Cancer Science, ISSN 1347-9032, 08/2017, Volume 108, Issue 8, pp. 1584 - 1593
Chemokine (CC motif) ligand 18 (CCL18) is involved in remodeling of the tumor microenvironment and plays critical roles in oncogenesis, invasiveness, and...
chemokine (CC motif) ligand 18 (CCL18) | epithelial–mesenchymal transition (EMT) | Cancer stem(‐like) cell (CSC) | oral squamous cell carcinoma (OSCC) | Slug | Cancer stem(-like) cell (CSC) | MIGRATION | DIAGNOSIS | HEAD | BREAST-CANCER METASTASIS | CCL18 PROMOTES | MARKERS | EPITHELIAL-MESENCHYMAL TRANSITION | epithelial-mesenchymal transition (EMT) | INVASION | ONCOLOGY | SIGNALING PATHWAY | Up-Regulation | Snail Family Transcription Factors - metabolism | Signal Transduction | TOR Serine-Threonine Kinases - metabolism | Neoplasm Invasiveness | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Mouth Neoplasms - metabolism | Chemokines, CC - genetics | Epithelial-Mesenchymal Transition | Cell Line, Tumor | Female | Aged | Mouth Neoplasms - genetics | Cell Movement | Chemokines, CC - metabolism | Immunohistochemistry | Squamous cell carcinoma | Metastasis | Analysis | Stem cells | TOR protein | Transcription factors | Mesenchyme | Kinases | Medical diagnosis | Cancer therapies | Metastases | Cell adhesion & migration | Proteins | Signal transduction | CCL18 protein | Tumorigenesis | Invasiveness | RNA-mediated interference | Rapamycin | Breast cancer | Oral squamous cell carcinoma | Cell lines | Ligands | Chemokines | Cell migration | Tumors | Original
chemokine (CC motif) ligand 18 (CCL18) | epithelial–mesenchymal transition (EMT) | Cancer stem(‐like) cell (CSC) | oral squamous cell carcinoma (OSCC) | Slug | Cancer stem(-like) cell (CSC) | MIGRATION | DIAGNOSIS | HEAD | BREAST-CANCER METASTASIS | CCL18 PROMOTES | MARKERS | EPITHELIAL-MESENCHYMAL TRANSITION | epithelial-mesenchymal transition (EMT) | INVASION | ONCOLOGY | SIGNALING PATHWAY | Up-Regulation | Snail Family Transcription Factors - metabolism | Signal Transduction | TOR Serine-Threonine Kinases - metabolism | Neoplasm Invasiveness | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Mouth Neoplasms - metabolism | Chemokines, CC - genetics | Epithelial-Mesenchymal Transition | Cell Line, Tumor | Female | Aged | Mouth Neoplasms - genetics | Cell Movement | Chemokines, CC - metabolism | Immunohistochemistry | Squamous cell carcinoma | Metastasis | Analysis | Stem cells | TOR protein | Transcription factors | Mesenchyme | Kinases | Medical diagnosis | Cancer therapies | Metastases | Cell adhesion & migration | Proteins | Signal transduction | CCL18 protein | Tumorigenesis | Invasiveness | RNA-mediated interference | Rapamycin | Breast cancer | Oral squamous cell carcinoma | Cell lines | Ligands | Chemokines | Cell migration | Tumors | Original
Journal Article
Progress In Modern Biomedicine, ISSN 1673-6273, 08/2012, Volume 12, Issue 22, pp. 4275 - 4280
Objective: To investigate the possible role of CCL18 in migration and metastasis of liver cancer by studying it's expression in serum. Methods: The levels of...
