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by Zhang, T and Gao, G and Chang, F
European Review for Medical and Pharmacological Sciences, ISSN 1128-3602, 2019, Volume 23, Issue 1, pp. 44 - 51
OBJECTIVE: The aim of this research is to explore the possible role of miR-152 in spinal cord injury and its underlying mechanism. MATERIALS AND METHODS: After... 
MiR-152 | Inflammation | Spinal cord injury | C-jun | REGENERATION | DOWN-REGULATION | PHARMACOLOGY & PHARMACY | MICRORNAS | NF-KAPPA-B | BRAIN
Journal Article
Journal Article
Journal Article
Science, ISSN 0036-8075, 1/1997, Volume 275, Issue 5296, pp. 90 - 94
Journal Article
Cell Proliferation, ISSN 0960-7722, 12/2017, Volume 50, Issue 6, pp. e12369 - n/a
Objectives Mesenchymal stem cell (MSC)‐mediated periodontal tissue regeneration is considered to be a promising method for periodontitis treatment. The... 
SIGNALING PATHWAY | NICHE | TISSUE REGENERATION | NECROSIS-FACTOR-ALPHA | EXTRACELLULAR-MATRIX | PROLIFERATION | MAPK | FIBRILLOGENESIS | EXPRESSION | FIBROBLASTS | CELL BIOLOGY | Tumor Necrosis Factor-alpha - metabolism | Mesenchymal Stromal Cells - drug effects | MAP Kinase Signaling System - physiology | Humans | Osteogenesis - drug effects | Cells, Cultured | JNK Mitogen-Activated Protein Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Cell Adhesion Molecules - metabolism | Periodontal Ligament - cytology | Tumor Necrosis Factor-alpha - pharmacology | Inflammation - metabolism | Mesenchymal Stromal Cells - cytology | MAP Kinase Signaling System - drug effects | Cell Differentiation - drug effects | Cell Movement | Genetic research | Phosphatases | Mitogens | Protein kinases | Analysis | Stem cells | Alkaline phosphatase | Transcription factors | Leukocyte migration | Mesenchyme | AKT protein | Kinases | Alizarin | Signal transduction | Biomedical materials | Periodontal ligament | Mineralization | Biocompatibility | Inhibition | Recombinant | Cbfa-1 protein | Tissue engineering | c-Jun protein | MAP kinase | Inflammation | Gene expression | Studies | Regeneration | Signaling | Periodontitis | Tumor necrosis factor | Skin & tissue grafts | Differentiation | Ligaments | Cell migration | Osteogenesis
Journal Article
Journal Article
Journal Article
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 11/2012, Volume 264, Issue 3, pp. 387 - 394
Acetaminophen (APAP) overdose is the most common cause of acute liver failure in the West. In mice, APAP hepatotoxicity can be rapidly induced with a single... 
Protein adducts | Mitochondria | Acetaminophen | Hepatotoxicity | Oxidant stress | c-Jun‐N-terminal kinase | C-Jun-N-terminal kinase | PEROXYNITRITE | COVALENT BINDING | HEPATIC-NECROSIS | MOUSE HEPATOCYTES | PERMEABILITY TRANSITION | CELL-DEATH | DNA FRAGMENTATION | GLUTATHIONE | PHARMACOLOGY & PHARMACY | TOXICOLOGY | c-Jun-N-terminal kinase | INDUCED HEPATOTOXICITY | Rats, Inbred F344 | Mitochondrial Proteins - genetics | MAP Kinase Kinase 4 - metabolism | Alanine Transaminase - genetics | Analgesics, Non-Narcotic - toxicity | Time Factors | Mitochondrial Proteins - metabolism | Glutamate Dehydrogenase - metabolism | Disease Models, Animal | Acetaminophen - toxicity | Mice, Inbred C57BL | Gene Expression Regulation | Antipyretics - toxicity | Rats | Mitochondria - metabolism | Mitochondria - drug effects | Glutamate Dehydrogenase - genetics | Rats, Sprague-Dawley | Animals | Chemical and Drug Induced Liver Injury - metabolism | MAP Kinase Kinase 4 - genetics | Protein Binding | Mice | Alanine Transaminase - metabolism | Oxidative Stress - drug effects | Oxidative stress | Liver diseases | Liver | Physiological aspects | Mitochondrial DNA | Comparative analysis | Index Medicus | Translocation | Oxidative metabolism | Toxicity | Data processing | Metabolism | Protein turnover | Necrosis | Proteins | c-Jun amino-terminal kinase | Models | Overdose | Intoxication | Injuries | OXIDATION | TRANSLOCATION | INJURIES | MITOCHONDRIA | RATS | TOXICITY | 60 APPLIED LIFE SCIENCES | NECROSIS | METABOLISM | OXIDIZERS | LIVER | MICE | PREFERRED SPECIES | protein adducts | oxidant stress | c-jun-N-terminal kinase | hepatotoxicity | mitochondria
Journal Article