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1. Full Text Adventitia and Perivascular Cells
by Majesky, Mark W
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2015, Volume 35, Issue 8, pp. e31 - e35
atherosclerosis | adipose tissue | arteries | stem cells | adventitia | PROGENITOR CELLS | DEFICIENT MICE | VEIN GRAFTS | MESENCHYMAL STEM-CELLS | INDUCED NEOINTIMAL HYPERPLASIA | VASCULAR FUNCTION | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | ADIPOSE-TISSUE | Vascular Remodeling | Vascular Diseases - pathology | Signal Transduction | Vascular Diseases - therapy | Humans | Adipose Tissue - pathology | Adipose Tissue - physiopathology | Cell Communication | Vascular Diseases - physiopathology | Arteries - pathology | Stem Cells - metabolism | Arteries - metabolism | Adventitia - metabolism | Adventitia - physiopathology | Adipose Tissue - metabolism | Animals | Arteries - physiopathology | Stem Cells - pathology | Adventitia - pathology | Vascular Diseases - metabolism
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2. Full Text NADPH Oxidase 4 Is Expressed in Pulmonary Artery Adventitia and Contributes to Hypertensive Vascular Remodeling
by Barman, Scott A and Chen, Feng and Su, Yunchao and Dimitropoulou, Christiana and Wang, Yusi and Catravas, John D and Han, Weihong and Orfi, Laszlo and Szantai-Kis, Csaba and Keri, Gyorgy and Szabadkai, Istvan and Barabutis, Nektarios and Rafikova, Olga and Rafikov, Ruslan and Black, Stephen M and Jonigk, Danny and Giannis, Athanassios and Asmis, Reto and Stepp, David W and Ramesh, Ganesan and Fulton, David J.R
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2014, Volume 34, Issue 8, pp. 1704 - 1715
OBJECTIVE—Pulmonary hypertension (PH) is a progressive disease arising from remodeling and narrowing of pulmonary arteries (PAs) resulting in high pulmonary... 
Adventitia | Fibroblast | NADPH oxidase | Pulmonary artery | pulmonary artery | ACTIVATION | adventitia | PROLIFERATION | FIBROBLASTS | NAD(P)H OXIDASE | fibroblast | CHRONIC HYPOXIA | MODELS | INFLAMMATION | NOX4 | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | MOLECULAR-MECHANISMS | Up-Regulation | Antihypertensive Agents - pharmacology | Fibroblasts - enzymology | Cell Proliferation | Reactive Oxygen Species - metabolism | Humans | Monocrotaline | NADPH Oxidases - metabolism | Pyrroles | Extracellular Matrix - metabolism | Male | Dose-Response Relationship, Drug | Extracellular Matrix - genetics | Transfection | Time Factors | HEK293 Cells | Adventitia - enzymology | NADPH Oxidases - genetics | Hypertrophy, Right Ventricular - pathology | Hypertension, Pulmonary - drug therapy | Indoles | Hypertension, Pulmonary - enzymology | Disease Models, Animal | Signal Transduction | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Familial Primary Pulmonary Hypertension | Hypertension, Pulmonary - genetics | Hypoxia - complications | Adventitia - drug effects | NADPH Oxidase 4 | Pulmonary Artery - drug effects | Fibroblasts - pathology | Hypertrophy, Right Ventricular - enzymology | Rats, Sprague-Dawley | Pulmonary Artery - enzymology | Animals | Hypertrophy, Right Ventricular - prevention & control | Adventitia - pathology | Mice | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Cell Movement | Index Medicus
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3. Full Text Adventures in the Adventitia
by Lehoux, Stephanie
Hypertension, ISSN 0194-911X, 05/2016, Volume 67, Issue 5, pp. 836 - 838
Adventitia | Humans
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4. Full Text Differentiated Smooth Muscle Cells Generate a Subpopulation of Resident Vascular Progenitor Cells in the Adventitia Regulated by Klf4
by Majesky, Mark W and Horita, Henrick and Ostriker, Allison and Lu, Sizhao and Regan, Jenna N and Bagchi, Ashim and Dong, Xiu Rong and Poczobutt, Joanna and Nemenoff, Raphael A and Weiser-Evans, Mary C.M
Circulation Research, ISSN 0009-7330, 01/2017, Volume 120, Issue 2, pp. 296 - 311
RATIONALE:The vascular adventitia is a complex layer of the vessel wall consisting of vasa vasorum microvessels, nerves, fibroblasts, immune cells, and... 
