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World Neurosurgery, ISSN 1878-8750, 12/2018, Volume 120, pp. e380 - e391
Glioblastoma (GBM) is the most common and fatal human brain tumor, with the worst prognosis. The aberrant microenvironment, enhanced by the activation of... 
Angiogenesis | Aspirin | HIF-1α | VEGF | Endothelial cells | Glioblastoma | GLIOMA-CELLS | SURGERY | APOPTOSIS | HIF-1 alpha | ACTIVATED PROTEIN-KINASE | RISK | PROLIFERATION | INTRACRANIAL HEMORRHAGE | CLINICAL NEUROLOGY | BREAST-CANCER | VENOUS THROMBOEMBOLISM | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | EXPRESSION | Humans | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Vascular Endothelial Growth Factor Receptor-2 - genetics | Mitogen-Activated Protein Kinase Kinases - metabolism | Glioblastoma - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glioblastoma - metabolism | Aspirin - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Real-Time Polymerase Chain Reaction | Cell Survival - drug effects | Vascular Endothelial Growth Factor Receptor-2 - drug effects | Signal Transduction | Endothelial Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | bcl-2-Associated X Protein - metabolism | Angiogenesis Inhibitors - pharmacology | Brain Neoplasms - genetics | bcl-2-Associated X Protein - drug effects | Blotting, Western | Antineoplastic Agents, Immunological - pharmacology | Drug Synergism | Vascular Endothelial Growth Factor Receptor-1 - drug effects | Mitogen-Activated Protein Kinase Kinases - drug effects | Proto-Oncogene Proteins c-bcl-2 - drug effects | Glioblastoma - blood supply | Hypoxia-Inducible Factor 1, alpha Subunit - drug effects | Sunitinib - pharmacology | ras Proteins - genetics | Bevacizumab - pharmacology | Mitogen-Activated Protein Kinase Kinases - genetics | ras Proteins - metabolism | Antineoplastic Agents, Alkylating - pharmacology | Brain Neoplasms - blood supply | Temozolomide - pharmacology | Brain Neoplasms - metabolism | Proto-Oncogene Proteins c-akt - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Proto-Oncogene Proteins c-bcl-2 - metabolism | Antineoplastic Agents - pharmacology | ras Proteins - drug effects | Vascular Endothelial Growth Factor A - drug effects | bcl-2-Associated X Protein - genetics | RNA, Messenger - drug effects | Vascular Endothelial Growth Factor Receptor-1 - genetics | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Endothelial Cells - drug effects | Proto-Oncogene Proteins c-akt - drug effects | Evaluation | Oncology, Experimental | Brain tumors | Development and progression | Research | Antineoplastic agents | Glioblastoma multiforme | Cells | Antimitotic agents | Prevention | Chemotherapy | Analysis | Lymphomas | Temozolomide | Angiogenesis inhibitors | Vascular endothelial growth factor | Mitogens | Protein kinases | Cancer | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 10/2007, Volume 110, Issue 8, pp. 2924 - 2930
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 08/2013, Volume 20, Issue 8, pp. 987 - 997
Non-proliferating oocytes within avascular regions of the ovary are exquisitely susceptible to chemotherapy. Early menopause and sterility are unintended... 
TAp73 | c-Abl | Imatinib | Ovarian follicles | Cisplatin | TAp63 | ovarian follicles | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | imatinib | QUALITY-CONTROL | P63 | FERTILITY | CELL BIOLOGY | CANCER-TREATMENT | GERM-CELLS | TUMOR-SUPPRESSOR | EPITHELIAL DEVELOPMENT | cisplatin | PROTECTS MOUSE OOCYTES | Proto-Oncogene Proteins c-abl - antagonists & inhibitors | Tumor Suppressor Protein p53 - antagonists & inhibitors | Apoptosis - drug effects | Oogenesis - physiology | bcl-2-Associated X Protein - physiology | Dose-Response Relationship, Drug | Tumor Suppressor Protein p53 - physiology | DNA Damage - physiology | Antineoplastic Agents - adverse effects | Oocytes - drug effects | Female | Models, Animal | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Nuclear Proteins - drug effects | Oogenesis - drug effects | Platinum - pharmacology | DNA Damage - drug effects | Proto-Oncogene Proteins c-abl - drug effects | Mice, Inbred C57BL | Cells, Cultured | Cisplatin - pharmacology | Pyrimidines - pharmacology | Mice, Inbred Strains | Imatinib Mesylate | Piperazines - pharmacology | Tumor Suppressor Protein p53 - drug effects | bcl-2-Associated X Protein - drug effects | Animals | Oocytes - physiology | Signal Transduction - drug effects | Cisplatin - adverse effects | Signal Transduction - physiology | Mice | Nuclear Proteins - physiology | Apoptosis - physiology | In Vitro Techniques | Platinum - adverse effects | Index Medicus | Original Paper
Journal Article
Journal Article
Journal Article
Journal Article
Annals of Surgical Oncology, ISSN 1068-9265, 9/2012, Volume 19, Issue 9, pp. 3097 - 3106
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2006, Volume 23, Issue 1, pp. 127 - 139
Journal Article
Neuroscience, ISSN 0306-4522, 2009, Volume 160, Issue 2, pp. 330 - 338
Abstract Maternal heroin abuse has been shown to result in teratogenic neurobehavioral defects in the offspring, but the underlying mechanisms remain largely... 
Neurology | Bcl-2 | Bax | prenatal exposure | heroin | caspase-3 | ACID | MOUSE | DEATH | DECARBOXYLASE IMMUNOREACTIVE CELLS | NEUROSCIENCES | HIPPOCAMPUS | FAS RECEPTOR | BEHAVIORAL DEFICITS | NEUROGENESIS | RAT-BRAIN | UP-REGULATION | Memory - drug effects | Prenatal Exposure Delayed Effects | Apoptosis - drug effects | Caspase 3 - metabolism | Neurons - cytology | Hippocampus - drug effects | Apoptosis Regulatory Proteins - drug effects | Maternal Exposure | Narcotics - adverse effects | Proto-Oncogene Proteins c-bcl-2 - metabolism | Reaction Time - drug effects | Caspase 3 - drug effects | Caspase 3 - genetics | Statistics, Nonparametric | Apoptosis Regulatory Proteins - genetics | Female | Spatial Behavior - drug effects | Neurons - drug effects | bcl-2-Associated X Protein - genetics | Hippocampus - embryology | bcl-2-Associated X Protein - metabolism | Hippocampus - cytology | Maze Learning - drug effects | bcl-2-Associated X Protein - drug effects | Apoptosis Regulatory Proteins - metabolism | Pregnancy | Animals | Analysis of Variance | Proto-Oncogene Proteins c-bcl-2 - drug effects | Heroin - adverse effects | Mice | Mice, Inbred BALB C | Proto-Oncogene Proteins c-bcl-2 - genetics | RNA, Messenger | Immunohistochemistry | Polymerase chain reaction | Drug abuse | Cell research | Neurosciences | Messenger RNA | Pregnant women | Neurons | Analysis | Apoptosis | Index Medicus
Journal Article