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Neuron, ISSN 0896-6273, 2005, Volume 47, Issue 6, pp. 817 - 831
The molecular mechanisms controlling the differentiation of neural progenitors into distinct subtypes of neurons during neocortical development are unknown.... 
AREA IDENTITY | NEOCORTICAL NEURONS | CELL FATE SPECIFICATION | CEREBRAL CORTICAL DEVELOPMENT | LAYER NEURONS | CENTRAL-NERVOUS-SYSTEM | SUBCORTICAL TARGETS | ZINC-FINGER GENE | SUBVENTRICULAR ZONE | NEUROSCIENCES | ZEBRAFISH FOREBRAIN | Membrane Glycoproteins - metabolism | Embryo, Mammalian | Homeodomain Proteins - metabolism | Nerve Tissue Proteins - deficiency | S100 Proteins - genetics | POU Domain Factors - genetics | Forkhead Transcription Factors - metabolism | In Situ Hybridization - methods | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | POU Domain Factors - metabolism | Animals, Newborn | DNA-Binding Proteins - physiology | Motor Neurons - physiology | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cell Cycle Proteins - metabolism | S100 Proteins - metabolism | In Situ Nick-End Labeling - methods | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Immunohistochemistry - methods | Annexin A2 - genetics | Cell Death - physiology | Green Fluorescent Proteins - biosynthesis | Mice | Annexin A2 - metabolism | Age Factors | Phosphopyruvate Hydratase - metabolism | Cell Movement - physiology | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | Amino Acids - metabolism | Motor Neurons - cytology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Membrane Proteins - metabolism | Bromodeoxyuridine - metabolism | Cell Differentiation - physiology | Pyramidal Tracts - physiology | Nerve Tissue Proteins - physiology | Neocortex - growth & development | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Dopamine and cAMP-Regulated Phosphoprotein 32 - metabolism | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Membrane Glycoproteins - genetics | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cell Count - methods | Neocortex - cytology | Receptors, Antigen, T-Cell, alpha-beta - metabolism | Neurosciences | Neurons | Developmental biology | Stem cells | Studies | Brain | Ultrasonic imaging | Transcription factors | Hybridization | Neurogenesis | Index Medicus
Journal Article
The EMBO Journal, ISSN 0261-4189, 07/2004, Volume 23, Issue 13, pp. 2544 - 2553
Bach1 is a transcriptional repressor of heme oxygenase‐1 and β‐globin genes, both of which are known to be transcriptionally induced by heme. To test the... 
heme oxygenase | Maf | globin | heme | oxidative stress | Globin | Oxidative stress | Heme | Heme oxygenase | CELLS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCTION | OXYGENASE-1 GENE | LEUCINE ZIPPER PROTEIN | CELL BIOLOGY | CADMIUM | SMALL MAF | TRANSCRIPTION FACTOR NF-E2 | BINDING | Immunohistochemistry | Heme - metabolism | Transcription Factors - chemistry | Humans | Leucine Zippers | Molecular Sequence Data | Globins - metabolism | Recombinant Fusion Proteins - metabolism | DNA-Binding Proteins - metabolism | Cell Nucleus - metabolism | Chromatin Immunoprecipitation | Heme Oxygenase-1 | Transcription, Genetic | Active Transport, Cell Nucleus | Hemin - pharmacology | Heme Oxygenase (Decyclizing) - metabolism | Dimerization | Heme Oxygenase (Decyclizing) - genetics | Genes, Reporter | Repressor Proteins - metabolism | Fibroblasts - metabolism | Amino Acid Sequence | Cell Line | Green Fluorescent Proteins - metabolism | Zinc Fingers | Gene Expression Regulation | Glutathione Transferase - metabolism | Alanine - metabolism | Nuclear Proteins - metabolism | Recombinant Fusion Proteins - chemistry | Transcription Factors - genetics | Fanconi Anemia Complementation Group Proteins | Amino Acid Motifs | MafK Transcription Factor | Membrane Proteins | Transcription Factors - metabolism | Karyopherins - metabolism | Escherichia coli - genetics | Plasmids | Fibroblasts - drug effects | Erythroid-Specific DNA-Binding Factors | Spectrophotometry | Basic-Leucine Zipper Transcription Factors | Amino Acid Substitution | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
Book
Cancer Cell, ISSN 1535-6108, 05/2012, Volume 21, Issue 5, pp. 601 - 613
The proto-oncogene is mis-expressed in various types of human brain tumors. To clarify how developmental and regional differences influence transformation, we... 
