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1. Full Text Amylin selectively signals onto POMC neurons in the arcuate nucleus of the hypothalamus
by Lutz, Thomas A and Coester, Bernd and Whiting, Lynda and Dunn-Meynell, Ambrose A and Boyle, Christina N and Bouret, Sebastien G and Levin, Barry E and Le Foll, Christelle
Diabetes, ISSN 0012-1797, 05/2018, Volume 67, Issue 5, pp. 805 - 817
Amylin phosphorylates ERK (p-ERK) in the area postrema to reduce eating and synergizes with leptin to phosphorylate STAT3 in the arcuate (ARC) and ventromedial... 
CALCITONIN-RECEPTOR | LEPTIN SENSITIVITY | GLUCOSE-HOMEOSTASIS | AREA POSTREMA NUCLEUS | GENE-RELATED PEPTIDE | ENERGY HOMEOSTASIS | ENDOCRINOLOGY & METABOLISM | DIET-INDUCED OBESITY | ACTIVATED NEURONS | NEUROPEPTIDE-Y SYSTEM | FEEDING CIRCUITS | Leptin - metabolism | Male | Islet Amyloid Polypeptide - genetics | Receptor Activity-Modifying Protein 3 - genetics | alpha-MSH - metabolism | MAP Kinase Signaling System | Neuropeptide Y - metabolism | Female | Neurons - metabolism | Animals, Newborn | Arcuate Nucleus of Hypothalamus - metabolism | Islet Amyloid Polypeptide - metabolism | Receptor Activity-Modifying Protein 1 - metabolism | Feeding Behavior | Ventromedial Hypothalamic Nucleus - metabolism | Receptor Activity-Modifying Protein 1 - genetics | Agouti-Related Protein - metabolism | Rats, Sprague-Dawley | Mice, Knockout | Pro-Opiomelanocortin - metabolism | Animals | Receptor Activity-Modifying Protein 3 - metabolism | Paraventricular Hypothalamic Nucleus - metabolism | Mice, Obese | Mice | Research | Pro-opiomelanocortin | Hypothalamus | Peptide hormones | Health aspects | Protein-protein interactions | Metabolism | 0701
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2. Full Text Changes in Melanocortin Expression and Inflammatory Pathways in Fetal Offspring of Nonhuman Primates Fed a High-Fat Diet
by Grayson, B. E and Levasseur, P. R and Williams, S. M and Smith, M. S and Marks, D. L and Grove, K. L
Endocrinology, ISSN 0013-7227, 04/2010, Volume 151, Issue 4, pp. 1622 - 1632
The hypothalamic melanocortin system, which controls appetite and energy expenditure, develops during the third trimester in primates. Thus, maternal nutrition... 
CORTICOTROPIN-RELEASING HORMONE | ALPHA-MSH | AGOUTI-RELATED PROTEIN | HYPOTHALAMIC NEUROPEPTIDE-Y | MESSENGER-RNA EXPRESSION | FEEDING-BEHAVIOR | ENDOCRINOLOGY & METABOLISM | ADRENAL HPA AXIS | ARCUATE NUCLEUS | DIABETIC MOTHER RATS | INDUCED OBESITY | Immunohistochemistry | Dietary Fats - metabolism | Microglia - metabolism | Melanocortins - metabolism | RNA, Messenger - genetics | Fetus - metabolism | Neuropeptide Y - genetics | RNA, Messenger - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Prenatal Nutritional Physiological Phenomena - physiology | Neuropeptide Y - metabolism | Pregnancy | Microscopy, Confocal | Receptors, Interleukin-1 Type I - metabolism | Inflammation - metabolism | Animals | Hypothalamus - metabolism | In Situ Hybridization | Animal Nutritional Physiological Phenomena | Interleukin-1beta - metabolism | Melanocortins - genetics | Female | Macaca | Adrenocorticotropic Hormone - metabolism
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3. Full Text Central Role of p53 in the Suntan Response and Pathologic Hyperpigmentation
by Cui, Rutao and Widlund, Hans R and Feige, Erez and Lin, Jennifer Y and Wilensky, Dara L and Igras, Viven E and D'Orazio, John and Fung, Claire Y and Schanbacher, Carl F and Granter, Scott R and Fisher, David E
Cell, ISSN 0092-8674, 2007, Volume 128, Issue 5, pp. 853 - 864
UV-induced pigmentation (suntanning) requires induction of α-melanocyte-stimulating hormone (α-MSH) secretion by keratinocytes. α-MSH and other bioactive... 
