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1. Full Text C‐Met as a potential target for the treatment of gastrointestinal cancer: Current status and future perspectives
by Bahrami, Afsane and Shahidsales, Soodabeh and Khazaei, Majid and Ghayour‐Mobarhan, Majid and Maftouh, Mina and Hassanian, Seyed Mahdi and Avan, Amir
Journal of cellular physiology, ISSN 0021-9541, 10/2017, Volume 232, Issue 10, pp. 2657 - 2673
upper gastrointestinal cancers | resistance to targeted agents | c‐Met inhibitors | c‐Met/HGF pathway | c-Met inhibitors | c-Met/HGF pathway | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Proto-Oncogene Proteins c-met - metabolism | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Humans | Drug Resistance, Neoplasm | Antineoplastic Agents - therapeutic use | Molecular Targeted Therapy | Hepatocyte Growth Factor - metabolism | Animals | Digestive System Neoplasms - drug therapy | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | Digestive System Neoplasms - pathology | Digestive System Neoplasms - enzymology | Care and treatment | Drug therapy | Gastrointestinal cancer | Drug approval | Cancer | Cell proliferation | Medical research | Therapy | Prognosis | Effectiveness | Clinical trials | Activation | Selectivity | Metastasis | Patients | Metastases | Anticancer properties | c-Met protein | Signal transduction | Signaling | Cell growth | Inhibitors | Pathways | Lungs | Reagents | Antitumor activity | Determinants | Aberration | Index Medicus
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2. Full Text MET is required for the recruitment of anti-tumoural neutrophils
by Finisguerra, Veronica and Di Conza, Giusy and Di Matteo, Mario and Serneels, Jens and Costa, Sandra and Thompson, A. A. Roger and Wauters, Els and Walmsley, Sarah and Prenen, Hans and Granot, Zvi and Casazza, Andrea and Mazzone, Massimiliano
Nature (London), ISSN 0028-0836, 06/2015, Volume 522, Issue 7556, pp. 349 - 353
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Neoplasms - metabolism | Proto-Oncogene Proteins c-met - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Humans | Middle Aged | Male | Neoplasm Metastasis | Transendothelial and Transepithelial Migration | Antineoplastic Agents - adverse effects | Gene Deletion | Neutrophils - secretion | Female | Hepatocyte Growth Factor | Antineoplastic Agents - pharmacology | Disease Models, Animal | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Neutrophils - drug effects | Neutrophils - immunology | Solubility | Inflammation - immunology | Disease Progression | Neoplasms - drug therapy | Proto-Oncogene Proteins c-met - genetics | Xenograft Model Antitumor Assays | Nitric Oxide - secretion | Animals | Neoplasms - immunology | Proto-Oncogene Proteins c-met - deficiency | Aged | Mice | Neoplasms - pathology | Physiological aspects | Health aspects | Cancer cells | Neutrophils | Hematopoietic growth factors | Index Medicus
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3. Full Text Reactive Neutrophil Responses Dependent on the Receptor Tyrosine Kinase c-MET Limit Cancer Immunotherapy
by Glodde, Nicole and Bald, Tobias and van den Boorn-Konijnenberg, Debby and Nakamura, Kyohei and O’Donnell, Jake S and Szczepanski, Sabrina and Brandes, Maria and Eickhoff, Sarah and Das, Indrajit and Shridhar, Naveen and Hinze, Daniel and Rogava, Meri and van der Sluis, Tetje C and Ruotsalainen, Janne J and Gaffal, Evelyn and Landsberg, Jennifer and Ludwig, Kerstin U and Wilhelm, Christoph and Riek-Burchardt, Monika and Müller, Andreas J and Gebhardt, Christoffer and Scolyer, Richard A and Long, Georgina V and Janzen, Viktor and Teng, Michele W.L and Kastenmüller, Wolfgang and Mazzone, Massimiliano and Smyth, Mark J and Tüting, Thomas and Hölzel, Michael
Immunity (Cambridge, Mass.), ISSN 1074-7613, 10/2017, Volume 47, Issue 4, pp. 789 - 802.e9
HGF | lymph node | cancer immunotherapy | plasticity | melanoma | c-MET | T cells | neutrophils | bone marrow | therapy resistance | Life Sciences & Biomedicine | Immunology | Science & Technology | Proto-Oncogene Proteins c-met - metabolism | Immunotherapy - methods | Interferon-gamma - metabolism | Signal Transduction - immunology | Proto-Oncogene Proteins c-met - immunology | Tumor Microenvironment - genetics | T-Lymphocytes - metabolism | Neoplasms, Experimental - immunology | Neutrophils - metabolism | Cell Proliferation - genetics | Mice, Inbred C57BL | Kaplan-Meier Estimate | Neoplasms, Experimental - therapy | Neutrophils - immunology | Gene Expression Profiling - methods | Gene Expression Regulation, Neoplastic - immunology | Mice, Transgenic | Signal Transduction - genetics | Mice, Knockout | Proto-Oncogene Proteins c-met - genetics | Tumor Microenvironment - immunology | Animals | Interferon-gamma - immunology | Cell Line, Tumor | T-Lymphocytes - immunology | Mice, Inbred BALB C | Neoplasms, Experimental - metabolism | Tyrosine | Care and treatment | Analysis | Immunotherapy | Phenols | Cancer | Animal models | Phosphorylation | Cytotoxicity | Lymphocytes T | Kinases | Lymph nodes | Metastases | Recruitment | c-Met protein | Vitiligo | Lymphocytes | Protein-tyrosine kinase receptors | Inhibition | Protein-tyrosine kinase | Antigens | Lymphatic system | Tumor cells | Neurons | Neutrophils | Melanoma | Leukocytes (neutrophilic) | Inflammation | Gene expression | Drainage | Serum levels | Signaling | Immunosuppression | Immune checkpoint | Infiltration | Microenvironments | Tumors | Index Medicus
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4. Full Text MET signalling: principles and functions in development, organ regeneration and cancer
by Bertotti, Andrea and Trusolino, Livio and Comoglio, Paolo M
Nature reviews. Molecular cell biology, ISSN 1471-0072, 12/2010, Volume 11, Issue 12, pp. 834 - 848
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Proto-Oncogene Proteins c-met - metabolism | Neoplasms - etiology | Humans | Models, Molecular | Animal Structures - physiology | Signal Transduction - genetics | Regeneration - physiology | Proto-Oncogene Proteins c-met - genetics | Proto-Oncogene Proteins c-met - physiology | Animals | Neoplasms - genetics | Models, Biological | Regeneration - genetics | Growth and Development - physiology | Signal Transduction - physiology | Growth and Development - genetics | Proto-Oncogene Proteins c-met - chemistry | Animal Structures - metabolism | Physiological aspects | Development and progression | Cell receptors | Cellular signal transduction | Research | Cancer | Index Medicus
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5. Full Text Thirty years of research on met receptor to move a biomarker from bench to bedside
by Furlan, Alessandro and Kherrouche, Zoulika and Montagne, Rémi and Copin, Marie-Christine and Tulasne, David
Cancer research (Chicago, Ill.), ISSN 0008-5472, 12/2014, Volume 74, Issue 23, pp. 6737 - 6744
Life Sciences & Biomedicine | Oncology | Science & Technology | Biomarkers - metabolism | Neoplasms - metabolism | Proto-Oncogene Proteins c-met - metabolism | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Humans | Antineoplastic Agents - therapeutic use | Neoplasms - drug therapy | Clinical Trials, Phase III as Topic | Animals | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | Ligands | Antineoplastic Agents - pharmacology | Protein Kinase Inhibitors - pharmacology | Index Medicus | Proto-Oncogene Proteins c-met/metabolism | Neoplasms/metabolism | Proto-Oncogene Proteins c-met/antagonists & inhibitors | Antineoplastic Agents/therapeutic use | Neoplasms/drug therapy | Biomarkers/metabolism | Signal Transduction/drug effects | Life Sciences | Antineoplastic Agents/pharmacology | Protein Kinase Inhibitors/therapeutic use | Protein Kinase Inhibitors/pharmacology | Cancer
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6. Full Text Significance of c‐MET overexpression in cytotoxic anticancer drug‐resistant small‐cell lung cancer cells
by Ozasa, Hiroaki and Oguri, Tetsuya and Maeno, Ken and Takakuwa, Osamu and Kunii, Eiji and Yagi, Yoshitaka and Uemura, Takehiro and Kasai, Daishi and Miyazaki, Mikinori and Niimi, Akio
Cancer science, ISSN 1347-9032, 08/2014, Volume 105, Issue 8, pp. 1032 - 1039
lung cancer | drug resistance | c‐MET inhibitor | c‐MET overexpression | gene amplification | C-MET overexpression | Gene amplification | Drug resistance | C-MET inhibitor | Lung cancer | Life Sciences & Biomedicine | Oncology | Science & Technology | Proto-Oncogene Proteins c-met - biosynthesis | Humans | Gene Expression Regulation, Neoplastic | In Situ Hybridization, Fluorescence | Small Cell Lung Carcinoma - genetics | Blotting, Western | Proto-Oncogene Proteins c-met - genetics | Small Cell Lung Carcinoma - metabolism | Drug Resistance, Neoplasm - genetics | Transfection | Cell Line, Tumor | RNA, Small Interfering | Real-Time Polymerase Chain Reaction | Apoptosis | Antimitotic agents | Antineoplastic agents | Cancer cells | Cytotoxicity | Kinases | Cancer therapies | c-Met protein | Proteins | Signal transduction | Epidermal growth factor | Antitumor agents | Protein-tyrosine kinase receptors | MET protein | Gefitinib | Growth factors | Protein-tyrosine kinase | Tyrosine | Immunoglobulins | Small cell lung carcinoma | Hepatocyte growth factor | siRNA | Gene expression | Medical prognosis | Protein expression | Gene loci | Mutation | Solid tumors | Cytotoxic agents | Index Medicus | c-MET inhibitor | Original | c-MET overexpression
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