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Science, ISSN 0036-8075, 6/2007, Volume 316, Issue 5830, pp. 1494 - 1497
Allergic contact dermatitis affects about 5% of men and 11% of women in industrialized countries and is one of the leading causes for occupational diseases. In... 
Receptors | Allergic contact dermatitis | Swelling | Agonists | Reports | Inflammation | Skin | Mice | Allergies | Endocannabinoids | Cannabinoid receptors | PROTEIN-1 | MULTIDISCIPLINARY SCIENCES | ANANDAMIDE | INVOLVEMENT | DEFICIENT | INVERSE AGONIST | NEUTROPHILS | CANNABINOID CB2 RECEPTOR | KNOCKOUT MICE | GENE ONTOLOGY | HYPERSENSITIVITY | Up-Regulation | Receptor, Cannabinoid, CB2 - physiology | Oligonucleotide Array Sequence Analysis | Receptor, Cannabinoid, CB2 - agonists | Skin - metabolism | Bornanes - administration & dosage | Bornanes - pharmacology | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Dermatitis, Allergic Contact - pathology | Receptor, Cannabinoid, CB2 - genetics | Receptor, Cannabinoid, CB1 - physiology | Arachidonic Acids - metabolism | Cannabinoid Receptor Modulators - physiology | Receptor, Cannabinoid, CB1 - agonists | Piperidines - pharmacology | Female | Dronabinol - pharmacology | Skin - pathology | Disease Models, Animal | Pyrazoles - pharmacology | Piperidines - administration & dosage | Cannabinoids - administration & dosage | Down-Regulation | Mice, Inbred C57BL | Chemokines - physiology | Dinitrofluorobenzene | Glycerides - metabolism | Polyunsaturated Alkamides - metabolism | Cannabinoids - pharmacology | Mice, Knockout | Pyrazoles - administration & dosage | Animals | Receptor, Cannabinoid, CB1 - genetics | Dronabinol - administration & dosage | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Dermatitis, Allergic Contact - physiopathology | Cannabinoids | Research | Histocompatibility | Risk factors | Skin diseases | Neurosciences | Rodents | Inflammatory diseases | Index Medicus
Journal Article
Neuropharmacology, ISSN 0028-3908, 08/2013, Volume 71, Issue 100, pp. 255 - 263
The G protein-coupled receptor 55 (GPR55) is a novel cannabinoid (CB) receptor, whose role in the gastrointestinal (GI) tract remains unknown. Here we studied... 
Cannabidiol | Gastrointestinal motility | Colon | Cannabinoid | GPR55 | ENDOCANNABINOID SYSTEM | MOUSE COLON | FUNCTIONAL GASTROINTESTINAL DISORDERS | NEUROSCIENCES | PATHOPHYSIOLOGY | IN-VITRO | CANNABINOID RECEPTOR | CB1 | LIGANDS | PHARMACOLOGY | PHARMACOLOGY & PHARMACY | IRRITABLE-BOWEL-SYNDROME | Colon - cytology | Tissue Banks | Receptor, Cannabinoid, CB2 - agonists | Humans | Colon - drug effects | Gastrointestinal Motility - drug effects | Cannabidiol - analogs & derivatives | Colon - innervation | Colonic Diseases, Functional - drug therapy | Male | Neurons - cytology | Myenteric Plexus - metabolism | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Molecular Targeted Therapy | Receptor, Cannabinoid, CB2 - genetics | Receptors, Cannabinoid - metabolism | Muscle, Smooth - drug effects | Receptor, Cannabinoid, CB1 - agonists | Cannabinoid Receptor Antagonists - pharmacology | Cannabidiol - therapeutic use | Neurons - metabolism | Neurons - drug effects | Receptors, Cannabinoid - chemistry | Receptors, Cannabinoid - genetics | Nerve Tissue Proteins - antagonists & inhibitors | Muscle, Smooth - innervation | Nerve Tissue Proteins - agonists | Gene Expression Regulation | Myenteric Plexus - drug effects | Cannabidiol - pharmacology | Myenteric Plexus - cytology | Muscle, Smooth - metabolism | Mice, Inbred Strains | Nerve Tissue Proteins - genetics | Colon - metabolism | Cannabinoids - pharmacology | Mice, Knockout | Nerve Tissue Proteins - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Cannabinoids - therapeutic use | Animals | Receptor, Cannabinoid, CB1 - genetics | Mice | Colonic Diseases, Functional - metabolism | Muscle, Smooth - cytology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | In Vitro Techniques | Chronic diseases | Physiological aspects | Medical colleges | G proteins | Membrane proteins | Index Medicus | G protein-coupled receptors | COX-2, cyclooxygenase-2 | CB2, cannabinoid-2 | GI, gastrointestinal | ECS, endocannabinoid system | cDNA, complementary DNA | MAGL, monoacylglycerol lipase | CBD, cannabidiol | CB1, cannabinoid-1 | GE, gastric emptying | LMMP, longitudinal muscle-myenteric plexus layer | GPR55, G protein-coupled receptor 55 | i.c.v., intracerebroventricular administration | i.p., intraperitoneal | FAAH, fatty acid amide hydrolase | EFS, electrical field stimulation | KRS, Krebs–Ringer solution | RT-PCR, reverse transcription polymerase chain reaction | PPARα, peroxisome proliferator-activated receptor-alpha | TRPV1, transient receptor potential vanilloid 1
Journal Article
ACS Chemical Neuroscience, ISSN 1948-7193, 07/2015, Volume 6, Issue 9, pp. 1546 - 1559
Synthetic cannabinoid (SC) designer drugs based on indole and indazole scaffolds and featuring L-valinamide or L-tert-leucinamide side chains are encountered... 
