSN 0142-9612, 2016, Volume 109, pp. 23 - 31
Journal Article
Journal of Controlled Release, ISSN 0168-3659, 06/2012, Volume 160, Issue 2, pp. 225 - 231
Journal Article
Molecular Vision, ISSN 1090-0535, 09/2014, Volume 20, pp. 1258 - 1270
Objective: Choroidal neovascularization (CNV) is a major cause of vision loss in retinal diseases such as age-related macular degeneration (AMD). Previously,... 
RETINAL NEOVASCULARIZATION | RECEPTOR SIGNALING PATHWAYS | TGF-BETA | THERAPY | INHIBITION | PIGMENT EPITHELIAL-CELLS | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | OPHTHALMOLOGY | MACULAR DEGENERATION | EXPRESSION | NEUROPILINS | Intravitreal Injections | Apoptosis - drug effects | Humans | Semaphorin-3A - metabolism | Case-Control Studies | MAP Kinase Kinase 4 - metabolism | Choroidal Neovascularization - pathology | Vitreous Body - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Mitogen-Activated Protein Kinase 1 - genetics | Transforming Growth Factor beta - antagonists & inhibitors | p38 Mitogen-Activated Protein Kinases - metabolism | Choroid - pathology | Choroid - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Choroid - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Choroid - blood supply | Semaphorin-3A - pharmacology | p38 Mitogen-Activated Protein Kinases - genetics | Smad Proteins - genetics | Choroidal Neovascularization - prevention & control | Gene Expression Regulation - drug effects | Cell Movement - drug effects | Transforming Growth Factor beta - pharmacology | Animals | Choroidal Neovascularization - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Vitreous Body - blood supply | MAP Kinase Kinase 4 - genetics | Vitreous Body - pathology | Cell Proliferation - drug effects | Mice | Choroidal Neovascularization - genetics | Smad Proteins - metabolism | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Vitreous Body - drug effects | Index Medicus
Journal Article
Journal Article
Graefe's Archive for Clinical and Experimental Ophthalmology, ISSN 0721-832X, 6/2012, Volume 250, Issue 6, pp. 839 - 848
Journal Article