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Publication DateNature, ISSN 0028-0836, 01/2015, Volume 518, Issue 7537, pp. 111 - 114
Proper positioning of organelles by cytoskeleton-based motor proteins underlies cellular events such as signalling, polarization and growth. For many...
MITOCHONDRIAL TRANSPORT | DENDRITIC SPINES | AMPA RECEPTORS | RECYCLING ENDOSOMES | MULTIDISCIPLINARY SCIENCES | GROWTH | NEURONS | TRAFFICKING | NEURODEGENERATION | AXONAL MITOCHONDRIA | LIVING CELLS | Optogenetics - methods | Endosomes - radiation effects | Axons - physiology | Dendritic Spines - metabolism | Cell Compartmentation - radiation effects | Endosomes - metabolism | Microtubules - metabolism | Biological Transport - radiation effects | Microtubules - radiation effects | Mitochondria - radiation effects | Peroxisomes - radiation effects | Intracellular Space - radiation effects | Dyneins - metabolism | Myosin Type V - metabolism | Cells, Cultured | Rats | Cytoskeleton - radiation effects | Mitochondria - metabolism | Hippocampus - cytology | Peroxisomes - metabolism | Kinesin - metabolism | Cell Compartmentation - physiology | Animals | Intracellular Space - metabolism | Kinesin - radiation effects | Axons - radiation effects | Cytoskeleton - metabolism | Dendritic Spines - radiation effects | Myosin Type V - radiation effects | Dyneins - radiation effects | Physiological aspects | Genetic aspects | Cell organelles | Research | Molecular motors (Biochemistry) | Formation | Proteins | Cytoskeleton | Mitochondria | Motility | Cellular biology | Light
MITOCHONDRIAL TRANSPORT | DENDRITIC SPINES | AMPA RECEPTORS | RECYCLING ENDOSOMES | MULTIDISCIPLINARY SCIENCES | GROWTH | NEURONS | TRAFFICKING | NEURODEGENERATION | AXONAL MITOCHONDRIA | LIVING CELLS | Optogenetics - methods | Endosomes - radiation effects | Axons - physiology | Dendritic Spines - metabolism | Cell Compartmentation - radiation effects | Endosomes - metabolism | Microtubules - metabolism | Biological Transport - radiation effects | Microtubules - radiation effects | Mitochondria - radiation effects | Peroxisomes - radiation effects | Intracellular Space - radiation effects | Dyneins - metabolism | Myosin Type V - metabolism | Cells, Cultured | Rats | Cytoskeleton - radiation effects | Mitochondria - metabolism | Hippocampus - cytology | Peroxisomes - metabolism | Kinesin - metabolism | Cell Compartmentation - physiology | Animals | Intracellular Space - metabolism | Kinesin - radiation effects | Axons - radiation effects | Cytoskeleton - metabolism | Dendritic Spines - radiation effects | Myosin Type V - radiation effects | Dyneins - radiation effects | Physiological aspects | Genetic aspects | Cell organelles | Research | Molecular motors (Biochemistry) | Formation | Proteins | Cytoskeleton | Mitochondria | Motility | Cellular biology | Light
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Loading RightsCell Reports, ISSN 2211-1247, 03/2016, Volume 14, Issue 11, pp. 2637 - 2652
Motor-cargo recruitment to microtubules is often the rate-limiting step of intracellular transport, and defects in this recruitment can cause neurodegenerative...
