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NATURE, ISSN 0028-0836, 07/2017, Volume 547, Issue 7662, pp. 179 - 179
Journal Article
The American Journal of Pathology, ISSN 0002-9440, 05/2019, Volume 189, Issue 5, pp. 1077 - 1090
Hepatoblastoma (HB) is the most common type of pediatric liver cancer. Activation of yes-associated protein (YAP) has been implicated in HB molecular... 
MAMMALIAN TARGET | RAPAMYCIN COMPLEX 1 | MALIGNANT-TUMORS | WNT/BETA-CATENIN | LIVER | HIPPO PATHWAY | GENOMIC ANALYSIS | YAP | CELL-PROLIFERATION | PATHOLOGY | BETA-CATENIN
Journal Article
Aging and Disease, ISSN 2152-5250, 04/2019, Volume 10, Issue 2, pp. 293 - 306
Journal Article
Gene Expression Patterns, ISSN 1567-133X, 09/2018, Volume 29, pp. 10 - 17
The Hippo signaling pathway regulates many cellular processes, but has been specifically associated with control organ size and tumor growth. Yes-associated... 
YAP1 | Cholangiocyte | Hepatocyte | Liver development | Hepatoblast | Endoderm | ORGAN SIZE CONTROL | TISSUE HOMEOSTASIS | HIPPO PATHWAY | CELL-PROLIFERATION | DEVELOPMENTAL BIOLOGY | BETA-CATENIN | FATE | IN-VIVO | GROWTH | GENETICS & HEREDITY | YAP ONCOPROTEIN | MICE | Liver cancer | Developmental biology | Liver | Genes | Medical genetics | Genetic engineering | Genetic transcription | Biomedical engineering | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of Cell Science, ISSN 0021-9533, 2015, Volume 128, Issue 4, pp. 790 - 803
Journal Article
Molecular Medicine Reports, ISSN 1791-2997, 05/2017, Volume 15, Issue 5, pp. 3035 - 3040
Wound healing is delayed in diabetes due to a number of factors, including impaired angiogenesis and poor dermal healing. The present study demonstrated that... 
Yes-associated protein | wound | substance P | diabetes | endothelial progenitor cells | Diabetes | Substance P | Wound | Endothelial progenitor cells | MEDICINE, RESEARCH & EXPERIMENTAL | ANGIOGENESIS | REGENERATION | HIPPO PATHWAY | YAP | MELLITUS | TAZ | REPAIR | ONCOLOGY | GROWTH | SKIN | Immunohistochemistry | Neovascularization, Physiologic - drug effects | Diabetes Mellitus, Experimental - drug therapy | Male | Substance P - pharmacology | Diabetes Mellitus, Type 2 - metabolism | Phosphoproteins - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Injections, Subcutaneous | Diabetes Mellitus, Experimental - metabolism | Wound Healing - drug effects | Bone Marrow - drug effects | Disease Models, Animal | Endothelial Progenitor Cells - drug effects | Random Allocation | Cell Movement - drug effects | Animals | Endothelial Progenitor Cells - pathology | Bone Marrow - pathology | Diabetes Mellitus, Experimental - pathology | Cell Proliferation - drug effects | Mice | Diabetes Mellitus, Type 2 - pathology | Adaptor Proteins, Signal Transducing - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Type 2 diabetes | Complications and side effects | Cellular proteins | Care and treatment | Wound healing | Genetic aspects | Health aspects | Antigens | Diabetes mellitus | Dermis | Kinases | Cell adhesion & migration | Angiogenesis | Cell growth | Rodents | Stem cells | Bone marrow | Skin | Localization | Chronic illnesses | Apoptosis
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 04/2018, Volume 115, Issue 17, pp. E4101 - E4110
During development, ventricular chamber maturation is a crucial step in the formation of a functionally competent postnatal heart. Defects in this process can... 
Cullin 7 | Hippo-YAP signaling | NEDD8 | Cardiomyopathy | Ventricular compaction | YES-ASSOCIATED PROTEIN | MULTIDISCIPLINARY SCIENCES | CARDIOMYOCYTE PROLIFERATION | NONCOMPACTION CARDIOMYOPATHY | DILATED CARDIOMYOPATHY | CLINICAL-FEATURES | ventricular compaction | COP9 SIGNALOSOME | HEART DEVELOPMENT | CARDIAC PROTEINOPATHY | CELL-CYCLE | MISFOLDED PROTEINS | cardiomyopathy | NEDD8 Protein - metabolism | Tumor Suppressor Proteins - metabolism | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | Myocardium - pathology | NEDD8 Protein - genetics | Phosphoproteins - genetics | Phosphoproteins - metabolism | Mice, Knockout | Cullin Proteins - genetics | Myocytes, Cardiac - pathology | Animals | Myocardium - metabolism | Tumor Suppressor Proteins - genetics | Adaptor Proteins, Signal Transducing - genetics | Cullin Proteins - metabolism | Myocytes, Cardiac - metabolism | Heart Ventricles - metabolism | Mice | Protein Processing, Post-Translational | Adaptor Proteins, Signal Transducing - metabolism | Heart Ventricles - pathology | Protein-Serine-Threonine Kinases - metabolism | Physiological aspects | Enzymes | Cellular signal transduction | Research | Cardiovascular research | Heart ventricles | Cell proliferation | Ubiquitin | Heart | Cardiac arrhythmia | Postpartum period | Activation | Lethality | Gestation | Kinases | Inactivation | Cullin | Proteins | Clonal deletion | Cell cycle | Deletion | Heart diseases | Ubiquitin-protein ligase | Hypoplasia | Phenotypes | Deactivation | Maturation | Cardiomyocytes | Substrates | Studies | Yes-associated protein | Signaling | Molecular modelling | Ventricle | Index Medicus | Biological Sciences | PNAS Plus
Journal Article