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1. Full Text MIR-146a modulates autoreactive Th17 cell differentiation and regulates organ-specific autoimmunity
by Li, Bo and Wang, Xi and Choi, In Young and Wang, Yu-Chen and Liu, Siyuan and Pham, Alexander T and Moon, Heesung and Smith, Drake J and Rao, Dinesh S and Boldin, Mark P and Yang, Lili
The Journal of clinical investigation, ISSN 0021-9738, 10/2017, Volume 127, Issue 10, pp. 3702 - 3716
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Autoimmunity | Myelin-Oligodendrocyte Glycoprotein - immunology | Myelin-Oligodendrocyte Glycoprotein - genetics | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Organ Specificity - immunology | Cell Differentiation - genetics | Interleukins - genetics | Organ Specificity - genetics | Autocrine Communication - immunology | Interleukins - immunology | Encephalomyelitis, Autoimmune, Experimental - genetics | Th17 Cells - pathology | Interleukin-6 - genetics | Multiple Sclerosis - genetics | MicroRNAs - immunology | Autocrine Communication - genetics | Mice, Knockout | Cell Differentiation - immunology | Animals | Interleukin-6 - immunology | Multiple Sclerosis - immunology | Th17 Cells - immunology | Mice | MicroRNAs - genetics | Research | Cell differentiation | T cells | Analysis | Multiple sclerosis | Oligodendrocyte-myelin glycoprotein | Disease | Cytokines | Myelin | Helper cells | Lymphocytes T | Inflammation | T cell receptors | Kinases | Gene expression | Experimental allergic encephalomyelitis | CD4 antigen | Interleukin 6 | T-cell receptor | Lymphocytes | Rheumatoid arthritis | MicroRNAs | Rodents | Stem cells | Interleukin 21 | Autocrine signalling | Autoimmune diseases | Index Medicus | Abridged Index Medicus
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2. Full Text Autocrine interleukin-10 mediates glucagon-like peptide-1 receptor-induced spinal microglial β-endorphin expression
by Wu, Hai-Yun and Tang, Xue-Qi and Mao, Xiao-Fang and Wang, Yong-Xiang
The Journal of neuroscience, ISSN 0270-6474, 11/2017, Volume 37, Issue 48, pp. 11701 - 11714
Spinal cord | β-endorphin | Autocrine | GLP-1 receptor | IL-10 | Microglia | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Microglia - metabolism | Spinal Cord - drug effects | Spinal Cord - metabolism | Rats, Wistar | Male | Autocrine Communication - drug effects | Exenatide | Dose-Response Relationship, Drug | Glucagon-Like Peptide-1 Receptor - genetics | Glucagon-Like Peptide-1 Receptor - biosynthesis | Spinal Cord - cytology | Animals, Newborn | Gene Expression | Microglia - drug effects | Autocrine Communication - physiology | Cells, Cultured | Rats | beta-Endorphin - biosynthesis | Peptides - pharmacology | Animals | Interleukin-10 - genetics | Interleukin-10 - biosynthesis | Venoms - pharmacology | Interleukin-10 - pharmacology | Glucagon-Like Peptide-1 Receptor - agonists | beta-Endorphin - genetics | Protein kinase A | Neuroprotection | Phosphorylation | Glucagon | Interleukin | Activation | Neuropathy | Kinases | Interleukin 6 | Signal transduction | Receptors | Interleukin 4 | Pathways | Rodents | Inhibition | Autocrine signalling | Glucagon-like peptide 1 | Pain perception | Cytokines | Markers | Stat3 protein | Inflammation | siRNA | Injection | Tumor necrosis factor-α | Cyclic AMP response element-binding protein | Gene expression | Interleukin 10 | Endorphins | Receptor mechanisms | Index Medicus | spinal cord | microglia | autocrine
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3. Full Text Melanoma cell–derived exosomes promote epithelial–mesenchymal transition in primary melanocytes through paracrine/autocrine signaling in the tumor microenvironment
by Xiao, Deyi and Barry, Samantha and Kmetz, Daniel and Egger, Michael and Pan, Jianmin and Rai, Shesh N and Qu, Jifu and McMasters, Kelly M and Hao, Hongying
Cancer letters, ISSN 0304-3835, 2016, Volume 376, Issue 2, pp. 318 - 327
