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European Journal of Heart Failure, ISSN 1388-9842, 04/2017, Volume 19, Issue 4, pp. 479 - 489
Hereditary haemoglobinopathies, mainly beta‐thalassemia and sickle cell disease, constitute the most common monogenic disorders in humans, and although once... 
Heart failure | Iron overload | Thalassaemia | Sickle cell disease | Cardiomyopathy | Pulmonary hypertension | VENTRICULAR DIASTOLIC FUNCTION | NATRIURETIC PEPTIDE LEVELS | CARDIAC & CARDIOVASCULAR SYSTEMS | PULMONARY-HYPERTENSION | T2-ASTERISK MAGNETIC-RESONANCE | MYOCARDIAL IRON | BETA-THALASSEMIA MAJOR | IRON OVERLOAD CARDIOMYOPATHY | SICKLE-CELL-DISEASE | NT-PROBNP LEVELS | COMBINED CHELATION-THERAPY | Anemia, Sickle Cell - physiopathology | Vascular Diseases - etiology | Humans | Cardiac Output | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | beta-Thalassemia - complications | Iron Overload - physiopathology | Transfusion Reaction - etiology | Iron Overload - etiology | Blood Transfusion | Myocardial Ischemia - physiopathology | Ventricular Dysfunction, Right - physiopathology | Heart Failure - etiology | Heart Valve Diseases - physiopathology | beta-Thalassemia - therapy | Anemia, Sickle Cell - complications | beta-Thalassemia - physiopathology | Ventricular Dysfunction, Left - etiology | Vascular Diseases - physiopathology | Heart Valve Diseases - etiology | Ventricular Dysfunction, Left - physiopathology | Hemoglobinopathies - therapy | Anemia, Sickle Cell - therapy | Hemoglobinopathies - complications | Transfusion Reaction - physiopathology | Myocardial Ischemia - etiology | Ventricular Dysfunction, Right - etiology | Hemoglobinopathies - physiopathology | Hypertension, Pulmonary - etiology
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2017, Volume 12, Issue 5, p. e0178353
Painful vaso-occlusive crisis (VOC), a complication of sickle cell disease (SCD), occurs when sickled red blood cells obstruct flow in the microvasculature. We... 
SYSTEM | CONSTRICTION | DEEP | ENDOTHELIAL DYSFUNCTION | MULTIDISCIPLINARY SCIENCES | SYMPATHETIC-NERVE ACTIVITY | CLINICAL SEVERITY | NITRIC-OXIDE | ANEMIA | Microvessels - physiopathology | Sickle Cell Trait - physiopathology | Biophysical Phenomena | Anemia, Sickle Cell - physiopathology | Humans | Middle Aged | beta-Thalassemia - physiopathology | Male | Autonomic Nervous System - physiopathology | Case-Control Studies | Young Adult | Vasoconstriction - physiology | Models, Biological | Pain - physiopathology | Adolescent | Adult | Female | Blood Pressure - physiology | Anemia, Sickle Cell - blood | Pain - blood | Complications and side effects | Sickle cell anemia | Vasoconstriction | Physiological aspects | Genetic aspects | Research | Biological markers | Pediatrics | Regulations | Baroreceptors | Arterioles | Nervous system | Blood | Risk factors | Autonomic nervous system | Hemoglobin | Outflow | Blood pressure | Anemia | Mortality | Blood vessels | Rats | Bioavailability | Diseases | Children & youth | Hospitals | Nitric oxide | Skin | Volatile organic compounds--VOCs | Blood diseases | Kidney transplantation | Transplants & implants | Electrophysiology | Oncology | Stimulation | Transplantation | Antagonists | Engineering | Biomedical materials | Control | Pain | Arginine | Rodents | Constrictions | Children | Capillaries | Cardiology | Sickle cell disease | Biomedical engineering | Reflexes | Hypertension | Stroke | Pain perception | Kidneys | Wood | Hematology | Blood cells | Markers | Polymerization | Pharmacology | Biophysics | Sympathetic nervous system | Blood flow | Flow | Medicine | Heart rate | Respiration | Cancer | VOCs | Volatile organic compounds
Journal Article
Haematologica, ISSN 0390-6078, 05/2007, Volume 92, Issue 5, pp. 658 - 665
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 03/2013, Volume 127, Issue 12, pp. 1317 - 1329
Journal Article
The American Journal of Medicine, ISSN 0002-9343, 2005, Volume 118, Issue 9, pp. 957 - 967
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, pp. e0172147 - e0172147
The risk and clinical significance of cardiac iron overload due to chronic transfusion varies with the underlying disease. Cardiac iron overload shortens the... 
MORTALITY | SURVIVAL | RESONANCE | MORBIDITY | CHELATION-THERAPY | DEPENDENT PATIENTS | MULTIDISCIPLINARY SCIENCES | DISEASE | ACCUMULATION | BETA-THALASSEMIA | CHILDREN | Heart - physiopathology | Thalassemia - blood | Myelodysplastic Syndromes - physiopathology | Anemia, Sickle Cell - physiopathology | Humans | Middle Aged | Male | Myelodysplastic Syndromes - blood | Iron Overload - physiopathology | Iron Overload - blood | Blood Transfusion | Iron Chelating Agents | Myocardium - metabolism | Adolescent | Adult | Female | Anemia, Sickle Cell - blood | Thalassemia - physiopathology | Child | Iron - blood | Sickle cell anemia | Cardiac patients | Analysis | Thalassemia | Iron | Research | Health aspects | Myelodysplastic syndromes | Blood transfusions | Heart | Erythropoiesis | Pediatrics | Transferrin | Nuclear magnetic resonance--NMR | Transfusion | Laboratories | Liver | Ferritin | Disorders | Macrophages | Blood | Myelodysplastic syndrome | Red blood cells | Chelation | Hemoglobin | Sickle cell disease | Heart diseases | Age | Life expectancy | Hematology | Anemia | Research & development--R&D | Magnetic resonance | Inflammation | Patients | Coronary artery disease | Chromatography | Hospitals | Life span | Scientific imaging | Hepcidin | Mass spectrometry | Index Medicus | Research & development | Nuclear magnetic resonance | R&D | NMR
Journal Article