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Nature Genetics, ISSN 1061-4036, 10/2004, Volume 36, Issue 10, pp. 1117 - 1121
Journal Article
2016, Methods molecular biology, ISBN 9781493963911, Volume 1481.
Web Resource
2016, Methods molecular biology, ISBN 9781493963911, Volume 1481.
Web Resource
2008, Methods in Molecular Biology, ISBN 9781603274685, Volume 469, xvi, 494
Book
Journal of Clinical Investigation, ISSN 0021-9738, 01/2017, Volume 127, Issue 1, pp. 137 - 152
Journal Article
Journal Article
2007, ISBN 0444528741, xiii, 248 p., [8] p. of plates
A superb compilation of reviews from leading experts in the field of Wnt signaling (signaling molecules that regulate cell-to-cell interactions during... 
Wnt genes | Embryology | Cellular signal transduction | Wnt proteins
Book
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 08/2017, Volume 21, Issue 8, pp. 1545 - 1554
Fibrosis in animal models and human diseases is associated with aberrant activation of the Wnt/β‐catenin pathway. Despite extensive research efforts, effective... 
Smad2/3 | myofibroblast proliferation | pulmonary fibrosis | Wnt/β‐catenin | Wnt/β-catenin | MEDICINE, RESEARCH & EXPERIMENTAL | TGF-BETA | MECHANISMS | BETA-CATENIN | PROTEIN CBP | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | PATHOGENESIS | INHIBITION | PATHWAY | Wnt/beta-catenin | IDIOPATHIC PULMONARY-FIBROSIS | PROMOTE | Cell Proliferation | Cadherins - metabolism | Vimentin - metabolism | Humans | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Smad3 Protein - metabolism | Wnt Proteins - metabolism | Actins - genetics | Myofibroblasts - metabolism | Smad3 Protein - genetics | Collagen Type I - genetics | Wnt Proteins - genetics | Vimentin - genetics | Smad2 Protein - genetics | Epithelial-Mesenchymal Transition | Cadherins - genetics | Fibroblasts - metabolism | A549 Cells | Cell Line | Myofibroblasts - pathology | Collagen Type I - metabolism | Signal Transduction | Gene Expression Regulation | Smad2 Protein - metabolism | Rats | Transforming Growth Factor beta1 - genetics | Fibroblasts - pathology | Rats, Sprague-Dawley | beta Catenin - metabolism | beta Catenin - genetics | Animals | Fibrosis | Vimentin | Animal models | Collagen (type I) | Wnt protein | Mesenchyme | Pathogenesis | Transforming growth factor-b | Lung | Activation | Proteins | β-catenin | Signal transduction | Molecular modelling | Smad2 protein | Effectors | Fibroblasts | Extracellular matrix | Autocrine signalling | Aberration | Index Medicus | Smad2 | β‐catenin | Wnt | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2009, Volume 4, Issue 4, pp. e5388 - e5388
Background: Understanding how the mechanical microenvironment influences cell fate, and more importantly, by what molecular mechanisms, will enhance not only... 
MULTIDISCIPLINARY SCIENCES | RNA, Small Interfering - genetics | Osteogenesis - physiology | Cadherins - metabolism | Stress, Mechanical | Receptor Tyrosine Kinase-like Orphan Receptors | Wnt-5a Protein | Wnt Proteins - metabolism | Adherens Junctions - metabolism | Mesenchymal Stromal Cells - cytology | Wnt Proteins - genetics | Base Sequence | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Cell Differentiation | Cell Line | Signal Transduction | Mesenchymal Stromal Cells - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | beta Catenin - metabolism | Biomechanical Phenomena | Animals | Receptor Protein-Tyrosine Kinases - genetics | Models, Biological | rho GTP-Binding Proteins - metabolism | Mice | Wnt Proteins - antagonists & inhibitors | Bioengineering | Transcription factors | Phosphorylation | Wnt protein | Transcription | Mesenchyme | Fluid flow | Homeostasis | Kinases | Regeneration (physiology) | Cell adhesion & migration | Proteins | β-catenin | Osteoporosis | Signal transduction | N-Cadherin | Cell fate | Loading | Biocompatibility | Bones | Bone density | Localization | Translocation | Heparan sulfate | Wound healing | Fluid dynamics | RhoA protein | Cadherin | Nuclear transport | Polymerase chain reaction | Regeneration | Molecular modelling | Morphology | Stem cells | Transduction | Bone | Mechanical stimuli | Mutation | Differentiation | Guanosinetriphosphatase | Index Medicus
Journal Article
Neuro-Oncology, ISSN 1522-8517, 02/2013, Volume 15, Issue 2, pp. 161 - 171
Background. Glioblastoma (GBM) is the most lethal and common type of primary brain tumor. Recent evidence suggests that a subpopulation of GBM cells... 
Glioblastoma stem cell | Wnt/bcatenin signaling | MET protooncogene |