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Journal Article
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2017, Volume 47, Issue 4, pp. 789 - 802.e9
Inhibitors of the receptor tyrosine kinase c-MET are currently used in the clinic to target oncogenic signaling in tumor cells. We found that concomitant c-MET... 
HGF | lymph node | cancer immunotherapy | plasticity | melanoma | c-MET | T cells | neutrophils | bone marrow | therapy resistance | MIGRATION | IMMUNE-RESPONSE | LUNG | ACTIVATION | T-CELL RESPONSES | INHIBITION | HEPATOCYTE GROWTH-FACTOR | LYMPH-NODES | IMMUNOLOGY | TUMOR-ASSOCIATED NEUTROPHILS | G-CSF | Proto-Oncogene Proteins c-met - metabolism | Immunotherapy - methods | Interferon-gamma - metabolism | Signal Transduction - immunology | Proto-Oncogene Proteins c-met - immunology | Tumor Microenvironment - genetics | T-Lymphocytes - metabolism | Neoplasms, Experimental - immunology | Neutrophils - metabolism | Cell Proliferation - genetics | Mice, Inbred C57BL | Kaplan-Meier Estimate | Neoplasms, Experimental - therapy | Neutrophils - immunology | Gene Expression Profiling - methods | Gene Expression Regulation, Neoplastic - immunology | Mice, Transgenic | Signal Transduction - genetics | Mice, Knockout | Proto-Oncogene Proteins c-met - genetics | Tumor Microenvironment - immunology | Animals | Interferon-gamma - immunology | Cell Line, Tumor | T-Lymphocytes - immunology | Mice, Inbred BALB C | Neoplasms, Experimental - metabolism | Tyrosine | Care and treatment | Analysis | Immunotherapy | Phenols | Cancer | Animal models | Phosphorylation | Cytotoxicity | Lymphocytes T | Kinases | Lymph nodes | Metastases | Recruitment | c-Met protein | Vitiligo | Lymphocytes | Protein-tyrosine kinase receptors | Inhibition | Protein-tyrosine kinase | Antigens | Lymphatic system | Tumor cells | Neurons | Neutrophils | Melanoma | Leukocytes (neutrophilic) | Inflammation | Gene expression | Drainage | Serum levels | Signaling | Immunosuppression | Immune checkpoint | Infiltration | Microenvironments | Tumors
Journal Article
Cancer science, ISSN 1347-9032, 2014, Volume 105, Issue 8, pp. 1032 - 1039
The c‐MET receptor tyrosine kinase is the receptor for hepatocyte growth factor. Recently, activation of the c‐MET/hepatocyte growth factor signaling pathway... 
lung cancer | drug resistance | c‐MET inhibitor | c‐MET overexpression | gene amplification | C-MET overexpression | Gene amplification | Drug resistance | C-MET inhibitor | Lung cancer | c-MET inhibitor | TYROSINE KINASE | PHOSPHATIDYLINOSITOL 3-KINASE | HEPATOCYTE GROWTH-FACTOR | OVARIAN-CANCER | FACTOR RECEPTOR | GEFITINIB RESISTANCE | c-MET overexpression | CHROMOSOMAL INSTABILITY | ONCOLOGY | COPY NUMBER | THERAPEUTIC INHIBITION | THYMIDYLATE-SYNTHASE | Proto-Oncogene Proteins c-met - biosynthesis | Humans | Gene Expression Regulation, Neoplastic | In Situ Hybridization, Fluorescence | Small Cell Lung Carcinoma - genetics | Blotting, Western | Proto-Oncogene Proteins c-met - genetics | Small Cell Lung Carcinoma - metabolism | Drug Resistance, Neoplasm - genetics | Transfection | Cell Line, Tumor | RNA, Small Interfering | Real-Time Polymerase Chain Reaction | Apoptosis | Antimitotic agents | Antineoplastic agents | Cancer cells | Cytotoxicity | Kinases | Cancer therapies | c-Met protein | Proteins | Signal transduction | Epidermal growth factor | Antitumor agents | Protein-tyrosine kinase receptors | MET protein | Gefitinib | Growth factors | Protein-tyrosine kinase | Tyrosine | Immunoglobulins | Small cell lung carcinoma | Hepatocyte growth factor | siRNA | Gene expression | Medical prognosis | Protein expression | Gene loci | Mutation | Solid tumors | Cytotoxic agents | Original
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 3/2006, Volume 103, Issue 11, pp. 4046 - 4051
Journal Article
Oncogene, ISSN 1476-5594, 2015, Volume 35, Issue 21, pp. 2687 - 2697
Antiangiogenic therapy resistance occurs frequently in patients with metastatic renal cell carcinoma (RCC). The purpose of this study was to understand the... 
ACTIVATION | METASTASIS | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | CELL BIOLOGY | RECEPTOR TYROSINE KINASES | CABOZANTINIB XL184 | ONCOLOGY | C-MET | GROWTH | GENETICS & HEREDITY | EXPRESSION | PROGRESSION | Proto-Oncogene Proteins c-met - metabolism | Humans | Drug Resistance, Neoplasm | Molecular Targeted Therapy | Sunitinib | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Carcinoma, Renal Cell - enzymology | Indoles - pharmacology | Carcinoma, Renal Cell - drug therapy | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Signal Transduction | Carcinoma, Renal Cell - pathology | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Angiogenesis Inhibitors - pharmacology | Proto-Oncogene Proteins - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Proto-Oncogene Proteins c-met - genetics | Xenograft Model Antitumor Assays | Pyrroles - pharmacology | Animals | Receptor Protein-Tyrosine Kinases - genetics | Mice, Nude | Kidney Neoplasms - enzymology | Kidney Neoplasms - pathology | Cell Proliferation - drug effects | Mice | Kidney Neoplasms - drug therapy | Molecular targeted therapy | Genetic aspects | Carcinoma, Renal cell | Drug therapy | Drug resistance | Properties | Phosphotransferases | Methods | Cancer therapies | Kidney cancer | Metastasis | Cabozantinib | Renal Cell Carcinoma | AXL | MET
Journal Article