Hematology, Oncology and Palliative Medicine | Microenvironment | Exosomes | Epithelial-to-mesenchymal transition | Paracrine/autocrine | Melanoma | Tumor progression | Life Sciences & Biomedicine | Oncology | Science & Technology | Exosomes - metabolism | Humans | Middle Aged | Tumor Microenvironment | Male | Melanocytes - metabolism | MicroRNAs - metabolism | Case-Control Studies | Transfection | Melanoma - genetics | Melanocytes - pathology | Epithelial-Mesenchymal Transition | Adult | Female | Melanoma - metabolism | Skin Neoplasms - pathology | Autocrine Communication | Paracrine Communication | Signal Transduction | Melanoma - secondary | Skin Neoplasms - metabolism | Skin Neoplasms - genetics | Cell Line, Tumor | Aged | MicroRNAs - genetics | Mitogen-Activated Protein Kinases - metabolism | Development and progression | Stem cells | Tumors | Medical colleges | Metastasis | Mitogens | Protein kinases | Cancer | Index Medicus | Paracrine | autocrine
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4. Full Text How Pulsatile Kisspeptin Stimulation and GnRH Autocrine Feedback Can Drive GnRH Secretion: A Modeling Investigation
by Lehnert, Jonas and Khadra, Anmar
Endocrinology (Philadelphia), ISSN 0013-7227, 05/2019, Volume 160, Issue 5, pp. 1289 - 1306
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Models, Theoretical | Autocrine Communication - physiology | Humans | Kisspeptins - administration & dosage | Autocrine Communication - drug effects | Luteinizing Hormone - metabolism | Algorithms | Animals | Time Factors | Gonadotropin-Releasing Hormone - metabolism | Neurons - physiology | Feedback, Physiological - drug effects | Neurons - metabolism | Feedback, Physiological - physiology | Neurons - drug effects | Electrophysiological Phenomena - drug effects | Physiological aspects | Control | Hormones, Sex | Gonadotropin releasing hormone | Autocrine mechanisms | Electric properties | Electrical properties | Neurons | Secretion | Firing rate | Stimulation | Hypothalamus | Hormones | Depolarization | Feedback | Mathematical models | Gonadotropin-releasing hormone | Kiss1 protein | Bursting | Autocrine signalling | Hyperpolarization | Locking | Index Medicus | Abridged Index Medicus
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5. Full Text Purinergic P2X4 receptors and mitochondrial ATP production regulate T cell migration
by Ledderose, Carola and Liu, Kaifeng and Kondo, Yutaka and Slubowski, Christian J and Dertnig, Thomas and Denicoló, Sara and Arbab, Mona and Hubner, Johannes and Konrad, Kirstin and Fakhari, Mahtab and Lederer, James A and Robson, Simon C and Visner, Gary A and Junger, Wolfgang G
The Journal of clinical investigation, ISSN 0021-9738, 08/2018, Volume 128, Issue 8, pp. 3583 - 3594
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Jurkat Cells | CD4-Positive T-Lymphocytes - cytology | Humans | Receptors, Purinergic P2X4 - immunology | Adenosine Triphosphate - genetics | Adenosine Triphosphate - immunology | Inflammation - immunology | Mitochondria - immunology | Autocrine Communication - genetics | CD4-Positive T-Lymphocytes - immunology | Cell Movement - immunology | Animals | Autocrine Communication - immunology | Mitochondria - genetics | Inflammation - genetics | Mice | Mice, Inbred BALB C | Receptors, Purinergic P2X4 - genetics | Mitochondria | Usage | Analysis | Transplantation of organs, tissues, etc | Research | T cells | Adenosine triphosphate | Cell migration | Chemokines | Transplants & implants | Leukocyte migration | Syngeneic grafts | Purine receptors | Lung transplantation | Lymphocytes T | Calcium influx | Inflammatory diseases | Cell adhesion & migration | Cell activation | Lymphocytes | Xenografts | Autocrine signalling | Immune system | Antigens | Lymphatic system | Immunomodulation | Therapeutic applications | Neutrophils | Inflammation | T cell receptors | SDF-1 protein | Calcium (mitochondrial) | CD4 antigen | Graft rejection | Lungs | Antigen-presenting cells | Index Medicus | Abridged Index Medicus
