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animals (137) 137
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wallerian degeneration (105) 105
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protection (12) 12
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Journal Article
Journal Article
Nature, ISSN 0028-0836, 04/2008, Volume 452, Issue 7189, pp. 887 - 891
Neurodegeneration can be triggered by genetic or environmental factors. Although the precise cause is often unknown, many neurodegenerative diseases share... 
NERVOUS-SYSTEM | AXON DEGENERATION | BIOSYNTHESIS | MULTIDISCIPLINARY SCIENCES | WALLERIAN DEGENERATION | DISEASE | MICE | WLD(S) | MODEL | AGGREGATION | DROSOPHILA | Spinocerebellar Ataxias - prevention & control | Ataxin-1 | Molecular Chaperones - metabolism | Nicotinamide-Nucleotide Adenylyltransferase - metabolism | Humans | Neurodegenerative Diseases - prevention & control | Cercopithecus aethiops | Drosophila Proteins - metabolism | Amide Synthases - metabolism | Drosophila - enzymology | Brain - metabolism | Nerve Tissue Proteins - toxicity | Ataxins | Nerve Tissue Proteins - chemistry | Nuclear Proteins - genetics | Disease Models, Animal | Spinocerebellar Ataxias - genetics | Neurodegenerative Diseases - pathology | Molecular Chaperones - genetics | Neurodegenerative Diseases - genetics | Nuclear Proteins - metabolism | Spinocerebellar Ataxias - pathology | Nuclear Proteins - chemistry | Nerve Tissue Proteins - genetics | Nuclear Proteins - toxicity | HSP70 Heat-Shock Proteins - metabolism | Nerve Tissue Proteins - metabolism | Animals | Spinocerebellar Ataxias - enzymology | Nicotinamide-Nucleotide Adenylyltransferase - genetics | Amide Synthases - genetics | Drosophila Proteins - genetics | Nerve Degeneration | COS Cells | Neurodegenerative Diseases - enzymology | Genetics | Neurosciences | Biochemistry | Insects | Cellular biology | Neurological disorders | Index Medicus
Journal Article
Cell Reports, ISSN 2211-1247, 02/2016, Volume 14, Issue 7, pp. 1673 - 1683
Journal Article
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 03/2017, Volume 14, Issue 1, p. 57
Journal Article
JOURNAL OF CELL BIOLOGY, ISSN 0021-9525, 02/2009, Volume 184, Issue 4, pp. 491 - 500
The slow Wallerian degeneration (Wld(S)) protein protects injured axons from degeneration. This unusual chimeric protein fuses a 70-amino acid N-terminal... 
VALOSIN-CONTAINING PROTEIN | SYNAPSES | MITOCHONDRIAL | RAT MUSCLE | MOUSE | NEURODEGENERATION | MODEL | WALLERIAN DEGENERATION WLD(S) | NEUROMUSCULAR-JUNCTIONS | TRANSGENIC MICE | CELL BIOLOGY
Journal Article
Nature Neuroscience, ISSN 1097-6256, 12/2001, Volume 4, Issue 12, pp. 1199 - 1206
Axons and their synapses distal to an injury undergo rapid Wallerian degeneration, but axons in the C57BL/Wld(S) mouse are protected. The degenerative and... 
