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1. Full Text p53 Dynamics Control Cell Fate
by Jeremy E. Purvis and Kyle W. Karhohs and Caroline Mock and Eric Batchelor and Alexander Loewer and Galit Lahav
Science, ISSN 0036-8075, 6/2012, Volume 336, Issue 6087, pp. 1440 - 1444
Cells transmit information through molecular signals that often show complex dynamical patterns. The dynamic behavior of the tumor suppressor p53 varies... 
Cell growth | Microscopy | DNA damage | Genes | REPORTS | Cellular senescence | Cell cycle | Cell lines | Lead | DNA repair | Apoptosis | DETERMINES | ACTIVATION | P53-MDM2 FEEDBACK LOOP | SPECIFICITY | TEMPORAL CONTROL | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | GENE-EXPRESSION | MDM2 | OSCILLATIONS | ANTAGONISTS | Cellular Senescence - genetics | Gamma Rays | Signal Transduction | Humans | Transcriptional Activation | Tumor Suppressor Protein p53 - metabolism | Apoptosis - genetics | Piperazines - metabolism | Imidazoles - pharmacology | Transcription Factors - genetics | DNA Breaks, Double-Stranded | Piperazines - pharmacology | Cell Cycle Checkpoints | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Models, Biological | DNA Repair | Tumor Suppressor Proteins - genetics | Cell Line, Tumor | Nuclear Proteins - genetics | Single-Cell Analysis | Promyelocytic Leukemia Protein | Imidazoles - metabolism | Cell proliferation | Tumor suppressor genes | DNA replication | Research | Health aspects | Proteins | Signal transduction | Gene expression | Index Medicus | Activated | Cellular | Dynamics | Deoxyribonucleic acid | Decisions | Constants | Activation
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2. Full Text ER stress regulates myeloid-derived suppressor cell fate through TRAIL-R-mediated apoptosis
by Condamine, Thomas and Kumar, Vinit and Ramachandran, Indu R and Youn, Je-In and Celis, Esteban and Finnberg, Niklas and El-Deiry, Wafik S and Winograd, Rafael and Vonderheide, Robert H and English, Nickolas R and Knight, Stella C and Yagita, Hideo and McCaffrey, Judith C and Antonia, Scott and Hockstein, Neil and Witt, Robert and Masters, Gregory and Bauer, Thomas and Gabrilovich, Dmitry I
Journal of Clinical Investigation, ISSN 0021-9738, 06/2014, Volume 124, Issue 6, pp. 2626 - 2639
Myeloid-derived suppressor cells (MDSCs) dampen the immune response thorough inhibition of T cell activation and proliferation and often are expanded in... 
MEDICINE, RESEARCH & EXPERIMENTAL | UNFOLDED PROTEIN RESPONSE | TUMOR-BEARING MICE | LIGAND TRAIL | RECEPTOR | ENDOPLASMIC-RETICULUM | TUMORICIDAL ACTIVITY | DEATH | ACCUMULATION | ANTI-DR5 ANTIBODY THERAPY | CANCER | Carcinoma, Non-Small-Cell Lung - pathology | Humans | Lung Neoplasms - metabolism | Mice, Inbred C57BL | Carcinoma, Non-Small-Cell Lung - metabolism | Lung Neoplasms - pathology | Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism | Carcinoma, Non-Small-Cell Lung - immunology | Lung Neoplasms - immunology | Animals | Apoptosis - immunology | Models, Biological | Myeloid Cells - immunology | Endoplasmic Reticulum Stress | Cell Line, Tumor | Myeloid Cells - metabolism | Female | Mice | Mice, Inbred BALB C | Myeloid Cells - pathology | Ligands (Biochemistry) | Genetic aspects | Stress (Physiology) | Properties | Gene expression | Endoplasmic reticulum | Apoptosis | Studies | Spleen | Homeostasis | Software | Cytoplasm | Cancer | Index Medicus | Abridged Index Medicus
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3. Full Text S-2-hydroxyglutarate regulates CD8 + T-lymphocyte fate
by Tyrakis, Petros A and Palazon, Asis and Macias, David and Lee, Kian L and Phan, Anthony T and Veliça, Pedro and You, Jia and Chia, Grace S and Sim, Jingwei and Doedens, Andrew and Abelanet, Alice and Evans, Colin E and Griffiths, John R and Poellinger, Lorenz and Goldrath, Ananda W and Johnson, Randall S
Nature, ISSN 0028-0836, 12/2016, Volume 540, Issue 7632, pp. 236 - 241
R-2-hydroxyglutarate accumulates to millimolar levels in cancer cells with gain-of-function isocitrate dehydrogenase 1/2 mutations. These levels of... 
