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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2005, Volume 102, Issue 12, pp. 4459 - 4464
Journal Article
11/2013
Nicotine Addiction is a chronic relapsing disorder. Relapse risk persists despite years of abstinence. Drug-associated cues have been demonstrated to induce... 
0347 | Extracellular-signal Regulated Kinase (ERK) | cAMP Response Element Binding Protein (CREB) | incubation of nicotine craving | nicotine addiction | 0419 | Fos
Dissertation
Journal of Biological Chemistry, ISSN 0021-9258, 06/2015, Volume 290, Issue 23, pp. 14391 - 14406
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2016, Volume 291, Issue 24, pp. 12771 - 12785
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2003, Volume 100, Issue 21, pp. 12147 - 12152
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2014, Volume 289, Issue 43, pp. 29739 - 29749
Background: mTOR integrates various intracellular and extracellular signals to regulate cell growth, proliferation, and survival. Results: PDK4 promotes... 
MAMMALIAN TARGET | RAPAMYCIN | Warburg Effect | TUBEROUS SCLEROSIS COMPLEX-1 | GROWTH-CONTROL | Mechanistic Target of Rapamycin (mTOR) | Pyruvate Dehydrogenase Kinase 4 | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | PYRUVATE-DEHYDROGENASE COMPLEX | RAT MODEL | MTOR | Ras Homolog Enriched in Brain | Signaling | cAMP-response Element-binding Protein (CREB) | DNA-Protein Interaction | Cellular Regulation | Tumorigenesis | UP-REGULATION | Protein Kinases - metabolism | Transcription, Genetic - drug effects | AMP-Activated Protein Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Ras Homolog Enriched in Brain Protein | Carcinogenesis - metabolism | Glycolysis - drug effects | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - antagonists & inhibitors | TOR Serine-Threonine Kinases - antagonists & inhibitors | Multiprotein Complexes - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Isoenzymes - metabolism | HEK293 Cells | Proto-Oncogene Proteins c-akt - metabolism | Tumor Suppressor Proteins - metabolism | Pyruvate Kinase - metabolism | PTEN Phosphohydrolase - metabolism | Neuropeptides - metabolism | Aerobiosis - drug effects | Carcinogenesis - pathology | Up-Regulation - drug effects | Animals | Monomeric GTP-Binding Proteins - metabolism | Signal Transduction - drug effects | Mice, Nude | Cyclic AMP Response Element-Binding Protein - metabolism | Dichloroacetic Acid - pharmacology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Cell Biology
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 34, pp. 13125 - 13133
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 1/2016, Volume 53, Issue 1, pp. 662 - 676
Journal Article
Journal of Invertebrate Pathology, ISSN 0022-2011, 01/2018, Volume 151, pp. 14 - 20
Cyclic AMP response element binding (CREB) proteins participate in the regulation of many biological processes. However, little is known about their role in... 
Immune response | Antheraea pernyi | cAMP response element binding protein | CELLS | ACTIVATION | LONG-TERM-MEMORY | PHOSPHORYLATION | TRANSCRIPTION | FACTOR CREB | INSECT | DROSOPHILA | COACTIVATORS | ZOOLOGY | GENE-EXPRESSION | Cyclic adenylic acid | DNA binding proteins | RNA | Protein kinases | Escherichia coli | Protein binding
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2013, Volume 288, Issue 45, pp. 32528 - 32538
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2012, Volume 32, Issue 11, pp. 2751 - 2760
OBJECTIVE—Migration of vascular smooth muscle cells (VSMCs) from the media into intima contributes to the development of atherosclerosis. Gene deletion... 