AUTOINHIBITION | DYNACTIN COMPLEX | TRACKING | AXON | LIS1 | DETYROSINATION | MICROTUBULE PLUS-END | MOTOR COORDINATION | BINDING | MOTILITY | CELL BIOLOGY | Phosphorylation | Fluorescence Recovery After Photobleaching | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Cercopithecus aethiops | Fluorescent Dyes - metabolism | Neoplasm Proteins - antagonists & inhibitors | Neurons - cytology | Microscopy, Video | Neoplasm Proteins - metabolism | Tubulin - metabolism | RNA Interference | Neurons - metabolism | Neoplasm Proteins - genetics | Dyneins - metabolism | Fluorescent Dyes - chemistry | Cells, Cultured | Axons - metabolism | Rats | Microtubule-Associated Proteins - antagonists & inhibitors | Tyrosine - metabolism | Animals | Lysosomal-Associated Membrane Protein 1 - metabolism | HeLa Cells | COS Cells | RNA, Small Interfering - metabolism | CLIP1 | DCTN1 | transport initiation | cytoplasmic dynein | live-cell imaging | microtubule detyrosination | in vitro reconstitution | p150Glued | dynactin | super-resolution microscopy
AUTOINHIBITION | DYNACTIN COMPLEX | TRACKING | AXON | LIS1 | DETYROSINATION | MICROTUBULE PLUS-END | MOTOR COORDINATION | BINDING | MOTILITY | CELL BIOLOGY | Phosphorylation | Fluorescence Recovery After Photobleaching | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Cercopithecus aethiops | Fluorescent Dyes - metabolism | Neoplasm Proteins - antagonists & inhibitors | Neurons - cytology | Microscopy, Video | Neoplasm Proteins - metabolism | Tubulin - metabolism | RNA Interference | Neurons - metabolism | Neoplasm Proteins - genetics | Dyneins - metabolism | Fluorescent Dyes - chemistry | Cells, Cultured | Axons - metabolism | Rats | Microtubule-Associated Proteins - antagonists & inhibitors | Tyrosine - metabolism | Animals | Lysosomal-Associated Membrane Protein 1 - metabolism | HeLa Cells | COS Cells | RNA, Small Interfering - metabolism | CLIP1 | DCTN1 | transport initiation | cytoplasmic dynein | live-cell imaging | microtubule detyrosination | in vitro reconstitution | p150Glued | dynactin | super-resolution microscopy
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Loading RightsAnnual Review of Neuroscience, ISSN 0147-006X, 2008, Volume 31, Issue 1, pp. 151 - 173
Many major human neurodegenerative diseases, including Alzheimer's disease, Parkinson's disease, and amyotrophic lateral sclerosis (ALS), display axonal...
Amyotrophic lateral sclerosis | Parkinson's disease | Huntington's disease | Alzheimer's disease | Charcot-Marie-tooth disease | Motor neuron disease | motor neuron disease | MARIE-TOOTH-DISEASE | ACTIVATED PROTEIN-KINASE | HEREDITARY SPASTIC PARAPLEGIA | GLYCOGEN-SYNTHASE KINASE-3 | AMYOTROPHIC-LATERAL-SCLEROSIS | MOTOR-NEURON DISEASE | MUTANT SUPEROXIDE-DISMUTASE | amyotrophic lateral sclerosis | HUNTINGTIN-ASSOCIATED PROTEIN-1 | NEUROSCIENCES | Charcot-Marie-Tooth disease | NUCLEOTIDE EXCHANGE FACTOR | CYCLIN-DEPENDENT KINASE-5 | Axonal Transport - genetics | Central Nervous System - metabolism | Humans | Central Nervous System - pathology | Charcot-Marie-Tooth Disease - genetics | Motor Neuron Disease - metabolism | Motor Neuron Disease - genetics | Parkinson Disease - metabolism | Motor Neuron Disease - physiopathology | Charcot-Marie-Tooth Disease - metabolism | Huntington Disease - physiopathology | Alzheimer Disease - physiopathology | Axons - metabolism | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - metabolism | Parkinson Disease - genetics | Parkinson Disease - physiopathology | Huntington Disease - metabolism | Animals | Neurodegenerative Diseases - physiopathology | Axons - pathology | Alzheimer Disease - metabolism | Huntington Disease - genetics | Central Nervous System - physiopathology | Charcot-Marie-Tooth Disease - physiopathology | Alzheimer Disease - genetics | Nervous system | Degeneration | Axonal transport | Analysis
