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2009, Advances in experimental medicine and biology, ISBN 9781441911445, Volume 656, xvi, 131
The Apc protein is a multifunctional participant in a diverse array of cellular functions. This book provides an overview of the functions performed by the Apc... 
Adenomatous Polyposis Coli | Colorectal Neoplasms | Colon (Anatomy) | metabolism | Adenomatous Polyposis Coli Protein | Tumor suppressor proteins | Genes, APC | Cancer
Book
09/2010, M.D. Anderson solid tumor oncology series, ISBN 9781441966025, Volume 5, 614
Colorectal cancer is the third most commonly diagnosed cancer in the US and the third most recently linked to cancer deaths. This is the first book to cover... 
Colon (Anatomy) | Genetic disorders | Rectum | Cancer
eBook
Journal of Clinical Oncology, ISSN 0732-183X, 10/2015, Volume 33, Issue 2, pp. 209 - 217
Journal Article
Pediatric Blood & Cancer, ISSN 1545-5009, 01/2018, Volume 65, Issue 1, pp. e26756 - n/a
Because children diagnosed with WNT‐activated medulloblastoma have a 10‐year overall survival rate of 95%, active long‐term follow‐up is critically important... 
Wnt signaling pathway | FAP | APC mutations | medulloblastoma | pilomatrixoma | PILOMATRICOMAS | ONCOLOGY | PEDIATRICS | BETA-CATENIN | HEMATOLOGY | CANCER | TUMORS | Hair Diseases - pathology | Hair Diseases - genetics | Cancer Survivors | Cerebellar Neoplasms - diagnosis | Humans | Wnt Proteins - metabolism | Adenomatous Polyposis Coli - pathology | Pilomatrixoma - diagnosis | Wnt Proteins - genetics | Hair Diseases - metabolism | Medulloblastoma - pathology | Skin Neoplasms - diagnosis | Pilomatrixoma - genetics | Germ-Line Mutation | Female | Cerebellar Neoplasms - pathology | Neoplasms, Second Primary - genetics | Adenomatous Polyposis Coli - genetics | Medulloblastoma - genetics | Child | Cerebellar Neoplasms - metabolism | Adenomatous Polyposis Coli Protein - genetics | Skin Neoplasms - pathology | Medulloblastoma - diagnosis | Medulloblastoma - metabolism | Pilomatrixoma - pathology | Cerebellar Neoplasms - genetics | Hair Diseases - diagnosis | Adenomatous Polyposis Coli - metabolism | Neoplasms, Second Primary - diagnosis | Skin Neoplasms - metabolism | Neoplasms, Second Primary - metabolism | Skin Neoplasms - genetics | Adenomatous Polyposis Coli Protein - metabolism | Pilomatrixoma - metabolism | Neoplasms, Second Primary - pathology | Adenomatous Polyposis Coli - diagnosis | Medulloblastoma | Polyposis, Familial | Genetic aspects | Wnt protein | Familial adenomatous polyposis | Adenomatous polyposis coli | Endoscopes | Point mutation | Children | Endoscopy | Mutation | Diagnosis | Polyps | Adenomatous polyposis coli protein | Cancer
Journal Article
Oncogene, ISSN 0950-9232, 12/2011, Volume 30, Issue 50, pp. 4977 - 4989
Familial adenomatous polyposis (FAP) is caused by germline mutations in the adenomatous polyposis coli (APC) gene. Two promoters, 1A and 1B, have been... 
