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Cell, ISSN 0092-8674, 10/2016, Volume 167, Issue 2, pp. 457 - 470.e13
Activated macrophages undergo metabolic reprogramming, which drives their pro-inflammatory phenotype, but the mechanistic basis for this remains obscure. Here,... 
succinate | reverse electron transport | toll-like receptors | innate immunity | immunometabolism | macrophage | succinate dehydrogenase | RESPIRATORY-CHAIN | DENDRITIC CELL | BIOCHEMISTRY & MOLECULAR BIOLOGY | REPERFUSION INJURY | ALPHA | IL-1-BETA | INTERLEUKIN-10 IL-10 | DIMETHYL FUMARATE | HYPOXIA | ELECTRON-TRANSPORT | CELL-ACTIVATION | CELL BIOLOGY | Mitochondria - enzymology | Reactive Oxygen Species - metabolism | Transcriptome | Malonates - pharmacology | Lipopolysaccharides - immunology | Oxidative Phosphorylation - drug effects | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Membrane Potential, Mitochondrial | Oxidation-Reduction - drug effects | Interleukin-10 - metabolism | Plant Proteins - metabolism | Macrophages - immunology | Oxidoreductases - metabolism | Mice, Inbred C57BL | Inflammation - immunology | Mitochondria - drug effects | Citric Acid Cycle | Macrophage Activation | Macrophages - metabolism | Succinate Dehydrogenase - genetics | Animals | Sequence Analysis, RNA | Succinic Acid - metabolism | Glycolysis | Inflammation - genetics | Succinate Dehydrogenase - metabolism | Mice | Oxidases | RNA sequencing | Glucose metabolism | Medical colleges | RNA | Gastrointestinal diseases | Genes | Research institutes | Macrophages | Gene expression | Mitogens
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2010, Volume 107, Issue 1, pp. 378 - 383
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 4/2010, Volume 189, Issue 2, pp. 211 - 221
Journal Article
Journal Article
Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 01/2012, Volume 56, Issue 1, pp. 324 - 331
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 08/2008, Volume 93, Issue 8, pp. 3199 - 3207
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2010, Volume 285, Issue 36, pp. 27879 - 27890
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 12/2010, Volume 191, Issue 7, pp. 1367 - 1380
Damage to mitochondria can lead to the depolarization of the inner mitochondrial membrane, thereby sensitizing impaired mitochondria for selective elimination... 
Depolarization | Mitochondria | Drosophila | Ubiquitins | Cell lines | HeLa cells | Antibodies | Parkinson disease | Opal | Standard deviation | PROTEOLYTIC CLEAVAGE | PINK1 | FUSION | PROTEIN-DEGRADATION | DISEASE | ENDOPLASMIC-RETICULUM | MUTATIONS | FISSION | REGULATES MITOCHONDRIAL MORPHOLOGY | DROSOPHILA | CELL BIOLOGY | RNA, Small Interfering - genetics | Protein Kinases - genetics | Microtubule-Associated Proteins - genetics | Humans | Autophagy - physiology | Mitochondrial Proteins - genetics | Membrane Potential, Mitochondrial - drug effects | Autophagy - drug effects | Mitochondrial Membrane Transport Proteins | Mitochondrial Proteins - metabolism | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Membrane Transport Proteins - metabolism | Membrane Proteins - metabolism | Ubiquitination - physiology | Nuclear Proteins - genetics | Membrane Fusion - drug effects | Fibroblasts - metabolism | Membrane Proteins - genetics | HCT116 Cells | Ubiquitin-Protein Ligases - metabolism | Adenosine Triphosphatases - metabolism | Dynamins | Nuclear Proteins - metabolism | Mitochondria - drug effects | Mice, Knockout | Leupeptins - pharmacology | Animals | GTP Phosphohydrolases - metabolism | GTP Phosphohydrolases - genetics | Models, Biological | Ubiquitin-Conjugating Enzymes - metabolism | Fibroblasts - drug effects | Cell Line, Tumor | Adenosine Triphosphatases - genetics | Mice | HeLa Cells | Proteasome Endopeptidase Complex - metabolism | Membrane Fusion - physiology | Mitochondria - physiology | Proteasome Inhibitors | Ubiquitin-Protein Ligases - genetics | Protein Binding - physiology | Proteins | Ligases | Research | Properties | Ubiquitin-proteasome system | Guanosine triphosphatase | Adenosine triphosphatase
Journal Article
Nature Cell Biology, ISSN 1465-7392, 02/2015, Volume 17, Issue 2, pp. 160 - 169
Journal Article