Amyotrophic lateral sclerosis | Parkinson's disease | Huntington's disease | Alzheimer's disease | Charcot-Marie-tooth disease | Motor neuron disease | motor neuron disease | MARIE-TOOTH-DISEASE | ACTIVATED PROTEIN-KINASE | HEREDITARY SPASTIC PARAPLEGIA | GLYCOGEN-SYNTHASE KINASE-3 | AMYOTROPHIC-LATERAL-SCLEROSIS | MOTOR-NEURON DISEASE | MUTANT SUPEROXIDE-DISMUTASE | amyotrophic lateral sclerosis | HUNTINGTIN-ASSOCIATED PROTEIN-1 | NEUROSCIENCES | Charcot-Marie-Tooth disease | NUCLEOTIDE EXCHANGE FACTOR | CYCLIN-DEPENDENT KINASE-5 | Axonal Transport - genetics | Central Nervous System - metabolism | Humans | Central Nervous System - pathology | Charcot-Marie-Tooth Disease - genetics | Motor Neuron Disease - metabolism | Motor Neuron Disease - genetics | Parkinson Disease - metabolism | Motor Neuron Disease - physiopathology | Charcot-Marie-Tooth Disease - metabolism | Huntington Disease - physiopathology | Alzheimer Disease - physiopathology | Axons - metabolism | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - metabolism | Parkinson Disease - genetics | Parkinson Disease - physiopathology | Huntington Disease - metabolism | Animals | Neurodegenerative Diseases - physiopathology | Axons - pathology | Alzheimer Disease - metabolism | Huntington Disease - genetics | Central Nervous System - physiopathology | Charcot-Marie-Tooth Disease - physiopathology | Alzheimer Disease - genetics | Nervous system | Degeneration | Axonal transport | Analysis
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Loading RightsScience, ISSN 0036-8075, 10/2010, Volume 330, Issue 6001, pp. 198 - 198
Amyloid-beta (A beta) peptides, derived from the amyloid precursor protein, and the microtubule-associated protein tau are key pathogenic factors in...
Axons | Mitochondria | Plasmids | Freight transport | Behavioral neuroscience | T tests | BREVIA | Freight | OVEREXPRESSION | MECHANISM | ALZHEIMERS-DISEASE | AMYLOID-BETA | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | TRAFFICKING | HIPPOCAMPAL-NEURONS
Axons | Mitochondria | Plasmids | Freight transport | Behavioral neuroscience | T tests | BREVIA | Freight | OVEREXPRESSION | MECHANISM | ALZHEIMERS-DISEASE | AMYLOID-BETA | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | TRAFFICKING | HIPPOCAMPAL-NEURONS
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Loading RightsAnnual Review of Biochemistry, ISSN 0066-4154, 2006, Volume 75, Issue 1, pp. 607 - 627
In contrast to most eukaryotic cells, neurons possess long, highly branched processes called axons and dendrites. In large mammals, such as humans, some axons...
Aging | Axons | Neurodegeneration | Motor proteins | Axonal pathology | BETA-SECRETASE ACTIVITY | HEREDITARY SPASTIC PARAPLEGIA | APP-LIKE PROTEIN-2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | INHIBITS NEURITE OUTGROWTH | neurodegeneration | CORTICAL CHOLINERGIC FIBERS | RAT SCIATIC-NERVE | axonal pathology | AMYLOID-PRECURSOR PROTEIN | NERVE GROWTH-FACTOR | axons | motor proteins | aging | KINESIN HEAVY-CHAIN | TRANSGENIC MICE | Dyneins - metabolism | Alzheimer Disease - physiopathology | Humans | Axons - metabolism | Axonal Transport - physiology | Alzheimer Disease - pathology | Microtubules - metabolism | Animals | Aging - physiology | Axons - pathology | Alzheimer Disease - metabolism | Axons - ultrastructure | Molecular Motor Proteins - metabolism | Causes of | Development and progression | Nervous system | Degeneration | Research | Axonal transport | Alzheimer's disease | Health aspects | Risk factors
Aging | Axons | Neurodegeneration | Motor proteins | Axonal pathology | BETA-SECRETASE ACTIVITY | HEREDITARY SPASTIC PARAPLEGIA | APP-LIKE PROTEIN-2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | INHIBITS NEURITE OUTGROWTH | neurodegeneration | CORTICAL CHOLINERGIC FIBERS | RAT SCIATIC-NERVE | axonal pathology | AMYLOID-PRECURSOR PROTEIN | NERVE GROWTH-FACTOR | axons | motor proteins | aging | KINESIN HEAVY-CHAIN | TRANSGENIC MICE | Dyneins - metabolism | Alzheimer Disease - physiopathology | Humans | Axons - metabolism | Axonal Transport - physiology | Alzheimer Disease - pathology | Microtubules - metabolism | Animals | Aging - physiology | Axons - pathology | Alzheimer Disease - metabolism | Axons - ultrastructure | Molecular Motor Proteins - metabolism | Causes of | Development and progression | Nervous system | Degeneration | Research | Axonal transport | Alzheimer's disease | Health aspects | Risk factors
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Loading RightsNeuron, ISSN 0896-6273, 10/2014, Volume 84, Issue 2, pp. 292 - 309
Axonal transport is essential for neuronal function, and many neurodevelopmental and neurodegenerative diseases result from mutations in the axonal transport...
