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Journal of Biological Chemistry, ISSN 0021-9258, 04/2013, Volume 288, Issue 17, pp. 11920 - 11929
Journal Article
Nature cell biology, ISSN 1476-4679, 2007, Volume 9, Issue 5, pp. 550 - 555
Journal Article
Nature communications, ISSN 2041-1723, 2019, Volume 10, Issue 1, pp. 1832 - 12
.... However, molecular details of the underlying mechanism are largely unknown. Using a photoactivatable ceramide probe, we here identify the voltage-dependent anion channels VDAC1 and VDAC2 as mitochondrial ceramide binding proteins... 
mitochondria | MEMBRANE | apoptosis | DEPENDENT ANION CHANNEL-1 | To be checked by Faculty | RADIATION-INDUCED APOPTOSIS | CELL-DEATH | SPHINGOLIPID METABOLISM | TRANSPORT | INHIBITION | Ceramides | VDAC2 | BAX | PROTEINS | Coarse-grain molecular dynamics simulations | EFFICIENT | MULTIDISCIPLINARY SCIENCES | Glutamic Acid - genetics | Humans | Voltage-Dependent Anion Channel 1 - isolation & purification | Voltage-Dependent Anion Channel 2 - genetics | Recombinant Proteins - isolation & purification | HEK293 Cells | Voltage-Dependent Anion Channel 2 - isolation & purification | Voltage-Dependent Anion Channel 1 - chemistry | Voltage-Dependent Anion Channel 2 - chemistry | Recombinant Proteins - metabolism | Ceramides - metabolism | HCT116 Cells | Voltage-Dependent Anion Channel 1 - metabolism | Recombinant Proteins - chemistry | Ceramides - chemistry | Recombinant Proteins - genetics | Binding Sites - genetics | Voltage-Dependent Anion Channel 2 - metabolism | Gene Knockout Techniques | Molecular Dynamics Simulation | Glutamic Acid - metabolism | HeLa Cells | Mitochondria - physiology | Voltage-Dependent Anion Channel 1 - genetics | Apoptosis | Glutamic Acid - chemistry | RNA, Small Interfering - metabolism | Electric potential | Membranes | Computer simulation | Mortality | Molecular dynamics | Lipids | Phospholipids | Mitochondria | Chemical modification | Anions | Colon cancer | Cell death | Anion channels | Voltage | Ceramide | Tumor suppressor genes | Ion channels | Colon | Binding sites | Glutamine
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, p. e0167908
...). Relationships between desminopathies and Voltage-dependent anion channel 1 (VDAC1) remain unclear. Here we successfully constructed the desminopathy rat model, evaluated with conventional stains, containing hematoxylin and eosin... 
BOUND HEXOKINASE | ATF2 | PROTEIN | BCL-XL | MULTIDISCIPLINARY SCIENCES | SUBCELLULAR-LOCALIZATION | VDAC | BAX | ACTIVATING TRANSCRIPTION FACTOR-2 | BCL-X(L) | CELL-DEATH | Humans | Middle Aged | bcl-X Protein - genetics | Male | Muscle Fibers, Skeletal - metabolism | Muscular Dystrophies - genetics | Case-Control Studies | Cardiomyopathies - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | HEK293 Cells | Adult | Female | bcl-2-Associated X Protein - genetics | Muscular Dystrophies - metabolism | Voltage-Dependent Anion Channel 1 - metabolism | bcl-2-Associated X Protein - metabolism | Rats | Cardiomyopathies - pathology | Mitochondria - metabolism | Muscular Dystrophies - pathology | Rats, Sprague-Dawley | Activating Transcription Factor 2 - metabolism | Animals | Cardiomyopathies - metabolism | Desmin - metabolism | Desmin - genetics | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Voltage-Dependent Anion Channel 1 - genetics | Apoptosis | Immunohistochemistry | Physiological aspects | Mitochondrial diseases | Research | Diagnosis | Risk factors | Electric potential | Neurosciences | Transcription factors | Bax protein | Bcl-2 protein | Cardiomyopathy | Kinases | Skin cancer | Proteins | Mitochondria | Anions | NADH | Bcl-x protein | Metabolites | Voltage | Tumor necrosis factor-TNF | Stains | Medical research | Enzymes | Muscles | Permeability | Metabolism | Patients | Desmin | Neurology | Pathology | Molecular modelling | Correlation analysis | Morphology | Nicotinamide adenine dinucleotide | Mutation | Protein interaction | Immunofluorescence | Endoplasmic reticulum | Major outer membrane protein | Myopathy | Localization
Journal Article
Circulation research, ISSN 1524-4571, 2008, Volume 103, Issue 9, pp. 983 - 991
Inhibition of glycogen synthase kinase (GSK)-3 reduces ischemia/reperfusion injury by mechanisms that involve the mitochondria. The goal of this study was to... 
VDAC | Mitochondria | GSK-3 | Adenine nucleotide transport | PERMEABILITY TRANSITION PORE | CARDIAC & CARDIOVASCULAR SYSTEMS | MECHANISM | mitochondria | PROTEIN-KINASE | REPERFUSION INJURY | ISCHEMIA | KINASE-3-BETA | adenine nucleotide transport | OPIOID-INDUCED CARDIOPROTECTION | CYTOCHROME-C | RESPIRATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Phosphorylation | Voltage-Dependent Anion Channels - drug effects | Maleimides - pharmacology | Myocardial Contraction - drug effects | Glycogen Synthase Kinase 3 beta | Male | Mitochondria, Heart - drug effects | Mitochondrial ADP, ATP Translocases - metabolism | Aminophenols - pharmacology | Myocardial Reperfusion Injury - enzymology | Mitochondrial Membranes - drug effects | Proton-Translocating ATPases - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Adenosine Triphosphate - metabolism | Indoles - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Mitochondrial Membrane Transport Proteins - metabolism | Adenosine Monophosphate - metabolism | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Ventricular Function, Left - drug effects | Enzyme Inhibitors - pharmacology | Mitochondria, Heart - enzymology | Rats | Cardiotonic Agents - pharmacology | Voltage-Dependent Anion Channel 2 - metabolism | Glycogen Synthase Kinase 3 - metabolism | Rats, Sprague-Dawley | Mitochondrial Membranes - metabolism | Animals | Adenine Nucleotides - metabolism | Voltage-Dependent Anion Channel 2 - drug effects | Adenosine Diphosphate - metabolism | Kinetics | Energy Metabolism - drug effects | Myocardial Reperfusion Injury - prevention & control | Voltage-Dependent Anion Channels - metabolism
Journal Article