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Cell death & disease, ISSN 2041-4889, 08/2017, Volume 8, Issue 8, pp. e3029 - e3029
...) and the hepatocyte growth factor receptor MET were also identified. The expression of these proteins was assessed... 
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Ephrin-A2 - metabolism | Proto-Oncogene Proteins c-met - metabolism | RNA, Small Interfering - genetics | Phosphorylation | Skin Neoplasms - drug therapy | Apoptosis - drug effects | Humans | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | Antineoplastic Agents - therapeutic use | Proto-Oncogene Proteins c-akt - genetics | Ribosomal Protein S6 Kinases, 90-kDa - metabolism | Melanoma - genetics | Cell Cycle Checkpoints - genetics | Microfilament Proteins - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Microfilament Proteins - genetics | Proto-Oncogene Proteins B-raf - metabolism | Melanoma - metabolism | Skin Neoplasms - pathology | Signal Transduction | CD13 Antigens - antagonists & inhibitors | Pyrimidinones - therapeutic use | Dasatinib - therapeutic use | Receptors, Cytoplasmic and Nuclear - genetics | Melanoma - pathology | CD13 Antigens - metabolism | Proto-Oncogene Proteins c-met - genetics | Skin Neoplasms - metabolism | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Drug Resistance, Neoplasm - genetics | CD13 Antigens - genetics | Microfilament Proteins - antagonists & inhibitors | Sulfonamides - therapeutic use | Cell Cycle Checkpoints - drug effects | Protein Kinase Inhibitors - therapeutic use | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Ribosomal Protein S6 Kinases, 90-kDa - genetics | Skin Neoplasms - genetics | Cell Line, Tumor | Indoles - therapeutic use | Pyridones - therapeutic use | Ephrin-A2 - genetics | Drug Resistance, Neoplasm - drug effects | RNA, Small Interfering - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Tyrosine | ETS protein | Hepatocyte growth factor | Melanoma | CD13 antigen | Mass spectroscopy | AKT protein | Dephosphorylation | Kinases | Gene expression | FLI1 protein | Metastases | c-Met protein | Protein expression | Protein-tyrosine kinase receptors | MET protein | Proteomes | Aminopeptidase | Protein-tyrosine kinase | Cell migration | EphA2 protein | Apoptosis | Index Medicus | Original
Journal Article
Nature genetics, ISSN 1546-1718, 07/2012, Volume 44, Issue 8, pp. 852 - 860
Human non-small cell lung cancers (NSCLCs) with activating mutations in EGFR frequently respond to treatment with EGFRtargeted tyrosine kinase inhibitors... 
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Erlotinib Hydrochloride | Proto-Oncogene Proteins c-met - metabolism | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Humans | Lung Neoplasms - metabolism | Middle Aged | Lung Neoplasms - pathology | Male | Intercellular Signaling Peptides and Proteins - metabolism | Epithelial-Mesenchymal Transition | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Adult | Female | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Proto-Oncogene Proteins - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Proto-Oncogene Proteins c-met - genetics | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | Receptor Protein-Tyrosine Kinases - genetics | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Aged | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Enzyme Activation | Mutation | Quinazolines - pharmacology | Erlotinib | Physiological aspects | Genetic aspects | Research | Lung cancer, Non-small cell | Drug therapy | Health aspects | Protein kinases | Studies | Medical research | Rodents | Lung cancer | Genomics | Kinases | Gene expression | Acquisitions & mergers | Tumors | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 3/2006, Volume 103, Issue 11, pp. 4046 - 4051
Journal Article
PloS one, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, pp. e40967 - e40967
MicroRNAs (miRNAs) are endogenous small non-coding RNAs that play central roles in diverse pathological processes. In this study, we investigated the effect of... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cyclin D2 - genetics | Humans | Gene Expression Regulation, Neoplastic | 3' Untranslated Regions - genetics | Male | Proto-Oncogene Proteins c-akt - genetics | Tumor Suppressor Protein p53 - genetics | MAP Kinase Signaling System - genetics | Melanoma - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Female | Uveal Neoplasms - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Genes, Tumor Suppressor | Microphthalmia-Associated Transcription Factor - biosynthesis | Melanoma - metabolism | Uveal Neoplasms - genetics | Uveal Neoplasms - pathology | Mitogen-Activated Protein Kinase 3 - genetics | Down-Regulation | Tumor Suppressor Protein p53 - metabolism | MicroRNAs - biosynthesis | Melanoma - pathology | Proto-Oncogene Proteins c-bcl-2 - biosynthesis | RNA, Neoplasm - biosynthesis | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Line, Tumor | RNA, Neoplasm - genetics | MicroRNAs - genetics | Cyclin D2 - biosynthesis | Microphthalmia-Associated Transcription Factor - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | MicroRNA | Analysis | Melanoma | Research | Tumor proteins | Tumors | Apoptosis | Transcription factors | Target recognition | Leukocyte migration | Laboratories | p53 Protein | Science | AKT protein | Metastasis | D2 protein | Cyclin D2 | Skin cancer | c-Met protein | Transfection | Cell cycle | miRNA | Tumorigenesis | Bioinformatics | Microphthalmia-associated transcription factor | Invasiveness | Extracellular signal-regulated kinase | Optometry | Gene expression | Ribonucleic acid--RNA | Hospitals | MicroRNAs | Tumor suppressor genes | Mutation | Cell migration | Index Medicus | RNA | Ribonucleic acid
Journal Article
JNCI : Journal of the National Cancer Institute, ISSN 1460-2105, 04/2011, Volume 103, Issue 8, pp. 645 - 661
.... MET mRNA, and protein expression and signal transduction were compared in treated and untreated cells, and the involvement of the DNA-damage sensor ataxia telangiectasia mutated (ATM... 