CYTOPLASMIC DYNEIN | SPINAL MUSCULAR-ATROPHY | SUBCORTICAL LAMINAR HETEROTOPIA | KINESIN SUPERFAMILY PROTEIN | MESSENGER-RNA | TUG-OF-WAR | RETROGRADE TRANSPORT | MICROTUBULE PLUS-END | AMYLOID PRECURSOR PROTEIN | NEUROSCIENCES | DENSE-CORE VESICLES | Animals | Humans | Axonal Transport - physiology | Signal Transduction - physiology | Axons - physiology | Neurons - metabolism | Neurodegenerative Diseases - metabolism | Organelles - metabolism | Proteins | Physiological aspects | Laws, regulations and rules | Transportation | Neurophysiology | Motility | Neurons | Morphology | Cytoskeleton | Mutation | Grants | Metabolism | Alzheimers disease | Experiments | slow axonal transport | signaling endosomes | organelle motility | autophagosomes | kinesin | mitochondria | cytoplasmic dynein | Fast axonal transport | organelle transport | lysosomes | microtubules | vesicular transport
CYTOPLASMIC DYNEIN | SPINAL MUSCULAR-ATROPHY | SUBCORTICAL LAMINAR HETEROTOPIA | KINESIN SUPERFAMILY PROTEIN | MESSENGER-RNA | TUG-OF-WAR | RETROGRADE TRANSPORT | MICROTUBULE PLUS-END | AMYLOID PRECURSOR PROTEIN | NEUROSCIENCES | DENSE-CORE VESICLES | Animals | Humans | Axonal Transport - physiology | Signal Transduction - physiology | Axons - physiology | Neurons - metabolism | Neurodegenerative Diseases - metabolism | Organelles - metabolism | Proteins | Physiological aspects | Laws, regulations and rules | Transportation | Neurophysiology | Motility | Neurons | Morphology | Cytoskeleton | Mutation | Grants | Metabolism | Alzheimers disease | Experiments | slow axonal transport | signaling endosomes | organelle motility | autophagosomes | kinesin | mitochondria | cytoplasmic dynein | Fast axonal transport | organelle transport | lysosomes | microtubules | vesicular transport
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Loading RightsNature Reviews Neuroscience, ISSN 1471-003X, 03/2013, Volume 14, Issue 3, pp. 157 - 157
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Loading RightsSCIENCE, ISSN 0036-8075, 10/2019, Volume 366, Issue 6462, pp. 199 - 199
The intracellular transport system in neurons is specialized to an extraordinary degree, enabling the delivery of critical cargo to sites in axons or dendrites...