Liver cancer | Overexpression | Migration | CCL18 protein | Chemokines | Enzyme-linked immunosorbent assay | Metastases
Liver cancer | Overexpression | Migration | CCL18 protein | Chemokines | Enzyme-linked immunosorbent assay | Metastases
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 07/2005, Volume 78, Issue 1, pp. 14 - 26
CC chemokine ligand 18 (CCL18) was originally discovered as pulmonary and activation‐regulated chemokine (PARC), dendritic cell (DC)‐chemokine 1 (DC‐CK1),...
macrophages | dendritic cells | homing | inflammatory | chemotaxis | Homing | Inflammatory | Dendritic cells | Macrophages | Chemotaxis | RHEUMATOID-ARTHRITIS | MATURE DENDRITIC CELLS | C-C CHEMOKINE | IMMUNOLOGY | OVARIAN-CARCINOMA | ACTIVATION-REGULATED CHEMOKINE | CELL BIOLOGY | HUMAN BLOOD MONOCYTES | GENE-EXPRESSION | TUMOR-ASSOCIATED MACROPHAGES | HEMATOLOGY | T-CELLS | MACROPHAGE INFLAMMATORY PROTEIN-1-ALPHA | Inflammation Mediators - immunology | Autoimmune Diseases - physiopathology | Dendritic Cells - immunology | Humans | Autoimmune Diseases - immunology | Inflammation - immunology | Receptors, Lymphocyte Homing - immunology | Inflammation - metabolism | Animals | Neoplasms - immunology | Chemokines, CC - genetics | Inflammation Mediators - metabolism | Inflammation - genetics | Chemokines, CC - immunology | Neoplasms - physiopathology | Chemotaxis, Leukocyte - immunology | Macrophages - immunology | Chemokines, CC - metabolism
macrophages | dendritic cells | homing | inflammatory | chemotaxis | Homing | Inflammatory | Dendritic cells | Macrophages | Chemotaxis | RHEUMATOID-ARTHRITIS | MATURE DENDRITIC CELLS | C-C CHEMOKINE | IMMUNOLOGY | OVARIAN-CARCINOMA | ACTIVATION-REGULATED CHEMOKINE | CELL BIOLOGY | HUMAN BLOOD MONOCYTES | GENE-EXPRESSION | TUMOR-ASSOCIATED MACROPHAGES | HEMATOLOGY | T-CELLS | MACROPHAGE INFLAMMATORY PROTEIN-1-ALPHA | Inflammation Mediators - immunology | Autoimmune Diseases - physiopathology | Dendritic Cells - immunology | Humans | Autoimmune Diseases - immunology | Inflammation - immunology | Receptors, Lymphocyte Homing - immunology | Inflammation - metabolism | Animals | Neoplasms - immunology | Chemokines, CC - genetics | Inflammation Mediators - metabolism | Inflammation - genetics | Chemokines, CC - immunology | Neoplasms - physiopathology | Chemotaxis, Leukocyte - immunology | Macrophages - immunology | Chemokines, CC - metabolism
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 12/2016, Volume 55, Issue 12, pp. 2051 - 2062
Non‐small cell lung cancer (NSCLC) comprises nearly 80% of lung cancers and the poor prognosis is due to its high invasiveness and metastasis. CC chemokine...
