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Cancer Science, ISSN 1347-9032, 01/2017, Volume 108, Issue 1, pp. 108 - 115
We previously reported that celecoxib, a selective COX ‐2 inhibitor, strongly inhibited human colon cancer cell proliferation by suppressing the Wnt/β‐catenin... 
2,5‐dimethylcelecoxib | Wnt/β‐catenin signaling pathway | celecoxib | 7L2 | colon cancer | TCF | 2,5-dimethylcelecoxib | TCF7L2 | Wnt/β-catenin signaling pathway | STEM-CELLS | CARDIOVASCULAR EVENTS | MUTYH-NULL MICE | BETA-CATENIN | Wnt/beta-catenin signaling pathway | COLON-CANCER | ONCOLOGY | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | COLORECTAL-CANCER | IN-VIVO | FAMILIAL ADENOMATOUS POLYPOSIS | COX-2 INHIBITORS | Transcription, Genetic - drug effects | Pyrazoles - therapeutic use | Humans | Body Weight - drug effects | Male | Wnt Proteins - metabolism | Transcription Factor 7-Like 2 Protein - metabolism | TCF Transcription Factors - metabolism | DNA Glycosylases - deficiency | Proteolysis - drug effects | Sulfonamides - blood | Pyrazoles - blood | Female | Pyrazoles - pharmacology | Intestinal Neoplasms - metabolism | DNA Glycosylases - genetics | Celecoxib - blood | Celecoxib - pharmacology | Celecoxib - therapeutic use | Sulfonamides - pharmacology | beta Catenin - metabolism | Blood Cell Count | Intestinal Neoplasms - pathology | Animals | Wnt Signaling Pathway - drug effects | Sulfonamides - therapeutic use | beta Catenin - antagonists & inhibitors | Cell Line, Tumor | Mice | Oxidative Stress - drug effects | Intestinal Neoplasms - drug therapy | Index Medicus | β‐catenin signaling pathway | Wnt | Original
Journal Article
Experimental Dermatology, ISSN 0906-6705, 11/2018, Volume 27, Issue 11, pp. 1237 - 1244
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2017, Volume 37, Issue 7, pp. 1772 - 1784
Triggering Receptor Expressed on Myeloid cells 2 (TREM2), which is expressed on myeloid cells including microglia in the CNS, has recently been identified as a... 
Akt/GSK3β signaling pathway | TREM2 | Cell survival | Alzheimer’s disease | Wnt/β-catenin signaling pathway | Microglia | NEURODEGENERATIVE DISEASE | NERVOUS-SYSTEM | CELLS | cell survival | ALZHEIMERS-DISEASE | AMYLOID PLAQUES | PROLIFERATION | BETA-CATENIN | NEUROSCIENCES | Wnt/beta-catenin signaling pathway | SIGNALING PATHWAY | PSEUDOMONAS-AERUGINOSA | Akt/GSK3 beta signaling pathway | IN-VIVO | Alzheimer's disease | microglia | Cell Cycle - genetics | Cyclin D1 - metabolism | Microglia - metabolism | Adjuvants, Immunologic - pharmacology | Membrane Glycoproteins - metabolism | Caspase 3 - metabolism | Cell Survival - genetics | Wnt Signaling Pathway - physiology | Male | Proteolysis - drug effects | Lithium Chloride - pharmacology | Thiadiazoles - pharmacology | Animals, Newborn | Cell Survival - drug effects | Brain - cytology | Microglia - drug effects | Gene Expression Regulation - genetics | Kainic Acid - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Excitatory Amino Acid Agonists - pharmacology | beta Catenin - metabolism | Membrane Glycoproteins - genetics | Gene Expression Regulation - drug effects | Animals | Wnt Signaling Pathway - drug effects | Cyclin D1 - genetics | Mice | Cell Cycle - drug effects | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Index Medicus | β-catenin signaling pathway | GSK3β signaling pathway | Akt | Wnt
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 02/2018, Volume 233, Issue 2, pp. 1574 - 1584
Knockdown of MACF1 dramatically reduced the nuclear translocation of β‐catenin, decreased the transcriptional activation of T cell factor 1 (TCF1), and... 
β‐catenin signaling | MACF1 | osteoblast | osteoblast differentiation | β-catenin signaling | Phosphorylation | Hepatocyte Nuclear Factor 1-alpha - genetics | Hepatocyte Nuclear Factor 1-alpha - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Cell Differentiation - genetics | Transfection | RNA Interference | Time Factors | Lithium Chloride - pharmacology | Microfilament Proteins - metabolism | Microfilament Proteins - genetics | Osteogenesis - genetics | Signal Transduction | Osteoblasts - drug effects | Osteogenesis - drug effects | beta Catenin - agonists | Gene Expression Regulation | Glycogen Synthase Kinase 3 beta - metabolism | beta Catenin - metabolism | beta Catenin - genetics | Phenotype | Animals | Cell Differentiation - drug effects | Mice | Osteoblasts - metabolism | 3T3 Cells | Core Binding Factor Alpha 1 Subunit - genetics | Alkaline phosphatase | Collagen (type I) | Lithium chloride | Lithium | Lymphocytes T | Kinases | Osteoblasts | Signal transduction | Cell activation | Actin | Mineralization | Transcription activation | Biocompatibility | Catenin | Hepatocyte nuclear factor 1 | Translocation | Cbfa-1 protein | Glycogen | Glycogen synthase kinase 3 | Crosslinking | Gene expression | Nuclear transport | Signaling | Molecular modelling | Osteoblastogenesis | Signal processing | Cytoskeleton | Transduction | Differentiation | Index Medicus
Journal Article
Journal Article
Journal Article
Cancer Science, ISSN 1347-9032, 03/2019, Volume 110, Issue 3, pp. 997 - 1011
The catalytic subunit p110δ of phosphoinositide 3‐kinase ( PI 3K) encoded by PIK 3 CD has been implicated in some human solid tumors. However, its roles in... 
β‐catenin signaling | PIK3CD | colorectal cancer | growth, invasion | β-catenin signaling | METASTASIS | GLYCOGEN-SYNTHASE KINASE-3 | PROLIFERATION | BETA-CATENIN | beta-catenin signaling | INHIBITION | PI3K | ONCOLOGY | WNT/BETA-CATENIN | ISOFORM P110-DELTA | PATHWAY | EXPRESSION | Index Medicus | Original
Journal Article
Hepatology, ISSN 0270-9139, 06/2018, Volume 67, Issue 6, pp. 2079 - 2081
The duality of β- and γ–catenin during liver injury has not been defined. It’s well known that loss of β-catenin plays a critical role in overall liver health... 
Adherens Junctions | gamma Catenin | Cadherins | beta Catenin | Index Medicus | β-catenin | Junctional integrity | liver disease | γ-catenin
Journal Article