EFFECTOR | MEMORY | METABOLISM | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | ALPHA-KETOGLUTARATE | MUTANT IDH | CELL-DIFFERENTIATION | ONCOMETABOLITE 2-HYDROXYGLUTARATE | CANCER | ANTIGEN | CD8-Positive T-Lymphocytes - cytology | Dioxygenases - metabolism | Oxygen - metabolism | Hypoxia - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glutarates - immunology | CD8-Positive T-Lymphocytes - metabolism | Receptors, Antigen, T-Cell - immunology | Lysine - metabolism | Homeostasis - drug effects | Protein Stability | Ketoglutaric Acids - metabolism | Von Hippel-Lindau Tumor Suppressor Protein - metabolism | Lymphocyte Activation | DNA - metabolism | DNA - chemistry | Animals | Cell Differentiation - drug effects | CD8-Positive T-Lymphocytes - drug effects | Glutarates - metabolism | Mice | Glutarates - pharmacology | Histones - metabolism | CD8-Positive T-Lymphocytes - immunology | DNA Methylation - drug effects | Genetic aspects | Immune response | Research | T cells | Health aspects | Immunological research | Index Medicus
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4. Full Text A T-bet gradient controls the fate and function of CCR6-RORγt + innate lymphoid cells
by Klose, Christoph S. N and Kiss, Elina A and Schwierzeck, Vera and Ebert, Karolina and Hoyler, Thomas and D'Hargues, Yannick and Göppert, Nathalie and Croxford, Andrew L and Waisman, Ari and Tanriver, Yakup and Diefenbach, Andreas
Nature, ISSN 0028-0836, 02/2013, Volume 494, Issue 7436, pp. 261 - 265
At mucosal surfaces, the immune system should not initiate inflammatory immune responses to the plethora of antigens constantly present in the environment, but... 
DEFENSE | ROR-GAMMA-T | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | NKP46(+) CELLS | HOST | DIFFERENTIATION | EXPRESSION | Enterocolitis - pathology | Enterocolitis - immunology | Antigens, Ly - genetics | Salmonella Infections - immunology | Salmonella Infections - metabolism | Intestinal Mucosa - cytology | Epithelium - immunology | Lymphocytes - immunology | Natural Cytotoxicity Triggering Receptor 1 - genetics | Intestinal Mucosa - immunology | Epithelium - microbiology | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Receptors, CCR6 - metabolism | Cell Differentiation | Interferon-gamma - genetics | Lymphocytes - metabolism | Epithelium - metabolism | Salmonella typhimurium - immunology | Salmonella typhimurium - pathogenicity | Mice, Inbred C57BL | Cells, Cultured | Mucus - secretion | Receptors, CCR6 - deficiency | Lymphocytes - cytology | Intestinal Mucosa - microbiology | Interleukin-23 - immunology | T-Box Domain Proteins - metabolism | Cell Lineage | Immunity, Innate - immunology | Animals | Interferon-gamma - immunology | Mice | Mice, Inbred BALB C | Enterocolitis - metabolism | Interferon-gamma - biosynthesis | Index Medicus
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5. Full Text GATA3 controls Foxp3 + regulatory T cell fate during inflammation in mice
by Wohlfert, Elizabeth A and Grainger, John R and Bouladoux, Nicolas and Konkel, Joanne E and Oldenhove, Guillaume and Ribeiro, Carolina Hager and Hall, Jason A and Yagi, Ryoji and Naik, Shruti and Bhairavabhotla, Ravikiran and Paul, William E and Bosselut, Remy and Wei, Gang and Zhao, Keji and Oukka, Mohamed and Zhu, Jinfang and Belkaid, Yasmine
Journal of Clinical Investigation, ISSN 0021-9738, 11/2011, Volume 121, Issue 11, pp. 4503 - 4515
Tregs not only keep immune responses to autoantigens in check, but also restrain those directed toward pathogens and the commensal microbiota. Control of... 
