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Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2010, Volume 127, Issue 1, pp. 200 - 207.e10
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 10/2011, Volume 166, Issue 1, pp. 64 - 71
Summary Tumour necrosis factor‐like weak inducer of apoptosis (TWEAK), a member of the tumour necrosis factor (TNF) family, has been implicated as a... 
tumour necrosis factor‐like weak inducer of apoptosis | endothelial cells | chemokines | Henoch–Schonlein purpura | Henoch-Schonlein purpura | Tumour necrosis factor-like weak inducer of apoptosis | Chemokines | Endothelial cells | INDUCED CELL-DEATH | tumour necrosis factor-like weak inducer of apoptosis | MULTIPLE PATHWAYS | IGA | ACTIVATION | KINASE | SKIN DISEASES | INDUCTION | IMMUNOLOGY | CENTRAL-NERVOUS-SYSTEM | NF-KAPPA-B | EXPRESSION | Inflammation - pathology | Apoptosis - drug effects | I-kappa B Proteins - immunology | Humans | Purpura, Schoenlein-Henoch - pathology | Child, Preschool | Male | NF-kappa B - metabolism | RNA, Messenger - biosynthesis | Inflammation - metabolism | Psoriasis - pathology | Psoriasis - metabolism | Tumor Necrosis Factors - pharmacology | Child | Psoriasis - immunology | NF-KappaB Inhibitor alpha | NF-kappa B - antagonists & inhibitors | Blotting, Western | Cytokine TWEAK | Signal Transduction - drug effects | Adolescent | Endothelial Cells - pathology | Interleukin-8 - immunology | Phosphorylation | Gene Expression - drug effects | NF-kappa B - immunology | RNA, Messenger - analysis | I-kappa B Proteins - metabolism | Case-Control Studies | Tumor Necrosis Factors - biosynthesis | Signal Transduction - immunology | Purpura, Schoenlein-Henoch - metabolism | Dermatitis, Atopic - immunology | Polymerase Chain Reaction | Adult | Female | Skin - pathology | Skin - immunology | Interleukin-8 - biosynthesis | Cells, Cultured | Dermatitis, Atopic - pathology | Inflammation - immunology | Tumor Necrosis Factors - immunology | Purpura, Schoenlein-Henoch - immunology | Endothelial Cells - immunology | Apoptosis - immunology | Chemokine CCL5 - biosynthesis | Dermatitis, Atopic - metabolism | Chemokine CCL5 - immunology | Skin - drug effects | Endothelial Cells - drug effects | Purpura (Pathology) | Psoriasis | RNA | Atopic dermatitis | Leukemia | Heat shock proteins | Medical law | Enzyme-linked immunosorbent assay | Endothelium | Necrosis | Apoptosis | Diseases | Drug therapy | Immune system | TWEAK protein | Leukocyte migration | mRNA | Leukocytes | Western blotting | Vasculitis | Cytokines | Data processing | Inflammation | Serum levels | Monocytes | NF- Kappa B protein | Infectious diseases | psoriasis vulgaris | Purpura | Tumor necrosis factor | Microvasculature | Skin | Cell migration | Translational Studies
Journal Article
Nephrology, ISSN 1320-5358, 02/2017, Volume 22, Issue 2, pp. 101 - 106
As one of the manifestations of patients with systemic lupus erythematosus, lupus nephritis (LN) has high morbidity and mortality. Although the explicit... 