CYTOPLASMIC DYNEIN | MICROTUBULE STABILIZATION | DYNEIN-DRIVEN TRANSPORT | MITOCHONDRIAL DYNAMICS | SPINAL MUSCULAR-ATROPHY | MULTIDISCIPLINARY SCIENCES | POLARIZED CARGO TRANSPORT | END-BINDING-PROTEIN | CORTICAL DEVELOPMENT | HIPPOCAMPAL-NEURONS | MOTOR-ADAPTER | Molecular machines | Cargo | Motor activity | Trafficking | Central nervous system | Cognitive ability | Lysosomes | Selectivity | mRNA | Service life assessment | Cytosol | Transportation systems | Complexity | Proteins | Mitochondria | Vesicles | Synaptic transmission | Dendrites | Cell body | Growth factors | Kinesin | Adaptor proteins | Motor neurons | Neurodegenerative diseases | Neurons | Molecular motors | Organelles | Trauma | Mathematical morphology | Axons | Chemotherapy | Microtubules | Regulatory mechanisms (biology) | Dynein | Neural stem cells | Mutation | Intracellular | Axonal transport | Synapses | Endosomes
CYTOPLASMIC DYNEIN | MICROTUBULE STABILIZATION | DYNEIN-DRIVEN TRANSPORT | MITOCHONDRIAL DYNAMICS | SPINAL MUSCULAR-ATROPHY | MULTIDISCIPLINARY SCIENCES | POLARIZED CARGO TRANSPORT | END-BINDING-PROTEIN | CORTICAL DEVELOPMENT | HIPPOCAMPAL-NEURONS | MOTOR-ADAPTER | Molecular machines | Cargo | Motor activity | Trafficking | Central nervous system | Cognitive ability | Lysosomes | Selectivity | mRNA | Service life assessment | Cytosol | Transportation systems | Complexity | Proteins | Mitochondria | Vesicles | Synaptic transmission | Dendrites | Cell body | Growth factors | Kinesin | Adaptor proteins | Motor neurons | Neurodegenerative diseases | Neurons | Molecular motors | Organelles | Trauma | Mathematical morphology | Axons | Chemotherapy | Microtubules | Regulatory mechanisms (biology) | Dynein | Neural stem cells | Mutation | Intracellular | Axonal transport | Synapses | Endosomes
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Loading RightsCell, ISSN 0092-8674, 01/2013, Volume 152, Issue 3, pp. 479 - 491
Fast axonal transport (FAT) requires consistent energy over long distances to fuel the molecular motors that transport vesicles. We demonstrate that glycolysis...
KINESIN | ENZYME-ACTIVITY | MICROTUBULES | DYNEIN | GLYCERALDEHYDE-3-PHOSPHATE DEHYDROGENASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | PROTEOMIC ANALYSIS | PROTEINS | CULTURED HIPPOCAMPAL-NEURONS | BRAIN | CELL BIOLOGY | Axonal Transport | Brain - cytology | Cells, Cultured | Axons - metabolism | Rats | Mitochondria - metabolism | Drosophila melanogaster - metabolism | Animals | Adenosine Triphosphate - metabolism | Glycolysis | Drosophila melanogaster - growth & development | Mice | Neurons - metabolism | Glyceraldehyde-3-Phosphate Dehydrogenases - metabolism | Glucose metabolism | Enzymes | Mitochondrial DNA | Neurons
KINESIN | ENZYME-ACTIVITY | MICROTUBULES | DYNEIN | GLYCERALDEHYDE-3-PHOSPHATE DEHYDROGENASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | PROTEOMIC ANALYSIS | PROTEINS | CULTURED HIPPOCAMPAL-NEURONS | BRAIN | CELL BIOLOGY | Axonal Transport | Brain - cytology | Cells, Cultured | Axons - metabolism | Rats | Mitochondria - metabolism | Drosophila melanogaster - metabolism | Animals | Adenosine Triphosphate - metabolism | Glycolysis | Drosophila melanogaster - growth & development | Mice | Neurons - metabolism | Glyceraldehyde-3-Phosphate Dehydrogenases - metabolism | Glucose metabolism | Enzymes | Mitochondrial DNA | Neurons
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Loading RightsNature Methods, ISSN 1548-7091, 07/2007, Volume 4, Issue 7, pp. 559 - 561
Neuronal mitochondria regulate synaptic physiology and cellular survival, and disruption of their function or transport causes neurological disease. We present...
