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Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 02/2008, Volume 134, Issue 2, pp. 511 - 522
Background & Aims: Loss of gastric parietal cells is a critical precursor to gastric metaplasia and neoplasia. However, the origin of metaplasia remains... 
Gastroenterology and Hepatology | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Metaplasia - metabolism | Humans | Parietal Cells, Gastric - metabolism | Stomach Neoplasms - metabolism | Gastric Mucosa - pathology | Precancerous Conditions - metabolism | Stomach Neoplasms - pathology | beta-Defensins | Fetal Proteins - metabolism | Gastric Mucosa - metabolism | Stomach Neoplasms - physiopathology | Minichromosome Maintenance Complex Component 3 | Atrophy | DNA-Binding Proteins - metabolism | Peptides - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Mucins - metabolism | Muscle Proteins - metabolism | Precancerous Conditions - pathology | Trefoil Factor-2 | Gastric Fundus - metabolism | Chief Cells, Gastric - metabolism | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Microtubule-Associated Proteins | Nuclear Proteins - metabolism | Gastrins - metabolism | Cell Transformation, Neoplastic - metabolism | Metaplasia - pathology | Mice, Knockout | Animals | Carrier Proteins - metabolism | Parietal Cells, Gastric - pathology | Epididymal Secretory Proteins - metabolism | Chief Cells, Gastric - pathology | Mice | Cell Transformation, Neoplastic - pathology | Gastric Fundus - pathology | Intrinsic Factor - metabolism | Vasoactive intestinal peptides | Analysis | Polypeptides | Index Medicus | Abridged Index Medicus
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 621 - 632
Background & Aims Epithelial cells that cover the intestinal mucosal surface maintain immune homeostasis and tolerance in the gastrointestinal tract. However,... 
Gastroenterology and Hepatology | IgA | Inflammatory Bowel Disease | T Cell | Immune Response | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Intestinal Mucosa - metabolism | Adaptor Protein Complex mu Subunits - metabolism | Epithelial Cells - metabolism | Adaptor Protein Complex beta Subunits - deficiency | Humans | Cell Membrane Permeability | Crohn Disease - metabolism | Cell Membrane - physiology | Ribonucleases - metabolism | Adaptor Protein Complex 1 - metabolism | Th17 Cells - metabolism | Intestinal Mucosa - immunology | alpha-Defensins - metabolism | Adaptor Protein Complex beta Subunits - immunology | Cell Membrane - metabolism | beta-Defensins - metabolism | Colon | Receptors, Cytokine - metabolism | Colitis - immunology | Adaptor Protein Complex 1 - deficiency | Lipocalins - metabolism | Adaptor Protein Complex 1 - immunology | Immunoglobulin A - metabolism | Signal Transduction | Acute-Phase Proteins - metabolism | Down-Regulation | Oncogene Proteins - metabolism | Homeostasis - immunology | Epithelial Cells - pathology | S100 Proteins - metabolism | Muramidase - metabolism | Intestinal Mucosa - microbiology | Mice, Knockout | Interleukin-17 - metabolism | Adaptor Protein Complex beta Subunits - metabolism | Animals | Colitis - microbiology | Proteins - metabolism | Ribonuclease, Pancreatic - metabolism | Lipocalin-2 | Cathelicidins - metabolism | Epithelial Cells - immunology | Receptors, Cytokine - immunology | Mice | Intestinal Mucosa - pathology | Medical colleges | Fc receptors | Gastrointestinal diseases | Homeostasis | Biological response modifiers | Immunoglobulin A | T cells | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 12/2010, Volume 107, Issue 52, pp. 22593 - 22598
We investigated the mechanisms by which T-cell cytokines are able to influence the Toll-like receptor (TLR)-induced, vitamin D-dependent antimicrobial pathway... 
T lymphocytes | Up regulation | Monocytes | Messenger RNA | Cytokines | Vitamin D | Mycobacterium tuberculosis | Antimicrobials | Small interfering RNA | Physiological regulation | Interleukin-4 | Interferon-γ | Innate immune response | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Steroid Hydroxylases - metabolism | Gene Expression - drug effects | Monocytes - cytology | Humans | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - genetics | Monocytes - metabolism | Antimicrobial Cationic Peptides - metabolism | Th1 Cells - metabolism | Interleukin-4 - pharmacology | T-Lymphocytes - metabolism | Calcitriol - metabolism | beta-Defensins - metabolism | Antimicrobial Cationic Peptides - genetics | Steroid Hydroxylases - genetics | beta-Defensins - genetics | Toll-Like Receptor 1 - metabolism | Cells, Cultured | 25-Hydroxyvitamin D3 1-alpha-Hydroxylase - metabolism | Vitamin D3 24-Hydroxylase | Toll-Like Receptor 2 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Th2 Cells - metabolism | Blotting, Western | Monocytes - drug effects | Signal Transduction - drug effects | Vitamin D - analogs & derivatives | Vitamin D - metabolism | Cytokines - pharmacology | Interferon-gamma - pharmacology | Immune response | Calcifediol | Regulation | Alfacalcidol | Research | T cells | Properties | Health aspects | Index Medicus | interleukin-4 | Biological Sciences | interferon-γ | innate immune response
Journal Article
The Journal of Immunology, ISSN 0022-1767, 01/2005, Volume 174, Issue 2, pp. 992 - 1002
The objective of this study was to examine the expression of TLR by human primary uterine epithelial cells (UEC) and to determine whether exposure to the TLR... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Toll-Like Receptor 1 | Tumor Necrosis Factor-alpha - metabolism | Toll-Like Receptor 3 | Epithelial Cells - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Membrane Glycoproteins - biosynthesis | Uterus - metabolism | Chemokines, CC | Interferon-beta - physiology | Uterus - immunology | RNA, Messenger - biosynthesis | Toll-Like Receptors | Inflammation Mediators - metabolism | Interferon-beta - genetics | Female | Chemokine CCL2 - metabolism | Receptors, Cell Surface - biosynthesis | Interleukin-8 - metabolism | Membrane Glycoproteins - immunology | Interleukin-6 - metabolism | Granulocyte Colony-Stimulating Factor - metabolism | Chemokine CCL4 | Poly I-C - pharmacology | beta-Defensins - genetics | Cytokines - metabolism | Antibodies, Monoclonal - pharmacology | Macrophage Inflammatory Proteins - metabolism | Cells, Cultured | Membrane Glycoproteins - agonists | Receptors, Cell Surface - agonists | Interferon-beta - biosynthesis | Immunity, Innate | Receptors, Cell Surface - immunology | Uterus - virology | Proteins - metabolism | Epithelial Cells - immunology | Epithelial Cells - virology | Uterus - cytology | Chemokines - metabolism | Chemokine CCL2 - antagonists & inhibitors | Toll-Like Receptor 10 | beta-Defensins - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Immunology and cell biology, ISSN 0818-9641, 11/2008, Volume 86, Issue 8, pp. 643 - 649
Journal Article
10.