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xanthohumol (403) 403
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Free radical biology & medicine, ISSN 0891-5849, 11/2015, Volume 88, Issue Pt B, pp. 417 - 426
In light of the emerging interplay between redox and metabolic signaling pathways we investigated the potential cross talk between nuclear factor E2-related... 
Xanthohumol | Nrf2 | ER stress | AMPK | HO-1 | PERK | LKB1 | MEF | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | AMP-Activated Protein Kinases - metabolism | Heme Oxygenase-1 - metabolism | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Oxidative Stress - physiology | Propiophenones - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Cross-Talk - physiology | Blotting, Western | Animals | Flow Cytometry | Fibroblasts - drug effects | NF-E2-Related Factor 2 - metabolism | Signal Transduction - physiology | Flavonoids - pharmacology | Membrane Proteins - metabolism | Mice | Real-Time Polymerase Chain Reaction | Unfolded Protein Response - physiology | Fibroblasts - metabolism | RNA | Glycogen | Synthesis | Index Medicus | ACC, acetyl-CoA carboxylase | CHO, Chinese hamster ovary | GSH, glutathione (reduced) | OCR, oxygen consumption rate | HO-1, heme oxygenase 1 | CaMKK, calcium calmodulin-dependent kinase kinase | Maf, small musculoaponeurotic fibrosarcoma | PERK, protein kinase RNA-like endoplasmic reticulum kinase | Xn, xanthohumol | Hrd1, synoviolin | TAK, transforming growth factor β-activated kinase | UPR, unfolded protein response | Hrd1 (HMG-CoA reductase degradation)-ubiquitin ligase | MEF, mouse embryonic fibroblasts | βTrcP1, β-transducin-repeat containing protein 1 | mTOR, mammalian target of rapamycin | ROS, reactive oxygen species | LKB1, liver kinase B1 | Keap1, Kelch-like ECH-associated protein | GSSG, oxidized glutathione | ARE, antioxidant response element | WT, wild type | GSK3β, glycogen synthase kinase 3β | Nrf2, nuclear factor E2-related factor 2 | AMPK, AMP-activated kinase | DCF, dichlorofluorescein | NADPH, nicotinamide adenine dinucleotide phosphate | DMSO, dimethyl sulfoxide | ER, endoplasmic reticulum | FCS, fetal calf serum
Journal Article
Current Alzheimer research, ISSN 1567-2050, 08/2019, Volume 16, Issue 9, p. 836
Background: Xanthohumol has been reported to have cytoprotection through activation of Nrf2−ARE signaling pathway and; it has capability of scavenging free... 
Neuroprotection | Signal transduction | Fibrillation | Free radicals | Oligomerization | Blood-brain barrier | Calcium | Xanthohumol | Neurodegeneration | Membrane permeability | Drug development | Alzheimer's disease
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 09/2008, Volume 7, Issue 9, pp. 2692 - 2702
The oncogenic Bcr-Abl tyrosine kinase activates various signaling pathways including phosphoinositide 3-kinase/Akt and nuclear factor-κB that mediate... 
NF-κB | chemoprevention | Xanthohumol | angiogenesis | ROS | Bcr-Abl | resistant cells | p53 | Life Sciences & Biomedicine | Oncology | Science & Technology
Journal Article
Phytochemistry (Oxford), ISSN 0031-9422, 05/2004, Volume 65, Issue 10, pp. 1317 - 1330
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 09/2018, Volume 19, Issue 9, p. 2729
Journal Article
Cancer, ISSN 1097-0142, 11/2007, Volume 110, Issue 9, pp. 2007 - 2011
BACKGROUND. Leukemias are dependent on Akt/NF‐κB activation and angiogenesis. METHODS. The antiangiogenic Akt/NF‐κB inhibitor xanthohumol (XN) has in vitro... 
AML | CML | NF‐κB | invasion | angiogenesis | Akt | xanthohumol | Angiogenesis | NF-κB | Xanthohumol | Invasion | Life Sciences & Biomedicine | Oncology | Science & Technology
Journal Article
International journal of cancer, ISSN 1097-0215, 04/2019, Volume 145, Issue 4, pp. 1007 - 1019
Esophageal cancer, a leading cause of cancer death worldwide, is associated with abnormal activation of the AKT signaling pathway. Xanthohumol, a prenylated... 
esophageal squamous cell carcinoma | xanthohumol | targeting AKT kinase activity | AKT signaling pathway | Life Sciences & Biomedicine | Oncology | Science & Technology | Esophageal Squamous Cell Carcinoma - metabolism | Cell Survival - drug effects | Apoptosis - drug effects | Humans | Propiophenones - pharmacology | G1 Phase - drug effects | Mice, SCID | Animals | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Esophageal Neoplasms - metabolism | Cell Line, Tumor | Female | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Flavonoids - pharmacology | Mice | Esophageal Squamous Cell Carcinoma - drug therapy | Proto-Oncogene Proteins c-akt - metabolism | Esophageal Neoplasms - drug therapy | Squamous cell carcinoma | Flavones | Growth | Glycogen | Bioflavonoids | Proteins | Cytochrome c | Prevention | Flavonoids | Chemotherapy | Synthesis | Analysis | Esophageal cancer | Cancer | TOR protein | Cell proliferation | Cytochrome | Cell culture | Biotechnology | Bax protein | Xenotransplantation | Clinical trials | AKT protein | Cyclin D1 | Kinases | Anticancer properties | Signal transduction | Cell cycle | Xenografts | Docking | Inhibition | G1 phase | Medical research | AKT1 protein | Diabetes mellitus | Glycogen synthase kinase 3 | Caspase | Poly(ADP-ribose) polymerase | Rapamycin | Ribosomal protein S6 | Esophagus | Signaling | Computer applications | Cytochromes | Tumors | Apoptosis | Index Medicus | Cancer Therapy and Prevention
Journal Article