GFP | NEURONS | FLUORESCENT PROTEIN | BIOCHEMICAL RESEARCH METHODS | TRANSGENIC MICE | Spectrometry, Fluorescence - methods | Axonal Transport | Luminescent Proteins - analysis | Animals | Neurons - ultrastructure | Axons - ultrastructure | Axons - metabolism | Mice | Mitochondria - metabolism | Neurons - metabolism | Mitochondria - chemistry | Fluorescence | Scientific imaging | Research methodology | Rodents | Neurological disorders
GFP | NEURONS | FLUORESCENT PROTEIN | BIOCHEMICAL RESEARCH METHODS | TRANSGENIC MICE | Spectrometry, Fluorescence - methods | Axonal Transport | Luminescent Proteins - analysis | Animals | Neurons - ultrastructure | Axons - ultrastructure | Axons - metabolism | Mice | Mitochondria - metabolism | Neurons - metabolism | Mitochondria - chemistry | Fluorescence | Scientific imaging | Research methodology | Rodents | Neurological disorders
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Loading Rightsneurogenetics, ISSN 1364-6745, 5/2012, Volume 13, Issue 2, pp. 169 - 179
Hereditary spastic paraplegia (HSP) is a neurodegenerative disorder preferentially affecting the longest corticospinal axons. More than 40 HSP genetic loci...
Human Genetics | Neurosciences | Mitochondria | Biomedicine | Hereditary spastic paraplegia (HSP) | KIF5A | Molecular Medicine | Motor neuron | Axonal transport | Axonal outgrowth | CYTOPLASMIC DYNEIN | COMPLEX | INTRACELLULAR-TRANSPORT | HEREDITARY SPASTIC PARAPLEGIA | CLINICAL NEUROLOGY | TARGETED DISRUPTION | CORTICAL-NEURONS | MUTATION | GENETICS & HEREDITY | KINESIN HEAVY-CHAIN | PROTEINS | Spastic Paraplegia, Hereditary - genetics | Axonal Transport - genetics | Kinesin - deficiency | Mice, Inbred C57BL | Cells, Cultured | Axons - metabolism | Mitochondria - metabolism | Mutation, Missense | Gene Knockout Techniques | Spastic Paraplegia, Hereditary - metabolism | Kinesin - metabolism | Motor Neurons - metabolism | Animals | Sensory Receptor Cells - cytology | Mitochondria - genetics | Motor Neurons - cytology | Mice | Sensory Receptor Cells - metabolism | Disease Models, Animal | Original
Human Genetics | Neurosciences | Mitochondria | Biomedicine | Hereditary spastic paraplegia (HSP) | KIF5A | Molecular Medicine | Motor neuron | Axonal transport | Axonal outgrowth | CYTOPLASMIC DYNEIN | COMPLEX | INTRACELLULAR-TRANSPORT | HEREDITARY SPASTIC PARAPLEGIA | CLINICAL NEUROLOGY | TARGETED DISRUPTION | CORTICAL-NEURONS | MUTATION | GENETICS & HEREDITY | KINESIN HEAVY-CHAIN | PROTEINS | Spastic Paraplegia, Hereditary - genetics | Axonal Transport - genetics | Kinesin - deficiency | Mice, Inbred C57BL | Cells, Cultured | Axons - metabolism | Mitochondria - metabolism | Mutation, Missense | Gene Knockout Techniques | Spastic Paraplegia, Hereditary - metabolism | Kinesin - metabolism | Motor Neurons - metabolism | Animals | Sensory Receptor Cells - cytology | Mitochondria - genetics | Motor Neurons - cytology | Mice | Sensory Receptor Cells - metabolism | Disease Models, Animal | Original
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Loading RightsNeuron, ISSN 0896-6273, 12/2017, Volume 96, Issue 6, pp. 1264 - 1271.e5
Microtubules are essential for polarized transport in neurons, but how their organization guides motor proteins to axons or dendrites is unclear. Because...
