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Human Brain Mapping, ISSN 1065-9471, 11/2017, Volume 38, Issue 11, pp. 5375 - 5390
Multiple sclerosis (MS) involves damage to white matter microstructures. This damage has been related to grey matter function as measured by standard,... 
white matter | diffusion tensor imaging | calibrated imaging | blood flow and metabolism | multiple sclerosis | SIGNAL | BRAIN ACTIVITY | BOLD HEMODYNAMIC-RESPONSE | OXYGEN-METABOLISM | FUNCTIONAL MRI | NEUROSCIENCES | NEUROIMAGING | HUMAN VISUAL-CORTEX | DIFFUSION TENSOR | AXONAL DEGENERATION | CEREBRAL-BLOOD-FLOW | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | FMRI | Cerebrovascular Circulation - physiology | White Matter - metabolism | Visual Cortex - diagnostic imaging | Humans | Middle Aged | Magnetic Resonance Imaging - methods | Male | Multiple Sclerosis - diagnostic imaging | Oxygen - metabolism | Diffusion Tensor Imaging - methods | Visual Cortex - pathology | Gray Matter - metabolism | Adult | Female | Multiple Sclerosis - metabolism | Severity of Illness Index | Brain Mapping - methods | Calibration | Gray Matter - diagnostic imaging | White Matter - pathology | White Matter - diagnostic imaging | Gray Matter - pathology | Multiple Sclerosis - pathology | Visual Cortex - metabolism | Cohort Studies | Physiological aspects | Neurosciences | Multiple sclerosis | Diagnostic imaging | Information management | Data entry | Neuroimaging | Brain | Nuclear magnetic resonance--NMR | Substantia grisea | Blood | Visual perception | Cerebral blood flow | Functional magnetic resonance imaging | Damage | Diffusion | Oxygen | Medical imaging | Oxygen metabolism | Cortex (occipital) | Brain mapping | Fatigue | Cortex (visual) | Metabolism | Substantia alba | Patients | Blood flow | Neurology | Magnetic resonance imaging | Neurological complications | Microstructure | WHITE MATTER | BLOOD FLOW AND METABOLISM | MULTIPLE SCLEROSIS | DIFFUSION TENSOR IMAGING | CALIBRATED IMAGING
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2015, Volume 112, Issue 9, pp. 2853 - 2858
Journal Article
Cerebrovascular Diseases, ISSN 1015-9770, 06/2016, Volume 42, Issue 1-2, pp. 106 - 109
Journal Article
Glia, ISSN 0894-1491, 2014, Volume 62, Issue 7, pp. 1125 - 1141
To ensure efficient energy supply to the high demanding brain, nutrients are transported into brain cells via specific glucose (GLUT) and monocarboxylate... 
neurodegeneration | nutrient transporters | reactive astrocytes | proliferator‐activated receptor gamma co‐activator 1‐alpha | Nutrient transporters | Neurodegeneration | Proliferator-activated receptor gamma co-activator 1-alpha | Reactive astrocytes | MOUSE-BRAIN | ACTIVATED RECEPTOR-GAMMA | NEUROSCIENCES | MCT2 | SKELETAL-MUSCLE | proliferator-activated receptor gamma co-activator 1-alpha | MITOCHONDRIAL CHANGES | LACTATE | NEURONS | ENERGY-METABOLISM | NF-KAPPA-B | EXPRESSION | Glutamate Plasma Membrane Transport Proteins - metabolism | Leukocytes - pathology | Microglia - metabolism | White Matter - metabolism | Humans | Middle Aged | Astrocytes - pathology | Male | Glucose Transporter Type 3 - metabolism | Brain - metabolism | Brain - blood supply | Monocarboxylic Acid Transporters - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Aged, 80 and over | Microglia - pathology | Adult | Female | White Matter - blood supply | Multiple Sclerosis - metabolism | Cell Line | Endothelial Cells - metabolism | Axons - metabolism | Multiple Sclerosis, Chronic Progressive - pathology | Multiple Sclerosis, Chronic Progressive - metabolism | White Matter - pathology | Transcription Factors - metabolism | Axons - pathology | Brain - pathology | Multiple Sclerosis - pathology | Aged | Endothelial Cells - pathology | Leukocytes - metabolism | Astrocytes - metabolism | Proteins | Glucose metabolism | Brain | Multiple sclerosis | Physiological aspects | Brain damage | Development and progression | Glucose | Dextrose
Journal Article
Annals of Neurology, ISSN 0364-5134, 11/2016, Volume 80, Issue 5, pp. 776 - 790
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2015, Volume 52, pp. 106 - 119
Highlights • Fingolimod (FTY720) improves long-term cognitive deficits after neonatal hyperoxia. • Hyperoxia-related long-term microstructural abnormalities... 
Psychiatry | Allergy and Immunology | White matter development | Oligodendrocyte | Fingolimod | Hyperoxia | Neonatal brain injury | SURVIVAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | RAT | IMMUNOLOGY | NEUROSCIENCES | CELL-DEATH | MULTIPLE-SCLEROSIS | OLIGODENDROCYTE PROGENITORS | FTY720 | INFANTS | ISCHEMIC BRAIN-INJURY | MODULATION | Lysophospholipids - metabolism | Microglia - metabolism | Oligodendroglia - metabolism | Receptors, Lysosphingolipid - antagonists & inhibitors | Rats, Wistar | White Matter - metabolism | Cognition Disorders - metabolism | Male | Brain - metabolism | Cognition Disorders - prevention & control | Oligodendroglia - drug effects | Microglia - pathology | Female | Sphingosine - metabolism | Receptors, Lysosphingolipid - metabolism | Fingolimod Hydrochloride - therapeutic use | Animals, Newborn | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cognition Disorders - pathology | Rats | White Matter - drug effects | Random Allocation | White Matter - pathology | Hyperoxia - pathology | Oligodendroglia - pathology | Pregnancy | Oxygen - administration & dosage | Sphingosine - analogs & derivatives | Animals | Diffusion Magnetic Resonance Imaging | Hyperoxia - drug therapy | Infants (Newborn) | Brain | Multiple sclerosis | Analysis | Injuries | Sphingosine | Index Medicus
Journal Article