MICROPHTHALMIA TRANSCRIPTION FACTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | SQUAMOUS-CELL CARCINOMA | PROOPIOMELANOCORTIN GENE-EXPRESSION | MELANOCORTIN-1 RECEPTOR | ULTRAVIOLET-B | PLASMA BETA-ENDORPHIN | SV40-TRANSFORMED CELLS | HUMAN MELANOCYTES | SKIN-CANCER | CELL BIOLOGY | Up-Regulation | Carcinoma, Basal Cell - genetics | Keratinocytes - radiation effects | Skin - metabolism | Humans | Transcriptional Activation | Male | Melanocytes - metabolism | RNA, Messenger - metabolism | alpha-MSH - metabolism | Melanocytes - radiation effects | beta-Endorphin - metabolism | Genes, p53 | Cell Culture Techniques | Pro-Opiomelanocortin - genetics | Foreskin - metabolism | Promoter Regions, Genetic | Mice, Inbred C57BL | RNA, Messenger - genetics | Tumor Suppressor Protein p53 - metabolism | Foreskin - pathology | Keratinocytes - cytology | Carcinoma, Basal Cell - metabolism | Ultraviolet Rays - adverse effects | Mice, Knockout | Skin Neoplasms - metabolism | Animals | Hyperpigmentation - metabolism | Keratinocytes - metabolism | Skin - radiation effects | Skin Neoplasms - genetics | Skin Pigmentation | Cell Line, Tumor | Mice | Apoptosis
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4. Full Text Lateral hypothalamic thyrotropin-releasing hormone neurons: Distribution and relationship to histochemically defined cell populations in the rat
by Horjales-Araujo, E and Hellysaz, A and Broberger, C
Neuroscience, ISSN 0306-4522, 2014, Volume 277, pp. 87 - 102
Highlights • Mapping of TRH neurons in the rat lateral hypothalamic area (LHA). • TRH neurons are distinct from other major peptide populations in the LHA. •... 
Neurology | arcuate nucleus | coexistence | neuropeptide | immunofluorescence | in situ hybridization | arousal | Coexistence | Immunofluorescence | In situ hybridization | Neuropeptide | Arcuate nucleus | Arousal | IMMUNOREACTIVE PV1 NUCLEUS | IMMUNOHISTOCHEMICAL LOCALIZATION | IN-SITU HYBRIDIZATION | CART NEURONS | NEUROSCIENCES | NEUROPEPTIDE-Y | IMMUNOCYTOCHEMICAL LOCALIZATION | MESSENGER-RNA | CENTRAL-NERVOUS-SYSTEM | MELANIN-CONCENTRATING HORMONE | CANINE NARCOLEPSY | Hypothalamic Area, Lateral - metabolism | Orexins | Enkephalins - metabolism | Hypothalamic Area, Lateral - cytology | Male | Neurons - cytology | Neuropeptides - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | RNA, Messenger - metabolism | Melanins - metabolism | Photomicrography | Rats, Sprague-Dawley | alpha-MSH - metabolism | Corticotropin-Releasing Hormone - metabolism | Neurotensin - metabolism | Urocortins - metabolism | Thyrotropin-Releasing Hormone - metabolism | Animals | In Situ Hybridization | Hypothalamic Hormones - metabolism | Fluorescent Antibody Technique | Pituitary Hormones - metabolism | Neurons - metabolism | Neurohormones | Brain | Thyrotropin | Neurons
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5. Full Text Identification of nesfatin-1 as a satiety molecule in the hypothalamus
by Tsuchiya, Takafumi and Oh-I, Shinsuke and Monden, Tsuyoshi and Satoh, Tetsurou and Okada, Shuichi and Eguchi, Hiroshi and Imaki, Toshihiro and Hashimoto, Koushi and Mori, Masatomo and Yamada, Masanobu and Yamamoto, Masanori and Adachi, Sachika and Inoue, Kinji and Horiguchi, Kazuhiko and Shimizu, Hiroyuki
Nature, ISSN 0028-0836, 10/2006, Volume 443, Issue 7112, pp. 709 - 712
The brain hypothalamus contains certain secreted molecules that are important in regulating feeding behaviour. Here we show that nesfatin, corresponding to... 