cAMP response element-binding protein | Toll-like receptor 2 | Migration | Vascular smooth muscle cells | Interleukin-6 | CHLAMYDIA-PNEUMONIAE | ATHEROSCLEROTIC LESION DEVELOPMENT | ACTIVATION | TOLL-LIKE-RECEPTOR-2 | PROLIFERATION | interleukin-6 | TOLL-LIKE RECEPTOR-2 | CREB | GROWTH | migration | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | vascular smooth muscle cells | EXPRESSION | PROGRESSION | toll-like receptor 2 | Cell Proliferation | Interleukin-6 - antagonists & inhibitors | Toll-Like Receptor 2 - genetics | Lipopeptides - pharmacology | Atherosclerosis - genetics | Muscle, Smooth, Vascular - immunology | rac GTP-Binding Proteins - metabolism | Toll-Like Receptor 2 - deficiency | RNA Interference | Time Factors | rac GTP-Binding Proteins - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Myocytes, Smooth Muscle - drug effects | Interleukin-6 - metabolism | Muscle, Smooth, Vascular - drug effects | Atherosclerosis - pathology | Atherosclerosis - immunology | Interleukin-6 - genetics | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Neuropeptides - metabolism | Chemotaxis - drug effects | Toll-Like Receptor 2 - metabolism | Atherosclerosis - metabolism | Mice, Knockout | Cyclic AMP Response Element-Binding Protein - genetics | Stress Fibers - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Ligands | Mice | Mitogen-Activated Protein Kinase 1 - metabolism | Phosphorylation | Toll-Like Receptor 2 - agonists | Muscle, Smooth, Vascular - metabolism | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Myocytes, Smooth Muscle - pathology | Dose-Response Relationship, Drug | Transfection | Neuropeptides - genetics | Myocytes, Smooth Muscle - metabolism | Promoter Regions, Genetic | Cell Survival | Mice, Inbred C57BL | Cells, Cultured | Antibodies, Neutralizing - pharmacology | Muscle, Smooth, Vascular - pathology | Animals | Cyclic AMP Response Element-Binding Protein - metabolism | Myocytes, Smooth Muscle - immunology | Protein Kinase Inhibitors - pharmacology | rac1 GTP-Binding Protein
Journal Article
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 07/2015, Volume 309, Issue 2, pp. R138 - R147
We reported that brain (pro) renin receptor (PRR) expression levels are elevated in DOCA-salt-induced hypertension; however, the underlying mechanism remained... 
Salt-sensitive hypertension | (Pro)renin receptor | Renin-angiotensin system | CAMP response element-binding protein | Central nervous system | SALT HYPERTENSIVE-RATS | CELLS | PHYSIOLOGY | RENIN | KINASE | central nervous system | HYPERTROPHY | INHIBITION | GENE | PATHWAY | salt-sensitive hypertension | SODIUM DEPLETION | (pro)renin receptor | UP-REGULATION | renin-angiotensin system | Up-Regulation | Phosphorylation | Desoxycorticosterone Acetate | Hypertension - drug therapy | RNA, Messenger - metabolism | Transcription Factor AP-1 - metabolism | CREB-Binding Protein - genetics | Proton-Translocating ATPases - metabolism | CREB-Binding Protein - metabolism | Proton-Translocating ATPases - genetics | Transfection | RNA Interference | Time Factors | Hypertension - chemically induced | Hypertension - genetics | Receptor, Angiotensin, Type 1 - drug effects | Binding Sites | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Disease Models, Animal | Cell Line | Promoter Regions, Genetic | Sodium Chloride | Angiotensin II - metabolism | Signal Transduction | Mice, Inbred C57BL | Receptors, Cell Surface - metabolism | Angiotensin-Converting Enzyme Inhibitors - administration & dosage | Hypertension - metabolism | Paraventricular Hypothalamic Nucleus - drug effects | Animals | Receptor, Angiotensin, Type 1 - metabolism | Paraventricular Hypothalamic Nucleus - metabolism | Renin-Angiotensin System - drug effects | Infusions, Intraventricular | Receptors, Cell Surface - genetics | Hypertension | Physiological aspects | Cyclic adenylic acid | Binding proteins | Health aspects | Brain | Enzyme inhibitors | Captopril | Angiotensin | Protein binding | Neural Control | Cardiovascular and Renal Integration
Journal Article