X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (826) 826
humans (709) 709
c-jun (701) 701
index medicus (662) 662
apoptosis (561) 561
phosphorylation (518) 518
cell biology (410) 410
biochemistry & molecular biology (405) 405
mice (399) 399
signal transduction (395) 395
jnk mitogen-activated protein kinases - metabolism (296) 296
kinases (289) 289
rats (287) 287
activation (279) 279
oncology (271) 271
jnk (268) 268
c-jun nh2-terminal kinase (267) 267
n-terminal kinase (265) 265
jnk mitogen-activated protein kinases (260) 260
jun nh2-terminal kinase (259) 259
proteins (257) 257
transcription factors (254) 254
male (253) 253
expression (244) 244
mitogen-activated protein kinases - metabolism (242) 242
proto-oncogene proteins c-jun - metabolism (215) 215
cells, cultured (211) 211
terminal kinase (210) 210
apoptosis - drug effects (206) 206
c-jun nh (204) 204
map kinase (196) 196
gene expression (193) 193
signal-transduction (190) 190
enzyme activation (189) 189
activated protein-kinase (184) 184
research (183) 183
gene-expression (176) 176
oxidative stress (173) 173
cell line, tumor (172) 172
c-jun protein (169) 169
physiology (169) 169
cell line (162) 162
cancer (156) 156
research article (153) 153
nf-kappa-b (148) 148
female (143) 143
protein kinases (137) 137
blotting, western (136) 136
protein-kinase (136) 136
mitogen-activated protein kinases (130) 130
signal transduction - drug effects (127) 127
inhibition (125) 125
medicine (125) 125
transcription factor ap-1 - metabolism (122) 122
enzyme inhibitors - pharmacology (121) 121
inflammation (121) 121
pathway (121) 121
physiological aspects (121) 121
stress (117) 117
induced apoptosis (115) 115
neurosciences (115) 115
extracellular signal-regulated kinase (111) 111
multidisciplinary sciences (111) 111
phosphorylation - drug effects (108) 108
calcium-calmodulin-dependent protein kinases - metabolism (107) 107
cells (107) 107
enzyme activation - drug effects (107) 107
health aspects (106) 106
rats, sprague-dawley (106) 106
rodents (106) 106
c-jun n-terminal kinase (105) 105
p38 mitogen-activated protein kinases (103) 103
signal transduction - physiology (101) 101
pharmacology & pharmacy (100) 100
biology (99) 99
genetic aspects (97) 97
cell death (96) 96
transfection (95) 95
proliferation (94) 94
induction (93) 93
science (93) 93
mitogen-activated protein kinase (92) 92
in-vitro (91) 91
apoptosis - physiology (89) 89
jnk mitogen-activated protein kinases - antagonists & inhibitors (88) 88
p38 (88) 88
immunology (85) 85
cell cycle (83) 83
death (82) 82
map kinase signaling system - drug effects (82) 82
p38 mitogen-activated protein kinases - metabolism (82) 82
proto-oncogene proteins c-jun - genetics (81) 81
biophysics (80) 80
map kinase signaling system (80) 80
tumor-necrosis-factor (80) 80
map kinase kinase 4 - metabolism (78) 78
signal-transduction pathway (78) 78
mice, inbred c57bl (77) 77
cell proliferation (76) 76
mice, knockout (76) 76
more...
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 02/2003, Volume 92, Issue 2, pp. 136 - 138
Stimulation of β-adrenergic receptors (βARs) causes apoptosis in adult rat ventricular myocytes (ARVMs). The role of reactive oxygen species (ROS) in mediating... 
Mitochondria | Reactive oxygen species | β-adrenergic receptors | terminal kinase | c-Jun NH | Apoptosis | c-Jun NH(2-)terminal kinase | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | mitochondria | beta-adrenergic receptors | reactive oxygen species | N-TERMINAL KINASE | apoptosis | PERIPHERAL VASCULAR DISEASE | RAT VENTRICULAR MYOCYTES | HEMATOLOGY | SUPEROXIDE-DISMUTASE | Catalase - pharmacology | Free Radical Scavengers - pharmacology | Norepinephrine - pharmacology | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Metalloporphyrins - pharmacology | Salicylates - pharmacology | Mitochondrial Membrane Transport Proteins | Prazosin - pharmacology | JNK Mitogen-Activated Protein Kinases | Receptors, Adrenergic, beta - drug effects | Receptors, Adrenergic, beta - metabolism | Myocytes, Cardiac - cytology | Catalase - genetics | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Bongkrekic Acid - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Caspase Inhibitors | Cytochrome c Group - metabolism | Ion Channels - antagonists & inhibitors | Animals | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Myocytes, Cardiac - metabolism | Mitogen-Activated Protein Kinases - genetics | Signal Transduction - physiology | Apoptosis - physiology | Catalase - biosynthesis | Organometallic Compounds - pharmacology | Mitogen-Activated Protein Kinases - metabolism
Journal Article
The Journal of cell biology, ISSN 1540-8140, 2004, Volume 164, Issue 3, pp. 385 - 394
.... Significantly, a major function of Krox-20 is to suppress the c-Jun NH2-terminal protein kinase (JNK... 