smooth muscle cells | vascular remodeling | adventitia | STEM-CELLS | CONTRIBUTE | CARDIAC & CARDIOVASCULAR SYSTEMS | INJURY | ATHEROSCLEROSIS | PTEN | VEIN GRAFTS | IDENTIFICATION | INACTIVATION | ORIGIN | GROWTH | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Adventitia - cytology | Adventitia - physiology | Myocytes, Smooth Muscle - physiology | Kruppel-Like Transcription Factors - physiology | Male | Mice, Transgenic | Muscle, Smooth, Vascular - cytology | Mice, Knockout | Pregnancy | Animals | Female | Stem Cells - physiology | Mice | Cell Differentiation - physiology | Muscle, Smooth, Vascular - physiology | CD34 antigen | Subpopulations | Phenotypes | Homeostasis | Smooth muscle | Macrophages | Progenitor cells | Hemopoiesis | Cell fate | Ataxin | KLF4 protein | Nerves | Stem cells | Fibroblasts | reprogramming | Stem Cells | vascular biology | Smooth muscle cells | progenitor cell | Kruppel-like factor 4 | Smooth Muscle Proliferation and Differentiation | Cellular Reprogramming
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5. Full Text Vasa Vasorum in Normal and Diseased Arteries
by Mulligan-Kehoe, Mary Jo and Simons, Michael
Circulation, ISSN 0009-7322, 06/2014, Volume 129, Issue 24, pp. 2557 - 2566
Vasa vasorum | Endothelial cells | Coronary arteries | PROGENITOR CELLS | endothelial cells | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROTIC PLAQUES | ANGIOGENESIS | CORONARY-ARTERIES | TO-MESENCHYMAL TRANSITION | coronary arteries | vasa vasorum | ENDOTHELIAL-CELLS | SAPHENOUS-VEIN | PERIPHERAL VASCULAR DISEASE | INTRAPLAQUE HEMORRHAGE | INFLAMMATORY RESPONSE | WOUND REPAIR | Atherosclerosis - physiopathology | Vasa Vasorum - physiology | Adventitia - physiology | Humans | Adventitia - blood supply | Adventitia - physiopathology | Animals | Arteries - physiology | Veins - physiopathology | Arteries - physiopathology | Homeostasis - physiology | Veins - physiology | Vasa Vasorum - physiopathology | Physiological aspects | Heart diseases | Arteries | Diseases
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6. Full Text CYLD Deubiquitinates Nicotinamide Adenine Dinucleotide Phosphate Oxidase 4 Contributing to Adventitial Remodeling
by Yu, Bing and Liu, Ziyi and Fu, Yi and Wang, Yingbao and Zhang, Lu and Cai, Zeyu and Yu, Fang and Wang, Xian and Zhou, Jun and Kong, Wei
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2017, Volume 37, Issue 9, pp. 1698 - 1709
OBJECTIVE—Transdifferentiation of adventitial fibroblasts (AFs) into myofibroblasts plays a critical role during the vascular remodeling that occurs during... 