ELMO1 | CC chemokine ligand 18 | matastasis | invasion | Nir1 | METASTASIS | CHEMOTAXIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | ONCOLOGY | INTEGRINS | LEADING CELLS | TUMOR-ASSOCIATED MACROPHAGES | CCL18 | EXPRESSION | PROGRESSION | MOTILITY | Humans | Middle Aged | Lung Neoplasms - pathology | Male | Chemokines, CC - analysis | Membrane Proteins - analysis | rac GTP-Binding Proteins - analysis | Calcium-Binding Proteins - immunology | Neoplasm Invasiveness - pathology | Lymphatic Metastasis - pathology | Adaptor Proteins, Signal Transducing - immunology | Female | Chemokines, CC - immunology | Carcinoma, Non-Small-Cell Lung - pathology | Lung - pathology | Signal Transduction | Calcium-Binding Proteins - analysis | Neoplasm Invasiveness - immunology | Lymphatic Metastasis - immunology | Membrane Proteins - immunology | rac GTP-Binding Proteins - immunology | Mice, SCID | Carcinoma, Non-Small-Cell Lung - immunology | Adaptor Proteins, Signal Transducing - analysis | Lung Neoplasms - immunology | Animals | Cell Line, Tumor | Mice | Cell Movement | Lung cancer, Non-small cell | Metastasis | Integrins | Ligands | Cancer therapies | Lung cancer | Chemokines | Cell adhesion & migration
ELMO1 | CC chemokine ligand 18 | matastasis | invasion | Nir1 | METASTASIS | CHEMOTAXIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | ONCOLOGY | INTEGRINS | LEADING CELLS | TUMOR-ASSOCIATED MACROPHAGES | CCL18 | EXPRESSION | PROGRESSION | MOTILITY | Humans | Middle Aged | Lung Neoplasms - pathology | Male | Chemokines, CC - analysis | Membrane Proteins - analysis | rac GTP-Binding Proteins - analysis | Calcium-Binding Proteins - immunology | Neoplasm Invasiveness - pathology | Lymphatic Metastasis - pathology | Adaptor Proteins, Signal Transducing - immunology | Female | Chemokines, CC - immunology | Carcinoma, Non-Small-Cell Lung - pathology | Lung - pathology | Signal Transduction | Calcium-Binding Proteins - analysis | Neoplasm Invasiveness - immunology | Lymphatic Metastasis - immunology | Membrane Proteins - immunology | rac GTP-Binding Proteins - immunology | Mice, SCID | Carcinoma, Non-Small-Cell Lung - immunology | Adaptor Proteins, Signal Transducing - analysis | Lung Neoplasms - immunology | Animals | Cell Line, Tumor | Mice | Cell Movement | Lung cancer, Non-small cell | Metastasis | Integrins | Ligands | Cancer therapies | Lung cancer | Chemokines | Cell adhesion & migration
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 04/2019, Volume 234, Issue 4, pp. 4608 - 4616
Background: Surgical stress has been suggested to facilitate colon cancer growth and metastasis. However, the precise mechanisms by which surgical trauma...
surgical | tregs and immunosuppressive environment | colon cancer | C‐C motif chemokine ligand 18 (CCL18) | C-C motif chemokine ligand 18 (CCL18) | SURVIVAL | SURGERY | PHYSIOLOGY | METASTASES | CELL BIOLOGY | IMPACT | DISEASE | CXC-CHEMOKINES | TUMOR-GROWTH | EXPRESSION | Correlation | Colorectal cancer | Transplantation | Lymphocytes T | Tissues | Blood | Bearing | Metastases | Recruitment | Angiogenesis | Colon cancer | Lymphocytes | Surgery | Peripheral blood | CD25 antigen | CCL18 protein | Colon | Immunoregulation | Trauma | CD4 antigen | Mice | Chemokines | Cancer | Tumors | Peritoneum | Original | Original s
surgical | tregs and immunosuppressive environment | colon cancer | C‐C motif chemokine ligand 18 (CCL18) | C-C motif chemokine ligand 18 (CCL18) | SURVIVAL | SURGERY | PHYSIOLOGY | METASTASES | CELL BIOLOGY | IMPACT | DISEASE | CXC-CHEMOKINES | TUMOR-GROWTH | EXPRESSION | Correlation | Colorectal cancer | Transplantation | Lymphocytes T | Tissues | Blood | Bearing | Metastases | Recruitment | Angiogenesis | Colon cancer | Lymphocytes | Surgery | Peripheral blood | CD25 antigen | CCL18 protein | Colon | Immunoregulation | Trauma | CD4 antigen | Mice | Chemokines | Cancer | Tumors | Peritoneum | Original | Original s
Journal Article
Journal of Dermatological Science, ISSN 0923-1811, 09/2018, Volume 91, Issue 3, pp. 317 - 324
The CC chemokine ligand 18 (CCL18) has a higher expression in some tumors, while the CCL18 level can be a marker of tumor progression and prognosis. We...
