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Journal Article
HUMAN MOLECULAR GENETICS, ISSN 0964-6906, 02/2014, Volume 23, Issue 4, pp. 889 - 905
Primary aldosteronism (PA) is the main cause of secondary hypertension, resulting from adrenal aldosterone-producing adenomas (APA) or bilateral hyperplasia.... 
CANCER-CELLS | FREQUENT EPIGENETIC INACTIVATION | ADRENAL-CORTEX | ZONA GLOMERULOSA | COLORECTAL-CANCER | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | KCNJ5 MUTATIONS | BETA-CATENIN | ADRENOCORTICAL TUMORS | SOMATIC MUTATIONS | Adrenal Cortex Neoplasms - complications | Aldosterone - biosynthesis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Hyperaldosteronism - metabolism | Mice, 129 Strain | Nuclear Receptor Subfamily 4, Group A, Member 2 - genetics | Hyperaldosteronism - etiology | Adult | Female | Membrane Proteins - metabolism | Adrenal Cortex Neoplasms - metabolism | Cytochrome P-450 CYP11B2 - metabolism | Wnt Signaling Pathway | Nuclear Receptor Subfamily 4, Group A, Member 1 - genetics | Membrane Proteins - genetics | Down-Regulation | Mice, Inbred C57BL | Aldosterone - blood | Aldosterone - secretion | Adrenocortical Adenoma - metabolism | Mice, Knockout | Animals | Cell Line, Tumor | Nuclear Receptor Subfamily 4, Group A, Member 1 - metabolism | Mice | Nuclear Receptor Subfamily 4, Group A, Member 2 - metabolism | Adrenocortical Adenoma - complications | Cytochrome P-450 CYP11B2 - genetics | Animal genetics | Cytochrome P-450 CYP11B2 | Hyperaldosteronism | Genomics | Nuclear Receptor Subfamily 4, Group A, Member 1 | Nuclear Receptor Subfamily 4, Group A, Member 2 | Aldosterone | Embryology and Organogenesis | Life Sciences | Genetics | Endocrinology and metabolism | Biochemistry, Molecular Biology | Adrenocortical Adenoma | Membrane Proteins | Human health and pathology | Adrenal Cortex Neoplasms | Development Biology | Molecular biology | Cancer
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