aneurysm | adventitia | NADPH oxidase | deubiquitinating enzymes | MYOFIBROBLAST | NEOINTIMAL HYPERPLASIA | INDUCED NOX4 EXPRESSION | KAPPA-B | MICROTUBULE DYNAMICS | TUMOR-SUPPRESSOR CYLD | IN-VITRO | ABDOMINAL AORTIC-ANEURYSM | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | PULMONARY-FIBROSIS | Aortic Aneurysm, Abdominal - chemically induced | Reactive Oxygen Species - metabolism | Humans | NADPH Oxidases - metabolism | Extracellular Matrix - metabolism | Half-Life | Male | Ubiquitination | RNA Interference | Time Factors | Proteolysis | Homocysteine - pharmacology | Adventitia - enzymology | NADPH Oxidases - genetics | Vascular Remodeling - drug effects | Interleukin-6 - metabolism | Transforming Growth Factor beta1 - pharmacology | Disease Models, Animal | Hyperhomocysteinemia - enzymology | Signal Transduction | Enzyme Stability | Genotype | Adventitia - drug effects | Myofibroblasts - drug effects | Ubiquitin Thiolesterase - genetics | Rats, Sprague-Dawley | Mice, Knockout | Phenotype | Aortic Aneurysm, Abdominal - enzymology | Cysteine Endopeptidases - genetics | Adventitia - pathology | COS Cells | Hyperhomocysteinemia - complications | Cell Movement | Cercopithecus aethiops | Myofibroblasts - enzymology | Dose-Response Relationship, Drug | Calcium Phosphates | Cysteine Endopeptidases - metabolism | Transfection | Cell Transdifferentiation - drug effects | HEK293 Cells | Ubiquitin Thiolesterase - metabolism | Cysteine Endopeptidases - deficiency | Chemokine CCL2 - metabolism | Aortic Aneurysm, Abdominal - pathology | Myofibroblasts - pathology | Mice, Inbred C57BL | Aortic Aneurysm, Abdominal - genetics | NADPH Oxidase 4 | Animals | Hyperhomocysteinemia - genetics
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7. Full Text C-Myb Regulates Proliferation and Differentiation of Adventitial Sca1+ Vascular Smooth Muscle Cell Progenitors by Transactivation of Myocardin
by Shikatani, Eric A and Chandy, Mark and Besla, Rickvinder and Li, Cedric C and Momen, Abdul and El-Mounayri, Omar and Robbins, Clinton S and Husain, Mansoor
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2016, Volume 36, Issue 7, pp. 1367 - 1376
Objective-Vascular smooth muscle cells (VSMCs) are believed to dedifferentiate and proliferate in response to vessel injury. Recently, adventitial progenitor... 
Myocardin | c-Myb genes | Cell differentiation | Smooth muscle cells | Progenitor cells | ARTERIAL INJURY | cell differentiation | STEM-CELLS | ATHEROSCLEROSIS | progenitor cells | NEOINTIMA FORMATION | VEIN GRAFTS | IDENTIFICATION | VESSEL WALL | OLIGONUCLEOTIDES | smooth muscle cells | myocardin | PERIPHERAL VASCULAR DISEASE | ACCUMULATION | HEMATOLOGY | EXPRESSION | Vascular Remodeling | Muscle, Smooth, Vascular - metabolism | Humans | Transcriptional Activation | Myocytes, Smooth Muscle - pathology | Proto-Oncogene Proteins c-myb - genetics | Adventitia - injuries | Stem Cells - metabolism | Adventitia - metabolism | Transfection | Time Factors | Ataxin-1 - metabolism | HEK293 Cells | Trans-Activators - genetics | Transcription, Genetic | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - drug effects | Nuclear Proteins - genetics | Binding Sites | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Transforming Growth Factor beta1 - pharmacology | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Promoter Regions, Genetic | Signal Transduction | Carotid Artery, Common - metabolism | Mice, Inbred C57BL | Genotype | Mice, Transgenic | Nuclear Proteins - metabolism | Adventitia - drug effects | Proto-Oncogene Proteins c-myb - metabolism | Muscle, Smooth, Vascular - injuries | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Cell Differentiation - drug effects | Stem Cells - drug effects | Stem Cells - pathology | Carotid Artery, Common - pathology | Trans-Activators - metabolism | Adventitia - pathology | Cell Proliferation - drug effects | Index Medicus
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8. Full Text Upregulated interleukins (IL-6, IL-10, and IL-13) in immunoglobulin G4-related aortic aneurysm patients
by Kasashima, Satomi, MD, PhD and Kawashima, Atsuhiro, MD, PhD and Zen, Yoh, MD, PhD and Ozaki, Satoru, PhD and Kasashima, Fuminori, MD, PhD and Endo, Masamitsu, MD, PhD and Matsumoto, Yasushi, MD, PhD and Kawakami, Kengo, MD, PhD
Journal of Vascular Surgery, ISSN 0741-5214, 2017, Volume 67, Issue 4, pp. 1248 - 1262
Abstract Objective Immunoglobulin (Ig) G4-related aortic aneurysms (IgG4-AAs) are a special aortic aneurysm among IgG4-related diseases (IgG4-RDs), which are... 