Neurons | Myelin | Schwann cells | Nerves | Cell cycle | Antibodies | Swiss 3T3 cells | Gene expression regulation | Cells | Apoptosis | JIP-1 | Neuregulin | Cgr2 | PNS | myelin | N-TERMINAL KINASE | neuregulin | INTERACTING PROTEIN-1 | egr2 | CELL BIOLOGY | PERIPHERAL-NERVE | IN-VIVO | GROWTH-FACTOR | CYCLE PROGRESSION | C-JUN | TRANSCRIPTION FACTOR | EMBRYONIC NERVES | SIGNAL-TRANSDUCTION PATHWAY | Sciatic Nerve - growth & development | Recombinant Fusion Proteins - metabolism | Schwann Cells - cytology | Myelin Sheath - metabolism | DNA-Binding Proteins - metabolism | MAP Kinase Kinase 7 | Mitogen-Activated Protein Kinase Kinases - metabolism | Neuregulin-1 | Early Growth Response Protein 2 | JNK Mitogen-Activated Protein Kinases | Sciatic Nerve - cytology | Schwann Cells - physiology | Animals, Newborn | Cell Survival | Down-Regulation | Rats | MAP Kinase Kinase Kinases - metabolism | DNA - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mice, Knockout | Cell Division - physiology | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Adaptor Proteins, Signal Transducing | Animals | Carrier Proteins - metabolism | Proto-Oncogene Proteins c-jun - metabolism | Myelin Sheath - genetics | Recombinant Fusion Proteins - genetics | Signal Transduction - physiology | Mice | Apoptosis - physiology | Transforming Growth Factor beta - metabolism | 3T3 Cells | MAP Kinase Kinase Kinase 1 | Mitogen-Activated Protein Kinases - metabolism | egr2; JIP-1; neuregulin; PNS; myelin
Journal Article
PLOS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, p. e0169267
...). AMs isolated from WT, I kappa B-kinase (IKK) beta(Delta Mye) (IKK beta was selectively deleted in macrophages), and JNK1... 
FIBROSIS | MECHANICAL VENTILATION | INFLAMMATION | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | Bronchoalveolar Lavage Fluid | Tumor Necrosis Factor-alpha - metabolism | Enzyme-Linked Immunosorbent Assay | Mice, Inbred C57BL | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Neutrophil Infiltration - genetics | Lung Injury - etiology | Neutrophil Infiltration - physiology | Animals | Lung Injury - microbiology | Pneumonia, Ventilator-Associated - microbiology | Pseudomonas aeruginosa - pathogenicity | Pneumonia, Ventilator-Associated - complications | Lung Injury - metabolism | Electrophoretic Mobility Shift Assay | Mice | Interleukin-6 - metabolism | Neutrophils - metabolism | Pneumonia, Ventilator-Associated - physiopathology | Nosocomial infection | Pneumonia | Transcription factors | Laboratories | Pathogenesis | Critical care | Drug resistance | Kinases | Macrophages | Experiments | Cell adhesion & migration | Interleukin 6 | Proteins | Signal transduction | Pathways | Bronchoalveolar lavage | Pseudomonas aeruginosa | Tumor necrosis factor-TNF | Alveoli | Injuries | Deoxyribonucleic acid--DNA | Ventilators | NF-κB protein | Ventilator-associated pneumonia | Cytokines | Neutrophils | Bronchus | Antimicrobial agents | JNK protein | Inflammation | Permeability | Tumor necrosis factor-α | Gene expression | Morbidity | Signaling | Hospitals | Lungs | Ventilation | Respiration | Colonization | Chemokines | Cancer | Deoxyribonucleic acid | DNA
Journal Article
eLife, ISSN 2050-084X, 2018, Volume 7
Breast cancer is the most commonly diagnosed malignancy in women. Analysis of breast cancer genomic DNA indicates frequent loss-of-function mutations in components of the cJUN NH2-terminal kinase (JNK) signaling pathway... 
BREAST-CANCER | ACTIVATION | GENE | LANDSCAPE | BIOLOGY | TRANSDUCTION | C-JUN | EXPRESSION | TUMORIGENESIS | SOMATIC MUTATIONS | REVEALS | Cell proliferation | Transcription factors | Animal models | Statistical analysis | Activator protein 1 | Genomes | Breast cancer | Malignancy | Gene expression | Epithelium | Kinases | Genomic instability | Proteins | Signal transduction | Etiology | Software | Mutation | Deoxyribonucleic acid--DNA | Tumors
Journal Article
Journal Article
The Journal of experimental medicine, ISSN 1540-9538, 2002, Volume 195, Issue 7, pp. 811 - 823
The c-Jun NH2-terminal kinase (JNK) signaling pathway is induced by cytokines and stress stimuli and is implicated in cell death and differentiation, but the specific function of this pathway depends on the cell type... 
T lymphocytes | c-Jun | IL-2Rα | AP-1 | MAP kinases | MEDICINE, RESEARCH & EXPERIMENTAL | APOPTOSIS | IL-2R alpha | PROTEIN | TRANSCRIPTION | PROLIFERATION | IMMUNOLOGY | SIGNAL-TRANSDUCTION | RECEPTOR-ALPHA-GENE | COSTIMULATORY SIGNALS | IN-VIVO | STRESS | EXPRESSION | Original
Journal Article
Journal Article
Journal of Pharmacological Sciences, ISSN 1347-8613, 2011, Volume 116, Issue 3, pp. 296 - 308
Journal Article
Journal Article