FUBINACA | JWH-018 | Cannabinoid | PINACA | THC | SR 144528 | SELECTIVE ANTAGONIST | CHEMISTRY, MEDICINAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | 1ST POTENT | RATS | ILLEGAL PRODUCTS | ALPHA-PVT | NEUROSCIENCES | RECEPTOR ANTAGONIST | INTERNATIONAL UNION | DERIVATIVES | SR141716A | Rats, Wistar | Receptor, Cannabinoid, CB2 - agonists | Body Temperature - drug effects | Indazoles - chemical synthesis | Humans | Indoles - chemical synthesis | Male | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Cannabinoid Receptor Agonists - chemistry | Cannabinoid Receptor Agonists - pharmacology | Receptor, Cannabinoid, CB2 - genetics | Dose-Response Relationship, Drug | Heart Rate - drug effects | Receptor, Cannabinoid, CB1 - agonists | Cannabinoid Receptor Antagonists - pharmacology | Indoles - pharmacology | Molecular Structure | Drug Evaluation, Preclinical | Membrane Potentials - drug effects | Designer Drugs - pharmacology | Indazoles - chemistry | Cannabinoid Receptor Agonists - chemical synthesis | Designer Drugs - chemical synthesis | Indazoles - pharmacology | Designer Drugs - chemistry | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Cell Line, Tumor | Mice | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Indoles - chemistry | Cohort Studies | Index Medicus
Journal Article
Neuropharmacology, ISSN 0028-3908, 08/2015, Volume 95, pp. 68 - 82
JWH-018 is a synthetic CB and CB agonist illegally marketed as products named “Spice” or “herbal blend” for its psychoactive effects which are much higher than... 
JWH-018 Cl | JWH-018 | JWH-018 Br | CB1 receptor | Δ9-THC | Synthetic cannabinoids | receptor | Δ | THC | CB | EVOKED ACTIVITY | MOUSE | RATS | RECEPTOR | INHALATION EXPOSURE | AGONISTS | NEUROSCIENCES | POTENT | Delta-THC | DELTA-TETRAHYDROCANNABINOL | PHARMACOLOGY & PHARMACY | BLOOD | Cannabinoid Receptor Agonists - metabolism | Cricetulus | Receptor, Cannabinoid, CB2 - agonists | Humans | Motor Activity - drug effects | Male | Hypothermia - chemically induced | Cannabinoid Receptor Agonists - chemistry | Cannabinoid Receptor Agonists - pharmacology | Receptor, Cannabinoid, CB2 - genetics | Seizures - chemically induced | Indoles - metabolism | Piperidines - pharmacology | Catalepsy - chemically induced | Pain Threshold - drug effects | Indoles - pharmacology | Cannabinoids - toxicity | Reflex, Abnormal - drug effects | Naphthalenes - metabolism | CHO Cells | Cannabinoid Receptor Agonists - toxicity | Pyrazoles - pharmacology | Binding, Competitive | Naphthalenes - chemistry | Cannabinoids - chemistry | Indoles - toxicity | Mice, Inbred ICR | Cannabinoids - pharmacology | Naphthalenes - pharmacology | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Halogenation | Naphthalenes - toxicity | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Indoles - chemistry | Animal experimentation | Marijuana | Neurosciences | Chemical properties | Dietary supplements | Index Medicus
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 06/2013, Volume 169, Issue 4, pp. 834 - 843
Background and Purpose GPR18 is a recently deorphaned lipid receptor that is activated by the endogenous lipid N-arachidonoyl glycine (NAGly) as well the... 