Malignancy | Overexpression | CCL18 | miR-128 | Malignant melanoma | INVASION | METASTASIS | CANCER CELLS | TUMOR-ASSOCIATED MACROPHAGES | EXPRESSION | CARCINOMA | DERMATOLOGY | Up-Regulation | Cadherins - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | Male | MicroRNAs - metabolism | Epithelial-Mesenchymal Transition - genetics | Antigens, CD - genetics | Cell Movement - genetics | Antigens, CD - metabolism | Melanoma - genetics | Female | 3' Untranslated Regions | Cadherins - genetics | Binding Sites | Melanoma - metabolism | Skin Neoplasms - pathology | Cell Proliferation - genetics | Melanoma - pathology | Disease Progression | Skin Neoplasms - metabolism | Skin Neoplasms - genetics | Chemokines, CC - genetics | Cell Line, Tumor | Aged | MicroRNAs - genetics | Chemokines, CC - metabolism | Genetic research | Development and progression | Analysis | Genes | Melanoma
Malignancy | Overexpression | CCL18 | miR-128 | Malignant melanoma | INVASION | METASTASIS | CANCER CELLS | TUMOR-ASSOCIATED MACROPHAGES | EXPRESSION | CARCINOMA | DERMATOLOGY | Up-Regulation | Cadherins - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | Male | MicroRNAs - metabolism | Epithelial-Mesenchymal Transition - genetics | Antigens, CD - genetics | Cell Movement - genetics | Antigens, CD - metabolism | Melanoma - genetics | Female | 3' Untranslated Regions | Cadherins - genetics | Binding Sites | Melanoma - metabolism | Skin Neoplasms - pathology | Cell Proliferation - genetics | Melanoma - pathology | Disease Progression | Skin Neoplasms - metabolism | Skin Neoplasms - genetics | Chemokines, CC - genetics | Cell Line, Tumor | Aged | MicroRNAs - genetics | Chemokines, CC - metabolism | Genetic research | Development and progression | Analysis | Genes | Melanoma
Journal Article
Oncotarget, ISSN 1949-2553, 03/2016, Volume 7, Issue 13, pp. 16262 - 16272
Chemokine (C-C motif) ligand 18 (CCL18) has been implicated in the pathogenesis and progression of various cancers; however, in oral squamous cell carcinoma...
Chemokine (C-C motif) ligand 18 (CCL18) | Akt | Oral squamous cell carcinoma | Proliferation | Invasion | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Cell Proliferation - physiology | Male | Mouth Neoplasms - metabolism | Head and Neck Neoplasms - metabolism | Head and Neck Neoplasms - pathology | Squamous Cell Carcinoma of Head and Neck | Animals | Heterografts | Mice, Nude | Neoplasm Invasiveness - pathology | Mouth Neoplasms - pathology | Adult | Female | Aged | Mice | Proto-Oncogene Proteins c-akt - metabolism | Chemokines, CC - metabolism
Chemokine (C-C motif) ligand 18 (CCL18) | Akt | Oral squamous cell carcinoma | Proliferation | Invasion | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Cell Proliferation - physiology | Male | Mouth Neoplasms - metabolism | Head and Neck Neoplasms - metabolism | Head and Neck Neoplasms - pathology | Squamous Cell Carcinoma of Head and Neck | Animals | Heterografts | Mice, Nude | Neoplasm Invasiveness - pathology | Mouth Neoplasms - pathology | Adult | Female | Aged | Mice | Proto-Oncogene Proteins c-akt - metabolism | Chemokines, CC - metabolism
Journal Article
Respirology, ISSN 1323-7799, 04/2017, Volume 22, Issue 3, pp. 542 - 550
Background and objective: The aim of this study was to investigate the prognostic value of four biomarkers, YKL-40, chemokine (C-C motif) ligand 18 (CCL18),...