MEDICINE, RESEARCH & EXPERIMENTAL | THYMOCYTE DEVELOPMENT | CYTOKINE PRODUCTION | TH2 CELLS | TGF-BETA | IN-VIVO | SUPPRESSIVE FUNCTION | RETINOIC-ACID | HELPER-CELLS | CONTROL INTESTINAL INFLAMMATION | TRANSCRIPTION FACTOR GATA-3 | Forkhead Transcription Factors - immunology | GATA3 Transcription Factor - genetics | Inflammation - pathology | T-Lymphocytes, Regulatory - metabolism | Up-Regulation | GATA3 Transcription Factor - immunology | Humans | Male | Gastrointestinal Tract - pathology | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Gastrointestinal Tract - immunology | Inflammation - metabolism | Female | Cytokines - genetics | Skin - pathology | Cytokines - immunology | Skin - immunology | Receptors, Antigen, T-Cell - metabolism | Mice, Inbred C57BL | Mice, Transgenic | Inflammation - immunology | GATA3 Transcription Factor - deficiency | Mice, Knockout | Animals | Cytokines - deficiency | Mice | Cytokines | Genetic aspects | Research | T cells | Properties | Genetic regulation | Gene expression | Index Medicus | Abridged Index Medicus
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6. Full Text Antigen Availability Determines CD8+ T Cell-Dendritic Cell Interaction Kinetics and Memory Fate Decisions
by Henrickson, Sarah E and Perro, Mario and Loughhead, Scott M and Senman, Balimkiz and Stutte, Susanne and Quigley, Michael and Alexe, Gabriela and Iannacone, Matteo and Flynn, Michael P and Omid, Shaida and Jesneck, Jonathan L and Imam, Sabrina and Mempel, Thorsten R and Mazo, Irina B and Haining, W. Nicholas and von Andrian, Ulrich H
Immunity, ISSN 1074-7613, 09/2013, Volume 39, Issue 3, pp. 496 - 507
T cells are activated by antigen (Ag)-bearing dendritic cells (DCs) in lymph nodes in three phases. The duration of the initial phase of transient, serial DC-T... 
EFFECTOR | ACTIVATION | TOLERANCE | LYMPH-NODES | IN-VIVO | DYNAMICS | DIFFERENTIATION | INDUCTION | IMMUNOLOGY | TRANSCRIPTION FACTOR | STOP SIGNAL | Lymphocytic choriomeningitis virus - immunology | Lymphocyte Activation | Dendritic Cells - immunology | Mice, Inbred C57BL | Cell Communication | Transcriptome - immunology | Adoptive Transfer | Lymph Nodes - immunology | Antigen-Presenting Cells - immunology | Mice, Knockout | Animals | Antigen Presentation | CD8-Positive T-Lymphocytes - metabolism | Cell Differentiation | Mice | CD8-Positive T-Lymphocytes - immunology | Dendritic Cells - metabolism | Immunologic Memory - immunology | T cell receptors | Immunology | Peptides | Gene expression | Lymphocytes | Index Medicus
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7. Full Text Extracellular-matrix tethering regulates stem-cell fate
by Trappmann, B and Gautrot, J.E and Connelly, J.T and Strange, D.G.T and Li, Y and Oyen, M.L and Stuart, M.A.C and Boehm, H and Li, B.J and Vogel, V and Spatz, J.P and Watt, F.M and Huck, W.T.S
Nature Materials, ISSN 1476-1122, 2012, Volume 11, Issue 7, pp. 642 - 649
To investigate how substrate properties influence stem-cell fate, we cultured single human epidermal stem cells on polydimethylsiloxane (PDMS) and... 