lupus nephritis | fibroblast growth factor‐inducible 14 | proinflammatory cytokines | glomerular basement membrane | tumour necrosis factor‐related weak inducer of apoptosis | tumour necrosis factor-related weak inducer of apoptosis | fibroblast growth factor-inducible 14 | APOPTOSIS | CELLS | RENAL-DISEASE | PROLIFERATION | MODEL | PATHOGENESIS | UROLOGY & NEPHROLOGY | TWEAK/FN14 PATHWAY | URINARY TWEAK | EXPRESSION | PROGRESSION | Lupus Nephritis - pathology | Receptors, Tumor Necrosis Factor - metabolism | Cell Proliferation | Kidney - drug effects | Kidney - pathology | Humans | Tumor Necrosis Factors - metabolism | Lupus Nephritis - drug therapy | TWEAK Receptor | Molecular Targeted Therapy | Cytokine TWEAK | Kidney - metabolism | Animals | Signal Transduction - drug effects | Receptors, Tumor Necrosis Factor - antagonists & inhibitors | Lupus Nephritis - physiopathology | Fibrosis | Inflammation Mediators - metabolism | Lupus Nephritis - metabolism | Kidney - physiopathology | Disease Models, Animal | Tumor Necrosis Factors - antagonists & inhibitors | Lupus | Nephritis | Fibroblast growth factors | Systemic lupus erythematosus | Tumors | Necrosis | Fibroblast growth factor | Animal models | Inflammation | Lupus nephritis | Morbidity | Angiogenesis | Signal transduction | Tumor necrosis factor | Fibroblasts | Gangrene | Monoclonal antibodies | Tumor necrosis factor-TNF | Growth factors | Apoptosis | Kidney transplantation
Journal Article
Journal Article
Journal Article
Neuroscience, ISSN 0306-4522, 2010, Volume 171, Issue 4, pp. 1256 - 1264
Abstract Tumor necrosis factor-like weak inducer of apoptosis (TWEAK) and its receptor Fibroblast growth factor-inducible 14 (Fn14) are expressed in neurons.... 
Neurology | poly(ADP-ribose) polymerase-1 (PARP-1) | fibroblast growth factor-inducible 14 (Fn14) | tumor necrosis factor-like weak inducer of apoptosis (TWEAK) | poly(ADP-ribose) polymers (PAR) | cerebral ischemia | Cerebral ischemia | Fibroblast growth factor-inducible 14 (Fn14) | Poly(ADP-ribose) polymers (PAR) | Poly(ADP-ribose) polymerase-1 (PARP-1) | Tumor necrosis factor-like weak inducer of apoptosis (TWEAK) | poly(ADP ribose) polymers (PAR) | NEUROVASCULAR UNIT | INCREASES | RECEPTOR | FN14 | tumor necrosis factor like weak inducer of apoptosis (TWEAK) | TWEAK/FN14 INTERACTION | NEUROSCIENCES | CELL-DEATH | PATHWAY ACTIVATION | poly(ADP ribose) polymerase 1 (PARP 1) | PLASMINOGEN-ACTIVATOR | ENDOTHELIAL-CELLS | NF-KAPPA-B | Infarction, Middle Cerebral Artery - physiopathology | Oxidants - pharmacology | Embryo, Mammalian | TWEAK Receptor | Cerebral Cortex - cytology | Dose-Response Relationship, Drug | Cell Death - genetics | Neurons - physiology | Receptors, Tumor Necrosis Factor - deficiency | Cell Death - drug effects | Neurons - drug effects | Tumor Necrosis Factors - pharmacology | Disease Models, Animal | Receptors, Tumor Necrosis Factor - metabolism | Gene Expression Regulation - genetics | Cells, Cultured | Hydrogen Peroxide - pharmacology | Signal Transduction - genetics | Infarction, Middle Cerebral Artery - pathology | Tumor Necrosis Factors - deficiency | Mice, Knockout | Gene Expression Regulation - drug effects | Glucose - deficiency | Tumor Necrosis Factor-alpha - pharmacology | Cytokine TWEAK | Poly(ADP-ribose) Polymerases - metabolism | Animals | Signal Transduction - drug effects | Hypoxia - pathology | Mice | Poly (ADP-Ribose) Polymerase-1 | Infarction, Middle Cerebral Artery - enzymology | Poly(ADP-ribose)polymerase-1 (PARP-1)
Journal Article
Journal Article