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The Journal of Immunology, ISSN 0022-1767, 09/2007, Volume 179, Issue 6, pp. 3926 - 3936
Journal Article
Bioorganic & Medicinal Chemistry Letters, ISSN 0960-894X, 07/2018, Volume 28, Issue 12, pp. 2210 - 2216
L. was well known as a traditional herbal medicine to treat inflammation and allergic skin diseases. The aim of this research was to indentify compounds with... 
Sanguisorba officinalis L | NF-κB | iNOS and COX-2 | Anti-allergic inflammatory | Rosaceae | ACTIVATION | CHEMISTRY, MEDICINAL | COX-2 | MAST-CELLS | CHEMISTRY, ORGANIC | TRITERPENE GLYCOSIDES | ETHANOL EXTRACT | ATOPIC-DERMATITIS | NF-kappa B | RBL-2H3 CELLS | NITRIC-OXIDE | iNOS | DEGRANULATION | ROOTS | NF-KAPPA-B | Tetradecanoylphorbol Acetate - pharmacology | Structure-Activity Relationship | Anti-Allergic Agents - pharmacology | Lipopolysaccharides - antagonists & inhibitors | Tetradecanoylphorbol Acetate - antagonists & inhibitors | Dose-Response Relationship, Drug | Inflammation - metabolism | Interleukin-4 - biosynthesis | Inflammation - drug therapy | Anti-Allergic Agents - isolation & purification | Enzyme Inhibitors - chemistry | Anti-Allergic Agents - chemistry | Molecular Structure | Nitric Oxide - biosynthesis | Interleukin-4 - antagonists & inhibitors | Nitric Oxide - antagonists & inhibitors | beta-N-Acetylhexosaminidases - metabolism | Enzyme Inhibitors - pharmacology | Rats | Enzyme Inhibitors - isolation & purification | Macrophages - metabolism | Animals | Lipopolysaccharides - pharmacology | Macrophages - drug effects | RAW 264.7 Cells | Mice | Sanguisorba - chemistry | Tumor Necrosis Factor-alpha - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Medical research | Nitric oxide | Analysis | Pharmacy | Medicine, Experimental | Drugstores | Skin diseases
Journal Article
Journal Article
Journal of Investigational Allergology and Clinical Immunology, ISSN 1018-9068, 2017, Volume 27, Issue 3, pp. 160 - 168
Journal Article
Journal Article
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 2015, Volume 309, Issue 9, pp. C593 - C599
Cardiovascular disease is the primary cause of morbidity and mortality in diabetes, and endothelial dysfunction is commonly seen in these patients. Increased... 
Endothelium-dependent hyperpolarization | Vascular rarefaction | GCA | Endothelial cell dysfunction | Diabetic vascular complications | N-ACETYLGLUCOSAMINE | diabetic vascular complications | ACTIVATION | PHYSIOLOGY | PROTEIN | ISOFORMS | GLCNACYLATION | PHOSPHORYLATION | NITRIC-OXIDE SYNTHASE | vascular rarefaction | endothelial cell dysfunction | CELL BIOLOGY | endothelium-dependent hyperpolarization | ROLES | Diabetes Mellitus, Experimental - enzymology | Coronary Vessels - physiopathology | Histone Acetyltransferases - biosynthesis | Humans | Antigens, Neoplasm - biosynthesis | Diabetes Mellitus, Experimental - genetics | Histone Acetyltransferases - genetics | Endothelium, Vascular - drug effects | Male | Coronary Artery Disease - enzymology | Endothelium, Vascular - enzymology | Vasodilation | Diabetic Angiopathies - physiopathology | Coronary Artery Disease - physiopathology | Diabetic Angiopathies - enzymology | beta-N-Acetylhexosaminidases - genetics | Diabetic Angiopathies - genetics | Diabetes Mellitus, Experimental - physiopathology | Diabetes Mellitus, Type 1 - enzymology | Antigens, Neoplasm - genetics | Coronary Vessels - drug effects | Coronary Vessels - enzymology | Signal Transduction | Diabetes Mellitus, Type 1 - physiopathology | Cells, Cultured | Endothelium, Vascular - physiopathology | Enzyme Inhibitors - pharmacology | Diabetes Mellitus, Type 1 - genetics | Enzyme Induction | Mice, Transgenic | beta-N-Acetylhexosaminidases - antagonists & inhibitors | Glycosylation | beta-N-Acetylhexosaminidases - biosynthesis | N-Acetylglucosaminyltransferases - metabolism | Connexins - metabolism | Animals | Histone Acetyltransferases - antagonists & inhibitors | Coronary Artery Disease - genetics | Hyaluronoglucosaminidase - antagonists & inhibitors | Protein Processing, Post-Translational | Hyaluronoglucosaminidase - biosynthesis | Endothelial Cells - enzymology | Hyaluronoglucosaminidase - genetics | Neovascularization, Physiologic | Endothelial Cells - drug effects | Complications and side effects | Care and treatment | Usage | Type 1 diabetes | Genetic engineering | Research | Cardiovascular diseases | Health aspects | Risk factors | Endothelium
Journal Article
FASEB Journal, ISSN 0892-6638, 2014, Volume 28, Issue 8, pp. 3325 - 3328
Dysfunctions in Wnt signaling increase beta-catenin stability and are associated with cancers, including colorectal cancer. In addition, beta-catenin... 
Wnt signaling | ETD-MS/MS | Cancer | LACTIC-ACID | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RISK | INCIDENT COLORECTAL-CANCER | CELL-GROWTH | TUMORS | CELL BIOLOGY | PATHWAY | BIOLOGY | cancer | GLCNAC GLYCOSYLATION | MUTATIONS | CARBOHYDRATE-METABOLISM | Intestinal Mucosa - metabolism | Phosphorylation | Threonine - chemistry | alpha Catenin - metabolism | Adenocarcinoma - etiology | Humans | Hyperglycemia - complications | Molecular Sequence Data | Male | Dietary Carbohydrates - metabolism | Neoplasm Proteins - metabolism | Adherens Junctions - metabolism | Acetylglucosamine - metabolism | Colorectal Neoplasms - etiology | Adenocarcinoma - metabolism | MCF-7 Cells | Proteolysis | HEK293 Cells | beta Catenin - chemistry | Protein Stability | Dietary Carbohydrates - toxicity | Colorectal Neoplasms - metabolism | Wnt Signaling Pathway | Amino Acid Sequence | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Enzyme Inhibitors - pharmacology | Neoplasm Proteins - chemistry | beta-N-Acetylhexosaminidases - antagonists & inhibitors | Glycosylation | beta Catenin - metabolism | Colon - metabolism | Adherens Junctions - pathology | Protein Interaction Mapping | Hyperglycemia - metabolism | Animals | beta-N-Acetylhexosaminidases - physiology | Glucose - metabolism | N-Acetylglucosaminyltransferases - antagonists & inhibitors | N-Acetylglucosaminyltransferases - physiology | Mice | Protein Processing, Post-Translational | Research Communications | ETD-MS
Journal Article
The Biochemical journal, ISSN 0264-6021, 2013, Volume 456, Issue 1, pp. 1 - 12
Protein O-GlcNAcylation is an abundant, dynamic and reversible type of protein post-translational modification in animals that has been implicated in... 
Animals | Signal Transduction - drug effects | Humans | N-Acetylglucosaminyltransferases - antagonists & inhibitors | beta-N-Acetylhexosaminidases - antagonists & inhibitors | Acetylglucosamine - metabolism | Acylation
Journal Article
Journal Article
Investigative ophthalmology & visual science, ISSN 0146-0404, 2012, Volume 53, Issue 4, pp. 2281 - 2291
Journal Article