Surgery | SURGERY | LARGE-VESSEL VASCULITIS | IGG4-RELATED DISEASE | CASTLEMANS-DISEASE | TGF-BETA | MAST-CELLS | SJOGRENS-SYNDROME | PERIPHERAL VASCULAR DISEASE | EXPRESSION | CHRONIC PERIAORTITIS | MIKULICZS-DISEASE | TH2 | Immunohistochemistry | T-Lymphocyte Subsets - immunology | Macrophages - chemistry | Up-Regulation | Aortic Aneurysm, Abdominal - immunology | Immunoglobulin G - blood | Humans | Middle Aged | Adventitia - immunology | Interleukin-10 - blood | Aortic Aneurysm, Abdominal - diagnostic imaging | Male | Antigens, CD - genetics | Aortic Aneurysm, Abdominal - blood | Case-Control Studies | Aortography - methods | Aorta, Abdominal - diagnostic imaging | In Situ Hybridization | Interleukin-6 - blood | T-Lymphocyte Subsets - chemistry | Aged, 80 and over | Female | Aorta, Abdominal - pathology | Interleukin-13 - blood | Macrophages - immunology | Endothelial Cells - chemistry | Interleukin-6 - genetics | Inflammation Mediators - blood | Adventitia - chemistry | Aortic Aneurysm, Abdominal - genetics | Biomarkers - blood | Antigens, Differentiation, Myelomonocytic - genetics | Computed Tomography Angiography | Aorta, Abdominal - immunology | Endothelial Cells - immunology | Aorta, Abdominal - chemistry | Adventitia - pathology | Aged | Antigens, CD34 - genetics | Receptors, Cell Surface - genetics
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9. Full Text STAT3-mediated MMP-2 expression is required for 15-HETE-induced vascular adventitial fibroblast migration
by Zhang, Li and Li, Yumei and Liu, Yumei and Wang, Xiaoyan and Chen, Minggang and Xing, Yan and Zhu, Daling
Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 05/2015, Volume 149, pp. 106 - 117
Vascular adventitial fibroblasts (VAFs) migration was involved in neointima formation, and increased 15-HETE levels contributed to vascular remodeling.... 
15-HETE | MMP-2 | Adventitia fibroblast | Migration | STAT3 | CELLS | ACTIVATION | ANGIOPLASTY | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOCRINOLOGY & METABOLISM | HYPOXIA | Up-Regulation | Vascular Remodeling | Rats, Wistar | Adventitia - cytology | Male | Adventitia - metabolism | Neointima - genetics | STAT3 Transcription Factor - metabolism | Fibroblasts - metabolism | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Mitogen-Activated Protein Kinase 8 - metabolism | Cells, Cultured | Fibroblasts - pathology | Neointima - metabolism | Matrix Metalloproteinase 2 - genetics | Hydroxyeicosatetraenoic Acids - metabolism | Animals | Cyclic AMP Response Element-Binding Protein - metabolism | Neointima - pathology | Adventitia - pathology | Fibroblasts - cytology | Enzyme Activation | Lung - blood supply | Cell Movement
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10. Full Text Varicella-zoster virus downregulates programmed death ligand 1 and major histocompatibility complex class i in human brain vascular adventitial fibroblasts, perineurial cells, and lung fibroblasts
by Jones, Dallas and Blackmon, Anna and Neff, C. Preston and Palmer, Brent E and Gilden, Don and Badani, Hussain and Nagel, Maria A
Journal of Virology, ISSN 0022-538X, 2016, Volume 90, Issue 23, pp. 10527 - 10534
Varicella-zoster virus (VZV) vasculopathy produces stroke, giant cell arteritis, and granulomatous aortitis, and it develops after virus reactivates from... 