SYSTEM | OBESITY | NUCLEOBINDIN | PATHWAY | CLONING | FOOD-INTAKE | NEFA | MULTIDISCIPLINARY SCIENCES | LEPTIN RECEPTOR | BINDING PROTEIN | MELANOCORTIN-4 RECEPTOR | Rats, Wistar | Leptin - metabolism | Injections, Intraventricular | Body Weight - drug effects | Male | Gene Expression Profiling | alpha-MSH - metabolism | DNA-Binding Proteins - metabolism | Dose-Response Relationship, Drug | Satiety Response - physiology | Receptors, Melanocortin - metabolism | Nerve Tissue Proteins - pharmacology | DNA-Binding Proteins - administration & dosage | Leptin - pharmacology | Nerve Tissue Proteins - administration & dosage | Calcium-Binding Proteins - chemistry | Calcium-Binding Proteins - metabolism | Receptors, Leptin | Satiety Response - drug effects | DNA-Binding Proteins - pharmacology | Nerve Tissue Proteins - cerebrospinal fluid | Appetite Regulation - drug effects | Rats | Receptors, Cell Surface - metabolism | Anorexia - metabolism | DNA-Binding Proteins - chemistry | Feeding Behavior - physiology | Calcium-Binding Proteins - administration & dosage | Feeding Behavior - drug effects | Obesity - metabolism | Nerve Tissue Proteins - metabolism | Rats, Zucker | Animals | Hypothalamus - metabolism | Signal Transduction - drug effects | Anorexia - prevention & control | Anorexia - chemically induced | Mice | Appetite Regulation - physiology | Calcium-Binding Proteins - pharmacology | Receptors, Cell Surface - genetics | Proteins | Brain | Signal transduction | Hormones | Gene expression | Rodents
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6. Full Text The central melanocortin system directly controls peripheral lipid metabolism
by Nogueiras, Ruben and Wiedmer, Petra and Perez-Tilve, Diego and Veyrat-Durebex, Christelle and Keogh, Julia M and Sutton, Gregory M and Pfluger, Paul T and Castaneda, Tamara R and Neschen, Susanne and Hofmann, Susanna M and Howles, Philip N and Morgan, Donald A and Benoit, Stephen C and Szanto, Ildiko and Schrott, Brigitte and Schürmann, Annette and Joost, Hans-Georg and Hammond, Craig and Hui, David Y and Woods, Stephen C and Rahmouni, Kamal and Butler, Andrew A and Farooqi, I. Sadaf and O'Rahilly, Stephen and Rohner-Jeanrenaud, Françoise and Tschöp, Matthias H
Journal of Clinical Investigation, ISSN 0021-9738, 11/2007, Volume 117, Issue 11, pp. 3475 - 3488
Disruptions of the melanocortin signaling system have been linked to obesity. We investigated a possible role of the central nervous melanocortin system... 
MEDICINE, RESEARCH & EXPERIMENTAL | WHITE ADIPOSE-TISSUE | FOOD-INTAKE | INSULIN-RESISTANCE | GENE-EXPRESSION | BRAIN-STEM | FRAMESHIFT MUTATION | LIPOPROTEIN-LIPASE | MELANOTAN-II | FAT MASS | NEUROPEPTIDE-Y | Central Nervous System - metabolism | Melanocortins - metabolism | Humans | Adipose Tissue - cytology | Adipocytes - cytology | Receptor, Melanocortin, Type 4 - metabolism | alpha-MSH - metabolism | Adipose Tissue - metabolism | Eating | Melanocyte-Stimulating Hormones - administration & dosage | Melanocyte-Stimulating Hormones - metabolism | Rats | Behavior, Animal - physiology | Lipid Metabolism | Receptor, Melanocortin, Type 4 - genetics | Rats, Sprague-Dawley | Receptors, Melanocortin | Mice, Knockout | Insulin - metabolism | Animals | Adipocytes - metabolism | Glucose - metabolism | Signal Transduction - physiology | Mice | alpha-MSH - analogs & derivatives | alpha-MSH - administration & dosage | Control | Genetic aspects | Pituitary hormones | Lipid metabolism | Research | Health aspects
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7. Full Text Obesity induces hypothalamic endoplasmic reticulum stress and impairs proopiomelanocortin (POMC) post-translational processing
by Çakir, Isin and Cyr, Nicole E and Perello, Mario and Litvinov, Bogdan Patedakis and Romero, Amparo and Stuart, Ronald C and Nillni, Eduardo A
Journal of Biological Chemistry, ISSN 0021-9258, 06/2013, Volume 288, Issue 24, pp. 17675 - 17688
It was shown previously that abnormal prohormone processing or inactive proconverting enzymes that are responsible for this processing cause profound obesity.... 
