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Annals of Biomedical Engineering, ISSN 0090-6964, 2011, Volume 39, Issue 9, pp. 2430 - 2444
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology: Journal of the American Heart Association, ISSN 1079-5642, 06/2004, Volume 24, Issue 6, pp. 1006 - 1014
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 04/2012, Volume 94, Issue 1, pp. 20 - 29
Vascular disease states are associated with endothelial dysfunction and increased production of reactive oxygen species derived from NADPH oxidases. However,... 
VCAM-1 | Macrophages | NADPH oxidase | Nox2 | Atherosclerosis | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | NITRIC-OXIDE SYNTHASE | ANGIOTENSIN-II | APOLIPOPROTEIN-E | CELL-ADHESION MOLECULE-1 | NAD(P)H OXIDASE | APOE(-/-) MICE | HUMAN ATHEROSCLEROSIS | TRANSGENIC MICE | Blood Pressure | Up-Regulation | Apolipoproteins E - deficiency | Membrane Glycoproteins - metabolism | Atherosclerosis - genetics | Humans | NADPH Oxidases - metabolism | Aortic Diseases - chemically induced | Atherosclerosis - enzymology | Time Factors | Superoxides - metabolism | NADPH Oxidases - genetics | Aortic Diseases - enzymology | Angiotensin II | Aortic Diseases - pathology | Disease Models, Animal | Atherosclerosis - pathology | Signal Transduction | Atherosclerosis - physiopathology | Atherosclerosis - chemically induced | Mice, Transgenic | Chemotaxis | NADPH Oxidase 2 | Disease Progression | Membrane Glycoproteins - genetics | Mice, Knockout | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Macrophages - metabolism | Animals | Apolipoproteins E - genetics | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - enzymology | Reactive oxygen species | Chemiluminescence | Superoxide | Endothelial cells | NAD(P)H oxidase | Endothelium | Cell activation | Vascular diseases | Arteriosclerosis | Angiotensin | CYBB protein | Cell adhesion | Aorta | Blood pressure | Original
Journal Article
Gene Regulation and Systems Biology, ISSN 1177-6250, 1/2016, Volume 10, pp. 95 - 103
The cellular and molecular mechanisms behind the process of atherosclerotic plaque destabilization are complex, and molecular data from aortic plaques are... 
ApoE | Network model | mice | Plaque destabilization | Network perturbation amplitude | Atherosclerosis
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2007, Volume 27, Issue 8, pp. 1706 - 1721
Journal Article
Journal Article
Journal Article
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 2017, Volume 57, Issue 4, pp. 1171 - 1183
Journal Article
FEBS Letters, ISSN 0014-5793, 12/2015, Volume 589, Issue 24, pp. 3998 - 4009
Homocysteine (Hcy) is an independent risk factor for atherosclerosis, but the underlying molecular mechanisms are not known. We investigated the effects of Hcy... 
DNA methylation | Foam cell | Homocysteine | Atherosclerosis | Fatty acid-binding protein 4 | atherosclerosis | homocysteine | free Cholesterol | DNMT1 | S-adenosylhomocysteine | AZC | azacytidine | S-adenosylmethionine | FABP4 | fatty acid-binding protein 4 | DNA methyltransferases 1 | HHcy | Hyperhomocysteinemia | ApoE | SAH | total Cholesterol | apolipoprotein E knockout | Cholesteryl Ester | SAM | Hcy | HYPERHOMOCYSTEINEMIA | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | BINDING PROTEIN 4 | ADIPOGENESIS | CELL BIOLOGY | BIOPHYSICS | DISEASE | PPAR-GAMMA | DIFFERENTIATION | EXPRESSION | Diet - adverse effects | Cholesterol - blood | Humans | Hyperhomocysteinemia - metabolism | Male | Monocytes - metabolism | Aorta - metabolism | Fatty Acid-Binding Proteins - metabolism | Apolipoproteins E - metabolism | Atherosclerosis - etiology | DNA Methylation | Hyperhomocysteinemia - physiopathology | Monocytes - pathology | Foam Cells - pathology | Aorta - enzymology | Foam Cells - metabolism | Genes, Reporter | Repressor Proteins - metabolism | Methionine - poisoning | Promoter Regions, Genetic | Hyperhomocysteinemia - pathology | Mice, Inbred C57BL | Gene Expression Regulation | Repressor Proteins - genetics | Foam Cells - enzymology | Cholesterol - metabolism | Hyperhomocysteinemia - etiology | Fatty Acid-Binding Proteins - genetics<