polarized transport | microtubule polarity | motor proteins | kinesin | microtubule orientation | neuronal polarity | dendrites | microtubules | axon | neurons | CARGO TRANSPORT | INTRACELLULAR-TRANSPORT | SUPERRESOLUTION MICROSCOPY | HIGH-DENSITY | AXON INITIAL SEGMENT | MECHANISMS | KINESIN-1 MOTOR DOMAIN | END-BINDING-PROTEIN | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | TUBULIN CODE | Proteins | Neurons | Kinesin | Myosin | Motors | Bundles | Axons | Insects | Microtubules | Optical properties | Cell cycle | Cytoskeleton | Polarity | Dendrites | Transport | Axon guidance | Axonal transport | Localization | Nanotechnology
polarized transport | microtubule polarity | motor proteins | kinesin | microtubule orientation | neuronal polarity | dendrites | microtubules | axon | neurons | CARGO TRANSPORT | INTRACELLULAR-TRANSPORT | SUPERRESOLUTION MICROSCOPY | HIGH-DENSITY | AXON INITIAL SEGMENT | MECHANISMS | KINESIN-1 MOTOR DOMAIN | END-BINDING-PROTEIN | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | TUBULIN CODE | Proteins | Neurons | Kinesin | Myosin | Motors | Bundles | Axons | Insects | Microtubules | Optical properties | Cell cycle | Cytoskeleton | Polarity | Dendrites | Transport | Axon guidance | Axonal transport | Localization | Nanotechnology
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Loading RightsThe EMBO Journal, ISSN 0261-4189, 02/2010, Volume 29, Issue 4, pp. 843 - 854
Cytoplasmic protein transport in axons (‘slow axonal transport’) is essential for neuronal homeostasis, and involves Kinesin‐1, the same motor for membranous...
axonal transport | chaperone | kinesin light chain | Hsc70 | tetratricopeptide repeat | Tetratricopeptide repeat | Kinesin light chain | Axonal transport | Chaperone | INTRACELLULAR-TRANSPORT | BIOCHEMISTRY & MOLECULAR BIOLOGY | MODEL | SQUID GIANT-AXON | CELL BIOLOGY | KINESIN | IN-VIVO | NEURONS | OPTIC-NERVE DAMAGE | PROTEINS | EXPRESSION | TRANSGENIC MICE | Creatine Kinase - chemistry | Microtubule-Associated Proteins - chemistry | Optic Nerve - pathology | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | HSC70 Heat-Shock Proteins - metabolism | Multiprotein Complexes | Molecular Sequence Data | Molecular Motor Proteins - genetics | Molecular Motor Proteins - metabolism | Protein Interaction Domains and Motifs | HSP40 Heat-Shock Proteins - chemistry | Recombinant Proteins - metabolism | Amino Acid Sequence | HSC70 Heat-Shock Proteins - genetics | HSP40 Heat-Shock Proteins - metabolism | HSP40 Heat-Shock Proteins - genetics | Axons - metabolism | Recombinant Proteins - chemistry | Axonal Transport - physiology | Mice, Transgenic | Recombinant Proteins - genetics | Molecular Motor Proteins - chemistry | Protein Transport | Decapodiformes | Animals | HSC70 Heat-Shock Proteins - chemistry | Creatine Kinase - metabolism | Mice | Kinetics | In Vitro Techniques | Proteins | Cellular biology | Molecular biology | Transgenic animals | Binding sites
axonal transport | chaperone | kinesin light chain | Hsc70 | tetratricopeptide repeat | Tetratricopeptide repeat | Kinesin light chain | Axonal transport | Chaperone | INTRACELLULAR-TRANSPORT | BIOCHEMISTRY & MOLECULAR BIOLOGY | MODEL | SQUID GIANT-AXON | CELL BIOLOGY | KINESIN | IN-VIVO | NEURONS | OPTIC-NERVE DAMAGE | PROTEINS | EXPRESSION | TRANSGENIC MICE | Creatine Kinase - chemistry | Microtubule-Associated Proteins - chemistry | Optic Nerve - pathology | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | HSC70 Heat-Shock Proteins - metabolism | Multiprotein Complexes | Molecular Sequence Data | Molecular Motor Proteins - genetics | Molecular Motor Proteins - metabolism | Protein Interaction Domains and Motifs | HSP40 Heat-Shock Proteins - chemistry | Recombinant Proteins - metabolism | Amino Acid Sequence | HSC70 Heat-Shock Proteins - genetics | HSP40 Heat-Shock Proteins - metabolism | HSP40 Heat-Shock Proteins - genetics | Axons - metabolism | Recombinant Proteins - chemistry | Axonal Transport - physiology | Mice, Transgenic | Recombinant Proteins - genetics | Molecular Motor Proteins - chemistry | Protein Transport | Decapodiformes | Animals | HSC70 Heat-Shock Proteins - chemistry | Creatine Kinase - metabolism | Mice | Kinetics | In Vitro Techniques | Proteins | Cellular biology | Molecular biology | Transgenic animals | Binding sites
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