PRO-NEUROPEPTIDE-Y | GLUCOSE-HOMEOSTASIS | MELANOCYTE-STIMULATING HORMONE | PARAVENTRICULAR NUCLEUS | MESSENGER-RNA | PROHORMONE CONVERTASES | ER STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTHYROTROPIN-RELEASING HORMONE | DIET-INDUCED OBESITY | LEPTIN RESISTANCE | eIF-2 Kinase - metabolism | Diet, High-Fat - adverse effects | Endoplasmic Reticulum - metabolism | Male | alpha-MSH - metabolism | Leptin - physiology | Obesity - etiology | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - genetics | Pro-Opiomelanocortin - genetics | Arcuate Nucleus of Hypothalamus - pathology | Adrenocorticotropic Hormone - metabolism | eIF-2 Kinase - genetics | Cell Line | Proprotein Convertase 2 - metabolism | Arcuate Nucleus of Hypothalamus - metabolism | Gene Expression Regulation | Rats | Suppressor of Cytokine Signaling Proteins - genetics | Rats, Sprague-Dawley | Obesity - metabolism | Obesity - pathology | Pro-Opiomelanocortin - metabolism | Suppressor of Cytokine Signaling 3 Protein | Protein Tyrosine Phosphatase, Non-Receptor Type 1 - metabolism | Animals | Endoplasmic Reticulum Stress | Mice | Protein Processing, Post-Translational | Suppressor of Cytokine Signaling Proteins - metabolism | Hypothalamus | Energy Metabolism | Metabolism | Protein Processing | Neuropeptide
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8. Full Text Central mechanisms of adiposity in adult female mice with androgen excess
by Nohara, Kazunari and Laque, Amanda and Allard, Camille and Münzberg, Heike and Mauvais‐Jarvis, Franck
Obesity, ISSN 1930-7381, 06/2014, Volume 22, Issue 6, pp. 1477 - 1484
Objective Androgen excess in women is associated with visceral adiposity. However, little is known on the mechanism through which androgen promotes visceral... 
NUTRITION & DIETETICS | ENERGY-EXPENDITURE | MESSENGER-RNA | INSULIN-RESISTANCE | FOOD-INTAKE | TISSUE | ENDOCRINOLOGY & METABOLISM | NEURONS | DORSOMEDIAL HYPOTHALAMUS | PREOPTIC AREA | LEPTIN RESISTANCE | SYMPATHETIC TONE | Body Composition | Up-Regulation | Body Weight | Dihydrotestosterone - administration & dosage | Ion Channels - genetics | Receptor, Melanocortin, Type 4 - metabolism | Mitochondrial Proteins - genetics | RNA, Messenger - metabolism | alpha-MSH - metabolism | Peptides, Cyclic - metabolism | Leptin - blood | Mitochondrial Proteins - metabolism | Arcuate Nucleus of Hypothalamus - drug effects | Androgens - blood | Dihydrotestosterone - blood | Female | Intra-Abdominal Fat - drug effects | Androgens - adverse effects | Hypothalamus - drug effects | Pro-Opiomelanocortin - genetics | Arcuate Nucleus of Hypothalamus - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Receptor, Melanocortin, Type 4 - genetics | Intra-Abdominal Fat - metabolism | Androgens - administration & dosage | Obesity - metabolism | Hyperphagia - pathology | Pro-Opiomelanocortin - metabolism | Animals | Hypothalamus - metabolism | Energy Metabolism | Ion Channels - metabolism | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Adiposity - physiology | Mice | Uncoupling Protein 1 | alpha-MSH - analogs & derivatives | Obesity | Androgens | Weight control | Laboratories | Rodents | adiposity | leptin resistance | Androgen | energy expenditure | dorsomedial hypothalamus | melanocortin
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9. Full Text Diet-Induced Obesity Causes Severe but Reversible Leptin Resistance in Arcuate Melanocortin Neurons
by Enriori, Pablo J and Evans, Anne E and Sinnayah, Puspha and Jobst, Erin E and Tonelli-Lemos, Luciana and Billes, Sonja K and Glavas, Maria M and Grayson, Bernadette E and Perello, Mario and Nillni, Eduardo A and Grove, Kevin L and Cowley, Michael A
Cell Metabolism, ISSN 1550-4131, 2007, Volume 5, Issue 3, pp. 181 - 194
Despite high leptin levels, most obese humans and rodents lack responsiveness to its appetite-suppressing effects. We demonstrate that leptin modulates... 