POLY(ADP-RIBOSE) POLYMERASE | CELLULAR REACTIONS | WILD-TYPE | NERVE | SPINAL-CORD INJURY | MOUSE | BCL-2 OVEREXPRESSION | C57BL/OLA MICE | MUTATION WLD(S) | NEUROSCIENCES | NEUROMUSCULAR-JUNCTIONS | Immunohistochemistry | Sciatic Nerve - injuries | Synaptic Vesicles - metabolism | Action Potentials - genetics | Synaptic Transmission - genetics | Neuromuscular Junction - metabolism | Nicotinamide-Nucleotide Adenylyltransferase - metabolism | Nervous System - metabolism | Cell Survival - genetics | Molecular Sequence Data | Muscle, Skeletal - innervation | Muscle, Skeletal - metabolism | Sciatic Nerve - ultrastructure | Recombinant Fusion Proteins - metabolism | Mutation - physiology | Sciatic Nerve - metabolism | Cell Nucleus - metabolism | Nervous System - pathology | Mice, Mutant Strains | Base Sequence - physiology | Motor Neurons - cytology | Axons - ultrastructure | Wallerian Degeneration - genetics | Muscle, Skeletal - growth & development | Wallerian Degeneration - metabolism | Wallerian Degeneration - physiopathology | Axons - metabolism | Mice, Transgenic | Fungal Proteins - genetics | Cell Nucleus - ultrastructure | Microscopy, Electron | Nerve Tissue Proteins - genetics | Neuromuscular Junction - ultrastructure | Motor Neurons - metabolism | Nerve Tissue Proteins - metabolism | Animals | Trauma, Nervous System | Saccharomyces cerevisiae Proteins | Nicotinamide-Nucleotide Adenylyltransferase - genetics | Recombinant Fusion Proteins - genetics | Mice | Ubiquitin-Conjugating Enzymes | Fungal Proteins - metabolism | Axons | Nervous system | Genetic aspects | Degeneration | Health aspects | Synapses | Wld protein | ubiquitin fusion degradation protein-2 | ubiquitination factor E4B
Journal Article
Journal of Molecular Neuroscience, ISSN 0895-8696, 2015, Volume 55, Issue 4, pp. 865 - 871
The axon-protective Wallerian degeneration slow (WLDS) protein can ameliorate the decline in axonal ATP levels after neurite transection. Here, we tested the... 
Axon injury | Bioenergetics | ATP | WLD | OPTIC-NERVE | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | WALLERIAN DEGENERATION | WLD(S) | WLDS | NEUROSCIENCES | NICOTINAMIDE ADENINE-DINUCLEOTIDE | PERIPHERAL-NERVE | RETINAL GANGLION-CELLS | NMNAT1 | METABOLISM | NAD BIOSYNTHESIS
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2004, Volume 24, Issue 9, pp. 2143 - 2155
Reactive astrocytes are prominent in the cellular response to spinal cord injury (SCI), but their roles are not well understood. We used a transgenic mouse... 
Blood-brain barrier | Neural degeneration | Glial fibrillary acid protein | Reactive astrocytes | Transgenic mice | Inflammation | Motor function | Spinal cord injury | spinal cord injury | blood-brain barrier | ADULT TRANSGENIC MICE | glial fibrillary acid protein | reactive astrocytes | NEUROSCIENCES | DELAYED WALLERIAN DEGENERATION | CELLULAR REACTIONS | RECOVERY | neural degeneration | transgenic mice | NEURITE OUTGROWTH | inflammation | FIBRILLARY ACIDIC PROTEIN | IN-VIVO | ABLATION | GENETIC INFLUENCES | motor function | MUTATION WLD(S) | Glial Fibrillary Acidic Protein - genetics | Inflammation - pathology | Neurons - pathology | Astrocytes - pathology | Blood-Brain Barrier - physiopathology | Motor Activity - drug effects | Bromodeoxyuridine | Wounds, Stab - pathology | Spinal Cord Injuries - pathology | Recovery of Function - physiology | Ganciclovir - pharmacology | Transgenes | Disease Models, Animal | Recombinant Fusion Proteins - biosynthesis | Astrocytes - drug effects | Antiviral Agents - pharmacology | Recovery of Function - genetics | Thymidine Kinase - genetics | Mice, Inbred C57BL | Nerve Crush | Mice, Transgenic | Cell Division - drug effects | Disease Progression | Oligodendroglia - pathology | Blood-Brain Barrier - pathology | Motor Activity - genetics | Astrocytes - physiology | Animals | Recombinant Fusion Proteins - genetics | Mice | Spinal Cord Injuries - physiopathology | Plasticity | Development | Repair
Journal Article
Journal Article