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Cancer Cell, ISSN 1535-6108, 11/2012, Volume 22, Issue 5, pp. 571 - 584
Journal Article
Gut, ISSN 0017-5749, 09/2014, Volume 63, Issue 9, pp. 1481 - 1489
Objective Pancreatic cancer is a leading cause of cancer-related death in the Western world. Current chemotherapy regimens have modest survival benefit. Thus,... 
MAMMALIAN TARGET | INHIBITION | METASTASIS | DUCTAL ADENOCARCINOMA | PHOSPHORYLATION | MOUSE MODEL | GROWTH | AKT | IMAGING PROLIFERATION | GASTROENTEROLOGY & HEPATOLOGY | GEMCITABINE | Pancreatic Neoplasms - metabolism | TOR Serine-Threonine Kinases - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Antineoplastic Agents - therapeutic use | Positron-Emission Tomography | Tumor Suppressor Protein p53 - genetics | Carcinoma, Pancreatic Ductal - diagnostic imaging | Carcinoma, Pancreatic Ductal - genetics | Pancreatic Neoplasms - drug therapy | TOR Serine-Threonine Kinases - antagonists & inhibitors | Mice, Mutant Strains | Proto-Oncogene Proteins p21(ras) - deficiency | Biomarkers, Tumor - metabolism | Biomarkers, Tumor - antagonists & inhibitors | PTEN Phosphohydrolase - genetics | PTEN Phosphohydrolase - deficiency | Sirolimus - therapeutic use | Drug Administration Schedule | Injections, Intraperitoneal | Pancreatic Neoplasms - genetics | Treatment Outcome | Tumor Suppressor Protein p53 - deficiency | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Protein Kinase Inhibitors - therapeutic use | Cell Line, Tumor | Mice | Mutation | Pancreatic Neoplasms - diagnostic imaging | Care and treatment | Chemotherapy | Usage | Pancreatic cancer | Rapamycin | Dosage and administration | Risk factors | Cancer | Studies | Medical research | Genotype & phenotype | Medical imaging | Clinical trials | Tomography | Metastasis | Gene expression | Cancer therapies | Tumors | Genetics | Cell Signalling | Pancreatic Cancer | Pancreas | 1506 | Pharmacogenomics
Journal Article
Science Translational Medicine, ISSN 1946-6234, 12/2010, Volume 2, Issue 62, pp. 62ra93 - 62ra93
Journal Article
Oncogene, ISSN 0950-9232, 08/2012, Volume 31, Issue 31, pp. 3635 - 3646
Journal Article
Nature, ISSN 0028-0836, 07/2017, Volume 547, Issue 7664, pp. 453 - 457
Plasticity of the cell state has been proposed to drive resistance to multiple classes of cancer therapies, thereby limiting their effectiveness(1-4). A... 
EPITHELIAL-MESENCHYMAL TRANSITION | METABOLISM | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | HUMAN-MELANOMA CELLS | SENSITIVITY | DEATH | INHIBITOR | EXPRESSION | SIGNATURE | REVEALS | Prostatic Neoplasms - metabolism | Cadherins - metabolism | Humans | Mesoderm - drug effects | Melanoma - enzymology | Male | Lipid Peroxides - metabolism | Zinc Finger E-box-Binding Homeobox 1 - genetics | Neoplasms - genetics | Mesoderm - enzymology | Cell Death | Epithelial-Mesenchymal Transition | Lipid Peroxidation - drug effects | Cell Transdifferentiation | Prostatic Neoplasms - drug therapy | Melanoma - metabolism | Glutathione Peroxidase - metabolism | Prostatic Neoplasms - pathology | Reproducibility of Results | Neoplasms - enzymology | Melanoma - pathology | Iron - metabolism | Neoplasms - drug therapy | Cell Lineage | Drug Resistance, Neoplasm - genetics | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Proteomics | Cell Line, Tumor | Prostatic Neoplasms - enzymology | Mesoderm - metabolism | Neoplasms - pathology | Mesoderm - pathology | Peroxidase | Research | Chemical properties | Chemical synthesis | Analysis | Cancer cells | Therapy | Biotechnology | Carcinoma | Mesenchyme | Genes | Lipid peroxidation | Lipids | Iron | Genomes | Phospholipids | Kinases | Cancer therapies | Proteins | Organoids | Dissipation | Peroxides | Lipid metabolism | Glutathione | Transfer RNA | Peroxidation | Glutathione peroxidase | Enzymes | Mortality | Melanoma | Selenocysteine | Tumor cell lines | Metabolism | Substrates | Cell death | Lipoxygenase | Prostate cancer | Prostate | Tumors | Apoptosis | Cancer | Index Medicus
Journal Article
Journal Article
Nature, ISSN 0028-0836, 09/2017, Volume 549, Issue 7673, pp. 533 - 537
High-grade gliomas (HGG) are a devastating group of cancers, and represent the leading cause of brain tumour-related death in both children and adults.... 
AUTISM | PEPTIDES | PHOSPHORYLATION | DISINTEGRIN | SUBTYPES | MULTIDISCIPLINARY SCIENCES | OLIGODENDROCYTE PRECURSOR CELLS | MUTATION | HIPPOCAMPAL | SELF-RENEWAL | CLEAVAGE | Neoplasm Transplantation | Neurons - pathology | Oligodendroglia - metabolism | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Nerve Tissue Proteins - deficiency | Brain Neoplasms - pathology | ADAM10 Protein - metabolism | Tumor Microenvironment | Male | Phosphatidylinositol 3-Kinases - metabolism | Brain Neoplasms - metabolism | Glioma - metabolism | Cell Adhesion Molecules, Neuronal - deficiency | Glioma - genetics | Membrane Proteins - deficiency | ADAM10 Protein - antagonists & inhibitors | Heterografts | Glioma - pathology | Adult | Cell Adhesion Molecules, Neuronal - metabolism | Membrane Proteins - metabolism | Neurons - metabolism | Oligodendroglia - cytology | Child | Signal Transduction | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Membrane Proteins - genetics | Brain Neoplasms - genetics | Nerve Tissue Proteins - genetics | Cell Adhesion Molecules, Neuronal - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Amyloid Precursor Protein Secretases - metabolism | Animals | Membrane Proteins - antagonists & inhibitors | Mice | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Gliomas | Health aspects | Research | TOR protein | Brain | Phosphorylation | Transcription | Brain tumors | Brain cancer | Genes | Glioblastoma | Genomes | Kinases | Glial stem cells | Proteins | Signal transduction | Cell growth | Proteolysis | Glioma cells | Xenografts | Children | Secretion | Adhesion | 1-Phosphatidylinositol 3-kinase | Oligodendroglioma | Signaling | Proteomics | Focal adhesion kinase
Journal Article