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Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 636 - 651
Cornelia de Lange syndrome (CdLS) is a genetically heterogeneous disorder that presents with extensive phenotypic variability, including facial dysmorphism,... 
SISTER-CHROMATID COHESION | INDIVIDUALS | MEDICINE, RESEARCH & EXPERIMENTAL | HYPERTRICHOSIS-CUBITI | VARIANTS | GENETIC-HETEROGENEITY | ACTIVATOR PROTEIN | MUTATIONS | IDENTIFICATION | HUMAN HOMOLOG | NIPBL REARRANGEMENTS | Myeloid-Lymphoid Leukemia Protein - biosynthesis | Histone Deacetylases - biosynthesis | Chondroitin Sulfate Proteoglycans - genetics | Humans | Transcriptome | Child, Preschool | Exonucleases | Infant | Male | Gene Expression Profiling | Exome | De Lange Syndrome - metabolism | Chromosomal Proteins, Non-Histone - biosynthesis | Cell Cycle Proteins - genetics | Adult | Chondroitin Sulfate Proteoglycans - biosynthesis | Child | De Lange Syndrome - genetics | Genome-Wide Association Study | Histone Deacetylases - genetics | Histone-Lysine N-Methyltransferase | Gene Expression Regulation | Repressor Proteins - genetics | Cell Cycle Proteins - biosynthesis | Codon, Nonsense | Chromosomal Proteins, Non-Histone - genetics | Proteins - genetics | Phenotype | Proteins - metabolism | Repressor Proteins - biosynthesis | Myeloid-Lymphoid Leukemia Protein - genetics | Adolescent | Heterozygote | De Lange Syndrome - pathology | De Lange syndrome | Gene mutations | Exome sequencing | Genetic research | Development and progression | Genetic transcription | Research | Diagnosis | Identification and classification | Methods | Proteins | Studies | Genotype & phenotype | Genomics | Zebrafish | Genomes | Mutation | Chromosomes | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2001, Volume 98, Issue 17, pp. 9742 - 9747
Short interfering RNAs (siRNAs) are double-stranded RNAs of ≈21-25 nucleotides that have been shown to function as key intermediaries in triggering... 
Biological Sciences | Double stranded RNA | Transfection | Delta cells | RNA | Somatic cells | Cell lines | Small interfering RNA | HeLa cells | Nucleotides | Gene expression | ZEBRAFISH | MESSENGER-RNA | GERM-LINE | MULTIDISCIPLINARY SCIENCES | DROSOPHILA CELLS | POLYMERASE | DEPENDENT PROTEIN-KINASE | DEGRADATION | C-ELEGANS | INTERFERENCE | TRANSGENE | Mammals - genetics | Green Fluorescent Proteins | Phosphorylation | Gene Silencing - drug effects | Species Specificity | Vertebrates - genetics | Humans | HeLa Cells - metabolism | Chloramphenicol O-Acetyltransferase - genetics | RNA, Antisense - pharmacology | Luminescent Proteins - biosynthesis | Muscle Proteins - biosynthesis | Cell Death | RNA, Helminth - physiology | Genes, Reporter | Calmodulin-Binding Proteins - genetics | Recombinant Fusion Proteins - biosynthesis | Caenorhabditis elegans Proteins | Cells, Cultured - drug effects | Caenorhabditis elegans - metabolism | Endoribonucleases - metabolism | Caenorhabditis elegans - genetics | RNA, Antisense - chemical synthesis | RNA, Double-Stranded - physiology | Helminth Proteins - biosynthesis | Invertebrates - genetics | Helminth Proteins - genetics | HeLa Cells - drug effects | Muscle Proteins - genetics | Animals | Calmodulin-Binding Proteins - biosynthesis | Ribonuclease III | RNA, Double-Stranded - pharmacology | Luminescent Proteins - genetics | Cells, Cultured - metabolism | Mice | Chloramphenicol O-Acetyltransferase - biosynthesis | Gene Silencing - physiology | Physiological aspects | Caenorhabditis elegans | Genetic aspects | Invertebrates | Vertebrates | Genetics | Ribonucleic acid--RNA | Genes | Ribonucleic acid | siRNAs | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 06/2017, Volume 36, Issue 25, pp. 3505 - 3514
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 04/2013, Volume 33, Issue 15, pp. 6524 - 6539
Journal Article
Anticancer Research, ISSN 0250-7005, 08/2016, Volume 36, Issue 8, pp. 3997 - 4005
Journal Article
Nature, ISSN 0028-0836, 02/2018, Volume 554, Issue 7690, pp. 112 - 117
Many craniofacial disorders are caused by heterozygous mutations in general regulators of housekeeping cellular functions such as transcription or ribosome... 