PHYSICS, CONDENSED MATTER | PHYSICS, APPLIED | GELS | MATERIALS SCIENCE, MULTIDISCIPLINARY | CHEMISTRY, PHYSICAL | KERATINOCYTES | ELASTICITY | ADHESION | FIBRILS | STIFFNESS | SUBSTRATE | FIBRONECTIN | TERMINAL DIFFERENTIATION | MORPHOLOGY | Mesenchymal Stromal Cells - drug effects | Extracellular Matrix - drug effects | Humans | Extracellular Matrix - metabolism | Collagen - chemistry | Acrylic Resins - pharmacology | Keratinocytes - cytology | Acrylic Resins - chemistry | Cell Culture Techniques - methods | Mesenchymal Stromal Cells - cytology | Dimethylpolysiloxanes - pharmacology | Cell Differentiation - drug effects | Dimethylpolysiloxanes - chemistry | Mechanical Phenomena | Infant, Newborn | Mechanical properties | Cell culture | Materials science | Biomedical materials | Collagen | Stem cells | Index Medicus | Elastic modulus | Collagens | Stiffness | Polyacrylamides | Differentiation | Kinases | Anchoring
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8. Full Text Loss of wnt/β‐catenin signaling causes cell fate shift of preosteoblasts from osteoblasts to adipocytes
by Song, Lige and Liu, Minlin and Ono, Noriaki and Bringhurst, F Richard and Kronenberg, Henry M and Guo, Jun
Journal of Bone and Mineral Research, ISSN 0884-0431, 11/2012, Volume 27, Issue 11, pp. 2344 - 2358
Wnt signaling is essential for osteogenesis and also functions as an adipogenic switch, but it is not known if interrupting wnt signaling via knockout of... 
OSTEOGENESIS | β‐CATENIN | ADIPOGENESIS | CANONICAL WNT SIGNALING | β-CATENIN | STEM-CELLS | ACTIVATED RECEPTOR-GAMMA | MESENCHYMAL PROGENITORS | BONE-MARROW | OSTEOPOROSIS | GLYCOGEN-SYNTHASE KINASE-3-BETA | beta-CATENIN | IN-VIVO | ENDOCRINOLOGY & METABOLISM | DIFFERENTIATION | Adipocytes - cytology | Adiposity | Gene Deletion | Integrases - metabolism | Transcription, Genetic | Osteoblasts - cytology | Adipogenesis - genetics | Osteogenesis - genetics | Wnt Signaling Pathway | Green Fluorescent Proteins - metabolism | Bone Marrow Cells - cytology | Cell Separation | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | beta Catenin - metabolism | Mice, Knockout | Transcription Factors - metabolism | Cell Lineage | Animals | Adipocytes - metabolism | Sp7 Transcription Factor | Mice | Osteoblasts - metabolism | Bone Marrow Cells - metabolism | Index Medicus | Adipose tissue | Wnt protein | catenin | Adipocytes | Gene deletion | Osteoblasts | Promoters | Myoblasts | Bone mass | Osteoblastogenesis | adipogenesis | Cell fate | Bone marrow | Differentiation | Osteogenesis
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9. Full Text Distinct Dendritic Cell Subsets Dictate the Fate Decision between Effector and Memory CD8+ T Cell Differentiation by a CD24-Dependent Mechanism
by Kim, Taeg S and Gorski, Stacey A and Hahn, Steven and Murphy, Kenneth M and Braciale, Thomas J
Immunity, ISSN 1074-7613, 03/2014, Volume 40, Issue 3, pp. 400 - 413
The contribution of different DC subsets to effector and memory CD8 T cell generation during infection and the mechanism by which DCs controls these fate... 
RESPONSES | LUNG | ACTIVATION | INFLUENZA-VIRUS INFECTION | RECEPTOR | RAGE | IMMUNOLOGY | HEAT-STABLE ANTIGEN | CD24 | EXPRESSION | GLYCATION END-PRODUCTS | CD8-Positive T-Lymphocytes - cytology | CD24 Antigen - metabolism | Dendritic Cells - immunology | Antigens, CD - metabolism | Lung - virology | Lymphocyte Activation - immunology | CD8-Positive T-Lymphocytes - metabolism | Female | Lung - metabolism | Receptor for Advanced Glycation End Products | Dendritic Cells - metabolism | Lymph Nodes - metabolism | Immunophenotyping | Integrin alpha Chains - metabolism | Lymph Nodes - virology | Lymph Nodes - immunology | Mice, Knockout | Cell Differentiation - immunology | Phenotype | Animals | CD8-Positive T-Lymphocytes - virology | Virus Release - immunology | Protein Binding | Immunologic Memory | Mice | CD11b Antigen - metabolism | CD8-Positive T-Lymphocytes - immunology | Lung - immunology | Receptors, Immunologic - metabolism | Lungs | Lymphocytes | Rodents | Colleges & universities | T cell receptors | Infections | Acquisitions & mergers | Index Medicus
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