mortality | chemokine (C-C motif) ligand 18 protein, human | pneumonia | YKL-40 protein, human | Pulmonary and Respiratory Medicine | biomarkers | chemokine (C‐C motif) ligand 18 protein, human | YKL‐40 protein, human | SURFACTANT PROTEIN-D | INJURY | PLASMA YKL-40 | RESPIRATORY SYSTEM | chemokine (C-C motif) ligand 18 protein | SERUM YKL-40 | DISEASE | YKL-40 protein | CHEMOKINE | human | EXPRESSION | PULMONARY-FIBROSIS | SEVERITY | Severity of Illness Index | Community-Acquired Infections - mortality | Calcitonin - blood | Intensive Care Units | Prognosis | C-Reactive Protein | Chemokines, CC - blood | Area Under Curve | Humans | Middle Aged | Pulmonary Surfactant-Associated Protein D - blood | Male | Hospitalization | Biomarkers - blood | Pneumonia - mortality | Mucin-1 - blood | Aged, 80 and over | Adult | Chitinase-3-Like Protein 1 - blood | Female | Pneumonia - blood | ROC Curve | Aged | Community-Acquired Infections - blood | Medicine, Experimental | Medical research | Health aspects | Analysis | Pneumonia | C-reactive protein | Mortality | Procalcitonin | Lung diseases | CCL18 protein | Multivariate analysis
mortality | chemokine (C-C motif) ligand 18 protein, human | pneumonia | YKL-40 protein, human | Pulmonary and Respiratory Medicine | biomarkers | chemokine (C‐C motif) ligand 18 protein, human | YKL‐40 protein, human | SURFACTANT PROTEIN-D | INJURY | PLASMA YKL-40 | RESPIRATORY SYSTEM | chemokine (C-C motif) ligand 18 protein | SERUM YKL-40 | DISEASE | YKL-40 protein | CHEMOKINE | human | EXPRESSION | PULMONARY-FIBROSIS | SEVERITY | Severity of Illness Index | Community-Acquired Infections - mortality | Calcitonin - blood | Intensive Care Units | Prognosis | C-Reactive Protein | Chemokines, CC - blood | Area Under Curve | Humans | Middle Aged | Pulmonary Surfactant-Associated Protein D - blood | Male | Hospitalization | Biomarkers - blood | Pneumonia - mortality | Mucin-1 - blood | Aged, 80 and over | Adult | Chitinase-3-Like Protein 1 - blood | Female | Pneumonia - blood | ROC Curve | Aged | Community-Acquired Infections - blood | Medicine, Experimental | Medical research | Health aspects | Analysis | Pneumonia | C-reactive protein | Mortality | Procalcitonin | Lung diseases | CCL18 protein | Multivariate analysis
Journal Article
European Journal of Cancer, ISSN 0959-8049, 2013, Volume 49, Issue 18, pp. 3900 - 3913
Abstract Chemokine (C–C motif) ligand 18 (CCL18), which is derived from tumour-associated macrophages (TAMs), plays a critical role in promoting breast cancer...
Hematology, Oncology and Palliative Medicine | F-actin | CCL18 | EMT | Molecular mechanisms | Nir1 | MIGRATION | SURVIVAL | MACROPHAGES | TRANSCRIPTION FACTOR SNAIL | EPITHELIAL-MESENCHYMAL TRANSITIONS | ONCOLOGY | INFLAMMATION | AKT2 | GROWTH | REPRESSOR | EXPRESSION | Humans | Middle Aged | Epithelial-Mesenchymal Transition - drug effects | Glycogen Synthase Kinase 3 beta | Phosphatidylinositol 3-Kinases - metabolism | Epithelial-Mesenchymal Transition - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Breast Neoplasms - metabolism | MCF-7 Cells | Aged, 80 and over | Adult | Female | Membrane Proteins - metabolism | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Snail Family Transcription Factors | Calcium-Binding Proteins - metabolism | Signal Transduction | Membrane Proteins - genetics | Neoplasm Invasiveness | Morpholines - pharmacology | Glycogen Synthase Kinase 3 - metabolism | Mice, SCID | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Chemokines, CC - genetics | Cell Line, Tumor | Protein Binding | Mice, Inbred NOD | Aged | Mice | Calcium-Binding Proteins - genetics | Chemokines, CC - metabolism
