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Cell, ISSN 0092-8674, 2008, Volume 132, Issue 1, pp. 125 - 136
Journal Article
Cardiovascular Research, ISSN 0008-6363, 11/2017, Volume 113, Issue 13, pp. 1615 - 1626
Journal Article
FASEB Journal, ISSN 0892-6638, 12/2013, Volume 27, Issue 12, pp. 4987 - 4997
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 07/2017, Volume 92, Issue 1, pp. 101 - 113
Macrophage activation is increased in diabetes and correlated with the onset and progression of vascular complications. To identify drugs that could inhibit... 
macrophages | fibrosis | inflammation | diabetes | PROTEIN | PERIPHERAL-BLOOD | NEPHROPATHY | MONOCYTIC CELLS | BETA | ALPHA-TOCOPHEROL SUPPLEMENTATION | CYTOKINE PRODUCTION | INSULIN-RESISTANCE | UROLOGY & NEPHROLOGY | PLASMA-GLUCOSE | Tumor Necrosis Factor-alpha - metabolism | Diabetes Mellitus, Experimental - drug therapy | Phosphorylation | Diabetic Cardiomyopathies - metabolism | Kidney - pathology | Streptozocin | Humans | Diabetes Mellitus, Experimental - genetics | Male | NF-kappa B - metabolism | beta-Arrestin 2 - metabolism | Receptors, Adrenergic, beta-2 - drug effects | Adrenergic beta-2 Receptor Agonists - pharmacology | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Protein Kinase C - metabolism | Myocardium - metabolism | Diabetic Cardiomyopathies - prevention & control | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - prevention & control | Adrenergic beta-2 Receptor Antagonists - pharmacology | Kidney - drug effects | Anti-Inflammatory Agents - pharmacology | Receptors, Adrenergic, beta-2 - genetics | Diabetic Nephropathies - metabolism | Diabetic Nephropathies - chemically induced | THP-1 Cells | beta-Arrestin 2 - genetics | Diabetic Nephropathies - genetics | Myocardium - pathology | Rats, Sprague-Dawley | Receptors, Adrenergic, beta-2 - metabolism | Diabetic Cardiomyopathies - genetics | Rats, Zucker | Macrophages - metabolism | Animals | NF-KappaB Inhibitor alpha - metabolism | Fibrosis | Macrophages - drug effects | Diabetic Cardiomyopathies - chemically induced | Macrophage Activation - drug effects
Journal Article
Cellular Signalling, ISSN 0898-6568, 03/2016, Volume 28, Issue 3, pp. 190 - 203
Cardiac myocyte oxidative stress and apoptosis are considered important mechanisms for the development of heart failure (HF). Chronic HF is characterized by... 
Heart failure | Oxidative stress | G protein-coupled receptor kinases | NADPH oxidase | CARDIAC-FUNCTION | ACTIVATION | ISOFORMS | MYOCYTES | ANGIOTENSIN-II | BETA-ARRESTIN | DEATH | LEVEL | DAMAGE | CELL BIOLOGY | HEART | Phosphorylation | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | Apoptosis - drug effects | NADPH Oxidases - metabolism | G-Protein-Coupled Receptor Kinase 2 - genetics | G-Protein-Coupled Receptor Kinase 2 - metabolism | Rhodopsin - metabolism | RNA Interference | Acetophenones - pharmacology | NADPH Oxidases - genetics | Receptors, Adrenergic, beta - metabolism | Cyclic AMP - metabolism | Cell Line | Receptors, Adrenergic, beta - chemistry | Angiotensin II - pharmacology | Myocytes, Cardiac - cytology | NADPH Oxidases - antagonists & inhibitors | Adrenergic beta-Agonists - pharmacology | Rats | Heart Failure - metabolism | Mitochondria - metabolism | Heart Failure - pathology | NADPH Oxidase 4 | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Microscopy, Confocal | Animals | Signal Transduction - drug effects | beta-Arrestins | Isoproterenol - pharmacology | Myocytes, Cardiac - metabolism | Arrestins | Oxidative Stress - drug effects | RNA, Small Interfering - metabolism | Oxidases | G proteins | Membrane proteins | Apoptosis | Stresses | Cellular | Receptors | Inhibitors | Cell death | Stimulation | Cardiac output | heart failure | oxidative stress
Journal Article
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 04/2018, Volume 314, Issue 4, pp. R574 - R583
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2016, Volume 113, Issue 50, pp. E8178 - E8186
Journal Article