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Journal
Journal of clinical densitometry, ISSN 1094-6950
Journal
Journal of bone and mineral metabolism, ISSN 1435-5604, 1988
Journal
Clinical reviews in bone and mineral metabolism, ISSN 1534-8644, 2002
Journal
International journal of osteoarchaeology, ISSN 1047-482X, 1991
Journal
Metabolic bone disease & related research, ISSN 0221-8747, 1984
Journal
Biology of blood and marrow transplantation, ISSN 1083-8791, 1995
Journal
Bone marrow transplantation, ISSN 0268-3369, 1986
Journal
Bone and mineral research, ISSN 0168-051X, 1983
Journal
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0175329
Teriparatide [human parathyroid hormone (1-34)], which exerts an anabolic effect on bone, is used for the treatment of osteoporosis in patients who are at a... 
HUMAN PARATHYROID HORMONE(1-34) | MARROW FIBROSIS | OVARIECTOMIZED CYNOMOLGUS MONKEYS | VERTEBRAL FRACTURES | THINNER CORTICES | DISTAL RADIUS | MULTIDISCIPLINARY SCIENCES | TURNOVER MARKERS | POSTMENOPAUSAL WOMEN | FIBROUS DYSPLASIA | MECHANICAL-PROPERTIES | Osteogenesis - physiology | X-Ray Microtomography | Bone and Bones - drug effects | Dose-Response Relationship, Drug | Collagen - drug effects | Bone and Bones - diagnostic imaging | Bone and Bones - metabolism | Biomechanical Phenomena - drug effects | Bone Resorption - metabolism | Female | Models, Animal | Bone Resorption - chemically induced | Porosity - drug effects | Imaging, Three-Dimensional | Bone and Bones - cytology | Rabbits | Bone Density Conservation Agents - adverse effects | Drug Administration Schedule | Teriparatide - administration & dosage | Teriparatide - blood | Osteogenesis - drug effects | Bone Density Conservation Agents - administration & dosage | Microscopy, Interference | Bone Density Conservation Agents - blood | Collagen - metabolism | Animals | Bone Resorption - pathology | Teriparatide - adverse effects | Microscopy, Fluorescence | Adaptations | Animal models | Spine | Bone (trabecular) | Bisphosphonates | Clinical outcomes | Bone resorption | Proteins | Osteoporosis | Alendronic acid | Bone growth | Computed tomography | Fibrous dysplasia | Bone marrow | Bone dysplasia | Tunneling | Skeleton | Bone density | Bone loss | Bone (cortical) | Drug therapy | Bone strength | Drug dosages | Age | Bone remodelling | Rats | Risk reduction | Endocrine therapy | Pharmacology | Frost | Bone turnover | Metabolism | Bone mass | Fractures | Osteodystrophy | Mice | Bone mineral density | Mutation | Porosity | Pharmacokinetics | Risk management | Kidney transplantation | Bone (cancellous)
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 3, p. e92368
The gut microbiota (GM) modulates the hosts metabolism and immune system. Probiotic bacteria are defined as live microorganisms which when administered in... 
C-REACTIVE PROTEIN | TRABECULAR BONE | WOMEN | MINERAL DENSITY | IMMUNE-SYSTEM | MULTIDISCIPLINARY SCIENCES | ESTROGEN-RECEPTOR-ALPHA | OSTEOPROTEGERIN | GUT MICROBIOTA | TNF-ALPHA | T-CELLS | Bone and Bones - pathology | Ovariectomy - adverse effects | T-Lymphocytes, Regulatory - metabolism | RANK Ligand - metabolism | Body Weight - drug effects | Bone and Bones - drug effects | Lactobacillus - drug effects | Bone Marrow - metabolism | Bone and Bones - metabolism | Inflammation Mediators - metabolism | Female | Minerals - metabolism | Bone Marrow - drug effects | Bone Resorption - etiology | Bone Resorption - blood | Cytokines - metabolism | Mice, Inbred C57BL | Bone Resorption - drug therapy | Biomarkers - blood | Probiotics - pharmacology | Lactobacillus - metabolism | Bone Resorption - prevention & control | Organ Size - drug effects | T-Lymphocytes, Regulatory - drug effects | Animals | Bone Marrow - pathology | Probiotics - therapeutic use | Mice | Osteoprotegerin - metabolism | Transforming Growth Factor beta - metabolism | Probiotics | Ovariectomy | Microbiota (Symbiotic organisms) | Physiological aspects | Bones | T cells | Density | Drinking water | Calcium | Lymphocytes T | Genomes | Arthritis | Immune status | Osteoprotegerin | Bone resorption | Osteoporosis | Microorganisms | Biomedical materials | Microbiota | Lymphocytes | Bone marrow | Bacteria | Tumor necrosis factor-TNF | Biocompatibility | Bone density | Bone loss | Bone (cortical) | Bone composition | Food | Immune system | Calcium (urinary) | Excretion | Gnotobiotics | Cytokines | Nutrition | Immunoregulation | Rheumatology | Inflammation | Tumor necrosis factor-α | IL-1β | Metabolism | Gene expression | Bone mineral content | Medicine | Serum levels | Osteoclastogenesis | Bone mass | Antibiotics | Womens health | Ligands | Endokrinologi och diabetes | Endocrinology and Diabetes
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2016, Volume 11, Issue 5, p. e0154680
The receptor-activator of nuclear kappaB ligand (RANKL) signaling pathway plays an important role in the regulation of bone growth and mediates the formation... 