Hematology, Oncology and Palliative Medicine | F-actin | CCL18 | EMT | Molecular mechanisms | Nir1 | MIGRATION | SURVIVAL | MACROPHAGES | TRANSCRIPTION FACTOR SNAIL | EPITHELIAL-MESENCHYMAL TRANSITIONS | ONCOLOGY | INFLAMMATION | AKT2 | GROWTH | REPRESSOR | EXPRESSION | Humans | Middle Aged | Epithelial-Mesenchymal Transition - drug effects | Glycogen Synthase Kinase 3 beta | Phosphatidylinositol 3-Kinases - metabolism | Epithelial-Mesenchymal Transition - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Breast Neoplasms - metabolism | MCF-7 Cells | Aged, 80 and over | Adult | Female | Membrane Proteins - metabolism | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Snail Family Transcription Factors | Calcium-Binding Proteins - metabolism | Signal Transduction | Membrane Proteins - genetics | Neoplasm Invasiveness | Morpholines - pharmacology | Glycogen Synthase Kinase 3 - metabolism | Mice, SCID | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Chemokines, CC - genetics | Cell Line, Tumor | Protein Binding | Mice, Inbred NOD | Aged | Mice | Calcium-Binding Proteins - genetics | Chemokines, CC - metabolism
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 04/2019, Volume 23, Issue 4, pp. 2689 - 2701
Metastasis is one of the primary causes for high mortality in patients with squamous cell carcinoma of the head and neck (SCCHN). Our previous study showed...
SCCHN | CCL18 | metastasis | EMT | MTDH | ASTROCYTE ELEVATED GENE-1 | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | MACROPHAGES | LIGAND 18 | CELL BIOLOGY | PROGNOSTIC MARKER | METADHERIN | CHEMOKINE | GLOBAL CANCER STATISTICS | EXPRESSION | PROGRESSION | Squamous cell carcinoma | Head | Leukocyte migration | Mesenchyme | Metastasis | Macrophages | Patients | Lymph nodes | Metastases | Proteins | Signal transduction | Signaling | Biomarkers | CCL18 protein | Neck | Serums | Cell migration | Enzyme-linked immunosorbent assay | Tumors | Recombinant | Original
SCCHN | CCL18 | metastasis | EMT | MTDH | ASTROCYTE ELEVATED GENE-1 | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | MACROPHAGES | LIGAND 18 | CELL BIOLOGY | PROGNOSTIC MARKER | METADHERIN | CHEMOKINE | GLOBAL CANCER STATISTICS | EXPRESSION | PROGRESSION | Squamous cell carcinoma | Head | Leukocyte migration | Mesenchyme | Metastasis | Macrophages | Patients | Lymph nodes | Metastases | Proteins | Signal transduction | Signaling | Biomarkers | CCL18 protein | Neck | Serums | Cell migration | Enzyme-linked immunosorbent assay | Tumors | Recombinant | Original
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 04/2019, Volume 9, Issue 1, pp. 5963 - 8
The chemokine (C-C motif) chemokine ligand 18 (CCL18) is a structural homolog of CCL3 primarily produced by monocyte-derived cells with an M2 phenotype....
HIV-1 | CYTOKINES | VIVO | CC-CHEMOKINE | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | MONOCYTES | PERIPHERAL-BLOOD | EXPRESSION | T-LYMPHOCYTES | ACTIVATION-REGULATED CHEMOKINE | Flow cytometry | Antiretroviral drugs | Phenotypes | Helper cells | Chronic infection | CCL3 protein | Infections | Lymphocytes T | Macrophages | Antiretroviral therapy | CD4 antigen | CCL22 protein | Monocytes | Cell activation | Lymphocytes | Human immunodeficiency virus--HIV | CD38 antigen | CCL18 protein | Plasma levels | CCL17 protein | Chemokines | Enzyme-linked immunosorbent assay
HIV-1 | CYTOKINES | VIVO | CC-CHEMOKINE | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | MONOCYTES | PERIPHERAL-BLOOD | EXPRESSION | T-LYMPHOCYTES | ACTIVATION-REGULATED CHEMOKINE | Flow cytometry | Antiretroviral drugs | Phenotypes | Helper cells | Chronic infection | CCL3 protein | Infections | Lymphocytes T | Macrophages | Antiretroviral therapy | CD4 antigen | CCL22 protein | Monocytes | Cell activation | Lymphocytes | Human immunodeficiency virus--HIV | CD38 antigen | CCL18 protein | Plasma levels | CCL17 protein | Chemokines | Enzyme-linked immunosorbent assay
Journal Article
Atherosclerosis, ISSN 0021-9150, 2008, Volume 204, Issue 2, pp. e15 - e20
Abstract Objective Using gene expression profiling, we aimed to identify genes that are predominantly expressed in human carotid atherosclerotic plaques. Such...