PATHOGENESIS | CELLS | PROTEIN | ENDONUCLEASE | RNA HELICASE DDX21 | DNA | MULTIDISCIPLINARY SCIENCES | PREVENTION | TREACHER-COLLINS-SYNDROME | NEURAL CREST | P53 | Xenopus | Chromatin - metabolism | Cell Nucleolus - pathology | Embryonic Stem Cells - cytology | Mandibulofacial Dysostosis - embryology | Humans | Protein Transport - drug effects | DNA-Directed RNA Polymerases - deficiency | RNA, Ribosomal - genetics | RNA Polymerase I - antagonists & inhibitors | Mandibulofacial Dysostosis - genetics | Cell Nucleus - metabolism | Skull - pathology | Nuclear Proteins - deficiency | Cell Nucleolus - drug effects | Stress, Physiological - drug effects | Neural Crest - enzymology | Benzothiazoles - pharmacology | Phenotype | Zebrafish Proteins - deficiency | Ribosomes - genetics | Mice | DNA Damage | HeLa Cells | Embryonic Stem Cells - metabolism | RNA Helicases - metabolism | Mandibulofacial Dysostosis - pathology | Neural Crest - pathology | Ribosomal Proteins - biosynthesis | Ribosomes - metabolism | DNA, Ribosomal - metabolism | Phosphoproteins - metabolism | Zebrafish - embryology | Cell Nucleus - pathology | Cell Nucleolus - metabolism | DEAD-box RNA Helicases - deficiency | DEAD-box RNA Helicases - metabolism | Nuclear Proteins - genetics | DNA, Ribosomal - genetics | Cell Nucleolus - genetics | Ribosomal Proteins - genetics | Tumor Suppressor Protein p53 - metabolism | RNA, Ribosomal - metabolism | Nuclear Proteins - metabolism | Phosphoproteins - genetics | Organ Specificity | DEAD-box RNA Helicases - genetics | Animals | Phosphoproteins - deficiency | RNA, Ribosomal - biosynthesis | Cell Nucleus - drug effects | Naphthyridines - pharmacology | Apoptosis | Nucleolus organizer region | Genetic aspects | Gene mutations | Observations | Neural crest | Craniofacial dysostosis | Chromatin | Transcription | Genomics | p53 Protein | Genes | DNA damage | Ribosomal DNA | Disorders | Homeostasis | Biosynthesis | Genomes | DNA-directed RNA polymerase | Proteins | DNA helicase | Nucleoli | Embryogenesis | Cell activation | RNA processing | Chemical synthesis | Deoxyribonucleic acid--DNA | Anemia | rRNA | RNA polymerase | Embryonic growth stage | Polymerase | DNA biosynthesis | Craniofacial growth | Ribonucleic acids | Stem cells | Mutation | Developmental disabilities | Tumors | RNA helicase | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 2011, Volume 124, Issue 6, pp. 720 - 730
BACKGROUND: Myocardial infarction leads to cardiac remodeling and development of heart failure. Insufficient myocardial capillary density after myocardial... 
microRNAs | heart failure | antagomir | angiogenesis | gene expression | myocardial infarction | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ZEBRAFISH | HEART-FAILURE | DOWN-REGULATION | KAPPA-B | HUMAN ENDOTHELIAL-CELLS | CONTROLS ANGIOGENESIS | CARDIAC-HYPERTROPHY | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | NEOVASCULARIZATION | HEMATOLOGY | Myocardial Infarction - genetics | Neovascularization, Physiologic - drug effects | MicroRNAs - antagonists & inhibitors | Capillaries - pathology | Apoptosis - drug effects | Male | Gene Expression Profiling | Zebrafish - embryology | Cell Hypoxia | Oligoribonucleotides - pharmacology | Heme Oxygenase-1 - genetics | GATA2 Transcription Factor - biosynthesis | Ventricular Remodeling | Laminin | RNA Interference | Myocardial Infarction - physiopathology | Drug Evaluation, Preclinical | Spheroids, Cellular | Heart Failure - etiology | GATA2 Transcription Factor - genetics | Cells, Cultured - drug effects | Zebrafish Proteins - biosynthesis | Heme Oxygenase-1 - biosynthesis | Proteoglycans | p21-Activated Kinases - genetics | Endothelial Cells - metabolism | Neovascularization, Physiologic - genetics | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | p21-Activated Kinases - biosynthesis | Arterioles - pathology | Myocardial Infarction - complications | Animals | RNA, Small Interfering - therapeutic use | Collagen | Cells, Cultured - metabolism | Mice | MicroRNAs - genetics | MicroRNAs - physiology | Zebrafish Proteins - genetics | Endothelial Cells - pathology | Drug Combinations | Heart failure | MicroRNA | Patient outcomes | Physiological aspects | Development and progression | Genetic aspects | Neovascularization | Research | Heart attack | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e55221 - e55221
Journal Article
Journal Article