GIANT-CELL TUMOR | DENOSUMAB | MULTIDISCIPLINARY SCIENCES | Osteosarcoma - drug therapy | RANK Ligand - metabolism | Humans | Male | Antineoplastic Agents - therapeutic use | Fibrous Dysplasia of Bone - drug therapy | RNA, Messenger - metabolism | Bone Neoplasms - pathology | Giant Cell Tumor of Bone - pathology | Chondrosarcoma - genetics | Chondrosarcoma - pathology | Bone Cysts, Aneurysmal - metabolism | Bone Neoplasms - genetics | Fibrous Dysplasia of Bone - genetics | Chondroma - drug therapy | Osteoclasts - pathology | Bone Cysts, Aneurysmal - pathology | Multiple Myeloma - genetics | Osteosarcoma - pathology | Bone Cysts, Aneurysmal - drug therapy | Chondroma - genetics | Gene Expression Regulation, Neoplastic | Chondroma - metabolism | Chondroma - pathology | Bone Neoplasms - metabolism | Denosumab - therapeutic use | Multiple Myeloma - drug therapy | Giant Cell Tumor of Bone - drug therapy | RNA, Messenger - antagonists & inhibitors | Female | Bone Neoplasms - drug therapy | Giant Cell Tumor of Bone - genetics | Osteosarcoma - metabolism | Fibrous Dysplasia of Bone - pathology | RNA, Messenger - genetics | Multiple Myeloma - metabolism | Organ Specificity | Chondrosarcoma - drug therapy | Fibrous Dysplasia of Bone - metabolism | Osteoclasts - metabolism | Bone Resorption - prevention & control | RANK Ligand - genetics | Multiple Myeloma - pathology | Giant Cell Tumor of Bone - metabolism | RANK Ligand - antagonists & inhibitors | Chondrosarcoma - metabolism | Osteosarcoma - genetics | Osteoclasts - drug effects | Bone Cysts, Aneurysmal - genetics | Bone resorption | Bones | Dosage and administration | Denosumab | Growth | Analysis | Immunohistochemistry | Chondrosarcoma | Multiple myeloma | Aneurysm | Bone tumors | Metastasis | Bone surgery | Metastases | Osteoprotegerin | Ewings sarcoma | Signal transduction | Bone growth | Biomedical materials | Osteosarcoma | Fibrous dysplasia | Bone dysplasia | Biocompatibility | Lesions | TRANCE protein | University graduates | Statistical analysis | Bone lesions | Gene expression | Osteoclastogenesis | Signaling | Bone cancer | Hospitals | Biopsy | Stromal cells | Orthopedics | Monoclonal antibodies | Ligands | Osteoclasts | Prostate cancer | Tumors
Journal Article
Bone, ISSN 8756-3282, 2013, Volume 54, Issue 1, pp. 83 - 91
Abstract Diastrophic dysplasia (DTD) is a chondrodysplasia caused by mutations in the SLC26A2 gene, leading to reduced intracellular sulfate pool in... 