Cardiovascular | CCL18 | Macrophages | Endarterectomy | Genomics | Atherosclerosis | Carotid Artery Diseases - immunology | Carotid Artery Diseases - surgery | Immunohistochemistry | Promoter Regions, Genetic | Coronary Disease - immunology | Chemokines, CC - blood | Oligonucleotide Array Sequence Analysis | Gene Frequency | Humans | Middle Aged | Gene Expression Profiling - methods | Male | Biomarkers - blood | Case-Control Studies | Endarterectomy, Carotid | Coronary Disease - genetics | Carotid Artery Diseases - genetics | Chemokines, CC - genetics | Polymerase Chain Reaction | Female | Aged | Polymorphism, Single Nucleotide | Macrophages - immunology | Proteins | Gene expression | Genetic | Macrophages/immunology | CC/blood/genetics | Single Nucleotide | Medicinsk genetik | Medical Genetics | Carotid Artery Diseases/genetics/immunology/surgery | Coronary Disease/genetics/immunology | Biological Markers/blood | Gene Expression Profiling/methods | Promoter Regions | Carotid | Chemokines | Polymorphism
Cardiovascular | CCL18 | Macrophages | Endarterectomy | Genomics | Atherosclerosis | Carotid Artery Diseases - immunology | Carotid Artery Diseases - surgery | Immunohistochemistry | Promoter Regions, Genetic | Coronary Disease - immunology | Chemokines, CC - blood | Oligonucleotide Array Sequence Analysis | Gene Frequency | Humans | Middle Aged | Gene Expression Profiling - methods | Male | Biomarkers - blood | Case-Control Studies | Endarterectomy, Carotid | Coronary Disease - genetics | Carotid Artery Diseases - genetics | Chemokines, CC - genetics | Polymerase Chain Reaction | Female | Aged | Polymorphism, Single Nucleotide | Macrophages - immunology | Proteins | Gene expression | Genetic | Macrophages/immunology | CC/blood/genetics | Single Nucleotide | Medicinsk genetik | Medical Genetics | Carotid Artery Diseases/genetics/immunology/surgery | Coronary Disease/genetics/immunology | Biological Markers/blood | Gene Expression Profiling/methods | Promoter Regions | Carotid | Chemokines | Polymorphism
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 03/2014, Volume 16, Issue 2, pp. R65 - R65
Introduction: Agonistic autoantibodies (Aabs) against the angiotensin II receptor type 1 (AT1R) and the endothelin receptor type A (ETAR) have been identified...