PROGENITORS | SOX9 EXPRESSION | ONCOLOGY | SONIC HEDGEHOG | MOUSE MODEL | EMBRYONIC LETHALITY | PROLIFERATION | GENERATION | MUTATIONS | PEDIATRIC MEDULLOBLASTOMA | ASTROCYTES | CELL BIOLOGY | Oncogene Proteins - genetics | Cell Proliferation | Humans | Zinc Finger Protein Gli2 | Brain Stem - metabolism | Hedgehog Proteins - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Glioma - pathology | Medulloblastoma - pathology | Cell Differentiation | N-Myc Proto-Oncogene Protein | Cerebellar Neoplasms - pathology | Prosencephalon - metabolism | Cerebellar Neoplasms - metabolism | Biomarkers - metabolism | Neuroectodermal Tumors, Primitive - metabolism | SOX9 Transcription Factor - metabolism | Transduction, Genetic | Signal Transduction | Oncogene Proteins - metabolism | Brain Neoplasms - genetics | Mice, Transgenic | Medulloblastoma - metabolism | Gestational Age | Cell Lineage | Mice, Nude | Brain Stem - embryology | Mice | Mutation | Brain Neoplasms - pathology | Brain Neoplasms - metabolism | Glioma - metabolism | Cerebellum - embryology | Hedgehog Proteins - genetics | Kruppel-Like Transcription Factors - metabolism | Female | Nuclear Proteins - genetics | Spheroids, Cellular | Proto-Oncogene Proteins - metabolism | Cerebellum - metabolism | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Neural Stem Cells - pathology | Animals | Cell Transformation, Neoplastic - pathology | Prosencephalon - embryology | Neural Stem Cells - metabolism | SOX9 Transcription Factor - genetics | Neuroectodermal Tumors, Primitive - pathology | Gliomas | Children's hospitals | Oncology, Experimental | Brain tumors | Stem cells | Transplantation | Universities and colleges | Research | Statistics | Tumors | Cancer | Index Medicus | N-MYC | glioma | SOX9 | medulloblastoma | neural stem cells | GFAP | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 08/2011, Volume 80, Issue 4, pp. 358 - 368
Enhanced transforming growth factor-β1 (TGF-β1) expression in renal cells promotes fibrosis and hypertrophy during the progression of diabetic nephropathy. The... 
TGF-β | fibrosis | diabetic nephropathy | gene expression | cell signaling | Up-Regulation | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Homeostasis | MicroRNAs - metabolism | Diabetes Mellitus, Type 2 - metabolism | Transfection | Time Factors | Kruppel-Like Transcription Factors - metabolism | Upstream Stimulatory Factors - metabolism | Diabetes Mellitus, Type 1 - chemically induced | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Binding Sites | Collagen Type IV - metabolism | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Collagen Type I - metabolism | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | Oligonucleotides - metabolism | Animals | Fibrosis | Diabetes Mellitus, Experimental - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Mutation | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 05/2008, Volume 10, Issue 5, pp. 593 - 601
Journal Article
Nature Medicine, ISSN 1078-8956, 04/2012, Volume 18, Issue 4, pp. 529 - 537
New cancer therapies are likely to arise from an in-depth understanding of the signaling networks influencing tumor initiation, progression and metastasis. We... 
MEDICINE, RESEARCH & EXPERIMENTAL | PROTEIN | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NOONAN-SYNDROME | CELL BIOLOGY | GENE | STEM-CELL | MYELOID-LEUKEMIA | DIFFERENTIATION | MUTATIONS | IN-VITRO PROPAGATION | DUCTAL CARCINOMA | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - metabolism | Autoantigens - metabolism | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Homeodomain Proteins - metabolism | Humans | Caspase 3 - metabolism | Receptor, ErbB-2 - metabolism | Ki-67 Antigen - metabolism | Gene Expression Profiling | Flow Cytometry | Time Factors | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Female | Membrane Proteins - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Gene Expression Regulation, Neoplastic - physiology | Computational Biology | Transcription Factors - genetics | Mice, SCID | src Homology Domains - physiology | Cell Adhesion Molecules - metabolism | Proto-Oncogene Proteins c-myc - metabolism | Disease Progression | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Animals | Breast Neoplasms - pathology | Cell Polarity - physiology | Doxycycline - pharmacology | Luminescent Proteins - genetics | Signal Transduction - physiology | Mice | Proto-Oncogene Proteins c-myc - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - genetics | Cell Transformation, Neoplastic - pathology | Luminescent Proteins - metabolism | Mitogen-Activated Protein Kinases - metabolism | RNA, Small Interfering - metabolism | Zinc Finger E-box-Binding Homeobox 1 | Transcription factors | Care and treatment | Phosphatases | Physiological aspects | Development and progression | Genetic aspects | Breast cancer | Research | Proteins | Signal transduction | Cellular biology | Gene expression | Index Medicus
Journal Article