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Circulation, ISSN 0009-7322, 08/2013, Volume 128, Issue 9, pp. 970 - 981
BACKGROUND—Sarcoplasmic reticulum (SR) Ca leak through ryanodine receptor type 2 (RyR2) dysfunction is of major pathophysiological relevance in human heart... 
Heart failure | Calcium calmodulin-dependent protein kinase type 2 | Sarcoplasmic reticulum | Protein kinases | Hypertrophy | heart failure | RYANODINE RECEPTOR PHOSPHORYLATION | CARDIAC & CARDIOVASCULAR SYSTEMS | CONTRACTILITY | hypertrophy | protein kinases | VENTRICULAR MYOCYTES | RELEASE | FAILING HEARTS | calcium-calmodulin-dependent protein kinase type 2 | sarcoplasmic reticulum | ATRIAL-FIBRILLATION | CAMKII | CALCIUM SPARKS | PKA PHOSPHORYLATION | PERIPHERAL VASCULAR DISEASE | HEART-FAILURE PROGRESSION | Phosphorylation | Calcium - metabolism | Humans | Middle Aged | Homeostasis | Cardiomegaly - pathology | Male | Ryanodine Receptor Calcium Release Channel - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - drug effects | Case-Control Studies | Myocardium - metabolism | Female | Sarcoplasmic Reticulum - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Cyclic AMP-Dependent Protein Kinases - drug effects | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Enzyme Inhibitors - pharmacology | Myocardium - pathology | Heart Failure - metabolism | Heart Failure - pathology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Disease Progression | Aged | Cardiomegaly - metabolism | Physiological aspects | Calcium channels | Research
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2014, Volume 289, Issue 47, pp. 32926 - 32936
Journal Article
Journal Article
Cell Metabolism, ISSN 1550-4131, 12/2013, Volume 18, Issue 6, pp. 803 - 815
A hallmark of obesity is selective suppression of hepatic insulin signaling (“insulin resistance”), but critical gaps remain in our understanding of the... 
SKELETAL-MUSCLE | LIPID-METABOLISM | TRIBBLES HOMOLOG | PHOSPHORYLATION | LIVER | ENDOCRINOLOGY & METABOLISM | RESISTANCE | TRB3 EXPRESSION | MICE | ENDOPLASMIC-RETICULUM STRESS | GLUCOSE-PRODUCTION | CELL BIOLOGY | Mitogen-Activated Protein Kinase 14 - antagonists & inhibitors | Humans | Intracellular Signaling Peptides and Proteins - metabolism | Hepatocytes - metabolism | Hepatocytes - cytology | Mitogen-Activated Protein Kinase 14 - metabolism | Palmitates - pharmacology | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Hepatocytes - drug effects | Protein-Serine-Threonine Kinases - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Insulin - pharmacology | Endoplasmic Reticulum Stress - drug effects | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Mitogen-Activated Protein Kinase 14 - genetics | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Mice, Knockout | Obesity - metabolism | Obesity - pathology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Insulin - metabolism | Animals | Mice, Obese | Mice | Type 2 diabetes | Obesity | Insulin resistance | Protein kinases | Insulin | Cells
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2009, Volume 106, Issue 7, pp. 2342 - 2347
Journal Article
Circulation Research, ISSN 0009-7330, 2013, Volume 112, Issue 4, pp. 664 - 674
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2015, Volume 290, Issue 8, pp. 4908 - 4927
synGAP is a neuron-specific Ras and Rap GTPase-activating protein (GAP) found in high concentrations in the postsynaptic density (PSD) fraction from the... 
NMDA RECEPTOR | DOMAIN | STIMULATION | INHIBITION | CALPAIN | CLONING | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUTATIONS | HIPPOCAMPAL-NEURONS | PLASTICITY | Oncogene Proteins - genetics | ras Proteins - genetics | Phosphorylation | ras GTPase-Activating Proteins - chemistry | rap1 GTP-Binding Proteins - chemistry | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | ras Proteins - metabolism | Neurons - cytology | GTPase-Activating Proteins - metabolism | Receptors, N-Methyl-D-Aspartate - genetics | Cyclin-Dependent Kinase 5 - chemistry | Cyclin-Dependent Kinase 5 - genetics | ras GTPase-Activating Proteins - genetics | Receptors, N-Methyl-D-Aspartate - chemistry | ras Proteins - chemistry | Proto-Oncogene Proteins p21(ras) - chemistry | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Proto-Oncogene Proteins p21(ras) - metabolism | rap1 GTP-Binding Proteins - metabolism | ras GTPase-Activating Proteins - metabolism | Oncogene Proteins - chemistry | Cells, Cultured | Oncogene Proteins - metabolism | Rats | Synapses - enzymology | GTPase-Activating Proteins - chemistry | Cyclin-Dependent Kinase 5 - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Animals | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - chemistry | Neurons - enzymology | GTPase-Activating Proteins - genetics | rap1 GTP-Binding Proteins - genetics | rap GTP-Binding Proteins | Ras Protein | Ras-related Protein 1 (Rap1) | Postsynaptic Density | Ca2 | Synaptic Plasticity | Small GTPase | Neurobiology | Mass Spectrometry (MS) | Cyclin-dependent Kinase 5 (CDK5) | Protein Kinase | Calmodulin-dependent Protein Kinase II (CaMKII) | Synaptic GTPase-activating Protein (synGAP)
Journal Article
Scientific Reports, ISSN 2045-2322, 02/2017, Volume 7, Issue 1, p. 43385
Sensory mismatch between actual motion information and anticipated sensory patterns (internal model) is the etiology of motion sickness (MS). Some evidence... 
BILATERAL VESTIBULAR DEAFFERENTATION | DORSAL HIPPOCAMPUS | PLACE CELLS | MULTIDISCIPLINARY SCIENCES | ELEMENT-BINDING PROTEIN | SPATIAL MEMORY | THETA-RHYTHM | KNOCKOUT MICE | SYNAPTIC PLASTICITY | PROTEIN-KINASE-II | ALPHA-CAMKII | Otolithic Membrane - metabolism | Nitriles - pharmacology | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - metabolism | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Male | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - genetics | CREB-Binding Protein - genetics | Otolithic Membrane - pathology | CA1 Region, Hippocampal - pathology | CREB-Binding Protein - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | Motion Sickness - physiopathology | CA1 Region, Hippocampal - metabolism | Otolithic Membrane - physiopathology | Phosphorylation - drug effects | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Disease Models, Animal | Motion Sickness - genetics | Butadienes - pharmacology | Mitogen-Activated Protein Kinase 3 - genetics | Motion Sickness - metabolism | Signal Transduction | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Motion Sickness - pathology | Gene Expression Regulation | Rats | Sulfonamides - pharmacology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Rats, Sprague-Dawley | Rotation | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - antagonists & inhibitors | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Mice | CA1 Region, Hippocampal - physiopathology | Benzylamines - pharmacology | Mitogen-Activated Protein Kinase 1 - metabolism | Phosphorylation | Ca2+/calmodulin-dependent protein kinase IV | Otoliths | Extracellular signal-regulated kinase | Retention | Cyclic AMP response element-binding protein | Habituation | Signal transduction | Etiology | Motion sickness | Ca2+/calmodulin-dependent protein kinase | Vestibular system | Hippocampus | Ca2+/calmodulin-dependent protein kinase II
Journal Article
Cell, ISSN 0092-8674, 12/2005, Volume 123, Issue 5, pp. 849 - 860
Journal Article
Journal Article