INTERFERON | GANGLIA | VIROLOGY | INFECTED ARTERIES | PATHWAY | AUTOIMMUNE-DISEASES | DNA | INFLAMMATION | TOLERANCE | PD-1 | EXPRESSION | Fibroblasts - virology | Herpesviridae Infections - etiology | Down-Regulation | Humans | Adventitia - immunology | Cells, Cultured | Adventitia - blood supply | Brain - virology | B7-H1 Antigen - genetics | Host-Pathogen Interactions - immunology | Histocompatibility Antigens Class I - metabolism | Brain - blood supply | B7-H1 Antigen - metabolism | Lung - virology | Neuroglia - immunology | Adventitia - virology | Fibroblasts - immunology | Herpesvirus 3, Human - pathogenicity | Neuroglia - virology | Brain - immunology | Lung - immunology | Herpesviridae Infections - immunology
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11. Full Text Distribution of macrophage polarization markers in human atherosclerosis
by Stöger, J. Lauran and Gijbels, Marion J.J and van der Velden, Saskia and Manca, Marco and van der Loos, Chris M and Biessen, Erik A.L and Daemen, Mat J.A.P and Lutgens, Esther and de Winther, Menno P.J
Atherosclerosis, ISSN 0021-9150, 2012, Volume 225, Issue 2, pp. 461 - 468
Abstract Objective Macrophages are decisive in the chronic inflammatory processes that drive atherogenesis. The purpose of this study was to explore the... 
Cardiovascular | Innate immunity | Foam cell | Macrophages | Coronary artery disease | Human pathology | RECRUITMENT | ACTIVATION | PHENOTYPE | PLAQUE | HETEROGENEITY | INFLAMMATION | RUPTURE | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | ADIPOSE-TISSUE | PLASTICITY | Immunohistochemistry | Humans | Adventitia - immunology | Transcriptome | RNA, Messenger - analysis | Male | Gene Expression Profiling | Inflammation Mediators - analysis | Carotid Artery Diseases - pathology | Aged, 80 and over | Female | Macrophages - immunology | Carotid Artery Diseases - immunology | Severity of Illness Index | Macrophages - classification | Macrophages - pathology | Biomarkers - analysis | Gene Expression Regulation | Plaque, Atherosclerotic | Disease Progression | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Fibrosis | Adventitia - pathology | Aged | Carotid Arteries - pathology | Rupture, Spontaneous | Atherosclerosis
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12. Full Text Inhibition of phosphorylated-STAT1 nuclear translocation and antiviral protein expression in human brain vascular adventitial fibroblasts infected with varicella-zoster virus
by Nagel, Maria A and James, Stephanie F and Traktinskiy, Igor and Wyborny, Ann and Choe, Alexander and Rempel, April and Baird, Nicholas L and Gilden, Don
Journal of Virology, ISSN 0022-538X, 2014, Volume 88, Issue 19, pp. 11634 - 11637
In varicella-zoster virus (VZV)-infected primary human brain vascular adventitial fibroblasts (BRAFs), levels of beta interferon (IFN-beta,) STAT1, and STAT2... 
HUMAN NEURONS | IRF3 | VIROLOGY | Phosphorylation | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Adventitia - blood supply | Brain - virology | Adventitia - metabolism | Brain - metabolism | Brain - blood supply | STAT1 Transcription Factor - metabolism | Blood Vessels - virology | Herpesvirus 3, Human - metabolism | Interferon-alpha - genetics | Interferon-beta - genetics | Adventitia - virology | Fibroblasts - metabolism | Fibroblasts - virology | Interferon-alpha - metabolism | Signal Transduction | Myxovirus Resistance Proteins - antagonists & inhibitors | Gene Expression Regulation | Fibroblasts - pathology | STAT1 Transcription Factor - genetics | Herpesvirus 3, Human - genetics | Protein Transport | Host-Pathogen Interactions | Interferon-beta - metabolism | STAT2 Transcription Factor - genetics | Brain - pathology | Adventitia - pathology | STAT2 Transcription Factor - metabolism | Primary Cell Culture | Myxovirus Resistance Proteins - genetics | Myxovirus Resistance Proteins - metabolism | Virus-Cell Interactions
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13. Full Text Biaxial deformation of collagen and elastin fibers in coronary adventitia
by Huan Chen and Mikhail N. Slipchenko and Yi Liu and Xuefeng Zhao and Ji-Xin Cheng and Yoram Lanir and Ghassan S. Kassab
Journal of Applied Physiology, ISSN 8750-7587, 12/2013, Volume 115, Issue 11, pp. 1683 - 1693
The microstructural deformation-mechanical loading relation of the blood vessel wall is essential for understanding the overall mechanical behavior of vascular... 
