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Journal Article
FASEB Journal, ISSN 0892-6638, 2014, Volume 28, Issue 8, pp. 3325 - 3328
Dysfunctions in Wnt signaling increase beta-catenin stability and are associated with cancers, including colorectal cancer. In addition, beta-catenin... 
Wnt signaling | ETD-MS/MS | Cancer | LACTIC-ACID | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RISK | INCIDENT COLORECTAL-CANCER | CELL-GROWTH | TUMORS | CELL BIOLOGY | PATHWAY | BIOLOGY | cancer | GLCNAC GLYCOSYLATION | MUTATIONS | CARBOHYDRATE-METABOLISM | Intestinal Mucosa - metabolism | Phosphorylation | Threonine - chemistry | alpha Catenin - metabolism | Adenocarcinoma - etiology | Humans | Hyperglycemia - complications | Molecular Sequence Data | Male | Dietary Carbohydrates - metabolism | Neoplasm Proteins - metabolism | Adherens Junctions - metabolism | Acetylglucosamine - metabolism | Colorectal Neoplasms - etiology | Adenocarcinoma - metabolism | MCF-7 Cells | Proteolysis | HEK293 Cells | beta Catenin - chemistry | Protein Stability | Dietary Carbohydrates - toxicity | Colorectal Neoplasms - metabolism | Wnt Signaling Pathway | Amino Acid Sequence | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Enzyme Inhibitors - pharmacology | Neoplasm Proteins - chemistry | beta-N-Acetylhexosaminidases - antagonists & inhibitors | Glycosylation | beta Catenin - metabolism | Colon - metabolism | Adherens Junctions - pathology | Protein Interaction Mapping | Hyperglycemia - metabolism | Animals | beta-N-Acetylhexosaminidases - physiology | Glucose - metabolism | N-Acetylglucosaminyltransferases - antagonists & inhibitors | N-Acetylglucosaminyltransferases - physiology | Mice | Protein Processing, Post-Translational | Index Medicus | Research Communications | ETD-MS
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 09/2010, Volume 12, Issue 9, pp. 876 - 885
Keratins 8 and 18 (K8 and K18) are heteropolymeric intermediate filament phosphoglycoproteins of simple-type epithelia. Mutations in K8 and K18 predispose the... 
CELLS | SIGNALING PATHWAYS | MITOTIC ARREST | KINASE-B | O-GLCNAC MODIFICATION | LIVER-DISEASE | NUCLEOCYTOPLASMIC PROTEINS | LINKED N-ACETYLGLUCOSAMINE | INTERMEDIATE-FILAMENTS | TRANSGENIC MICE | CELL BIOLOGY | Liver - pathology | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Apoptosis - genetics | Glycosylation - drug effects | Pancreas - injuries | Phosphorylation - genetics | Keratins - metabolism | Liver - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Antibodies, Monoclonal - immunology | Protein-Serine-Threonine Kinases - metabolism | Chemical and Drug Induced Liver Injury - prevention & control | Antibodies, Monoclonal - pharmacology | Liver - metabolism | Enzyme Inhibitors - pharmacology | Pancreas - pathology | Acetylglucosamine - pharmacology | Mice, Transgenic | Pancreas - metabolism | Mice, Knockout | Phenylcarbamates - pharmacology | fas Receptor - agonists | Chemical and Drug Induced Liver Injury - metabolism | Models, Biological | Cytoskeleton - metabolism | Cytosol - metabolism | Mice | Keratins - genetics | Protein Binding - physiology | Cytosol - drug effects | Hepatocytes - metabolism | Proto-Oncogene Proteins c-akt - genetics | fas Receptor - immunology | Acetylglucosamine - analogs & derivatives | Chemical and Drug Induced Liver Injury - pathology | Hepatocytes - drug effects | Keratins - chemistry | Pancreas - drug effects | beta-N-Acetylhexosaminidases - antagonists & inhibitors | Chemical and Drug Induced Liver Injury - genetics | Glycogen Synthase Kinase 3 - metabolism | Mice, Inbred Strains | Streptozocin - pharmacology | Keratin-18 - genetics | HSP70 Heat-Shock Proteins - metabolism | Protein Kinase C-delta - metabolism | Animals | Keratin-8 - metabolism | Keratin-18 - metabolism | Oximes - pharmacology | Amino Acid Substitution - genetics | Keratin | Liver diseases | Gene mutations | Physiological aspects | Glycosylation | Genetic aspects | Research | Health aspects | Risk factors | Index Medicus
Journal Article
Neurotoxicity Research, ISSN 1029-8428, 7/2016, Volume 30, Issue 1, pp. 53 - 66
Complement-associated factors are implicated in pathogen presentation, neurodegeneration, and microglia resolution of tissue injury. To characterize complement... 
