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Journal of cellular physiology, ISSN 0021-9541, 10/2017, Volume 232, Issue 10, pp. 2657 - 2673
upper gastrointestinal cancers | resistance to targeted agents | c‐Met inhibitors | c‐Met/HGF pathway | c-Met inhibitors | c-Met/HGF pathway | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Proto-Oncogene Proteins c-met - metabolism | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Humans | Drug Resistance, Neoplasm | Antineoplastic Agents - therapeutic use | Molecular Targeted Therapy | Hepatocyte Growth Factor - metabolism | Animals | Digestive System Neoplasms - drug therapy | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | Digestive System Neoplasms - pathology | Digestive System Neoplasms - enzymology | Care and treatment | Drug therapy | Gastrointestinal cancer | Drug approval | Cancer | Cell proliferation | Medical research | Therapy | Prognosis | Effectiveness | Clinical trials | Activation | Selectivity | Metastasis | Patients | Metastases | Anticancer properties | c-Met protein | Signal transduction | Signaling | Cell growth | Inhibitors | Pathways | Lungs | Reagents | Antitumor activity | Determinants | Aberration | Index Medicus
Journal Article
Nature (London), ISSN 0028-0836, 06/2015, Volume 522, Issue 7556, pp. 349 - 353
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Neoplasms - metabolism | Proto-Oncogene Proteins c-met - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Humans | Middle Aged | Male | Neoplasm Metastasis | Transendothelial and Transepithelial Migration | Antineoplastic Agents - adverse effects | Gene Deletion | Neutrophils - secretion | Female | Hepatocyte Growth Factor | Antineoplastic Agents - pharmacology | Disease Models, Animal | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Neutrophils - drug effects | Neutrophils - immunology | Solubility | Inflammation - immunology | Disease Progression | Neoplasms - drug therapy | Proto-Oncogene Proteins c-met - genetics | Xenograft Model Antitumor Assays | Nitric Oxide - secretion | Animals | Neoplasms - immunology | Proto-Oncogene Proteins c-met - deficiency | Aged | Mice | Neoplasms - pathology | Physiological aspects | Health aspects | Cancer cells | Neutrophils | Hematopoietic growth factors | Index Medicus
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 10/2017, Volume 47, Issue 4, pp. 789 - 802.e9
HGF | lymph node | cancer immunotherapy | plasticity | melanoma | c-MET | T cells | neutrophils | bone marrow | therapy resistance | Life Sciences & Biomedicine | Immunology | Science & Technology | Proto-Oncogene Proteins c-met - metabolism | Immunotherapy - methods | Interferon-gamma - metabolism | Signal Transduction - immunology | Proto-Oncogene Proteins c-met - immunology | Tumor Microenvironment - genetics | T-Lymphocytes - metabolism | Neoplasms, Experimental - immunology | Neutrophils - metabolism | Cell Proliferation - genetics | Mice, Inbred C57BL | Kaplan-Meier Estimate | Neoplasms, Experimental - therapy | Neutrophils - immunology | Gene Expression Profiling - methods | Gene Expression Regulation, Neoplastic - immunology | Mice, Transgenic | Signal Transduction - genetics | Mice, Knockout | Proto-Oncogene Proteins c-met - genetics | Tumor Microenvironment - immunology | Animals | Interferon-gamma - immunology | Cell Line, Tumor | T-Lymphocytes - immunology | Mice, Inbred BALB C | Neoplasms, Experimental - metabolism | Tyrosine | Care and treatment | Analysis | Immunotherapy | Phenols | Cancer | Animal models | Phosphorylation | Cytotoxicity | Lymphocytes T | Kinases | Lymph nodes | Metastases | Recruitment | c-Met protein | Vitiligo | Lymphocytes | Protein-tyrosine kinase receptors | Inhibition | Protein-tyrosine kinase | Antigens | Lymphatic system | Tumor cells | Neurons | Neutrophils | Melanoma | Leukocytes (neutrophilic) | Inflammation | Gene expression | Drainage | Serum levels | Signaling | Immunosuppression | Immune checkpoint | Infiltration | Microenvironments | Tumors | Index Medicus
Journal Article
Nature reviews. Molecular cell biology, ISSN 1471-0072, 12/2010, Volume 11, Issue 12, pp. 834 - 848
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Proto-Oncogene Proteins c-met - metabolism | Neoplasms - etiology | Humans | Models, Molecular | Animal Structures - physiology | Signal Transduction - genetics | Regeneration - physiology | Proto-Oncogene Proteins c-met - genetics | Proto-Oncogene Proteins c-met - physiology | Animals | Neoplasms - genetics | Models, Biological | Regeneration - genetics | Growth and Development - physiology | Signal Transduction - physiology | Growth and Development - genetics | Proto-Oncogene Proteins c-met - chemistry | Animal Structures - metabolism | Physiological aspects | Development and progression | Cell receptors | Cellular signal transduction | Research | Cancer | Index Medicus
Journal Article
Cancer research (Chicago, Ill.), ISSN 0008-5472, 12/2014, Volume 74, Issue 23, pp. 6737 - 6744
Life Sciences & Biomedicine | Oncology | Science & Technology | Biomarkers - metabolism | Neoplasms - metabolism | Proto-Oncogene Proteins c-met - metabolism | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Humans | Antineoplastic Agents - therapeutic use | Neoplasms - drug therapy | Clinical Trials, Phase III as Topic | Animals | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | Ligands | Antineoplastic Agents - pharmacology | Protein Kinase Inhibitors - pharmacology | Index Medicus | Proto-Oncogene Proteins c-met/metabolism | Neoplasms/metabolism | Proto-Oncogene Proteins c-met/antagonists & inhibitors | Antineoplastic Agents/therapeutic use | Neoplasms/drug therapy | Biomarkers/metabolism | Signal Transduction/drug effects | Life Sciences | Antineoplastic Agents/pharmacology | Protein Kinase Inhibitors/therapeutic use | Protein Kinase Inhibitors/pharmacology | Cancer
Journal Article
Cancer science, ISSN 1347-9032, 08/2014, Volume 105, Issue 8, pp. 1032 - 1039
lung cancer | drug resistance | c‐MET inhibitor | c‐MET overexpression | gene amplification | C-MET overexpression | Gene amplification | Drug resistance | C-MET inhibitor | Lung cancer | Life Sciences & Biomedicine | Oncology | Science & Technology | Proto-Oncogene Proteins c-met - biosynthesis | Humans | Gene Expression Regulation, Neoplastic | In Situ Hybridization, Fluorescence | Small Cell Lung Carcinoma - genetics | Blotting, Western | Proto-Oncogene Proteins c-met - genetics | Small Cell Lung Carcinoma - metabolism | Drug Resistance, Neoplasm - genetics | Transfection | Cell Line, Tumor | RNA, Small Interfering | Real-Time Polymerase Chain Reaction | Apoptosis | Antimitotic agents | Antineoplastic agents | Cancer cells | Cytotoxicity | Kinases | Cancer therapies | c-Met protein | Proteins | Signal transduction | Epidermal growth factor | Antitumor agents | Protein-tyrosine kinase receptors | MET protein | Gefitinib | Growth factors | Protein-tyrosine kinase | Tyrosine | Immunoglobulins | Small cell lung carcinoma | Hepatocyte growth factor | siRNA | Gene expression | Medical prognosis | Protein expression | Gene loci | Mutation | Solid tumors | Cytotoxic agents | Index Medicus | c-MET inhibitor | Original | c-MET overexpression
Journal Article