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Journal Article
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2017, Volume 76, Issue 6
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2017, Volume 76, Issue Suppl 2, p. 6
Osteoarthritis (OA) has long been viewed as a degenerative “wear-and-tear” disease of cartilage. There is, however, increasing evidence to confirm that... 
Chemical sensors | Pathogenesis | Innate immunity | Arthritis | Inflammation | Anti-inflammatory agents | Interleukin 17 | Calcium influx | Transient receptor potential proteins | Pain | Rheumatoid arthritis | Rodents | Interleukin 1 | Chondrocytes | Sensory neurons | Ankyrin | Osteoarthritis | Cartilage diseases
Journal Article
PLOS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 11, p. e27957
Background: Understanding the key elements of signaling of chondroprogenitor cells at the earliest steps of differentiation may substantially improve our... 
INACTIVATION | CALCIUM OSCILLATIONS | CHONDROCYTES | KV1.5 | SODIUM-CHANNELS | MULTIDISCIPLINARY SCIENCES | GROWTH | ACTIVATED POTASSIUM CHANNEL | PROLIFERATION | ION CHANNELS | DIFFERENTIATION | Tetraethylammonium - pharmacology | Chondrocytes - cytology | Chondrogenesis - drug effects | Chondrogenesis - genetics | Cell Count | Cytosol - drug effects | Extracellular Space - drug effects | Cartilage - drug effects | Gene Expression Profiling | Cell Lineage - drug effects | Chondrocytes - drug effects | Extracellular Space - metabolism | Potassium Channels, Voltage-Gated - metabolism | Cartilage - cytology | Sodium Channels - metabolism | Cell Membrane - metabolism | Tetrodotoxin - pharmacology | Cell Membrane - drug effects | Chondrocytes - metabolism | Potassium Channels, Voltage-Gated - genetics | Membrane Potentials - drug effects | Cartilage - embryology | Ions | Cartilage - metabolism | Chick Embryo | Gene Expression Regulation - drug effects | Patch-Clamp Techniques | Animals | Calcium Signaling - drug effects | Cell Differentiation - drug effects | Cytosol - metabolism | Kinetics | Ion Channel Gating - drug effects | Ammonium | Regulators | Thymidine | Confocal | Cartilage | Signal transduction | Embryology | Cell growth | Sodium channels (voltage-gated) | Physiology | Potassium channels (voltage-gated) | Membrane potential | Chondrogenesis | Calcium channels (voltage-gated) | Cultures | Anatomy & physiology | Gene expression | Joint diseases | Embryos | Polymerase chain reaction | Stem cells | Chondrocytes | Hypoxia | Tetrodotoxin | Cell proliferation | Electric potential | Plasma | Mesenchyme | Science | Oscillations | Confocal microscopy | Biology | Phase transitions | Limb buds | Depolarization | Calcium signalling | Windows (intervals) | Lymphocytes | Rodents | Ion channels | Localization | Calcium channels | Tissue engineering | Data processing | Histology | Biophysics | Tea | Microscopy | Differentiation | Potassium | Apoptosis
Journal Article
Current Osteoporosis Reports, ISSN 1544-1873, 12/2016, Volume 14, Issue 6, pp. 260 - 268
Worldwide, osteoarthritis (OA) is one of the leading causes of chronic pain, for which adequate relief is not available. Ongoing peripheral input from the... 
Pain | Medicine & Public Health | Orthopedics | GPCRs | Nerve growth factor | Targets | Ion channels | Epidemiology | Osteoarthritis | KNEE | PRIMARY AFFERENTS | GENE-RELATED PEPTIDE | INTRAARTICULAR INJECTION | SODIUM-CHANNELS | SENSING ION CHANNELS | RAT MODEL | NERVE GROWTH-FACTOR | ARTICULAR-CARTILAGE | ENDOCRINOLOGY & METABOLISM | JOINT PAIN | Arthralgia - etiology | Transient Receptor Potential Channels - antagonists & inhibitors | Calcium Channels - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Calcium Channel Blockers - therapeutic use | Molecular Targeted Therapy | Osteoarthritis - drug therapy | Receptors, Calcitonin Gene-Related Peptide - metabolism | Nerve Growth Factor - antagonists & inhibitors | Voltage-Gated Sodium Channel Blockers - therapeutic use | Receptors, Chemokine - antagonists & inhibitors | Transient Receptor Potential Channels - metabolism | Osteoarthritis - complications | Nerve Growth Factor - metabolism | Acid Sensing Ion Channel Blockers - therapeutic use | Receptors, Calcitonin Gene-Related Peptide - antagonists & inhibitors | Receptors, Chemokine - metabolism | Osteoarthritis - metabolism | Receptors, Bradykinin - metabolism | Arthralgia - metabolism | Receptors, G-Protein-Coupled - antagonists & inhibitors | Bradykinin Receptor Antagonists - therapeutic use | Arthralgia - drug therapy | Voltage-Gated Sodium Channels - metabolism | Acid Sensing Ion Channels - metabolism | Calcium channels | Care and treatment | Analysis | Chronic pain | Nerve Growth Factor
Journal Article