eye | GPR | mouse | intraocular pressure | cannabinoid | THC | GPR18 | RAT | ANANDAMIDE | INTRAOCULAR-PRESSURE | IDENTIFICATION | ARRIVE GUIDELINES | MESSENGER-RNA | CANNABINOID RECEPTOR AGONISTS | PHARMACOLOGY & PHARMACY | RETINA | N-ARACHIDONOYL GLYCINE | CB1 RECEPTORS | Endocannabinoids - metabolism | Administration, Ophthalmic | Glycine - analogs & derivatives | Glycine - metabolism | Intraocular Pressure - drug effects | Receptors, G-Protein-Coupled - metabolism | Epithelium, Corneal - cytology | Male | Eye Proteins - agonists | Receptors, G-Protein-Coupled - agonists | Cannabinoid Receptor Agonists - pharmacology | Receptor, Cannabinoid, CB2 - genetics | Resorcinols - administration & dosage | Ciliary Body - metabolism | Anterior Eye Segment - metabolism | Receptors, Cannabinoid - metabolism | Resorcinols - pharmacology | Arachidonic Acids - metabolism | Arachidonic Acids - administration & dosage | Cannabinoid Receptor Antagonists - pharmacology | Epithelium, Corneal - drug effects | Eye Proteins - antagonists & inhibitors | Eye Proteins - genetics | Glycine - administration & dosage | Receptors, Cannabinoid - chemistry | Receptors, Cannabinoid - genetics | Resorcinols - metabolism | Ciliary Body - cytology | Mice, Inbred C57BL | Anterior Eye Segment - cytology | Cannabinoid Receptor Agonists - administration & dosage | Mice, Knockout | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Eye Proteins - metabolism | Signal Transduction - drug effects | Ciliary Body - drug effects | Receptors, G-Protein-Coupled - antagonists & inhibitors | Epithelium, Corneal - metabolism | Anterior Eye Segment - drug effects | Ligands | Cannabinoid Receptor Antagonists - administration & dosage | Mice | Receptors, G-Protein-Coupled - genetics | Immunohistochemistry | Glycine | Analysis | Glaucoma | Medical research | Index Medicus | Cannabinoids 2012, Part Two | Themed Section
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, pp. e0167240 - e0167240
Tamoxifen (Tam) is a selective estrogen receptor (ER) modulator (SERM) that is an essential drug to treat ER-positive breast cancer. Aside from known actions... 
SIGNAL-TRANSDUCTION | IN-VITRO | ALLOSTERIC MODULATORS | HUMAN-BREAST-CANCER | MULTIDISCIPLINARY SCIENCES | INVERSE AGONIST | CELL-GROWTH | RAT-BRAIN | ADENYLATE-CYCLASE | ESTROGEN-RECEPTOR | CB1 RECEPTOR | Cannabinoid Receptor Agonists - metabolism | Cricetulus | Receptor, Cannabinoid, CB2 - agonists | Humans | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Cannabinoid Receptor Agonists - pharmacology | Isomerism | Breast Neoplasms - metabolism | Indoles - metabolism | Cyclohexanols - metabolism | Receptor, Cannabinoid, CB1 - agonists | Colforsin - metabolism | Tamoxifen - chemistry | Cannabinoid Receptor Antagonists - pharmacology | Female | Indoles - pharmacology | Cyclohexanols - pharmacology | Cyclic AMP - metabolism | CHO Cells | Binding, Competitive | Tamoxifen - metabolism | Cricetinae | Colforsin - pharmacology | Selective Estrogen Receptor Modulators - chemistry | Adenylyl Cyclases - metabolism | Breast Neoplasms - drug therapy | Selective Estrogen Receptor Modulators - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Guanosine 5'-O-(3-Thiotriphosphate) - metabolism | Animals | Cannabinoid Receptor Antagonists - metabolism | Tamoxifen - pharmacology | Tamoxifen - analogs & derivatives | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Selective Estrogen Receptor Modulators - pharmacology | GTP-Binding Proteins - metabolism | Cell receptors | Patient outcomes | Drug metabolism | Development and progression | Genetic aspects | Breast cancer | Tamoxifen | Drug therapy | Health aspects | Cytochrome | Drugs | Estrogens | Estrogen receptors | Drug development | Isomers | Proteins | Adenylate cyclase | Osteoporosis | Receptors | Toxicology | Cell growth | Metabolites | Cannabinoid CB2 receptors | Cannabinoid receptors | Medical treatment | Cytochrome P450 | Pharmacology | Cannabinoid CB1 receptors | FDA approval | Medicine | Studies | Inverse agonists | Affinity | Selective estrogen receptor modulators | Cancer | Index Medicus
Journal Article
Molecules, ISSN 1420-3049, 08/2018, Volume 23, Issue 9, p. 2183
Journal Article
Neuropharmacology, ISSN 0028-3908, 09/2012, Volume 63, Issue 4, pp. 653 - 666
Cannabinoids have been shown to exert neuroprotective effects in a plethora of neurodegenerative conditions. Over the past decade, some studies demonstrate... 