Life Sciences & Biomedicine | Oncology | Science & Technology | Humans | Neoplasm Invasiveness - prevention & control | Radiation Tolerance | Receptors, Growth Factor - antagonists & inhibitors | NF-kappa B - metabolism | RNA, Messenger - metabolism | NF-kappa B - radiation effects | Receptors, Growth Factor - genetics | Proto-Oncogene Proteins c-met - radiation effects | Signal Transduction - radiation effects | Protein-Serine-Threonine Kinases - metabolism | Radiation, Ionizing | Tumor Suppressor Proteins - metabolism | DNA-Binding Proteins - radiation effects | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Cell Cycle Proteins - metabolism | Cell Cycle Proteins - radiation effects | Receptors, Growth Factor - radiation effects | Proto-Oncogene Proteins c-met - drug effects | Ataxia Telangiectasia Mutated Proteins | Phosphorylation - radiation effects | Cell Line, Tumor | Mice | RNA, Small Interfering | Tumor Suppressor Proteins - radiation effects | Receptors, Growth Factor - metabolism | Neoplasms - metabolism | Proto-Oncogene Proteins c-met - metabolism | Apoptosis - radiation effects | Up-Regulation - radiation effects | Transcription, Genetic - radiation effects | Transplantation, Heterologous | Sulfones - pharmacology | DNA-Binding Proteins - metabolism | Chromatin Immunoprecipitation | Protein-Serine-Threonine Kinases - radiation effects | Tumor Suppressor Proteins - genetics | Polymerase Chain Reaction | Cell Cycle Proteins - genetics | Indoles - pharmacology | Neoplasms - radiotherapy | Gene Expression Regulation, Neoplastic - drug effects | In Situ Nick-End Labeling | Enzyme-Linked Immunosorbent Assay | Radiation-Sensitizing Agents - pharmacology | Gene Silencing | Protein-Serine-Threonine Kinases - genetics | DNA-Binding Proteins - genetics | Cell Survival - radiation effects | Cell Movement - radiation effects | Proto-Oncogene Proteins c-met - genetics | Blotting, Northern | Animals | NF-kappa B - genetics | Protein Kinase Inhibitors - pharmacology | Neoplasms - pathology | Receptors, Growth Factor - drug effects | DNA Damage - radiation effects | Mitogen-Activated Protein Kinases - metabolism | Ionizing radiation | Physiological aspects | Genetic aspects | Cancer invasiveness | Research | Gene expression | Radiotherapy | Health aspects | Index Medicus
Journal Article
The New England journal of medicine, ISSN 1533-4406, 10/2010, Volume 363, Issue 18, pp. 1693 - 1703
...,7 and non-small-cell lung cancer.8 In non-small-cell lung cancer, EML4-ALK is an aberrant fusion gene that encodes a cytoplasmic chimeric protein... 
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Lung Neoplasms - drug therapy | Pyrazoles - therapeutic use | Microtubule-Associated Proteins - genetics | Humans | Middle Aged | Receptors, Growth Factor - antagonists & inhibitors | Lung Neoplasms - pathology | Male | Protein Kinase Inhibitors - adverse effects | Protein-Tyrosine Kinases - genetics | Pyridines - adverse effects | Serine Endopeptidases - genetics | Cell Cycle Proteins - genetics | Adult | Female | Pyrazoles - adverse effects | Pyridines - therapeutic use | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Pyridines - administration & dosage | Administration, Oral | Carcinoma, Non-Small-Cell Lung - genetics | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Kaplan-Meier Estimate | In Situ Hybridization, Fluorescence | Receptor Protein-Tyrosine Kinases | Disease Progression | Protein Kinase Inhibitors - administration & dosage | Pyrazoles - administration & dosage | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Protein-Tyrosine Kinases - antagonists & inhibitors | Tyrosine | Care and treatment | Lung cancer, Small cell | Genetic aspects | Diagnosis | Health aspects | Phosphotransferases | Lymphomas | Pharmaceutical industry | Lung cancer | Tumors | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2012, Volume 57, Issue 6, pp. 1292 - 1298
Journal Article