Adventitia | Deformation | Microstructure | Collagen | Elastin | Multiphoton microscopy | SPORT SCIENCES | PHYSIOLOGY | microstructure | adventitia | FIBROUS CONNECTIVE TISSUES | collagen | CONSTITUTIVE MODEL | 2-LAYER MODEL | MICROSCOPY | elastin | MECHANICAL-PROPERTIES | MICROMECHANICS | multiphoton microscopy | ARTERIAL-WALL | 2ND-HARMONIC GENERATION | deformation | MEDIA | EXTRACELLULAR-MATRIX | Adventitia - metabolism | Heart - physiopathology | Collagen - metabolism | Elastin - metabolism | Coronary Vessels - pathology | Animals | Swine | Adventitia - pathology | Coronary Vessels - metabolism
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14. Full Text Contribution of acid sphingomyelinase to angiotensin II-induced vascular adventitial remodeling via membrane rafts/Nox2 signal pathway
by Li, Xin and Li, Xiao-Xue and Wang, Hui-Fang and Xu, Ming
Life Sciences, ISSN 0024-3205, 02/2019, Volume 219, pp. 303 - 310
Vascular adventitial fibroblasts (AFs) in the vascular remodeling during atherosclerosis are increasing arousing attention. Acid sphingomyelinase (ASM) is a... 
Ceramide | Vascular adventitial fibroblast | Nox2 | Ang II | Acid sphingomyelinase | MEDICINE, RESEARCH & EXPERIMENTAL | NADPH OXIDASE | ACTIVATION | FUSION | RETENTION | ATHEROSCLEROSIS | FIBROBLASTS | LIPOPROTEIN | PHARMACOLOGY & PHARMACY | EXPRESSION | Angiotensin II - pharmacology | Immunoprecipitation | Membrane Microdomains - metabolism | Adventitia - physiology | Gene Silencing | Rats | Male | Adventitia - drug effects | Rats, Sprague-Dawley | Blotting, Western | NADPH Oxidase 2 - physiology | Adventitia - metabolism | Microscopy, Confocal | Animals | NADPH Oxidase 2 - metabolism | Sphingomyelin Phosphodiesterase - metabolism | Signal Transduction - physiology | Vascular Remodeling - physiology | Sphingomyelin Phosphodiesterase - physiology | Vascular Remodeling - drug effects
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15. Full Text Tracking Adventitial Fibroblast Contribution to Disease: A Review of Current Methods to Identify Resident Fibroblasts
by Kuwabara, Jill T and Tallquist, Michelle D
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2017, Volume 37, Issue 9, pp. 1598 - 1607
Cells present in the adventitia, or outermost layer of the blood vessel, contribute to the progression of vascular diseases, such as atherosclerosis,... 
aorta | adventitia | fibroblast | hypertension | mice | PROGENITOR CELLS | CARDIAC FIBROBLAST | SONIC HEDGEHOG | VEIN GRAFTS | INTERNAL ELASTIC LAMINA | VASCULAR SMOOTH-MUSCLE | MESENCHYMAL STEM-CELLS | IN-VIVO | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | HEMATOLOGY | PERIVASCULAR FIBROBLASTS | Biomarkers - metabolism | Cardiovascular Diseases - metabolism | Humans | Genotype | Mice, Transgenic | Cardiovascular Diseases - pathology | Fibroblasts - pathology | Cardiovascular Diseases - genetics | Adventitia - metabolism | Cell Lineage | Phenotype | Animals | Adventitia - pathology | Fibroblasts - metabolism | perivascular
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