Neuroinflammation | CB2 | CB1 | WIN55212-2 | PPAR-γ | Beta-amyloid | NITRIC-OXIDE SYNTHASE | ALZHEIMERS-DISEASE | NEUROSCIENCES | PPAR-gamma | CELL-DEATH | CASPASE-3 ACTIVATION | CORTICAL-NEURONS | SIGNALING PATHWAY | DNA FRAGMENTATION | IN-VIVO | PHARMACOLOGY & PHARMACY | INFLAMMATORY RESPONSE | NF-KAPPA-B | Receptor, Cannabinoid, CB2 - agonists | Neuroglia - pathology | Injections, Intraventricular | Male | PPAR gamma - metabolism | Alzheimer Disease - pathology | Cannabinoid Receptor Agonists - therapeutic use | Neuroglia - immunology | Nootropic Agents - antagonists & inhibitors | Cannabinoid Receptor Antagonists - pharmacology | Neurons - metabolism | Alzheimer Disease - immunology | Morpholines - therapeutic use | Receptors, Cannabinoid - chemistry | Disease Models, Animal | Benzoxazines - administration & dosage | Nerve Tissue Proteins - agonists | Alzheimer Disease - drug therapy | Rats | Hippocampus - pathology | Random Allocation | PPAR gamma - antagonists & inhibitors | Signal Transduction - drug effects | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Naphthalenes - therapeutic use | Neurons - pathology | Rats, Wistar | Hippocampus - drug effects | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Cannabinoid Receptor Agonists - chemistry | Neuroglia - drug effects | Receptors, Cannabinoid - metabolism | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | Receptor, Cannabinoid, CB1 - agonists | Morpholines - antagonists & inhibitors | Benzoxazines - antagonists & inhibitors | Neurons - drug effects | Nerve Tissue Proteins - antagonists & inhibitors | Hippocampus - immunology | Morpholines - administration & dosage | Neurons - immunology | Nootropic Agents - therapeutic use | Cannabinoid Receptor Agonists - administration & dosage | Benzoxazines - therapeutic use | Nerve Tissue Proteins - metabolism | Hippocampus - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Nootropic Agents - administration & dosage | Naphthalenes - administration & dosage | Alzheimer Disease - metabolism | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | PPAR gamma - agonists | Neuroglia - metabolism | Naphthalenes - antagonists & inhibitors | Index Medicus
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2015, Volume 77, Issue 5, pp. 475 - 487
Abstract Background Mixed cannabinoid receptor 1 and 2 (CB1 and CB2 ) agonists such as Δ9 -tetrahydrocannabinol (Δ9 -THC) can produce tolerance, physical... 
Psychiatry | Tolerance | CB2 | Precipitated withdrawal | Chemotherapy-induced neuropathic pain | Knockout mouse | Side effect | DORSAL-HORN NEURONS | MECHANICAL ALLODYNIA | PSYCHIATRY | VENTRAL ROOT TRANSECTION | CB2 RECEPTOR | INDUCED PERIPHERAL NEUROPATHY | SPINAL-CORD | NECROSIS-FACTOR-ALPHA | NERVE INJURY | NEUROSCIENCES | ACID AMIDE HYDROLASE | PRIMARY SENSORY NEURONS | Hyperalgesia - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Spinal Cord - drug effects | Receptor, Cannabinoid, CB2 - agonists | Male | Cannabinoid Receptor Agonists - pharmacology | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Paclitaxel - toxicity | Receptor, Cannabinoid, CB1 - agonists | Cannabinoid Receptor Antagonists - pharmacology | Female | Indoles - pharmacology | Chemokine CCL2 - metabolism | Chromones - pharmacology | Dronabinol - pharmacology | Disease Models, Animal | Mice, Inbred C57BL | Random Allocation | Hyperalgesia - physiopathology | Mice, Knockout | Antineoplastic Agents, Phytogenic - toxicity | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Hyperalgesia - drug therapy | Spinal Cord - physiopathology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Cannabinoids | Neurosciences | Paclitaxel | Index Medicus
Journal Article