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Clinica Chimica Acta, ISSN 0009-8981, 01/2015, Volume 438, pp. 401 - 414
The link between vascular calcification (VC) and increased mortality is now well established. Over time, as clinical importance of this phenomenon has begun to... 
Vascular calcification | Bone morphogenetic protein-2 | Fetuin-A | Fibroblast growth factor-23 | Matrix Gla protein | Osteoprotegerin | MAINTENANCE HEMODIALYSIS-PATIENTS | PERITONEAL-DIALYSIS PATIENTS | RENAL-TRANSPLANT RECIPIENTS | HUMAN ATHEROSCLEROTIC PLAQUES | SMOOTH-MUSCLE-CELLS | MEDICAL LABORATORY TECHNOLOGY | CHRONIC KIDNEY-DISEASE | MATRIX-GLA-PROTEIN | CORONARY-ARTERY-DISEASE | Dyslipidemias - genetics | Diabetes Complications | Dyslipidemias - complications | Diabetes Mellitus - genetics | Humans | Vascular Calcification - genetics | Fibroblast Growth Factors - genetics | Renal Insufficiency, Chronic - complications | Renal Insufficiency, Chronic - metabolism | Fibroblast Growth Factors - metabolism | Vascular Calcification - metabolism | Vascular Calcification - etiology | Bone Morphogenetic Protein 2 - metabolism | Osteoprotegerin - genetics | Renal Insufficiency, Chronic - genetics | Vascular Calcification - physiopathology | Extracellular Matrix Proteins - metabolism | Biomarkers - metabolism | Calcium-Binding Proteins - metabolism | Bone Morphogenetic Protein 2 - genetics | Risk Assessment | Signal Transduction | Extracellular Matrix Proteins - genetics | Gene Expression Regulation | Diabetes Mellitus - metabolism | Renal Insufficiency, Chronic - physiopathology | Diabetes Mellitus - physiopathology | Dyslipidemias - metabolism | alpha-2-HS-Glycoprotein - metabolism | Osteoprotegerin - metabolism | alpha-2-HS-Glycoprotein - genetics | Calcium-Binding Proteins - genetics | Dyslipidemias - physiopathology | Calcification | Biological markers | Bone morphogenetic proteins | Life Sciences | Cellular Biology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 02/2015, Volume 35, Issue 2, pp. 399 - 408
OBJECTIVE—Vascular and valvular calcifications are pathological processes regulated by resident cells, and depending on a complex interplay between... 
aortic valve | multivesicular bodies | calcification of | gene expression | vascular calcification | AORTIC-STENOSIS | MATRIX VESICLES | VALVE | aortic valve, calcification of | CARTILAGE | DISEASE | VITAMIN-K | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | FETUIN-A | INSIGHTS | Calcinosis - genetics | Aortic Valve Stenosis - genetics | Calcium - metabolism | Humans | Middle Aged | Vascular Calcification - genetics | Actins - metabolism | Osteocalcin - genetics | Male | Aorta - metabolism | Case-Control Studies | Aortic Valve - pathology | Coronary Vessels - metabolism | Aortic Valve Stenosis - prevention & control | Vascular Calcification - metabolism | Coronary Artery Disease - pathology | Aged, 80 and over | Adult | Female | Calcinosis - metabolism | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Coronary Vessels - pathology | Osteocalcin - metabolism | Tissue Culture Techniques | Coronary Artery Disease - metabolism | Extracellular Matrix Proteins - genetics | Coronary Artery Disease - prevention & control | Gene Expression Regulation | Vascular Calcification - prevention & control | Vascular Calcification - pathology | Aortic Valve - metabolism | Aorta - pathology | Proteins - genetics | Aortic Valve Stenosis - pathology | Proteins - metabolism | Coronary Artery Disease - genetics | alpha-2-HS-Glycoprotein - metabolism | Aortic Valve Stenosis - metabolism | Aged | Calcinosis - prevention & control | Calcinosis - pathology | Calcium-Binding Proteins - genetics
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 2017, Volume 26, Issue 11, pp. 2156 - 2163
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23730
Genetic background may play an important role in the process of SARS-CoV infection and SARS development. We found several proteins that could interact with the... 
HEALTH-CARE WORKERS | ALPHA-2-HS GLYCOPROTEIN | FETUIN | SYNDROME (SARS)-ASSOCIATED CORONAVIRUS | MANNOSE-BINDING LECTIN | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | NUCLEOCAPSID PROTEIN | SYNDROME CORONAVIRUS INFECTION | POLYMORPHISM | ACUTE RESPIRATORY SYNDROME | Cell Line | Promoter Regions, Genetic | Severe Acute Respiratory Syndrome - virology | Humans | Linkage Disequilibrium - genetics | Genotype | Male | SARS Virus - pathogenicity | Hep G2 Cells | Young Adult | Adolescent | Polymorphism, Single Nucleotide - genetics | Severe Acute Respiratory Syndrome - genetics | alpha-2-HS-Glycoprotein - metabolism | Adult | Female | Genetic Variation - genetics | alpha-2-HS-Glycoprotein - genetics | Severe Acute Respiratory Syndrome - blood | Cytochrome | Drugs | Biotechnology | Transcription | Laboratories | Liver | Genes | Linkage disequilibrium | Sex | Viruses | Smooth muscle | Infections | Single-nucleotide polymorphism | Macrophages | Epidemiology | Proteins | Confidence intervals | Cell cycle | Genotypes | Pathogens | Statistical analysis | Liver diseases | Deactivation | Hematology | Cytochrome P450 | Severe acute respiratory syndrome | Health risks | Glycoprotein | Glycoproteins | Leukocytes (neutrophilic) | Inflammation | Genetic diversity | Medicine | Leukotriene B4 | Engineering research | Rheumatoid arthritis | Proteomics | Cations | Diabetes | Gene mapping | Viral infections
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 11/2013, Volume 273, Issue 1, pp. 219 - 226
Bone is a target for high affinity aryl hydrocarbon receptor (AHR) ligands, such as dioxins. Although bone morphology, mineral density and strength are... 