Q-PCR | mutation | promoter | familial adenomatous ployposis | APC | METHYLATION | HYPERMETHYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | BETA-CATENIN | CLINICAL CHARACTERIZATION | REGION | CELL BIOLOGY | GERM-LINE MUTATIONS | ONCOLOGY | COLORECTAL-CANCER | GENETICS & HEREDITY | COLI GENE | EXPRESSION | TRUNCATING MUTATIONS | Adenomatous Polyposis Coli Protein - genetics | Intestinal Mucosa - metabolism | Adenomatous Polyposis Coli Protein - biosynthesis | Adenoma - genetics | Humans | Middle Aged | Gene Silencing | Male | Adenomatous Polyposis Coli - pathology | Promoter Regions, Genetic - genetics | Adenoma - metabolism | Adenomatous Polyposis Coli - metabolism | Sweden | Alleles | Adenoma - pathology | Germ-Line Mutation | Adult | Female | Registries | Aged | Adenomatous Polyposis Coli - genetics | Gene Expression Regulation, Neoplastic - genetics | Intestinal Mucosa - pathology | Oncology | Mutation | Genomics | Cancer | Transcription | Familial adenomatous polyposis | Gene regulation | Mucosa | adenomatous polyposis coli protein | Adenoma | Gene deletion | Blood | Promoters | Gene silencing | adenomatous polyposis coli | Models | Tumorigenesis | Original | Q–PCR | pathology | Dermatologi och venereologi | Dermatology and Venereal Diseases | Genetic | Adenomatous Polyposis Coli Protein | genetics | biosynthesis | Intestinal Mucosa | Promoter Regions | Adenomatous Polyposis Coli | Gene Expression Regulation | metabolism | Cancer and Oncology | Neoplastic | Cancer och onkologi
Journal Article
2008, ISBN 9780470059517, xviii, 441
"This is essential reading for everyone who would like to update their knowledge of the pulmonary epithelium. It offers a comparison between laboratory animal... 
Pulmonary endothelium | Pathophysiology | Epithelium | Respiratory organs
Book
Life Sciences, ISSN 0024-3205, 10/2015, Volume 139, pp. 145 - 152
Colorectal cancer (CRC) is the third leading cause of death in both men and women in North America. Despite chemotherapeutic efforts, CRC is associated with a... 
Colorectal carcinogenesis | Base excision repair | DNA damage | Adenomatous polyposis coli | CANCER-CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | BREAST EPITHELIAL-CELLS | LONG-PATCH | NUCLEAR ANTIGEN | MISMATCH REPAIR | DNA-POLYMERASE-BETA | APC TUMOR-SUPPRESSOR | ALKYLATING-AGENTS | APURINIC APYRIMIDINIC SITES | PHARMACOLOGY & PHARMACY | STRAND DISPLACEMENT SYNTHESIS | Dacarbazine - therapeutic use | Rectum - pathology | Humans | Colon - drug effects | Antineoplastic Agents - therapeutic use | Molecular Targeted Therapy | Adenomatous Polyposis Coli - pathology | Carcinogenesis - metabolism | Adenomatous Polyposis Coli Protein - analysis | Rectum - drug effects | Dacarbazine - analogs & derivatives | DNA Polymerase beta - metabolism | Adenomatous Polyposis Coli - genetics | Adenomatous Polyposis Coli Protein - genetics | DNA Repair - drug effects | Colon - pathology | Rectum - metabolism | Carcinogenesis - genetics | Models, Molecular | Flap Endonucleases - analysis | Flap Endonucleases - metabolism | Colon - metabolism | Adenomatous Polyposis Coli - metabolism | Carcinogenesis - drug effects | Carcinogenesis - pathology | DNA Polymerase beta - analysis | Animals | Adenomatous Polyposis Coli - drug therapy | Adenomatous Polyposis Coli Protein - metabolism | Mutation | Chemotherapy | Colon cancer | Mortality | Development and progression | Polyposis, Familial | Carcinogenesis | Cancer | Index Medicus | base excision repair | colorectal carcinogenesis
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2018, Volume 13, Issue 9, p. e0203845
Journal Article
BMC Medical Genetics, ISSN 1471-2350, 10/2018, Volume 19, Issue 1, pp. 188 - 6
BackgroundFamilial adenomatous polyposis (FAP) is an autosomal dominant hereditary syndrome characterised by the development of hundreds to thousands of... 
Massively parallel sequencing | Germline mutation | Genetic testing | Familial adenomatous polyposis | APC gene | GENE-MUTATIONS | POPULATION