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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2014, Volume 34, Issue 8, pp. 1704 - 1715
OBJECTIVE—Pulmonary hypertension (PH) is a progressive disease arising from remodeling and narrowing of pulmonary arteries (PAs) resulting in high pulmonary... 
Adventitia | Fibroblast | NADPH oxidase | Pulmonary artery | pulmonary artery | ACTIVATION | adventitia | PROLIFERATION | FIBROBLASTS | NAD(P)H OXIDASE | fibroblast | CHRONIC HYPOXIA | MODELS | INFLAMMATION | NOX4 | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | MOLECULAR-MECHANISMS | Up-Regulation | Antihypertensive Agents - pharmacology | Fibroblasts - enzymology | Cell Proliferation | Reactive Oxygen Species - metabolism | Humans | Monocrotaline | NADPH Oxidases - metabolism | Pyrroles | Extracellular Matrix - metabolism | Male | Dose-Response Relationship, Drug | Extracellular Matrix - genetics | Transfection | Time Factors | HEK293 Cells | Adventitia - enzymology | NADPH Oxidases - genetics | Hypertrophy, Right Ventricular - pathology | Hypertension, Pulmonary - drug therapy | Indoles | Hypertension, Pulmonary - enzymology | Disease Models, Animal | Signal Transduction | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Familial Primary Pulmonary Hypertension | Hypertension, Pulmonary - genetics | Hypoxia - complications | Adventitia - drug effects | NADPH Oxidase 4 | Pulmonary Artery - drug effects | Fibroblasts - pathology | Hypertrophy, Right Ventricular - enzymology | Rats, Sprague-Dawley | Pulmonary Artery - enzymology | Animals | Hypertrophy, Right Ventricular - prevention & control | Adventitia - pathology | Mice | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Cell Movement | Index Medicus
Journal Article
Hypertension, ISSN 0194-911X, 05/2016, Volume 67, Issue 5, pp. 836 - 838
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2017, Volume 37, Issue 9, pp. 1698 - 1709
OBJECTIVE—Transdifferentiation of adventitial fibroblasts (AFs) into myofibroblasts plays a critical role during the vascular remodeling that occurs during... 
aneurysm | adventitia | NADPH oxidase | deubiquitinating enzymes | MYOFIBROBLAST | NEOINTIMAL HYPERPLASIA | INDUCED NOX4 EXPRESSION | KAPPA-B | MICROTUBULE DYNAMICS | TUMOR-SUPPRESSOR CYLD | IN-VITRO | ABDOMINAL AORTIC-ANEURYSM | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | PULMONARY-FIBROSIS | Aortic Aneurysm, Abdominal - chemically induced | Reactive Oxygen Species - metabolism | Humans | NADPH Oxidases - metabolism | Extracellular Matrix - metabolism | Half-Life | Male | Ubiquitination | RNA Interference | Time Factors | Proteolysis | Homocysteine - pharmacology | Adventitia - enzymology | NADPH Oxidases - genetics | Vascular Remodeling - drug effects | Interleukin-6 - metabolism | Transforming Growth Factor beta1 - pharmacology | Disease Models, Animal | Hyperhomocysteinemia - enzymology | Signal Transduction | Enzyme Stability | Genotype | Adventitia - drug effects | Myofibroblasts - drug effects | Ubiquitin Thiolesterase - genetics | Rats, Sprague-Dawley | Mice, Knockout | Phenotype | Aortic Aneurysm, Abdominal - enzymology | Cysteine Endopeptidases - genetics | Adventitia - pathology | COS Cells | Hyperhomocysteinemia - complications | Cell Movement | Cercopithecus aethiops | Myofibroblasts - enzymology | Dose-Response Relationship, Drug | Calcium Phosphates | Cysteine Endopeptidases - metabolism | Transfection | Cell Transdifferentiation - drug effects | HEK293 Cells | Ubiquitin Thiolesterase - metabolism | Cysteine Endopeptidases - deficiency | Chemokine CCL2 - metabolism | Aortic Aneurysm, Abdominal - pathology | Myofibroblasts - pathology | Mice, Inbred C57BL | Aortic Aneurysm, Abdominal - genetics | NADPH Oxidase 4 | Animals | Hyperhomocysteinemia - genetics
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2016, Volume 36, Issue 7, pp. 1367 - 1376
Objective-Vascular smooth muscle cells (VSMCs) are believed to dedifferentiate and proliferate in response to vessel injury. Recently, adventitial progenitor... 
Myocardin | c-Myb genes | Cell differentiation | Smooth muscle cells | Progenitor cells | ARTERIAL INJURY | cell differentiation | STEM-CELLS | ATHEROSCLEROSIS | progenitor cells | NEOINTIMA FORMATION | VEIN GRAFTS | IDENTIFICATION | VESSEL WALL | OLIGONUCLEOTIDES | smooth muscle cells | myocardin | PERIPHERAL VASCULAR DISEASE | ACCUMULATION | HEMATOLOGY | EXPRESSION | Vascular Remodeling | Muscle, Smooth, Vascular - metabolism | Humans | Transcriptional Activation | Myocytes, Smooth Muscle - pathology | Proto-Oncogene Proteins c-myb - genetics | Adventitia - injuries | Stem Cells - metabolism | Adventitia - metabolism | Transfection | Time Factors | Ataxin-1 - metabolism | HEK293 Cells | Trans-Activators - genetics | Transcription, Genetic | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - drug effects | Nuclear Proteins - genetics | Binding Sites | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Transforming Growth Factor beta1 - pharmacology | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Promoter Regions, Genetic | Signal Transduction | Carotid Artery, Common - metabolism | Mice, Inbred C57BL | Genotype | Mice, Transgenic | Nuclear Proteins - metabolism | Adventitia - drug effects | Proto-Oncogene Proteins c-myb - metabolism | Muscle, Smooth, Vascular - injuries | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Cell Differentiation - drug effects | Stem Cells - drug effects | Stem Cells - pathology | Carotid Artery, Common - pathology | Trans-Activators - metabolism | Adventitia - pathology | Cell Proliferation - drug effects | Index Medicus
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 2017, Volume 67, Issue 4, pp. 1248 - 1262
Journal Article
Journal Article
Journal Article