Tissue quality | Aryl hydrocarbon receptor | Bone microstructure | Micro-computed tomography | Bone strength | Nanoindentation | RESISTANT RAT STRAINS | LONG-TERM EXPOSURE | OSTEOBLASTS IN-VITRO | COMPUTED-TOMOGRAPHY | SEAL HALICHOERUS-GRYPUS | MINERAL DENSITY | AH-RECEPTOR | ARYLHYDROCARBON RECEPTOR | 2,3,7,8-TETRACHLORODIBENZO-P-DIOXIN TCDD | PHARMACOLOGY & PHARMACY | TOXICOLOGY | 3,3',4,4',5-PENTACHLOROBIPHENYL PCB126 | Collagen Type I - blood | Vesicular Transport Proteins - metabolism | Body Weight - drug effects | Male | Bone Remodeling - drug effects | Bone and Bones - drug effects | Collagen Type II - metabolism | Collagen Type X - metabolism | Bone and Bones - metabolism | Peptide Fragments - blood | Receptors, Aryl Hydrocarbon - metabolism | Female | Nuclear Proteins - genetics | Osteogenesis - genetics | Mice, Inbred C57BL | Vesicular Transport Proteins - genetics | Gene Expression Regulation | Receptors, Aryl Hydrocarbon - genetics | Collagen Type X - genetics | Nuclear Proteins - metabolism | Procollagen - blood | Biomarkers - blood | Collagen Type II - genetics | Mice, Knockout | Phenotype | Animals | Vascular Endothelial Growth Factor B - metabolism | Polychlorinated Dibenzodioxins - toxicity | alpha-2-HS-Glycoprotein - metabolism | Vascular Endothelial Growth Factor B - genetics | Mice | alpha-2-HS-Glycoprotein - genetics | Herbicides | Bones | Genetic aspects | Density | Dioxin | Analysis | Index Medicus | DIOXIN | HYDROCARBONS | PHENOTYPE | TOXICITY | 60 APPLIED LIFE SCIENCES | COMPUTERIZED TOMOGRAPHY | SKELETON | TRABECULAR BONE | LIGANDS | GENES | MICE | RECEPTORS | MICROSTRUCTURE | Farmakologi och toxikologi | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Fysiologi
Journal Article
Journal Article
Atherosclerosis, ISSN 0021-9150, 2015, Volume 241, Issue 1, pp. 130 - 137
Abstract Objective Adipose Tissue (AT) dysregulation contributes to the pro-inflammatory state and insulin resistance of Metabolic Syndrome (MetS). We examined... 
Cardiovascular | Adipose tissue | TLR | Metabolic syndrome | Lipopolysaccharide binding protein | Fetuin A | CARDIAC & CARDIOVASCULAR SYSTEMS | MARKER | ENDOTOXIN | MONOCYTES | HUMANS | OBESITY | PATTERN | INSULIN-RESISTANCE | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | RECEPTOR-ACTIVITY | EXPRESSION | Up-Regulation | alpha-2-HS-Glycoprotein - analysis | Carrier Proteins - secretion | Humans | Middle Aged | Male | Acute-Phase Proteins - genetics | RNA, Messenger - metabolism | Case-Control Studies | HMGB1 Protein - genetics | HMGB1 Protein - secretion | Young Adult | Metabolic Syndrome - blood | Subcutaneous Fat - metabolism | Adiposity | Subcutaneous Fat - secretion | HMGB1 Protein - metabolism | Metabolic Syndrome - physiopathology | Adult | Female | Adipogenesis | Disease Models, Animal | Carrier Proteins - blood | Acute-Phase Proteins - secretion | Mice, Inbred C57BL | alpha-2-HS-Glycoprotein - secretion | Membrane Glycoproteins - blood | Obesity - physiopathology | Biomarkers - blood | 3T3-L1 Cells | Lipopolysaccharide Receptors - blood | Membrane Glycoproteins - genetics | Obesity - metabolism | Carrier Proteins - genetics | Membrane Glycoproteins - secretion | Animals | Toll-Like Receptor 4 - blood | Metabolic Syndrome - diagnosis | Metabolic Syndrome - genetics | Adipocytes - metabolism | Toll-Like Receptor 2 - blood | alpha-2-HS-Glycoprotein - metabolism | Mice, Obese | Aged | Mice | alpha-2-HS-Glycoprotein - genetics | HMGB1 Protein - blood | Adipose tissues | Medical colleges | Insulin resistance | Mitogens | Atherosclerosis | Protein binding
Journal Article