HUMDISEASE | MOLNEURO | ENERGY-BALANCE | POMC NEURONS | AGOUTI-RELATED PROTEIN | GLUCOSE-HOMEOSTASIS | DB/DB MICE | MESSENGER-RNA EXPRESSION | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | RECEPTOR GENE | FAT DIET | HYPOTHALAMIC LEPTIN | CELL BIOLOGY | Body Composition | Melanocortins - metabolism | Arcuate Nucleus of Hypothalamus - cytology | Male | alpha-MSH - metabolism | Dose-Response Relationship, Drug | Neuropeptide Y - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Dietary Fats - administration & dosage | Leptin - administration & dosage | Leptin - pharmacology | Neurons - metabolism | Signal Transduction | Arcuate Nucleus of Hypothalamus - metabolism | Mice, Inbred C57BL | Obesity - metabolism | Gene Expression Regulation - drug effects | Pro-Opiomelanocortin - metabolism | Animals | Hypothalamus - metabolism | Diet | Weight Loss | Mice | In Vitro Techniques | RNA, Messenger | Agouti-Related Protein | Physiological aspects | Obesity | Neurons | Leptin
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10. Full Text Cellular Warriors at the Battle of the Bulge
by Jean Marx
Science, ISSN 0036-8075, 2/2003, Volume 299, Issue 5608, pp. 846 - 849
The tools of molecular genetics have yielded a rush of new information about molecular signals that regulate body weight. Some guide short-term decisions, such... 
Appetite | Obesity | Enzymes | Receptors | Neurons | Arcuate nucleus | News | Hormones | Lipid metabolism | Diabetes | Insulin | MULTIDISCIPLINARY SCIENCES | Body Weight | Peptide YY - metabolism | Caloric Restriction | Humans | Leptin - metabolism | Protein Tyrosine Phosphatases - metabolism | alpha-MSH - metabolism | Brain - metabolism | Neuropeptide Y - metabolism | Peptide Hormones - metabolism | Protein Tyrosine Phosphatases - genetics | Ghrelin | Neurons - physiology | Obesity - etiology | Fatty Acids - metabolism | Arcuate Nucleus of Hypothalamus - metabolism | Intercellular Signaling Peptides and Proteins | Leptin - genetics | Longevity | Stearoyl-CoA Desaturase - genetics | Obesity - metabolism | Pro-Opiomelanocortin - metabolism | Insulin - metabolism | Animals | Proteins - metabolism | Anti-Obesity Agents | Protein Tyrosine Phosphatase, Non-Receptor Type 1 | Receptors, Pituitary Hormone - metabolism | Stearoyl-CoA Desaturase - metabolism | Receptor, Insulin - metabolism | Mice | Agouti-Related Protein | Research | Peptide hormones | Reducing diets | Cellular biology
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11. Full Text Immunohistological pointers to a possible role for excessive cathelicidin (LL‐37) expression by apocrine sweat glands in the pathogenesis of hidradenitis suppurativa/acne inversa
by Emelianov, V.U and Bechara, F.G and Gläser, R and Langan, E.A and Taungjaruwinai, W.M and Schröder, J.M and Meyer, K.C and Paus, R
British Journal of Dermatology, ISSN 0007-0963, 05/2012, Volume 166, Issue 5, pp. 1023 - 1034
Summary Background  The cause of follicular occlusion, a key early event in the pathogenesis of hidradenitis suppurativa (HS), also known as acne inversa,... 
