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PAIN, ISSN 0304-3959, 05/2019, Volume 160, pp. S17 - S22
Peripheral neuropathy is the most common neurodegenerative disease affecting hundreds of millions of patients worldwide and is an important cause of chronic... 
ACTIVATION | PROTEIN | Sarmoptosis | DLK kinase | SELF-DESTRUCTION | WALLERIAN DEGENERATION | INJURY | NMNAT2 | NEUROSCIENCES | CLINICAL NEUROLOGY | CELL-DEATH | NAD | SYNAPTIC GROWTH | SARM1 | ANESTHESIOLOGY | NEUROMUSCULAR-JUNCTION | PHR1 ligase | LEUCINE-ZIPPER KINASE
Journal Article
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 10/2018, Volume 115, Issue 42, pp. 10654 - 10659
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2012, Volume 32, Issue 12, pp. 4145 - 4155
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2010, Volume 30, Issue 18, pp. 6375 - 6386
Journal Article
Journal Article
12/2016, ISBN 0128036907, 344
Neuroprotection in Alzheimer's Disease offers a translational point-of-view from both basic and clinical standpoints, putting it on the cusp for further... 
Alzheimer's disease | Biology, life sciences | Nervous system | Degeneration | Neuroprotective agents
eBook
Neurobiology of Disease, ISSN 0969-9961, 2008, Volume 31, Issue 3, pp. 316 - 326
Abstract We examined the potential protective effect of BDNF against β-amyloid-induced neurotoxicity in vitro and in vivo in rats. In neuronal cultures, BDNF... 
Neurology | Neuroprotection | TrkB | BDNF | Somatostatin | Push-pull perfusion | Indusium griseum | Dentate gyrus hilus | β-Amyloid peptides | Corpus callosum | NEURONAL APOPTOSIS | NEUROTROPHIC FACTOR | ALZHEIMERS-DISEASE | CORPUS-CALLOSUM ATROPHY | PARIETAL CORTEX | CELL LOSS | NEUROSCIENCES | indusium griseum | MESSENGER-RNA | neuroprotection | somatostatin | push-pull perfusion | dentate gyrus hilus | HIPPOCAMPAL CONTINUATION | corpus callosum | beta-amyloid peptides | DENTATE GYRUS | Neuroprotective Agents - therapeutic use | Corpus Callosum - metabolism | Peptide Fragments - toxicity | Male | Wallerian Degeneration - prevention & control | Receptor, trkB - genetics | Hippocampus - drug effects | Nerve Degeneration - chemically induced | Brain - metabolism | Dose-Response Relationship, Drug | Receptors, Somatostatin - genetics | Wallerian Degeneration - drug therapy | Brain-Derived Neurotrophic Factor - pharmacology | Neuroprotective Agents - pharmacology | Nerve Degeneration - prevention & control | Amyloid beta-Peptides - metabolism | Female | Receptors, Somatostatin - metabolism | Brain-Derived Neurotrophic Factor - therapeutic use | Disease Models, Animal | Wallerian Degeneration - chemically induced | Alzheimer Disease - physiopathology | Peptide Fragments - metabolism | Amyloid beta-Peptides - toxicity | Cells, Cultured | Alzheimer Disease - drug therapy | Rats | Corpus Callosum - drug effects | Treatment Outcome | Rats, Sprague-Dawley | Brain - drug effects | Hippocampus - metabolism | Amyloid beta-Peptides - antagonists & inhibitors | Animals | Peptide Fragments - antagonists & inhibitors | Corpus Callosum - pathology | Alzheimer Disease - metabolism | Receptors, Somatostatin - drug effects | Brain - pathology | Receptor, trkB - metabolism | Nerve Degeneration - drug therapy | Growth factors | Index Medicus
Journal Article
NeuroImage, ISSN 1053-8119, 04/2019, Volume 189, pp. 180 - 191
White matter abnormalities, revealed by Diffusion Tensor Imaging (DTI), are observed in patients with Alzheimer's Disease (AD), representing neural network... 
Retrograde degeneration | EpoD | Amyloid beta | p301L tau | Diffusion tensor imaging | Retinal ganglion cell | NEUROFIBRILLARY TANGLES | ALZHEIMERS-DISEASE | WALLERIAN DEGENERATION | PATHOLOGY | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | NEUROIMAGING | EPOTHILONE D | MAGNETIC-RESONANCE | DIFFUSION | WHITE-MATTER HYPERINTENSITIES | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | SYNTHETIC TAU FIBRILS | Tubulin Modulators - pharmacology | Amyloid beta-Peptides - pharmacology | Peptide Fragments - administration & dosage | Epothilones - pharmacology | Tauopathies - pathology | White Matter - drug effects | Nerve Degeneration - diagnostic imaging | Peptide Fragments - pharmacology | Geniculate Bodies - drug effects | Nerve Degeneration - chemically induced | Diffusion Tensor Imaging | White Matter - diagnostic imaging | Animals | Nerve Degeneration - prevention & control | Tauopathies - diagnostic imaging | Epothilones - administration & dosage | Tubulin Modulators - administration & dosage | Mice | Tauopathies - chemically induced | Amyloid beta-Peptides - administration & dosage | Tauopathies - drug therapy | Nerve Degeneration - drug therapy | Retinal Ganglion Cells - drug effects | Disease Models, Animal | Medical colleges | Neurosciences | Neural networks | Anisotropy | Physiological aspects | Models | Genetic engineering | Alzheimer's disease | Phosphorylation | Peptides | Cognitive ability | Retina | Visual system | Neurodegeneration | Drug dosages | Multimedia | Medical imaging | Optic nerve | Neurodegenerative diseases | Transgenic mice | Histology | Injection | Substantia alba | Patients | Optic tract | Studies | Pathology | Retinal ganglion cells | Axons | Brain research | Magnetic resonance imaging | Tau protein | Lateral geniculate nucleus | Laboratory animals | Alzheimers disease | Animal cognition
Journal Article
Neurochemical Research, ISSN 0364-3190, 8/2019, Volume 44, Issue 8, pp. 1964 - 1976
Journal Article