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6. Full Text Increased autocrine interleukin‐6 production is significantly associated with worse clinical outcome in patients with chronic lymphocytic leukemia
by Wang, Hua‐Qing and Jia, Li and Li, Yu‐Ting and Farren, Timothy and Agrawal, Samir G and Liu, Feng‐Ting
Journal of cellular physiology, ISSN 0021-9541, 08/2019, Volume 234, Issue 8, pp. 13994 - 14006
autocrine IL‐6 | chronic lymphocytic leukemia | tumor microenvironment | clinical outcome | autocrine IL-6 | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Autocrine Communication | Multivariate Analysis | Signal Transduction | Humans | NF-kappa B - metabolism | Treatment Outcome | Mitochondria - metabolism | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | Disease-Free Survival | Leukemia, Lymphocytic, Chronic, B-Cell - metabolism | Interleukin-6 - biosynthesis | Tumor Cells, Cultured | STAT3 Transcription Factor - metabolism | Apoptosis | Chronic lymphocytic leukemia | Care and treatment | Chemotherapy | Interleukins | Patient outcomes | B cells | Cancer | Chronic lymphatic leukemia | Cell survival | Cytokines | Leukemia | Interleukin | Agent orange | Stat3 protein | Lymphatic leukemia | Survival | Patients | Clinical outcomes | Clonal deletion | Lymphocytes B | Medical prognosis | Janus kinase 2 | Cascades | Remission | Autocrine signalling | Index Medicus | Original | Original s
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7. Full Text Tumor angiogenesis is enforced by autocrine regulation of high-mobility group box 1
by van Beijnum, J. R and Nowak-Sliwinska, P and van den Boezem, E and Hautvast, P and Buurman, W. A and Griffioen, A. W
Oncogene, ISSN 0950-9232, 01/2013, Volume 32, Issue 3, pp. 363 - 374
chicken embryo chorioallantoic membrane | high-mobility group box 1 | VEGF | autocrine | tumor angiogenesis | Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Autocrine Communication | Colorectal Neoplasms - blood supply | Humans | Gene Expression Regulation, Neoplastic | Neovascularization, Pathologic | Chorioallantoic Membrane - metabolism | Chick Embryo | Receptors, Immunologic - antagonists & inhibitors | Phenotype | Animals | Endothelial Cells - cytology | HMGB1 Protein - metabolism | Colorectal Neoplasms - pathology | Receptor for Advanced Glycation End Products | Colorectal Neoplasms - metabolism | Cell Movement | Care and treatment | Chromosomal proteins | Physiological aspects | Genetic aspects | Neovascularization | Research | Health aspects | Endothelium | Cancer | Angiogenesis | Cancer therapies | Tumors | Index Medicus | Platelet-derived growth factor | Migration | photodynamic therapy | Antibodies | Paracrine signalling | Matrix metalloproteinase | Drug resistance | angiogenesis inhibitors | HMGB1 protein | Feedback | Toll-like receptors | Autocrine signalling | Vascular endothelial growth factor | Advanced glycosylation end products | Cytokines | Data processing | Inflammation | Growth factor receptors | TLR4 protein | Gene expression | Embryos | Endothelial cells | Chorioallantoic membrane | Solid tumors | Integrins
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8. Full Text Complement activation, regulation, and molecular basis for complement‐related diseases
by Bajic, Goran and Degn, Søren E and Thiel, Steffen and Andersen, Gregers R
The EMBO journal, ISSN 0261-4189, 11/2015, Volume 34, Issue 22, pp. 2735 - 2757
proteolytic regulation | structural biology | innate immunity | complement | inflammation | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Complement Activation - genetics | Inflammation - pathology | Animals | Autocrine Communication - immunology | Proteolysis | Humans | Inflammation - genetics | Inflammation - immunology | Complement Activation - immunology | Autocrine Communication - genetics | Pathogens | Genetics | Immunology | Molecular biology | Index Medicus | Review
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