MELANOCYTE-STIMULATING HORMONE | MIGRATION INHIBITORY FACTOR | MULTIFUNCTIONAL MODULATOR | MICROBICIDAL PROTEIN PSORIASIN | ANTIMICROBIAL PEPTIDES | IN-SITU | HUMAN KERATINOCYTES | ANTIBACTERIAL PEPTIDE LL-37 | INFLAMMATORY MEDIATORS | SKIN INFLAMMATION | DERMATOLOGY | Immunohistochemistry | S100 Calcium Binding Protein A7 | Tumor Necrosis Factor-alpha - metabolism | Epidermis - metabolism | Up-Regulation | Hidradenitis Suppurativa - etiology | Dermis - metabolism | Hidradenitis Suppurativa - metabolism | Humans | Middle Aged | Male | S100 Proteins - metabolism | Muramidase - metabolism | Antimicrobial Cationic Peptides - metabolism | alpha-MSH - metabolism | Case-Control Studies | Young Adult | Sweat Glands - metabolism | Chemotactic Factors - metabolism | Adult | Female | beta-Defensins - metabolism | Interleukin-8 - metabolism | Occlusion | Sweat gland | Leukocyte migration | Pathogenesis | Secretion | Acne | Menopause | Dermis | Epidermis | Inflammation | Epithelium | Macrophage migration inhibitory factor | Tumor necrosis factor | Migration inhibitory factor | Antimicrobial peptides | Tumor necrosis factor-TNF | Skin diseases | Immunoreactivity | Sweat | Lysozyme | Tumors
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12. Full Text Hypothalamic POMC neurons promote cannabinoid-induced feeding
by Koch, Marco and Varela, Luis and Kim, Jae Geun and Kim, Jung Dae and Hernández-Nuño, Francisco and Simonds, Stephanie E and Castorena, Carlos M and Vianna, Claudia R and Elmquist, Joel K and Morozov, Yury M and Rakic, Pasko and Bechmann, Ingo and Cowley, Michael A and Szigeti-Buck, Klara and Dietrich, Marcelo O and Gao, Xiao-Bing and Diano, Sabrina and Horvath, Tamas L
Nature, ISSN 0028-0836, 03/2015, Volume 519, Issue 7541, pp. 45 - 50
Hypothalamic pro-opiomelanocortin (POMC) neurons promote satiety. Cannabinoid receptor 1 (CB1R) is critical for the central regulation of food intake. Here we... 
MITOCHONDRIAL DYNAMICS | FOOD-INTAKE | MULTIDISCIPLINARY SCIENCES | PROOPIOMELANOCORTIN NEURONS | ENDOCANNABINOID SYSTEM | UNCOUPLING PROTEIN-2 | DIET-INDUCED OBESITY | ARCUATE NUCLEUS | CB1 RECEPTORS | MELANOCORTIN SYSTEM | NEUROPEPTIDE-Y | Male | Eating - physiology | Satiety Response - physiology | beta-Endorphin - metabolism | Receptor, Cannabinoid, CB1 - agonists | Mitochondrial Proteins - metabolism | Neurons - metabolism | Hypothalamus - drug effects | Neurons - drug effects | Satiety Response - drug effects | Hypothalamus - physiology | Mice, Inbred C57BL | Mitochondria - metabolism | Mitochondria - drug effects | Cannabinoids - pharmacology | Pro-Opiomelanocortin - metabolism | Eating - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Animals | Hypothalamus - cytology | Ion Channels - metabolism | Naloxone - pharmacology | alpha-MSH - secretion | Mice | Uncoupling Protein 2 | Energy Metabolism - drug effects | Animal feeding behavior | Cannabinoids | Usage | Analysis | Rodents | Research | Pro-opiomelanocortin | Health aspects | Proteins | Marijuana | Hunger | Peptides | Neurons
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