Orthopedics | Proteoglycan | Bone histomorphometry | Osteoclasts | Animal models | Diastrophic dysplasia | Osteoblasts | COLLAGEN | TRANSPORTER | MOUSE | CARTILAGE | MURINE MODEL | CHONDRODYSPLASIAS | MUTATION | ENDOCRINOLOGY & METABOLISM | LEADS | MICE | PLATE | Bone and Bones - pathology | Bone Development - physiology | Humans | Male | Bone Remodeling - physiology | Parathyroid Hormone - blood | Dwarfism - blood | Calcium - blood | Dwarfism - metabolism | Mice, Mutant Strains | Bone and Bones - metabolism | Cell Differentiation | Dwarfism - physiopathology | Sulfur - metabolism | Bone Resorption - physiopathology | Osteoclasts - pathology | Bone Density | Bone Resorption - blood | Collagen - ultrastructure | Mice, Inbred C57BL | Organ Size | Proteoglycans - metabolism | Bone Resorption - complications | Osteoclasts - metabolism | Aging - urine | Aging - pathology | Aging - blood | Collagen - metabolism | Dwarfism - complications | Bone and Bones - physiopathology | Animals | Bone Resorption - pathology | Bone and Bones - ultrastructure | Mice | Phosphates | Parathyroid hormone | Analysis | Collagen | Bone densitometry | Bones | Macrophage colony stimulating factor | Sulfates | Density | Proteoglycans | Bone remodelling | Fibrils | Bone turnover | Chondrodystrophy | Cartilage | Bone growth | Ionizing radiation | Bone mass | Cross-linking | Chondrocytes | Bone dysplasia | Fibroblasts | Skeleton | Bone matrix | BMC, bone mineral content | BrdU, 5-bromo-2′-deoxyuridine | TRAP, tartrate resistant acid phosphatase | DEXA, dual energy X-ray absorptiometry | DLS | PTH, parathyroid hormone | BER, bone elongation rate | DTDST, diastrophic dysplasia sulfate transporter | P, postnatal day | PBS, phosphate buffer saline | BFR, bone formation rate | BMD, bone mineral density | DPD, deoxypyridinoline | RANK-L, receptor activator of nuclear factor kappa-B ligand | MAR, mineral apposition rate | M-CSF, macrophage colony-stimulating factor | BS, double labeled surface per bone surface | SLC26A2, solute carrier family 26 member 2 | Original Full Length | CTX, C-terminal telopeptides of type I collagen | DTD, diastrophic dysplasia | FCS, fetal calf serum
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, p. e0171276
Osteoporosis is a metabolic skeletal disease characterized by an imbalance between osteoclast-mediated bone resorption and osteoblast-mediated bone formation.... 
MINERAL DENSITY | THERAPY | ADIPONECTIN | CATHEPSIN-K | MULTIDISCIPLINARY SCIENCES | ESTROGEN | FRACTURES | BODY-COMPOSITION | DIFFERENTIATION | POSTMENOPAUSAL WOMEN | OSTEOPOROSIS | Bone and Bones - pathology | Ovariectomy - adverse effects | Raloxifene Hydrochloride - pharmacology | Osteoporosis - drug therapy | High-Energy Shock Waves | PPAR gamma - metabolism | Osteoporosis - etiology | Bone and Bones - drug effects | Bone Resorption - genetics | Adiponectin - genetics | Bone and Bones - metabolism | Bone Resorption - metabolism | Adiponectin - metabolism | Female | Transcription, Genetic | Bone and Bones - radiation effects | Osteogenesis - genetics | Disease Models, Animal | Bone Resorption - etiology | Osteogenesis - drug effects | Rats | Osteogenesis - radiation effects | Bone Density Conservation Agents - pharmacology | Bone Resorption - prevention & control | Animals | Osteoporosis - pathology | Biomarkers | Shock waves | Usage | Growth | Bones | Raloxifene | Dosage and administration | Research | Drug therapy | Health aspects | Osteopenia | Alkaline phosphatase | Neurosciences | Femur | Mesenchyme | Spine | Estrogen receptors | Veterinary medicine | Bone surgery | Adiponectin | Osteoprotegerin | Bone resorption | Osteoporosis | Biomedical materials | Bone growth | Rodents | Surgery | Computer architecture | Bone marrow | Biocompatibility | Bone loss | Ultrasound | Public health | Vertebrae | Cathepsin K | Ovariectomy | Bone remodelling | Metabolism | Gene expression | Osteoblastogenesis | Tumor necrosis factor | Parathyroid hormone | Womens health | Pharmacy | Weight reduction | Stem cells | Bone mineral density | Clinical medicine | Synergistic effect | Osteogenesis
Journal Article