INTERSTITIAL LUNG-DISEASE | PULMONARY | SCLERODERMA | INFLAMMATION | AGONISTIC ANTIBODIES | MATERNAL AUTOANTIBODIES | AT1 RECEPTOR | RHEUMATOLOGY | NF-KAPPA-B | II TYPE-1 | T-CELLS | Chemotaxis, Leukocyte | Interleukin-18 - immunology | Enzyme-Linked Immunosorbent Assay | Humans | Middle Aged | Scleroderma, Systemic - metabolism | Male | Monocytes - metabolism | Monocytes - immunology | Receptor, Endothelin A - immunology | Scleroderma, Systemic - immunology | Autoantibodies - immunology | Interleukin-18 - biosynthesis | Flow Cytometry | Receptor, Angiotensin, Type 1 - metabolism | Chemokines, CC - biosynthesis | Adult | Female | Receptor, Endothelin A - metabolism | Chemokines, CC - immunology | Receptor, Angiotensin, Type 1 - immunology | Real-Time Polymerase Chain Reaction | Cell Movement | Medicine, Experimental | Medical research | Respiratory tract diseases | Endothelin | Analysis
INTERSTITIAL LUNG-DISEASE | PULMONARY | SCLERODERMA | INFLAMMATION | AGONISTIC ANTIBODIES | MATERNAL AUTOANTIBODIES | AT1 RECEPTOR | RHEUMATOLOGY | NF-KAPPA-B | II TYPE-1 | T-CELLS | Chemotaxis, Leukocyte | Interleukin-18 - immunology | Enzyme-Linked Immunosorbent Assay | Humans | Middle Aged | Scleroderma, Systemic - metabolism | Male | Monocytes - metabolism | Monocytes - immunology | Receptor, Endothelin A - immunology | Scleroderma, Systemic - immunology | Autoantibodies - immunology | Interleukin-18 - biosynthesis | Flow Cytometry | Receptor, Angiotensin, Type 1 - metabolism | Chemokines, CC - biosynthesis | Adult | Female | Receptor, Endothelin A - metabolism | Chemokines, CC - immunology | Receptor, Angiotensin, Type 1 - immunology | Real-Time Polymerase Chain Reaction | Cell Movement | Medicine, Experimental | Medical research | Respiratory tract diseases | Endothelin | Analysis
Journal Article
Journal of Translational Medicine, ISSN 1479-5876, 02/2017, Volume 15, Issue 1, p. 43
Background: Niemann-Pick disease type C (NP-C) is a rare, autosomal recessive neurodegenerative disease caused by mutations in either the NPC1 or NPC2 genes....
Screening | 7-ketocholesterol | Chitotriosidase | NP-C suspicion index | CCL18/PARC | Diagnosis | Niemann-Pick disease type C | MEDICINE, RESEARCH & EXPERIMENTAL | PHENOTYPE | IDENTIFICATION | MARKED ELEVATION | GAUCHER-DISEASE | PERFORMANCE LIQUID-CHROMATOGRAPHY | DISRUPTION | GENE | CHOLESTEROL HOMEOSTASIS | MUTATIONS | RAPID DIAGNOSIS | Demography | Prospective Studies | Chemokines, CC - blood | Niemann-Pick Disease, Type C - enzymology | Humans | Middle Aged | Child, Preschool | Infant | Male | Young Adult | Filipin | Computer Simulation | Adolescent | Oxysterols | Adult | Family | Female | Mutation | Niemann-Pick Disease, Type C - diagnosis | Niemann-Pick Disease, Type C - blood | Child | Hexosaminidases - blood | Niemann-Pick disease | Care and treatment | Cytokines | Molecular diagnostic techniques | Innovations | Genetic aspects | Identification and classification
Screening | 7-ketocholesterol | Chitotriosidase | NP-C suspicion index | CCL18/PARC | Diagnosis | Niemann-Pick disease type C | MEDICINE, RESEARCH & EXPERIMENTAL | PHENOTYPE | IDENTIFICATION | MARKED ELEVATION | GAUCHER-DISEASE | PERFORMANCE LIQUID-CHROMATOGRAPHY | DISRUPTION | GENE | CHOLESTEROL HOMEOSTASIS | MUTATIONS | RAPID DIAGNOSIS | Demography | Prospective Studies | Chemokines, CC - blood | Niemann-Pick Disease, Type C - enzymology | Humans | Middle Aged | Child, Preschool | Infant | Male | Young Adult | Filipin | Computer Simulation | Adolescent | Oxysterols | Adult | Family | Female | Mutation | Niemann-Pick Disease, Type C - diagnosis | Niemann-Pick Disease, Type C - blood | Child | Hexosaminidases - blood | Niemann-Pick disease | Care and treatment | Cytokines | Molecular diagnostic techniques | Innovations | Genetic aspects | Identification and classification
Journal Article