Neurochemistry | Neurosciences | C1q | Synapse stripping | Neurobiology | Cell Biology | M1/M2 polarization | Microglia | Neurology | Biomedicine | Tumor necrosis factor | Interleukin 1 | Pharmacology/Toxicology | GRANULE NEURONS | ALZHEIMERS-DISEASE | BRAIN MICROGLIA | NEUROSCIENCES | MESSENGER-RNA | APOPTOTIC CELLS | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | RAT-BRAIN | SYNAPTIC TERMINALS | CEREBRAL-ISCHEMIA | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Apoptosis - drug effects | Arginase - metabolism | Male | Synapses - pathology | Nerve Degeneration - metabolism | Complement System Proteins - metabolism | Lectins - metabolism | C-Reactive Protein - metabolism | Synapses - metabolism | Dentate Gyrus - drug effects | Interleukin-1beta - metabolism | Inflammation Mediators - metabolism | Interleukin-10 - metabolism | Trimethyltin Compounds - toxicity | Interleukin-6 - metabolism | Synaptophysin - metabolism | Synapses - drug effects | Microglia - drug effects | beta-N-Acetylhexosaminidases - metabolism | Nerve Tissue Proteins - metabolism | Mossy Fibers, Hippocampal - metabolism | Animals | Mossy Fibers, Hippocampal - pathology | Mice | Transforming Growth Factor beta - metabolism | Paired Box Transcription Factors - metabolism | Nitric Oxide Synthase Type II - metabolism | Messenger RNA | Interleukins | Neurons | Environmental health | Index Medicus
Journal Article
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 01/2014, Volume 306, Issue 1, pp. L23 - L34
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 10/2014, Volume 229, Issue 10, pp. 1557 - 1568
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2012, Volume 287, Issue 19, pp. 15395 - 15408
Cellular O-linked N-acetylglucosamine (O-GlcNAc) levels are modulated by two enzymes: uridine diphosphate-N-acetyl-D-glucosamine: polypeptidyltransferase (OGT)... 
STRUCTURAL INSIGHTS | NUCLEAR | GLCNACYLATION | MECHANISM | TETRATRICOPEPTIDE REPEATS | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CYTOSOLIC PROTEINS | GLYCOSYLATION | NUCLEOCYTOPLASMIC PROTEINS | BETA | Phosphorylation | Membrane Glycoproteins - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - metabolism | Humans | N-Acetylglucosaminyltransferases - genetics | Substrate Specificity | tau Proteins - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - genetics | Acetylglucosamine - metabolism | Nuclear Pore Complex Proteins - genetics | tau Proteins - genetics | Mass Spectrometry | Protein-Arginine N-Methyltransferases - genetics | beta-N-Acetylhexosaminidases - genetics | Recombinant Proteins - metabolism | Nuclear Pore Complex Proteins - metabolism | beta-N-Acetylhexosaminidases - metabolism | Serine - genetics | Glycosylation | Recombinant Proteins - genetics | Serine - metabolism | Blotting, Western | N-Acetylglucosaminyltransferases - metabolism | Membrane Glycoproteins - genetics | Protein-Arginine N-Methyltransferases - metabolism | Proteins - genetics | Proteins - metabolism | Adaptor Proteins, Signal Transducing - genetics | Protein Processing, Post-Translational | Kinetics | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Index Medicus | Glycoside Hydrolases | Carbohydrate Glycoconjugate | Glycosyltransferases | Glycobiology and Extracellular Matrices | Carbohydrate | Glycoconjugate | Carbohydrate Biosynthesis | O-GlcNAc
Journal Article
Brain, ISSN 0006-8950, 05/2011, Volume 134, Issue 5, pp. 1293 - 1314
Amyotrophic lateral sclerosis is a relentless and devastating adult-onset neurodegenerative disease with no known cure. In mice with amyotrophic lateral... 
IL-4 | FoxP3 | regulatory T lymphocytes | inflammation | microglia | MOTOR-NEURONS | WILD-TYPE MICROGLIA | SPINAL-CORD | ANTIINFLAMMATORY CYTOKINE | NEUROSCIENCES | CLINICAL NEUROLOGY | INHERITED ALS | IMMUNE-SYSTEM | MOUSE MODEL | ANIMAL-MODEL | ALTERNATIVE ACTIVATION | PARKINSONS-DISEASE | T-Lymphocytes, Regulatory - metabolism | Superoxide Dismutase - genetics | Toll-Like Receptor 2 - genetics | Age Factors | Amyotrophic Lateral Sclerosis - physiopathology | Humans | Middle Aged | Male | RNA, Messenger - metabolism | DNA-Binding Proteins - deficiency | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Lectins - metabolism | Flow Cytometry | Forkhead Transcription Factors - metabolism | Spinal Cord - pathology | Adult | Female | beta-N-Acetylhexosaminidases - genetics | Receptors, Chemokine - genetics | Cytokines - genetics | Disease Models, Animal | Cytokines - metabolism | Gene Expression Regulation - genetics | beta-N-Acetylhexosaminidases - metabolism | Amyotrophic Lateral Sclerosis - genetics | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Cells, Cultured | Gene Expression Regulation - physiology | Mice, Transgenic | Toll-Like Receptor 2 - metabolism | Amyotrophic Lateral Sclerosis - blood | Coculture Techniques - methods | Mutation - genetics | Disease Progression | Microglia | Amyotrophic Lateral Sclerosis - pathology | Animals | Analysis of Variance | Interleukin-2 Receptor alpha Subunit - metabolism | Lectins - genetics | Aged | Mice | CX3C Chemokine Receptor 1 | CD4 Antigens - metabolism | Index Medicus | Abridged Index Medicus | Amyotrophic lateral sclerosis | Original
Journal Article